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Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2011, Volume 121, Issue 11, pp. 4393 - 4408
Journal Article
Circulation, ISSN 0009-7322, 04/2014, Volume 129, Issue 15, pp. 1586 - 1597
BACKGROUND—Pericytes and their crosstalk with endothelial cells are critical for the development of a functional microvasculature and vascular remodeling. It... 
cell communication | transforming growth factor beta | fibroblast growth factor 2 | pericytes | endothelilal cells | pulmonary arterial hypertension | interleukin-6 | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | BONE MORPHOGENETIC PROTEIN | MICROVASCULAR PERICYTES | INHIBITION | ARTERIAL-HYPERTENSION | ADAMS-OLIVER SYNDROME | PERIPHERAL VASCULAR DISEASE | MICE | DIFFERENTIATION | CONTRIBUTES | MOLECULAR-MECHANISMS | Pericytes - drug effects | Rats, Wistar | Humans | Middle Aged | Pericytes - cytology | Fibroblast Growth Factor 2 - pharmacology | Hypertension, Pulmonary - physiopathology | Cell Communication - physiology | Male | Pericytes - physiology | Retinal Vessels - physiology | Microcirculation - physiology | Pulmonary Circulation - physiology | Interleukin-6 - physiology | Retinal Vessels - cytology | Adult | Female | Fibroblast Growth Factor 2 - physiology | Cell Differentiation - physiology | Endothelial Cells - physiology | Muscle, Smooth, Vascular - physiology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Cells, Cultured | Rats | Mice, Transgenic | Muscle, Smooth, Vascular - cytology | Neovascularization, Physiologic - physiology | Animals | Interleukin-6 - pharmacology | Cell Differentiation - drug effects | Endothelial Cells - cytology | Cell Communication - drug effects | Mice | Hypertension, Pulmonary - pathology | Endothelial Cells - drug effects | Physiological aspects | Smooth muscle | Fibroblast growth factors | Research | Pulmonary hypertension | Interleukin-6 | Index Medicus | Abridged Index Medicus
Journal Article
PLoS Medicine, ISSN 1549-1277, 01/2008, Volume 5, Issue 1, pp. 0123 - 0138
Background Important support functions, including promotion of tumor growth, angiogenesis, and invasion, have been attributed to the different cell types... 
HUMAN BREAST-CARCINOMA | MEDICINE, GENERAL & INTERNAL | TGF-BETA | GROWTH-FACTOR-RECEPTOR | SQUAMOUS CARCINOGENESIS | TYPE-16 TRANSGENIC MICE | FACTOR-BETA-RECEPTOR | CELL INVASION | MOUSE MODEL | TISSUE STROMA | CERVICAL-CANCER | Fibroblast Growth Factor 7 - physiology | Fibroblasts - enzymology | Uterine Cervical Neoplasms - blood supply | Humans | Carcinoma - virology | Neoplasm Proteins - antagonists & inhibitors | Antineoplastic Agents - therapeutic use | Receptors, Platelet-Derived Growth Factor - physiology | Proto-Oncogene Proteins c-kit - biosynthesis | Neovascularization, Pathologic - physiopathology | Neoplasm Proteins - genetics | Carcinoma - drug therapy | Estradiol - administration & dosage | Receptors, Platelet-Derived Growth Factor - genetics | Mice, Transgenic | Piperazines - therapeutic use | Uterine Cervical Neoplasms - physiopathology | Pyrimidines - pharmacology | Fibroblast Growth Factor 7 - biosynthesis | Piperazines - pharmacology | Carcinoma - physiopathology | Stromal Cells - secretion | Fibroblast Growth Factor 2 - genetics | Neovascularization, Pathologic - drug therapy | Fibroblasts - drug effects | Carcinoma - blood supply | Mice | Fibroblasts - secretion | Epithelial Cells - metabolism | Neoplasm Proteins - physiology | Platelet-Derived Growth Factor - genetics | Human papillomavirus 16 | Paracrine Communication - physiology | Stromal Cells - enzymology | Stromal Cells - drug effects | Female | Proto-Oncogene Proteins c-kit - genetics | Antineoplastic Agents - pharmacology | Fibroblast Growth Factor 2 - physiology | Receptors, Platelet-Derived Growth Factor - antagonists & inhibitors | Neoplasm Proteins - biosynthesis | Pericytes - metabolism | Fibroblast Growth Factor 2 - biosynthesis | Uterine Cervical Neoplasms - drug therapy | Mice, Inbred Strains | Imatinib Mesylate | Fibroblast Growth Factor 7 - genetics | Animals | Estradiol - toxicity | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Paracrine Communication - drug effects | Uterine Cervical Neoplasms - virology | Protein Kinase Inhibitors - pharmacology | Benzamides | Platelet-Derived Growth Factor - physiology | Platelet-Derived Growth Factor - antagonists & inhibitors | Index Medicus | Oncology | Gynecological | Chemotherapy | Cancer | Proteins | Medical research | Human papillomavirus | Cervical cancer | Tumors
Journal Article
STEM CELLS, ISSN 1066-5099, 09/2007, Volume 25, Issue 9, pp. 2206 - 2214
We have utilized a serum‐ and stromal cell‐free “spin embryoid body (EB)” differentiation system to investigate the roles of four growth factors, bone... 
