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Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2015, Volume 43, Issue 3, pp. 475 - 487
Interleukin-17 (IL-17) induces pathology in autoimmunity and infections; therefore, constraint of this pathway is an essential component of its regulation. We... 
negative regulation | Regnase-1 | fungal immunity | autoimmunity | signal transduction | IL-17 | Autoimmunity | Signal transduction | Fungal immunity | Negative regulation | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | IL-17 RECEPTOR | TARGET GENES | CANDIDA-ALBICANS | C/EBP-BETA | GENE-EXPRESSION | HOST-DEFENSE | NECROSIS-FACTOR-ALPHA | MESSENGER-RNA STABILITY | T-CELL | IMMUNOLOGY | Candidiasis - genetics | Oncogene Proteins - genetics | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Immunoblotting | Male | Receptors, Interleukin-17 - genetics | Ribonucleases - metabolism | Host-Pathogen Interactions - immunology | Inflammation - metabolism | Lipocalins - genetics | Adaptor Proteins, Signal Transducing - immunology | Pneumonia - immunology | Candida albicans - physiology | Encephalomyelitis, Autoimmune, Experimental - genetics | Receptors, Interleukin-17 - metabolism | Interleukin-6 - metabolism | Pneumonia - genetics | Interleukin-6 - genetics | Oncogene Proteins - metabolism | Signal Transduction - genetics | Candida albicans - immunology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Interleukin-17 - metabolism | Lipocalin-2 | Lipocalins - immunology | Interleukin-6 - immunology | Pneumonia - metabolism | Ribonucleases - genetics | Encephalomyelitis, Autoimmune, Experimental - metabolism | Receptors, Interleukin-17 - immunology | Interleukin-17 - immunology | Acute-Phase Proteins - genetics | Candidiasis - microbiology | Signal Transduction - immunology | HEK293 Cells | Female | Candidiasis - immunology | Nuclear Proteins - genetics | Oncogene Proteins - immunology | Lipocalins - metabolism | Cell Line | Acute-Phase Proteins - metabolism | Mice, Inbred C57BL | Cells, Cultured | Nuclear Proteins - metabolism | Inflammation - immunology | Nuclear Proteins - immunology | Animals | Ribonucleases - immunology | Adaptor Proteins, Signal Transducing - genetics | Inflammation - genetics | Adaptor Proteins, Signal Transducing - metabolism | Acute-Phase Proteins - immunology | Medical colleges | Nucleases | Interleukins | RNA | Cellular signal transduction | Inflammation | Proteins | Cytokines | Rodents | Fibroblasts | Infections | Kinases | Laboratory animals | Gene expression | Experiments
Journal Article
Arthritis research & therapy, ISSN 1478-6354, 2013, Volume 15, Issue 5, pp. R168 - R168
Introduction: B lymphocytes might play a pathogenic role in dermal fibrosis in systemic sclerosis (SSc). B-cell activating factor (BAFF), a key cytokine for... 
BAFF | ADHESION | SCLERODERMA | TIMP-1 | AUTOIMMUNITY | SKIN FIBROSIS | DEPLETION | MECHANISMS | RHEUMATOLOGY | INTERLEUKIN-6 | IL-6 | Gene Expression - drug effects | Chemokine CCL2 - immunology | Skin - metabolism | Antibodies, Anti-Idiotypic - pharmacology | Coculture Techniques | Humans | Transforming Growth Factor beta1 - metabolism | Actins - metabolism | Matrix Metalloproteinase 9 - immunology | Fibrosis - metabolism | Actins - immunology | Immunoglobulin M - immunology | Actins - genetics | Transforming Growth Factor beta1 - immunology | Matrix Metalloproteinase 9 - genetics | Fibrosis - immunology | Gene Expression - immunology | B-Cell Activating Factor - immunology | Chemokine CCL2 - metabolism | Collagen - genetics | Interleukin-6 - metabolism | B-Lymphocytes - metabolism | Skin - pathology | Fibroblasts - metabolism | Skin - immunology | B-Lymphocytes - cytology | Fibrosis - genetics | Enzyme-Linked Immunosorbent Assay | Collagen - immunology | Scleroderma, Systemic - metabolism | Cells, Cultured | Scleroderma, Systemic - genetics | Antibodies, Anti-Idiotypic - immunology | Scleroderma, Systemic - immunology | Fibroblasts - pathology | Reverse Transcriptase Polymerase Chain Reaction | Muscle, Smooth - chemistry | Collagen - metabolism | Tissue Inhibitor of Metalloproteinase-1 - genetics | B-Cell Activating Factor - metabolism | B-Lymphocytes - immunology | Interleukin-6 - immunology | Fibroblasts - immunology | B-Cell Activating Factor - pharmacology | Tissue Inhibitor of Metalloproteinase-1 - immunology | Computer software industry | Cytokines | RNA | Systemic scleroderma | Scleroderma (Disease) | Fibroblasts | Bone morphogenetic proteins | B cells | Transforming growth factors | Blood donors | Life Sciences | Human health and pathology | Rhumatology and musculoskeletal system
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 07/2009, Volume 16, Issue 7, pp. 991 - 1005
Cross-presentation of cell-associated antigen is important in the priming of CD8(+) T-cell responses to proteins that are not expressed by antigen-presenting... 
