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Molecular and cellular biology, ISSN 0270-7306, 2007, Volume 27, Issue 13, pp. 4953 - 4967
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
CELLS | OXIDATIVE STRESS | ACTIVATED PROTEIN-KINASE | PGC-1-ALPHA | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | GENE-EXPRESSION | TRANSCRIPTIONAL COACTIVATOR | DIFFERENTIATION | MICROARRAY DATA | TRANSGENIC MICE | CELL BIOLOGY | Gene Expression Regulation, Enzymologic - drug effects | Acetyltransferases - metabolism | Multienzyme Complexes - metabolism | Adipocytes - drug effects | Glucose Intolerance | AMP-Activated Protein Kinases | Oxidative Phosphorylation - drug effects | Adipose Tissue, White - cytology | Body Composition - drug effects | Isoenzymes - metabolism | Adenosine Triphosphate - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Trans-Activators - genetics | Food Deprivation | p38 Mitogen-Activated Protein Kinases - metabolism | Homeostasis - drug effects | Phosphorylation - drug effects | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Isoenzymes - genetics | Hydrogen Peroxide - pharmacology | Protein-Serine-Threonine Kinases - genetics | Mitochondria - metabolism | Multienzyme Complexes - genetics | Macrophages - cytology | Mitochondria - drug effects | Feeding Behavior - drug effects | Polyamines - metabolism | Macrophages - metabolism | Organ Size - drug effects | Animals | Adipocytes - metabolism | Fibroblasts - drug effects | Adipose Tissue, White - enzymology | Adipose Tissue, White - growth & development | Glucose - metabolism | Macrophages - drug effects | Trans-Activators - metabolism | Mice | Transcription Factors | Adipose Tissue, White - drug effects | Energy Metabolism - drug effects | White/cytology/drug effects/enzymology/growth | Hydrogen Peroxide/pharmacology | Macrophages/cytology/drug effects/metabolism | Mitochondria/drug effects/metabolism | Fibroblasts/drug effects/metabolism | p38 Mitogen-Activated Protein Kinases/metabolism | MEDICIN OCH HÄLSOVETENSKAP | Homeostasis/drug effects | Multienzyme Complexes/genetics/metabolism | Protein-Serine-Threonine Kinases/genetics/metabolism | Trans-Activators/genetics/metabolism | Enzymologic/drug effects | Glucose/metabolism | Adipose Tissue | Feeding Behavior/drug effects | Organ Size/drug effects | Oxidative Phosphorylation/drug effects | MEDICAL AND HEALTH SCIENCES | development | Acetyltransferases/metabolism | Adipocytes/drug effects/metabolism | Gene Expression Regulation | Adenosine Triphosphate/metabolism | Body Composition/drug effects | Polyamines/metabolism | Energy Metabolism/drug effects | Isoenzymes/genetics/metabolism | Phosphorylation/drug effects
Journal Article
Autophagy, ISSN 1554-8627, 09/2011, Volume 7, Issue 9, pp. 1045 - 1051
Multiple stress pathways result in the induction of autophagy and apoptosis. Current methods (e.g., protein gel blot, microscopy) do not offer quantitative... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Autophagy | Multispectral imaging cytometry | Influenza | New methodology | Apoptosis | influenza | CROSS-TALK | new methodology | autophagy | BECLIN 1 | multispectral imaging cytometry | apoptosis | CELL-DEATH | CELL BIOLOGY | Leukocytes, Mononuclear - metabolism | Image Cytometry - methods | Apoptosis - drug effects | Apoptosis - radiation effects | Autophagy - radiation effects | Microtubule-Associated Proteins - metabolism | Humans | Autophagy - drug effects | Lysosomes - radiation effects | Chloroquine - pharmacology | Lysosomes - metabolism | Ultraviolet Rays | Time Factors | Leukocytes, Mononuclear - radiation effects | Signal Transduction - radiation effects | Fibroblasts - metabolism | Lysosomes - drug effects | Leukocytes, Mononuclear - drug effects | Animals | Fibroblasts - radiation effects | Signal Transduction - drug effects | Embryo, Mammalian - cytology | Fibroblasts - drug effects | Leukocytes, Mononuclear - cytology | Fibroblasts - cytology | Mice | Autophagy/radiation effects | Signal Transduction/radiation effects | Image Cytometry/methods | Autophagy/drug effects | Life Sciences | Lysosomes/drug effects | Immunology | Fibroblasts/metabolism | Leukocytes, Mononuclear/radiation effects | Fibroblasts/drug effects | Leukocytes, Mononuclear/metabolism | Apoptosis/radiation effects | Leukocytes, Mononuclear/drug effects | Fibroblasts/cytology | Apoptosis/drug effects | Fibroblasts/radiation effects | Lysosomes/metabolism | Leukocytes, Mononuclear/cytology | Signal Transduction/drug effects | Chloroquine/pharmacology | Embryo, Mammalian/cytology | Lysosomes/radiation effects | Microtubule-Associated Proteins/metabolism
Journal Article
Journal Article
Journal of Endodontics, ISSN 0099-2399, 2014, Volume 40, Issue 3, pp. 387 - 392
.... We aimed to evaluate the effect of these biomaterials on odontogenic differentiation of human dental pulp stem cells (DPSCs... 
