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Scientific Reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 368 - 15
Regenerative therapy to replace missing teeth is a critical area of research. Functional bioengineered teeth have been produced by the organ germ method using... 
IGF-I | STEM-CELLS | HORMONE | PROTEIN | MULTIDISCIPLINARY SCIENCES | DISTINCT | FACTOR-I | BONE | EXPRESSION | ORGAN | ENAMEL KNOT | Morphogenesis | Cell proliferation | Fibroblast growth factor | Germ cells | Dental enamel | Mesenchyme | Insulin-like growth factor I | Enamel | Teeth | Insulin-like growth factors | Insulin | Fibroblast growth factor 4
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2016, Volume 22, Issue 7, pp. 800 - 806
Type 2 diabetes (T2D) is among the most common and costly disorders worldwide(1). The goal of current medical management for T2D is to transiently ameliorate... 
MEDICINE, RESEARCH & EXPERIMENTAL | HOMEOSTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MODEL | SUPPRESSION | ASTROCYTES | BALANCE | CELL BIOLOGY | INSULIN | TANYCYTES | METABOLISM | INTRAVENOUS GLUCOSE-TOLERANCE | IN-VIVO | Body Composition | Ependymoglial Cells - metabolism | Injections, Intraventricular | Male | Muscle, Skeletal - metabolism | Diabetes Mellitus, Type 2 - metabolism | Neoplasm Proteins - metabolism | Deoxyglucose | Neoplasm Proteins - drug effects | Brain - metabolism | Adipose Tissue - metabolism | Receptor, Insulin - genetics | Diet, High-Fat | Muscle, Skeletal - drug effects | Myocardium - metabolism | Hypothalamus - drug effects | Diabetes Mellitus, Experimental - metabolism | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Glucose Tolerance Test | Heat-Shock Proteins - drug effects | Heat-Shock Proteins - metabolism | Liver - metabolism | Proto-Oncogene Proteins c-fos - metabolism | Rats | Ependymoglial Cells - drug effects | Receptor, Insulin - antagonists & inhibitors | Remission Induction | Blotting, Western | Proto-Oncogene Proteins c-fos - drug effects | Mice, Knockout | Blood Glucose - drug effects | Brain - drug effects | Hyperglycemia - metabolism | Rats, Zucker | Animals | Hypothalamus - metabolism | Hypothalamus - cytology | Fibroblast Growth Factor 1 - pharmacology | Heart - drug effects | Mice, Obese | Mice | Blood Glucose - metabolism | Adipose Tissue - drug effects | Forkhead Box Protein O1 - genetics | Carbon Radioisotopes | Type 2 diabetes | Molecular targeted therapy | Care and treatment | Innovations | Development and progression | Genetic aspects | Fibroblast growth factors | Health aspects | Fibroblasts | Diabetes | Drug therapy | Rodents | Index Medicus | brain | diabetes | fibroblast growth factor
Journal Article
Kidney International, ISSN 0085-2538, 01/2018, Volume 93, Issue 1, pp. 95 - 109
Inflammation plays a central role in the etiology of diabetic nephropathy, a global health issue. We observed a significant reduction in the renal expression... 
cytokines | fibrosis | inflammation | mesangial cells | diabetic nephropathy | PATHWAYS | ACTIVATION | JNK | KINASE | FIBROBLAST-GROWTH-FACTOR | PATHOGENESIS | OBESITY | INSULIN-RESISTANCE | UROLOGY & NEPHROLOGY | NF-KAPPA-B | EXPRESSION | Diabetes Mellitus, Experimental - drug therapy | Kidney - pathology | Rats, Wistar | Diabetic Nephropathies - etiology | Humans | JNK Mitogen-Activated Protein Kinases - metabolism | Male | NF-kappa B - metabolism | Diabetes Mellitus, Type 1 - complications | Diabetic Nephropathies - blood | Diabetes Mellitus, Experimental - blood | Kidney - metabolism | Inflammation Mediators - metabolism | Diabetes Mellitus, Experimental - complications | Diabetic Nephropathies - prevention & control | Diabetes Mellitus, Type 2 - complications | Cell Line | Diabetic Nephropathies - pathology | Kidney - drug effects | Cytokines - metabolism | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Recombinant Proteins - pharmacology | Diabetes Mellitus, Type 1 - drug therapy | Blood Glucose - drug effects | Fibroblast Growth Factor 1 - blood | Diabetes Mellitus, Type 2 - blood | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Diabetes Mellitus, Type 1 - blood | Fibroblast Growth Factor 1 - pharmacology | Macrophages - drug effects | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Index Medicus
Journal Article
Journal Article
by Chen, LL and Li, XH and Wang, YY and Song, TT and Li, HT and Xie, LB and Li, LC and Chen, XW and Ma, LK and Chen, Y and Lv, Y and Li, XW and Ge, RS
FRONTIERS IN ENDOCRINOLOGY, ISSN 1664-2392, 03/2019, Volume 10, pp. 118 - 118
Fibroblast growth factor 1 (FGF1) is reported to be expressed in the testis. How FGF1 affects stem Leydig cell development remains unclear. Here, we report the... 
differentiation | proliferation | testosterone | SELECTIVE DESTRUCTION | RECEPTOR | FIBROBLAST-GROWTH-FACTOR | IDENTIFICATION | GLUCOSE-HOMEOSTASIS | fibroblast growth factor 1 | ENDOCRINOLOGY & METABOLISM | SULFONATE EDS | EXPRESSION | stem leydig cell | Fibroblast growth factors | Research | Gene expression | Leydig cells | Stem cells
Journal Article
DIABETES, ISSN 0012-1797, 07/2019, Volume 68, Issue 7, pp. 1462 - 1472
Fibroblast growth factor 1 (FGF1) has been shown to reverse hyperglycemia in diabetic rodent models through peripheral and central administration routes.... 
INJECTION | GROWTH-FACTOR FAMILY | BETA-CELLS | ENDOCRINOLOGY & METABOLISM | SERUM | CEREBROSPINAL-FLUID | FGF1 | Fibroblast growth factor | Animal models | Nutrient deficiency | Fibroblast growth factor 1 | Secretion | Diabetes mellitus | Glucose | Insulin | Cell density | Hyperglycemia | Food intake | Fibroblasts | Diabetes | Pancreas | Growth factors | Structure-function relationships
Journal Article
Journal Article
CIRCULATION-ARRHYTHMIA AND ELECTROPHYSIOLOGY, ISSN 1941-3149, 05/2017, Volume 10, Issue 5
Background TGF-(1) (transforming growth factor-(1)) importantly contributes to cardiac fibrosis by controlling differentiation, migration, and collagen... 
MIGRATION | gap junctions | VENTRICULAR FIBROBLASTS | CARDIAC & CARDIOVASCULAR SYSTEMS | ATRIAL | myofibroblast | FIBROBLAST-MYOCYTE INTERACTIONS | RECEPTOR | PROLIFERATION | myocytes | transforming growth factors | fibroblasts | CONTRACTION | ion channel | ION CHANNELS | DIFFERENTIATION | K+ CHANNELS | cardiac
Journal Article