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Nature Immunology, ISSN 1529-2908, 05/2012, Volume 13, Issue 5, pp. 499 - 510
Journal Article
Infection and Immunity, ISSN 0019-9567, 12/2015, Volume 83, Issue 12, pp. 4740 - 4749
Interferon (IFN)-inducible guanylate binding proteins (GBPs) mediate cell-autonomous host resistance to bacterial pathogens and promote inflammasome... 
IFN | AIM2 INFLAMMASOME | IMMUNITY | INFECTIOUS DISEASES | CASPASE-1 | SUSCEPTIBILITY | TRACHOMATIS | INNATE | PYROPTOSIS | IMMUNOLOGY | HOST-RESISTANCE | PROTECTS | Interleukin-18 - immunology | Caspases - immunology | NLR Family, Pyrin Domain-Containing 3 Protein | Chlamydia trachomatis - immunology | Interleukin-1beta - genetics | GTP-Binding Proteins - genetics | Chlamydia muridarum - immunology | Chlamydia Infections - immunology | Chlamydia Infections - pathology | Vacuoles - immunology | Chlamydia Infections - genetics | Inclusion Bodies - microbiology | Fibroblasts - microbiology | Interferon-gamma - genetics | Carrier Proteins - immunology | Macrophages - immunology | Macrophages - microbiology | Chlamydia trachomatis - genetics | Inclusion Bodies - immunology | Vacuoles - microbiology | DNA-Binding Proteins - immunology | GTP-Binding Proteins - immunology | Signal Transduction | Caspases - genetics | Mice, Inbred C57BL | Chlamydia muridarum - genetics | Gene Expression Regulation | Interleukin-1beta - immunology | DNA-Binding Proteins - genetics | Inflammasomes - genetics | Mice, Knockout | Host-Pathogen Interactions | Carrier Proteins - genetics | Animals | Chlamydia Infections - microbiology | Chlamydia muridarum - pathogenicity | Inflammasomes - immunology | Interferon-gamma - immunology | Chlamydia trachomatis - pathogenicity | Fibroblasts - immunology | Mice | Primary Cell Culture | Interleukin-18 - genetics | Index Medicus | Cellular Microbiology | Pathogen-Host Cell Molecular Interactions | Spotlight
Journal Article
Immunity, ISSN 1074-7613, 09/2015, Volume 43, Issue 3, pp. 475 - 487
Interleukin-17 (IL-17) induces pathology in autoimmunity and infections; therefore, constraint of this pathway is an essential component of its regulation. We... 
negative regulation | Regnase-1 | fungal immunity | autoimmunity | signal transduction | IL-17 | Autoimmunity | Signal transduction | Fungal immunity | Negative regulation | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | IL-17 RECEPTOR | TARGET GENES | CANDIDA-ALBICANS | C/EBP-BETA | GENE-EXPRESSION | HOST-DEFENSE | NECROSIS-FACTOR-ALPHA | MESSENGER-RNA STABILITY | T-CELL | IMMUNOLOGY | Candidiasis - genetics | Oncogene Proteins - genetics | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Immunoblotting | Male | Receptors, Interleukin-17 - genetics | Ribonucleases - metabolism | Host-Pathogen Interactions - immunology | Inflammation - metabolism | Lipocalins - genetics | Adaptor Proteins, Signal Transducing - immunology | Pneumonia - immunology | Candida albicans - physiology | Encephalomyelitis, Autoimmune, Experimental - genetics | Receptors, Interleukin-17 - metabolism | Interleukin-6 - metabolism | Pneumonia - genetics | Interleukin-6 - genetics | Oncogene Proteins - metabolism | Signal Transduction - genetics | Candida albicans - immunology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Interleukin-17 - metabolism | Lipocalin-2 | Lipocalins - immunology | Interleukin-6 - immunology | Pneumonia - metabolism | Ribonucleases - genetics | Encephalomyelitis, Autoimmune, Experimental - metabolism | Receptors, Interleukin-17 - immunology | Interleukin-17 - immunology | Acute-Phase Proteins - genetics | Candidiasis - microbiology | Signal Transduction - immunology | HEK293 Cells | Female | Candidiasis - immunology | Nuclear Proteins - genetics | Oncogene Proteins - immunology | Lipocalins - metabolism | Cell Line | Acute-Phase Proteins - metabolism | Mice, Inbred C57BL | Cells, Cultured | Nuclear Proteins - metabolism | Inflammation - immunology | Nuclear Proteins - immunology | Animals | Ribonucleases - immunology | Adaptor Proteins, Signal Transducing - genetics | Inflammation - genetics | Adaptor Proteins, Signal Transducing - metabolism | Acute-Phase Proteins - immunology | Medical colleges | Nucleases | Interleukins | RNA | Cellular signal transduction | Inflammation | Proteins | Cytokines | Rodents | Fibroblasts | Infections | Kinases | Laboratory animals | Gene expression | Experiments | Index Medicus
Journal Article
Arthritis Research and Therapy, ISSN 1478-6354, 10/2013, Volume 15, Issue 5, pp. R168 - R168
Introduction: B lymphocytes might play a pathogenic role in dermal fibrosis in systemic sclerosis (SSc). B-cell activating factor (BAFF), a key cytokine for... 
