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Nature (London), ISSN 1476-4687, 2015, Volume 526, Issue 7572, pp. 263 - 267
.... In late-stage colorectal cancer, the most commonly used targeted therapies are the monoclonal antibodies cetuximab and panitumumab, which prevent epidermal growth factor receptor (EGFR) activation(1... 
PANITUMUMAB | THERAPY | MULTIDISCIPLINARY SCIENCES | SEQUENCE | ACQUIRED-RESISTANCE | SOMATIC MUTATION | IDENTIFICATION | GENE COPY NUMBER | ACTIVATING MUTATIONS | BREAST | INSIGHTS | Receptor, Epidermal Growth Factor - genetics | Receptor, ErbB-2 - genetics | Proto-Oncogene Proteins p21(ras) - genetics | Colorectal Neoplasms - genetics | Genomics | Humans | Antibodies, Monoclonal - therapeutic use | Antineoplastic Agents - therapeutic use | Cetuximab - therapeutic use | Molecular Targeted Therapy | Receptor, Fibroblast Growth Factor, Type 1 - genetics | MAP Kinase Kinase 1 - genetics | Colorectal Neoplasms - drug therapy | Female | Antineoplastic Agents - pharmacology | Insulin Receptor Substrate Proteins - genetics | Colorectal Neoplasms - metabolism | Antibodies, Monoclonal - pharmacology | Cetuximab - pharmacology | DNA Copy Number Variations - genetics | Mutation - genetics | Genome, Human - genetics | Receptor, Epidermal Growth Factor - chemistry | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Exome - genetics | Animals | Receptor, Platelet-Derived Growth Factor alpha - genetics | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Mice | Drug Resistance, Neoplasm - drug effects | Care and treatment | Gene mutations | Colorectal cancer | Drug metabolism | Genetic aspects | Identification and classification | Health aspects | Proteins | Epidermal growth factor | Genes | Clinical trials | Mutation | Kinases | Cancer therapies | Tumors
Journal Article
PloS one, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e61426
.... Platelets can stimulate angiogenesis and promote gastric ulcer healing. We compared the expressions of proangiogenic growth factors and their receptors in the gastric... 
THERAPY | PLATELETS | MULTIDISCIPLINARY SCIENCES | INJURY | PREVALENCE | RELEASE | LIVER-DISEASE | PEPTIC-ULCER | PORTAL-HYPERTENSION | Demography | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Humans | Middle Aged | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Male | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Receptor, Platelet-Derived Growth Factor beta - genetics | Vascular Endothelial Growth Factor A - genetics | Platelet-Derived Growth Factor - genetics | Intercellular Signaling Peptides and Proteins - blood | Vascular Endothelial Growth Factor Receptor-2 - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Stomach Ulcer - complications | Angiogenesis Inducing Agents - metabolism | Fibroblast Growth Factor 2 - metabolism | Female | Stomach Ulcer - genetics | Liver Cirrhosis - genetics | Receptor, Platelet-Derived Growth Factor beta - metabolism | Vascular Endothelial Growth Factor Receptor-1 - genetics | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Liver Cirrhosis - complications | RNA, Messenger - genetics | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Receptors, Cell Surface - metabolism | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Platelet-Derived Growth Factor - metabolism | Platelet Count | Stomach Ulcer - blood | Fibroblast Growth Factor 2 - genetics | Receptor, Platelet-Derived Growth Factor alpha - genetics | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Receptors, Cell Surface - genetics | RNA | Peptic ulcer | Comparative analysis | Vascular endothelial growth factor | Health aspects | Liver cirrhosis | Growth factors | Fibroblast growth factor | Platelet-derived growth factor | Pathogenesis | Mucosa | mRNA | Angiogenesis | Receptors | Blood platelets | Rodents | Gastroenterology | Nonsteroidal anti-inflammatory drugs | Endoscopy | Fibroblast growth factor receptor 1 | Fibroblast growth factor receptor 2 | Deoxyribonucleic acid--DNA | Fibroblast growth factor 2 | Thrombocytopenia | Liver diseases | Patients | Medicine | Hospitals | Biopsy | Ulcers | Platelets | Deoxyribonucleic acid | DNA
Journal Article
The oncologist (Dayton, Ohio), ISSN 1549-490X, 2015, Volume 20, Issue 6, pp. 660 - 673
Journal Article
International journal of molecular sciences, ISSN 1661-6596, 09/2018, Volume 19, Issue 9, p. 2487
.... Signaling via vascular endothelial growth factor (VEGF)-C, VEGF-D, and VEGF receptor (VEGFR)-3 is a central molecular mechanism for lymphangiogenesis... 