Endocrinology & Metabolism | Dentistry | differentiation | calcium-enriched mixture | stem cell | pulp regeneration | mineral trioxide aggregate | CEM cement | human | dental pulp capping | Biomaterials | ENRICHED MIXTURE CEMENT | TREATMENT OUTCOMES | TISSUE | PULPOTOMY | PERMANENT MOLARS | IN-VITRO | MATRIX PROTEIN-1 | DENTISTRY, ORAL SURGERY & MEDICINE | HUMAN ODONTOBLASTS | IRREVERSIBLE PULPITIS | Bone Morphogenetic Protein 2 - drug effects | Phosphoproteins - drug effects | Humans | Sialoglycoproteins - drug effects | Transforming Growth Factor beta1 - drug effects | Cell Culture Techniques | Calcium Compounds - pharmacology | Fibroblast Growth Factor 4 - drug effects | Silicates - pharmacology | Microscopy, Electron, Scanning | Bone Morphogenetic Protein 4 - drug effects | Calcification, Physiologic - drug effects | Odontogenesis - drug effects | Dentinogenesis - drug effects | Cells, Cultured | Dental Pulp - cytology | Cementogenesis - drug effects | Biocompatible Materials - pharmacology | Cell Adhesion - drug effects | Extracellular Matrix Proteins - drug effects | Cell Shape - drug effects | Oxides - pharmacology | Cytokines - drug effects | Cell Differentiation - drug effects | Stem Cells - drug effects | Root Canal Filling Materials - pharmacology | Silicate Cement - pharmacology | Aluminum Compounds - pharmacology | Cell Proliferation - drug effects | Drug Combinations | Biological products | Cytokines | Gene expression | Stem cells
Journal Article
Biomaterials, ISSN 0142-9612, 2011, Volume 32, Issue 32, pp. 8161 - 8171
... healing, cartilage/bone regeneration and the sustained release of drugs due to similar physical properties to natural tissue [1–6] . Numerous researchers... 
Advanced Basic Science | Dentistry | Bone tissue regeneration | Hydrogel | Hyaluronic acid | Photopolymerization | Simvastatin | NANOPARTICLES | CELLS | IN-VITRO | MATERIALS SCIENCE, BIOMATERIALS | ENGINEERING, BIOMEDICAL | Cell Death - radiation effects | Methacrylates - chemistry | RNA, Messenger - metabolism | Simvastatin - pharmacology | Spectroscopy, Fourier Transform Infrared | Tissue Scaffolds - chemistry | Polymerization - drug effects | Light | Surface Properties - drug effects | Methacrylates - chemical synthesis | Rheology - radiation effects | Cell Death - drug effects | Surface Properties - radiation effects | Osteogenesis - genetics | Bone Regeneration - drug effects | Cell Line | Cell Survival - drug effects | Rabbits | Calcification, Physiologic - drug effects | Polymerization - radiation effects | Osteogenesis - drug effects | RNA, Messenger - genetics | Hyaluronic Acid - chemistry | Calcification, Physiologic - radiation effects | Osteogenesis - radiation effects | Reverse Transcriptase Polymerase Chain Reaction | Cell Survival - radiation effects | Animals | Fibroblasts - radiation effects | Fibroblasts - drug effects | Cell Proliferation - drug effects | Fibroblasts - cytology | Mice | Kinetics | Hydrogels - chemistry | Rheology - drug effects | Cell Proliferation - radiation effects | Biological products | Antilipemic agents | Biomedical engineering
Journal Article
Cell metabolism, ISSN 1550-4131, 2017, Volume 26, Issue 3, pp. 493 - 508.e4
Type 2 cytokines are important signals triggering biogenesis of thermogenic beige adipocytes in white adipose tissue (WAT) during cold acclimation. However,... 