BAFF | ADHESION | SCLERODERMA | TIMP-1 | AUTOIMMUNITY | SKIN FIBROSIS | DEPLETION | MECHANISMS | RHEUMATOLOGY | INTERLEUKIN-6 | IL-6 | Gene Expression - drug effects | Chemokine CCL2 - immunology | Skin - metabolism | Antibodies, Anti-Idiotypic - pharmacology | Coculture Techniques | Humans | Transforming Growth Factor beta1 - metabolism | Actins - metabolism | Matrix Metalloproteinase 9 - immunology | Fibrosis - metabolism | Actins - immunology | Immunoglobulin M - immunology | Actins - genetics | Transforming Growth Factor beta1 - immunology | Matrix Metalloproteinase 9 - genetics | Fibrosis - immunology | Gene Expression - immunology | B-Cell Activating Factor - immunology | Chemokine CCL2 - metabolism | Collagen - genetics | Interleukin-6 - metabolism | B-Lymphocytes - metabolism | Skin - pathology | Fibroblasts - metabolism | Skin - immunology | B-Lymphocytes - cytology | Fibrosis - genetics | Enzyme-Linked Immunosorbent Assay | Collagen - immunology | Scleroderma, Systemic - metabolism | Cells, Cultured | Scleroderma, Systemic - genetics | Antibodies, Anti-Idiotypic - immunology | Scleroderma, Systemic - immunology | Fibroblasts - pathology | Reverse Transcriptase Polymerase Chain Reaction | Muscle, Smooth - chemistry | Collagen - metabolism | Tissue Inhibitor of Metalloproteinase-1 - genetics | B-Cell Activating Factor - metabolism | B-Lymphocytes - immunology | Interleukin-6 - immunology | Fibroblasts - immunology | B-Cell Activating Factor - pharmacology | Tissue Inhibitor of Metalloproteinase-1 - immunology | Computer software industry | Cytokines | RNA | Systemic scleroderma | Scleroderma (Disease) | Fibroblasts | Bone morphogenetic proteins | B cells | Transforming growth factors | Blood donors | Index Medicus | Life Sciences | Human health and pathology | Rhumatology and musculoskeletal system
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 09/2009, Volume 86, Issue 3, pp. 655 - 662
A prototypic mechanism for enhancement of inflammation by an endogenous molecule is described. The nuclear protein HMGB1 has previously been demonstrated to... 
innate cell‐mediated immunity | cell activation | Innate cell-mediated immunity | Cell activation | CELLS | ACTIVATION | RECEPTOR | IMMUNOLOGY | innate cell-mediated immunity | HUMAN MONOCYTES | CELL BIOLOGY | GROUP CHROMOSOMAL-PROTEINS | AMPHOTERIN | DNA | HIGH-MOBILITY-GROUP-BOX-1 | MOBILITY GROUP BOX-1 | HEMATOLOGY | BINDING | Coculture Techniques | Cytokines - analysis | Humans | Synovial Membrane - immunology | HMGB1 Protein - immunology | HMGB1 Protein - isolation & purification | Lipopolysaccharides - immunology | HMGB1 Protein - genetics | Recombinant Proteins - isolation & purification | Toll-Like Receptor 9 - immunology | Signal Transduction - immunology | HMGB1 Protein - pharmacology | Leukocytes, Mononuclear - immunology | Fibroblasts - metabolism | Inflammation Mediators - immunology | Cell Survival - drug effects | Biotinylation | Leukocytes, Mononuclear - drug effects | Oligodeoxyribonucleotides - immunology | Cells, Cultured | Interleukin-1beta - immunology | Rats | Inflammation - immunology | Toll-Like Receptor 4 - immunology | Recombinant Proteins - pharmacology | Synovial Membrane - metabolism | Arthritis, Rheumatoid - pathology | Animals | Recombinant Proteins - immunology | Signal Transduction - drug effects | Lipopolysaccharides - pharmacology | Interleukin-6 - biosynthesis | Toll-Like Receptor 2 - immunology | Fibroblasts - immunology | Ligands | Toll-Like Receptor 1 - immunology | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 09/2015, Volume 195, Issue 5, pp. 1939 - 1943
Detection of intracellular DNA triggers activation of the stimulator of IFN genes dependent IFN-stimulatory DNA (ISD) pathway, which is essential for antiviral... 