Vascular endothelial growth factor-C | Lymphangiogenesis | Transforming growth factor-β | Fibrosis | TISSUE-GROWTH-FACTOR | TYPE-1 DIABETIC-PATIENTS | PERITONEAL MESOTHELIAL CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | vascular endothelial growth factor-C | MONOCLONAL-ANTIBODY | FACTOR RECEPTOR-3 | CHEMISTRY, MULTIDISCIPLINARY | SOLUTE TRANSPORT | transforming growth factor-beta | LYMPHATIC ABSORPTION RATE | ULTRAFILTRATION FAILURE | DIALYSIS PATIENTS | lymphangiogenesis | fibrosis | GLYCATION END-PRODUCTS | Epithelial Cells - metabolism | Humans | Peritoneal Fibrosis - genetics | Nephrogenic Fibrosing Dermopathy - metabolism | Renal Insufficiency, Chronic - metabolism | Lymphatic Vessels - metabolism | Peritoneal Fibrosis - metabolism | Renal Insufficiency, Chronic - genetics | Vascular Endothelial Growth Factor D - genetics | Macrophages - pathology | Signal Transduction | Gene Expression Regulation | Nephrogenic Fibrosing Dermopathy - genetics | Epithelial Cells - pathology | Nephrogenic Fibrosing Dermopathy - pathology | Peritoneal Fibrosis - pathology | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Vascular Endothelial Growth Factor C - genetics | Renal Insufficiency, Chronic - pathology | Macrophages - metabolism | Vascular Endothelial Growth Factor D - metabolism | Animals | Transforming Growth Factor beta - genetics | Vascular Endothelial Growth Factor C - metabolism | Lymphatic Vessels - pathology | Connective Tissue Growth Factor - genetics | Lymphangiogenesis - genetics | Transforming Growth Factor beta - metabolism | Connective Tissue Growth Factor - metabolism | Vascular Endothelial Growth Factor Receptor-3 - genetics | Lymphatic system | Epithelial cells | Transforming growth factor-a | Blood vessels | Connective tissue growth factor | Macrophages | Endothelial cells | Peritoneal dialysis | Vascular endothelial growth factor receptors | Connective tissues | Signal transduction | Signaling | Etiology | Capillaries | Vascular endothelial growth factor | Growth factors | Peritoneum | Nephrology | Homeostasis | Mitochondrial DNA | Proteins | Fibroblasts | Cytokines | Inflammation | Permeability | Diabetes | Kidney diseases | transforming growth factor-β
Journal Article
PloS one, ISSN 1932-6203, 2010, Volume 5, Issue 11, p. e14117
... EGFR is dominant for growth signaling, but not in cell lines where EGFR signaling is absent. A luciferase reporter containing... 
Fibroblast Growth Factor 7 - pharmacology | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Coculture Techniques | Humans | Lung Neoplasms - metabolism | Fibroblast Growth Factor 2 - pharmacology | Lung Neoplasms - pathology | Extracellular Signal-Regulated MAP Kinases - metabolism | Receptor, Fibroblast Growth Factor, Type 3 - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Fibroblasts - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Drug Resistance, Neoplasm - genetics | Signal Transduction - drug effects | Fibroblasts - drug effects | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Fibroblasts - cytology | Protein Kinase Inhibitors - pharmacology | Quinazolines - pharmacology | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Tyrosine | Phenols | Fibroblast growth factors | RNA | Analysis | Luciferase | Fibroblast growth factor | Biotechnology | Transcription | Gene regulation | Lung cancer | Biology | Kinases | Cell adhesion & migration | Morphogenesis | Epidermal growth factor | Fibroblast growth factor receptor 7 | Gefitinib | Protein-tyrosine kinase | Fibroblast growth factor receptor 2 | Fibroblast growth factor 2 | Epidermal growth factor receptors | Extracellular signal-regulated kinase | Non-small cell lung carcinoma | Gene expression | Src protein | Self sufficiency | Pulmonary hypertension | Studies | Signaling | Inhibitors | Monoclonal antibodies | Ligands | Mutation | Tumors | Fibroblast growth factor receptors
Journal Article
Annals of oncology, ISSN 0923-7534, 2014, Volume 25, Issue 3, pp. 552 - 563
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2012, Volume 109, Issue 45, pp. 18273 - 18280
During cardiogenesis, Fibroblast Growth Factor (Fgf10) is expressed in the anterior second heart field... 