adipose remodeling | macrophages | adipocyte precursor cells | beiging | adaptive thermogenesis | eotaxin | eosinophils | type 2 immunity | ANTIDIABETIC ACTIONS | CELLS | ENERGY-EXPENDITURE | ADIPOCYTES | ADIPONECTIN | EOSINOPHILS | ENDOCRINOLOGY & METABOLISM | INSULIN SENSITIVITY | EOTAXIN | GROWTH-FACTOR 21 | FGF21 | CELL BIOLOGY | Eosinophils - metabolism | Eosinophils - drug effects | Sympathetic Nervous System - drug effects | Adaptation, Physiological - drug effects | Glucuronidase - metabolism | Adipose Tissue, White - metabolism | Adipocytes - cytology | Male | Autocrine Communication - drug effects | Stem Cells - cytology | Adipocytes - drug effects | Extracellular Signal-Regulated MAP Kinases - metabolism | Stem Cells - metabolism | Sympathetic Nervous System - metabolism | Immunity - drug effects | Fibroblast Growth Factors - metabolism | Cold Temperature | Chemokine CCL11 - metabolism | Mice, Inbred C57BL | Fibroblast Growth Factors - deficiency | Recombinant Proteins - pharmacology | Mice, Knockout | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Adipocytes - metabolism | Stem Cells - drug effects | Adipose Tissue, Beige - drug effects | Macrophages - drug effects | Cell Proliferation - drug effects | Thermogenesis - drug effects | Adipose Tissue, Beige - metabolism | Adipose Tissue, White - drug effects | Adipose tissues | Fibroblast growth factors | Immune response | Macrophages | Pharmaceutical biotechnology
Journal Article
International Endodontic Journal, ISSN 0143-2885, 05/2012, Volume 45, Issue 5, pp. 401 - 412
Seifrtová M, Havelek R, Ćmielová J, Jiroutová A, Soukup T, Brůčková L, Mokrý J, English D, Řezáčová M. The response of human ectomesenchymal dental pulp stem... 
apoptosis | Mitogen‐activated protein kinases | human dermal fibroblasts | cisplatin | dental pulp stem cells | p53 | Human dermal fibroblasts | Dental pulp stem cells | Cisplatin | Apoptosis | Mitogen-activated protein kinases | P53 | CANCER-CELLS | LONG-TERM CULTURE | OVARIAN-CARCINOMA CELLS | INDUCED APOPTOSIS | SENSITIVITY | SPONTANEOUS DIFFERENTIATION | CHEMOTHERAPEUTIC-AGENTS | DNA | DENTISTRY, ORAL SURGERY & MEDICINE | ACTIVATED PROTEIN-KINASES | Mutagens - toxicity | Phosphorylation | Skin - cytology | Cyclin-Dependent Kinase Inhibitor p21 - drug effects | Ectoderm - drug effects | Extracellular Signal-Regulated MAP Kinases - drug effects | Apoptosis - drug effects | Humans | Caspase 9 - drug effects | Caspase 8 - drug effects | Cisplatin - toxicity | Caspase 3 - drug effects | Cell Culture Techniques | JNK Mitogen-Activated Protein Kinases - drug effects | Cell Survival - drug effects | Mesenchymal Stromal Cells - drug effects | Caspase 7 - drug effects | Cytostatic Agents - toxicity | Dental Pulp - cytology | Dental Pulp - drug effects | Enzyme Activation - drug effects | Tumor Suppressor Protein p53 - drug effects | Ectoderm - cytology | p38 Mitogen-Activated Protein Kinases - drug effects | MAP Kinase Signaling System - drug effects | Serine - drug effects | Fibroblasts - drug effects | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Cell Cycle - drug effects | Skin - drug effects | Proteins | Antimitotic agents | Analysis | Stem cells | Universities and colleges | Antineoplastic agents | Tumor proteins | Protein kinases | Phosphotransferases
Journal Article
PloS one, ISSN 1932-6203, 2010, Volume 5, Issue 5, p. e10431
... their relevance for pre-clinical drug discovery, disease modeling and basic research. Primary and non-transformed prostate epithelial cells, but also several PrCa lines, formed well... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | GENE-EXPRESSION SIGNATURE | STEM-CELLS | MAMMARY EPITHELIA | METASTASIS | BIOLOGY | TUMOR-CELL INVASION | DIFFERENTIATION | CULTURE MODEL | LINES | Laminin - pharmacology | Epithelial Cells - drug effects | Humans | Mesoderm - drug effects | Spheroids, Cellular - pathology | Male | Antineoplastic Agents - therapeutic use | Phosphatidylinositol 3-Kinases - metabolism | Spheroids, Cellular - enzymology | Neoplasm Proteins - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | RNA, Messenger - metabolism | Prostate - pathology | Prostatic Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Prostate - drug effects | Antineoplastic Agents - pharmacology | Collagen - pharmacology | Spheroids, Cellular - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Proto-Oncogene Proteins c-akt - metabolism | Principal Component Analysis | Prostatic Neoplasms - drug therapy | Epithelium - drug effects | Prostatic Neoplasms - pathology | Epithelium - pathology | Neoplasm Invasiveness | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | RNA, Messenger - genetics | Epithelial Cells - pathology | Cell Shape - drug effects | Phenotype | Proteoglycans - pharmacology | Signal Transduction - drug effects | Models, Biological | Prostatic Neoplasms - enzymology | Cell Proliferation - drug effects | TOR Serine-Threonine Kinases | Cell Transformation, Neoplastic - pathology | Mesoderm - pathology | Drug Combinations | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Epigenetic inheritance | Growth | Oncology, Experimental | Genes | Research | Gene expression | Ionizing radiation | Stem cells | Physiological aspects | Models | Drug discovery | Drug therapy | Prostate cancer | Integrins | Cancer | Cell culture | Biotechnology | Transformation | Motility | Leukocyte migration | Mesenchyme | Epithelial cells | Homeostasis | AKT protein | Metastasis | Drug resistance | Tissues | Ovarian cancer | Cell adhesion & migration | Metastases | Rodents | Fibroblasts | Extracellular matrix | Basal lamina | Lipid metabolism | Medical research | Invasiveness | Phenotypic plasticity | Tumor cell lines | Metabolism | Spheroids | 1-Phosphatidylinositol 3-kinase | Signaling | Interferon | Three dimensional models | Prostate | Cell migration
Journal Article
Aging cell, ISSN 1474-9718, 2015, Volume 14, Issue 4, pp. 644 - 658
...‐related chronic diseases. Here, we describe the rationale for identification and validation of a new class of drugs termed senolytics, which selectively kill senescent cells... 
dasatinib | plasminogen‐activated inhibitor | dependence receptors | quercetin | ephrins | PI3K delta | p21 | Dasatinib | Dependence receptors | Ephrins | Plasminogen-activated inhibitor | Quercetin | P21 | ENDOTHELIAL DYSFUNCTION | PLASMINOGEN-ACTIVATOR INHIBITOR-1 | EXPRESSION PROFILES | plasminogen-activated inhibitor | CELLULAR SENESCENCE | CANCER-THERAPY | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | LUNG-CANCER | SET ENRICHMENT ANALYSIS | GENE-EXPRESSION | IONIZING-RADIATION | TUMOR-GROWTH | Carotid Arteries - drug effects | Endonucleases - genetics | Plasminogen Activator Inhibitor 2 - genetics | Transcriptome | Gene Expression Profiling | Adipocytes - drug effects | Aging - genetics | Osteoporosis - metabolism | Ephrins - metabolism | Plasminogen Activator Inhibitor 2 - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Osteoporosis - genetics | Intervertebral Disc - pathology | Fibroblasts - metabolism | Endothelial Cells - metabolism | Intervertebral Disc - chemistry | Fibroblasts - pathology | Mesenchymal Stem Cells - pathology | Mice, Knockout | Dasatinib - pharmacology | Osteoporosis - pathology | Ephrins - genetics | Fibroblasts - drug effects | Mice | Endothelial Cells - pathology | bcl-X Protein - metabolism | Aging - metabolism | Aging - drug effects | bcl-X Protein - genetics | Cellular Senescence - drug effects | Phosphatidylinositol 3-Kinases - metabolism | Endonucleases - metabolism | DNA-Binding Proteins - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Mesenchymal Stem Cells - drug effects | Mesenchymal Stem Cells - metabolism | Heart - physiopathology | Cellular Senescence - genetics | Osteoporosis - prevention & control | DNA-Binding Proteins - genetics | Adipocytes - pathology | Aging - pathology | Phosphatidylinositol 3-Kinases - genetics | Animals | Quercetin - pharmacology | Adipocytes - metabolism | Heart - drug effects | Carotid Arteries - pathology | Intervertebral Disc - drug effects | Drug Combinations | Endothelial Cells - drug effects | Original
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2015, Volume 24, Issue 2, pp. 315 - 324
Summary Objective The aetiology of OA is not fully understood although several adipokines such as leptin are known mediators of disease progression. Since... 