CYCLIC GMP-AMP | INTRACELLULAR DNA | LUPUS | DENDRITIC CELLS | GENE | DNA EXONUCLEASE TREX1 | CYTOSOLIC DNA | INNATE IMMUNE SENSOR | MUTATIONS | IMMUNOLOGY | RETROVIRUSES | Nucleotides, Cyclic - metabolism | Phosphoproteins - immunology | Exodeoxyribonucleases - immunology | Interferon-beta - immunology | Nervous System Malformations - immunology | Gene Expression - drug effects | Autoantibodies - blood | Humans | Immunoblotting | Interferons - immunology | Phosphoproteins - metabolism | Inflammation - metabolism | Nervous System Malformations - metabolism | Autoimmune Diseases of the Nervous System - genetics | Gene Expression - immunology | Interferon-beta - genetics | Exodeoxyribonucleases - genetics | Nucleotidyltransferases - metabolism | Autoimmune Diseases of the Nervous System - metabolism | Nervous System Malformations - genetics | Macrophages - immunology | Disease Models, Animal | Fibroblasts - metabolism | Interferons - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Inflammation - immunology | Phosphoproteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Autoimmune Diseases of the Nervous System - immunology | Autoantibodies - immunology | Macrophages - metabolism | Animals | Interferon-beta - metabolism | Embryo, Mammalian - cytology | Fibroblasts - drug effects | Exodeoxyribonucleases - metabolism | Inflammation - genetics | Nucleotidyltransferases - genetics | Fibroblasts - immunology | Macrophages - drug effects | Nucleotidyltransferases - immunology | Nucleotides, Cyclic - immunology | Index Medicus | Abridged Index Medicus
Journal Article
eLife, ISSN 2050-084X, 12/2012, Volume 2012, Issue 1, pp. e00047 - e00047
Innate immunity is the first immunological defence against pathogens. During virus infection detection of nucleic acids is crucial for the inflammatory... 
NEUTROPHIL EXTRACELLULAR TRAPS | INTRACELLULAR DNA | DAI DLM-1/ZBP1 | MAMMALIAN DNA | IN-VIVO | BIOLOGY | V(D)J RECOMBINATION | DEPENDENT PROTEIN-KINASE | CYTOSOLIC DNA | AUTOIMMUNE-DISEASE | CYTOPLASMIC DNA | Embryo, Mammalian | Interferon Type I - immunology | Interferon Regulatory Factor-3 - genetics | DNA-Binding Proteins - deficiency | Vaccinia virus - immunology | DNA-Activated Protein Kinase - genetics | Nuclear Proteins - deficiency | Herpesvirus 1, Human - immunology | Influenza A virus - immunology | Nuclear Proteins - genetics | DNA-Activated Protein Kinase - deficiency | Cytokines - immunology | Interferon Regulatory Factor-3 - immunology | Fibroblasts - virology | DNA-Binding Proteins - immunology | Signal Transduction | Gene Expression Regulation - immunology | Membrane Proteins - genetics | DNA, Viral - metabolism | Protein-Serine-Threonine Kinases - genetics | V(D)J Recombination | Membrane Proteins - immunology | DNA-Binding Proteins - genetics | Immunity, Innate | Nuclear Proteins - immunology | Mice, Knockout | DNA, Viral - immunology | Animals | DNA Repair - immunology | Interferon Type I - genetics | Fibroblasts - immunology | Protein-Serine-Threonine Kinases - immunology | Mice | Mice, Inbred BALB C | Primary Cell Culture | DNA-Activated Protein Kinase - immunology | Cytokines - biosynthesis | Protein kinase C | Severe combined immunodeficiency | Transcription | Genes | Viruses | Innate immunity | Infections | Kinases | DNA repair | Proteins | Fibroblasts | Sensors | Localization | Deoxyribonucleic acid--DNA | RNA viruses | Fractionation | Statistical analysis | Cytokines | V(D)J recombination | Inflammation | RNA polymerase | Pattern recognition | DNA viruses | DNA-dependent protein kinase | Interferon regulatory factor 3 | Cytoplasm | Chemokines | Index Medicus
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 07/2009, Volume 16, Issue 7, pp. 991 - 1005
Cross-presentation of cell-associated antigen is important in the priming of CD8(+) T-cell responses to proteins that are not expressed by antigen-presenting... 