Heart | Transgenic animals | Heart ventricles | Genes | Myocardium | Promoter regions | Transgenes | Embryos | Progenitor cells | Binding sites | Transcriptional regulation | Mouse embryo | TRANSCRIPTION FACTORS | CELLS | ARTERIAL POLE | MOUSE HEART | transcriptional regulation | MULTIDISCIPLINARY SCIENCES | OUTFLOW TRACT | mouse embryo | ENHANCER | ISL1 | DIFFERENTIATION | EXPRESSION | FGF10 | Protein Binding - genetics | LIM-Homeodomain Proteins - metabolism | Heart - embryology | Homeodomain Proteins - metabolism | Molecular Sequence Data | Chromosomes, Artificial, Bacterial - genetics | Gene Expression Regulation, Developmental | Base Pairing - genetics | Base Sequence | Myocardium - metabolism | Transcription, Genetic | Fibroblast Growth Factor 10 - genetics | Binding Sites | Fibroblast Growth Factor 10 - metabolism | Transgenes - genetics | Mice, Transgenic | Transcription Factors - genetics | T-Box Domain Proteins - genetics | Down-Regulation - genetics | Homeodomain Proteins - genetics | Regulatory Sequences, Nucleic Acid - genetics | T-Box Domain Proteins - metabolism | Transcription Factors - metabolism | Homeobox Protein Nkx-2.5 | LIM-Homeodomain Proteins - genetics | Animals | Genes, Switch - genetics | Models, Biological | Mice | Physiological aspects | Fibroblast growth factors | Genetic aspects | Research | Genetic transcription | Genetic regulation | Health aspects | Heart muscle | Regulatory Sequences, Nucleic Acid | Cellular Biology | Life Sciences | Chromosomes, Artificial, Bacterial | Genes, Switch | Down-Regulation | Base Pairing | Fibroblast Growth Factor 10 | Homeodomain Proteins | Protein Binding | LIM-Homeodomain Proteins | Transcription Factors | T-Box Domain Proteins | Biological Sciences
Journal Article
Nature communications, ISSN 2041-1723, 2011, Volume 2, Issue 1, p. 229
The fibroblast growth factor receptor 2-IIIb (FGFR2b) and the vascular endothelial growth factor receptor 2 (VEGFR2... 
FACTOR-RECEPTOR | LIGANDS | HB-EGF | MULTIDISCIPLINARY SCIENCES | ECTODOMAIN | KINASE | TACE | ALPHA-CONVERTING-ENZYME | MICE | POTENTIAL ROLE | KERATINOCYTE MIGRATION | ADAM17 Protein | Receptor, Epidermal Growth Factor - genetics | Phosphorylation | Cell Proliferation | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Humans | Transcriptional Activation | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor Receptor-2 - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Fibroblast Growth Factor 7 - metabolism | Receptor, Epidermal Growth Factor - metabolism | Transfection | Mitogen-Activated Protein Kinase 1 - genetics | src-Family Kinases - metabolism | Female | Fetus | p38 Mitogen-Activated Protein Kinases - metabolism | Endothelial Cells - physiology | Cell Line | Gene Expression | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Epidermal Growth Factor - genetics | Intercellular Signaling Peptides and Proteins - genetics | Vascular Endothelial Growth Factor Receptor-2 - metabolism | p38 Mitogen-Activated Protein Kinases - genetics | Epidermal Growth Factor - metabolism | Mice, Transgenic | Keratinocytes - cytology | Fibroblast Growth Factor 7 - genetics | Phosphatidylinositol 3-Kinases - genetics | Heparin-binding EGF-like Growth Factor | ADAM Proteins - metabolism | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Endothelial Cells - cytology | Mice | ADAM Proteins - genetics | src-Family Kinases - genetics | Fetal Blood | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Cell Movement | Keratinocytes - physiology | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2014, Volume 111, Issue 45, pp. E4869 - E4877
The human FGF receptors (FGFRs) play critical roles in various human cancers, and several FGFR inhibitors are currently under clinical investigation.... 
Structure-based drug design | Drug discovery | Cancer drug resistance | Kinase inhibitor | structure-based drug design | WILD-TYPE | MULTIDISCIPLINARY SCIENCES | BCR-ABL | GROWTH-FACTOR RECEPTORS | DRUG-RESISTANCE | kinase inhibitor | LUNG-CANCER | GENE FUSIONS | cancer drug resistance | SELECTIVE INHIBITOR | drug discovery | THERAPEUTIC TARGET | FACTOR RECEPTOR 4 | REGULATES PROLIFERATION | Receptor, Fibroblast Growth Factor, Type 4 - chemistry | Receptor, Fibroblast Growth Factor, Type 4 - metabolism | Receptor, Fibroblast Growth Factor, Type 2 - chemistry | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Humans | ErbB Receptors - genetics | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Crystallography, X-Ray | Structure-Activity Relationship | Mutation, Missense | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Protein Kinase Inhibitors - chemistry | Neoplasms - genetics | Receptor, Fibroblast Growth Factor, Type 2 - antagonists & inhibitors | Antineoplastic Agents - pharmacology | Receptor, Fibroblast Growth Factor, Type 4 - genetics | Binding Sites | ErbB Receptors - antagonists & inhibitors | ErbB Receptors - metabolism | Neoplasms - enzymology | Antineoplastic Agents - chemistry | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Neoplasms - drug therapy | Drug Resistance, Neoplasm - genetics | ErbB Receptors - chemistry | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Receptor, Fibroblast Growth Factor, Type 1 - chemistry | Neoplasms - pathology | Receptor, Fibroblast Growth Factor, Type 4 - antagonists & inhibitors | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Amino Acid Substitution | Drug Resistance, Neoplasm - drug effects | Biological Sciences | PNAS Plus
Journal Article