Fibroblast growth factor 2 | Bone morphogenetic protein 4 | Embryonic stem cells | Hematopoiesis | Vascular endothelial growth factor | Stem cell factor | stem cell factor | embryonic stem cells | MOUSE | COCULTURE | MURINE | CELL & TISSUE ENGINEERING | CELL BIOLOGY | PRIMITIVE STREAK | hematopoiesis | IN-VITRO | COMMITMENT | GENE | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | vascular endothelial growth factor | fibroblast growth factor 2 | STROMAL CELLS | HEMATOLOGY | bone morphogenetic protein 4 | ES CELLS | MESODERM INDUCTION | Bone Morphogenetic Protein 4 | Embryonic Stem Cells - cytology | Bone Morphogenetic Proteins - physiology | Homeodomain Proteins - metabolism | Humans | Cells, Cultured | Fibroblast Growth Factor 2 - pharmacology | Primitive Streak - metabolism | Homeodomain Proteins - genetics | Hematopoiesis - drug effects | Drug Synergism | Animals | Cell Differentiation - drug effects | Gene Expression Regulation, Developmental | Stem Cell Factor - pharmacology | Hematopoiesis - physiology | Fibroblast Growth Factor 2 - physiology | Mice | Culture Media, Serum-Free - pharmacology | Bone Morphogenetic Proteins - pharmacology | Stem Cell Factor - physiology | Vascular Endothelial Growth Factor A - pharmacology | Vascular Endothelial Growth Factor A - physiology | Drug Combinations | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2016, Volume 11, Issue 3, pp. e0150500 - e0150500
Neointima forming after stent implantation consists of vascular smooth muscle cells (VSMCs) in 90%. Growth factors TGF-β1, PDGFB, EGF, bFGF and VEGF-A play an... 
Coronary Artery Disease - surgery | Genetic Predisposition to Disease - genetics | Proto-Oncogene Proteins c-sis - genetics | Epidermal Growth Factor - physiology | Epidermal Growth Factor - genetics | Humans | Middle Aged | Transforming Growth Factor beta - physiology | Polymorphism, Single Nucleotide - physiology | Male | Coronary Restenosis - genetics | Vascular Endothelial Growth Factor A - genetics | Proto-Oncogene Proteins c-sis - physiology | Transforming Growth Factor beta - genetics | Fibroblast Growth Factor 2 - genetics | Coronary Artery Disease - genetics | Polymorphism, Single Nucleotide - genetics | Female | Aged | Fibroblast Growth Factor 2 - physiology | Stents - adverse effects | Vascular Endothelial Growth Factor A - physiology | Surgical implants | Angiography | Laboratories | Genomics | Genes | Metals | Stenosis | Heterozygotes | Smooth muscle | Cardiovascular disease | Arthritis | Gene polymorphism | Genotype & phenotype | Epidermal growth factor | Coding | Implants | Fibroblasts | Cardiology | Vascular endothelial growth factor | Growth factors | Heart diseases | Stents | Genotypes | Injury analysis | Fibroblast growth factor 2 | Medical research | Transforming growth factor-b1 | Coronary artery | Muscles | Implantation | Dentistry | Coronary artery disease | Patients | Homozygotes | Medicine | Polymerase chain reaction | Restenosis | Restriction fragment length polymorphism | Coronary vessels | Molecular biology | Polymorphism | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/2009, Volume 119, Issue 3, pp. 512 - 523
Pulmonary hypertension (PH) is a progressive, lethal lung disease characterized by pulmonary artery SMC (PA-SMC) hyperplasia. leading to right-sided heart... 
MEDICINE, RESEARCH & EXPERIMENTAL | INHIBITION | SUBENDOTHELIAL EXTRACELLULAR-MATRIX | FACTOR-2 | GENE-EXPRESSION | RATS | MONOCROTALINE | FIBROBLAST-GROWTH-FACTOR | SMOOTH-MUSCLE-CELLS | LUNG-CANCER CELLS | FACTOR RECEPTOR | RNA, Small Interfering - genetics | Humans | Hypertension, Pulmonary - physiopathology | Fibroblast Growth Factor 1 - genetics | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Muscle, Smooth, Vascular - physiopathology | Hypertension, Pulmonary - prevention & control | Endothelium, Vascular - physiology | In Situ Hybridization | Cell Division | Fibroblast Growth Factor 1 - physiology | Fibroblast Growth Factor 2 - physiology | Muscle, Smooth, Vascular - physiology | Disease Models, Animal | RNA, Messenger - genetics | Cells, Cultured | Endothelium, Vascular - physiopathology | Gene Expression Regulation | Rats | Lung - physiopathology | Pulmonary Artery - physiopathology | Lung - physiology | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Disease Progression | Pulmonary Artery - physiology | Animals | Fibroblast Growth Factor 2 - genetics | Hypertension, Pulmonary - pathology | Physiological aspects | Development and progression | Fibroblast growth factors | Genetic aspects | Research | Pulmonary hypertension | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Biological Psychiatry, ISSN 0006-3223, 2016, Volume 80, Issue 6, pp. 479 - 489
Journal Article