autophagy | DENDRITIC CELLS | PROAPOPTOTIC BAX | BIOCHEMISTRY & MOLECULAR BIOLOGY | apoptosis | TUMOR-CELLS | DEATH | MATURATION | CELL BIOLOGY | CALRETICULIN EXPOSURE | cross-priming | HEAT-SHOCK PROTEINS | IMMUNE-RESPONSES | APOPTOTIC CELLS | CASPASE ACTIVATION | Atg5 | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Dendritic Cells - immunology | Humans | bcl-2 Homologous Antagonist-Killer Protein - genetics | Apoptosis - genetics | Interferon Type I - immunology | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Gene Knockdown Techniques | Autophagy - immunology | Lymphocyte Activation - immunology | CD8-Positive T-Lymphocytes - metabolism | Calreticulin - metabolism | Interferon Type I - metabolism | Influenza A virus - immunology | Autophagy - genetics | Dendritic Cells - metabolism | bcl-2-Associated X Protein - genetics | Fibroblasts - metabolism | Fibroblasts - virology | Mice, Inbred C57BL | bcl-2-Associated X Protein - metabolism | bcl-2 Homologous Antagonist-Killer Protein - immunology | Antigen Presentation - immunology | Mice, Knockout | Microtubule-Associated Proteins - immunology | bcl-2-Associated X Protein - immunology | Animals | Apoptosis - immunology | Autophagy-Related Protein 5 | Cross-Priming - immunology | Fibroblasts - immunology | Major Histocompatibility Complex - immunology | Mice | Mice, Inbred BALB C | Calreticulin - immunology | CD8-Positive T-Lymphocytes - immunology | Orthomyxoviridae Infections - virology | Orthomyxoviridae Infections - immunology | RNA, Small Interfering - metabolism | Index Medicus | Life Sciences | Immunology
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2017, Volume 127, Issue 12, pp. 4415 - 4420
Primary immunodeficiencies are often monogenic disorders characterized by vulnerability to specific infectious pathogens. Here, we performed whole-exome... 
MEDICINE, RESEARCH & EXPERIMENTAL | IMMUNITY | ACTIVATOR | INTERFERON-GAMMA | MYCOBACTERIAL DISEASE | ALPHA/BETA | MENDELIAN SUSCEPTIBILITY | CLINICAL-FEATURES | DEFICIENCY | SIGNAL TRANSDUCER | INBORN-ERRORS | Bacteremia - immunology | Genetic Diseases, Inborn - microbiology | STAT2 Transcription Factor - immunology | Humans | Genetic Diseases, Inborn - genetics | Genetic Diseases, Inborn - virology | Male | Cytomegalovirus Infections - immunology | Cytomegalovirus Infections - genetics | Bacteremia - microbiology | Streptococcal Infections - genetics | Phosphorylation - genetics | Female | Phosphorylation - immunology | Receptor, Interferon alpha-beta - genetics | Fibroblasts - microbiology | Mycobacterium Infections, Nontuberculous - immunology | Immunologic Deficiency Syndromes - immunology | Receptors, Interferon - genetics | Fibroblasts - virology | Genetic Diseases, Inborn - immunology | Viremia - genetics | Viridans Streptococci - immunology | Bacteremia - genetics | Immunologic Deficiency Syndromes - virology | STAT1 Transcription Factor - genetics | Immunologic Deficiency Syndromes - microbiology | STAT1 Transcription Factor - immunology | Cytomegalovirus - immunology | Mycobacterium abscessus - immunology | Receptor, Interferon alpha-beta - immunology | Receptors, Interferon - immunology | Viremia - virology | STAT2 Transcription Factor - genetics | Immunologic Deficiency Syndromes - genetics | Fibroblasts - immunology | Viremia - immunology | Streptococcal Infections - immunology | Mutation | Mycobacterium Infections, Nontuberculous - genetics | Virus diseases | Usage | Immunotherapy | Genetic aspects | Disease susceptibility | Research | Mutation (Biology) | Stat2 protein | Cytomegalovirus | Phosphorylation | Viremia | Immunodeficiency | Stop codon | C-Terminus | Gene deletion | Bacteremia | Nuclear transport | α-Interferon | Clonal deletion | Fibroblasts | Interferon | Codons | Stat1 protein | Viral infections | Index Medicus | Abridged Index Medicus
Journal Article