Rheumatology | Smad1 | Human chondrocytes | Hip osteoarthritis | β-Catenin | Dedifferentiation | Leptin | COLLAGEN | RHEUMATOLOGY | DIFFERENTIATED PHENOTYPE | HYPERTROPHY | IN-VITRO | SYNOVIAL FIBROBLASTS | HUMAN OSTEOARTHRITIC CARTILAGE | SIGNALING PATHWAY | beta-Catenin | ARTICULAR CHONDROCYTES | ORTHOPEDICS | KNEE OSTEOARTHRITIS | EXPRESSION | Receptors, Transforming Growth Factor beta - genetics | Humans | Leptin - metabolism | Middle Aged | Male | Lymphotoxin-alpha - metabolism | RNA, Messenger - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Lymphotoxin-alpha - genetics | Cartilage, Articular - metabolism | Smad5 Protein - metabolism | Smad2 Protein - genetics | Aged, 80 and over | Smad2 Protein - drug effects | Protein-Serine-Threonine Kinases - metabolism | Collagen Type X - drug effects | SOX9 Transcription Factor - metabolism | Matrix Metalloproteinase 13 - genetics | Smad2 Protein - metabolism | Prednisolone - pharmacology | beta Catenin - metabolism | Matrix Metalloproteinase 13 - metabolism | Glycogen Synthase Kinase 3 - genetics | Receptors, Transforming Growth Factor beta - drug effects | Activin Receptors, Type II - genetics | Protein-Serine-Threonine Kinases - drug effects | Activin Receptors, Type II - metabolism | Osteoarthritis, Hip - metabolism | Glycogen Synthase Kinase 3 - drug effects | Activin Receptors, Type II - drug effects | Chondrocytes - drug effects | Collagen Type X - metabolism | beta Catenin - drug effects | Smad1 Protein - genetics | Adult | Female | Smad5 Protein - genetics | Chondrocytes - metabolism | RNA, Messenger - drug effects | Cell Dedifferentiation - drug effects | Cartilage, Articular - cytology | Lymphotoxin-alpha - drug effects | Protein-Serine-Threonine Kinases - genetics | Collagen Type X - genetics | SOX9 Transcription Factor - drug effects | Matrix Metalloproteinase 13 - drug effects | Glycogen Synthase Kinase 3 - metabolism | beta Catenin - genetics | Osteoarthritis, Hip - genetics | Smad5 Protein - drug effects | Core Binding Factor Alpha 1 Subunit - drug effects | Wnt Signaling Pathway - drug effects | Receptors, Transforming Growth Factor beta - metabolism | Wnt Signaling Pathway - genetics | Glucocorticoids - pharmacology | Smad1 Protein - metabolism | Aged | Smad1 Protein - drug effects | Cartilage, Articular - drug effects | In Vitro Techniques | Cell Dedifferentiation - genetics | Core Binding Factor Alpha 1 Subunit - genetics
Journal Article
Nature communications, ISSN 2041-1723, 2018, Volume 9, Issue 1, pp. 4728 - 16
...ARTICLE The critical role of AMPK in driving Akt activation under stress, tumorigenesis and drug resistance Fei Han1,2, Chien-Feng Li3,4, Zhen Cai1,2, Xian... 
OXIDATIVE STRESS | ENERGY STRESS | PHOSPHORYLATION | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | GROWTH-FACTOR | GLUCOSE DEPRIVATION | CELL-SURVIVAL | CANCER PROGRESSION | PC12 CELLS | HYPOXIA | Human Umbilical Vein Endothelial Cells - metabolism | Calcium - metabolism | Humans | Carcinogenesis - metabolism | Glycolysis - drug effects | Neovascularization, Pathologic - pathology | Ubiquitination - drug effects | Adenylate Kinase - metabolism | Stress, Physiological - drug effects | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Fibroblasts - metabolism | Cell Survival - drug effects | ErbB Receptors - metabolism | Enzyme Activation - drug effects | Phosphoserine - metabolism | S-Phase Kinase-Associated Proteins - metabolism | Disease Progression | Carcinogenesis - drug effects | Carcinogenesis - pathology | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Fibroblasts - drug effects | Calcium-Calmodulin-Dependent Protein Kinase Kinase - metabolism | Cell Line, Tumor | Mice | Epidermal Growth Factor - pharmacology | Drug Resistance, Neoplasm - drug effects | Stresses | Phosphorylation | Animal models | Cell survival | AKT protein | Activation | Breast cancer | Glucose | Drug resistance | 1-Phosphatidylinositol 3-kinase | Ubiquitination | Epidermal growth factor | Skp2 protein | Calcium-binding protein | Tumorigenesis | Growth factors | Calmodulin | Ubiquitin-protein ligase | Calcium ions | Cancer | Tumors
Journal Article