autophagy | DENDRITIC CELLS | PROAPOPTOTIC BAX | BIOCHEMISTRY & MOLECULAR BIOLOGY | apoptosis | TUMOR-CELLS | DEATH | MATURATION | CELL BIOLOGY | CALRETICULIN EXPOSURE | cross-priming | HEAT-SHOCK PROTEINS | IMMUNE-RESPONSES | APOPTOTIC CELLS | CASPASE ACTIVATION | Atg5 | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Dendritic Cells - immunology | Humans | bcl-2 Homologous Antagonist-Killer Protein - genetics | Apoptosis - genetics | Interferon Type I - immunology | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Gene Knockdown Techniques | Autophagy - immunology | Lymphocyte Activation - immunology | CD8-Positive T-Lymphocytes - metabolism | Calreticulin - metabolism | Interferon Type I - metabolism | Influenza A virus - immunology | Autophagy - genetics | Dendritic Cells - metabolism | bcl-2-Associated X Protein - genetics | Fibroblasts - metabolism | Fibroblasts - virology | Mice, Inbred C57BL | bcl-2-Associated X Protein - metabolism | bcl-2 Homologous Antagonist-Killer Protein - immunology | Antigen Presentation - immunology | Mice, Knockout | Microtubule-Associated Proteins - immunology | bcl-2-Associated X Protein - immunology | Animals | Apoptosis - immunology | Autophagy-Related Protein 5 | Cross-Priming - immunology | Fibroblasts - immunology | Major Histocompatibility Complex - immunology | Mice | Mice, Inbred BALB C | Calreticulin - immunology | CD8-Positive T-Lymphocytes - immunology | Orthomyxoviridae Infections - virology | Orthomyxoviridae Infections - immunology | RNA, Small Interfering - metabolism | Life Sciences | Immunology
Journal Article
Journal of leukocyte biology, ISSN 0741-5400, 2009, Volume 86, Issue 3, pp. 655 - 662
A prototypic mechanism for enhancement of inflammation by an endogenous molecule is described. The nuclear protein HMGB1 has previously been demonstrated to... 
innate cell‐mediated immunity | cell activation | Innate cell-mediated immunity | Cell activation | CELLS | ACTIVATION | RECEPTOR | IMMUNOLOGY | innate cell-mediated immunity | HUMAN MONOCYTES | CELL BIOLOGY | GROUP CHROMOSOMAL-PROTEINS | AMPHOTERIN | DNA | HIGH-MOBILITY-GROUP-BOX-1 | MOBILITY GROUP BOX-1 | HEMATOLOGY | BINDING | Coculture Techniques | Cytokines - analysis | Humans | Synovial Membrane - immunology | HMGB1 Protein - immunology | HMGB1 Protein - isolation & purification | Lipopolysaccharides - immunology | HMGB1 Protein - genetics | Recombinant Proteins - isolation & purification | Toll-Like Receptor 9 - immunology | Signal Transduction - immunology | HMGB1 Protein - pharmacology | Leukocytes, Mononuclear - immunology | Fibroblasts - metabolism | Inflammation Mediators - immunology | Cell Survival - drug effects | Biotinylation | Leukocytes, Mononuclear - drug effects | Oligodeoxyribonucleotides - immunology | Cells, Cultured | Interleukin-1beta - immunology | Rats | Inflammation - immunology | Toll-Like Receptor 4 - immunology | Recombinant Proteins - pharmacology | Synovial Membrane - metabolism | Arthritis, Rheumatoid - pathology | Animals | Recombinant Proteins - immunology | Signal Transduction - drug effects | Lipopolysaccharides - pharmacology | Interleukin-6 - biosynthesis | Toll-Like Receptor 2 - immunology | Fibroblasts - immunology | Ligands | Toll-Like Receptor 1 - immunology
Journal Article
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 2014, Volume 63, Issue 6, pp. 1241 - 1250
Connective tissue growth factor (CTGF) is involved in the pathogenesis of various fibrotic disorders. However, its role in the heart is not clear. To... 
Hypertension | Inflammation | S100 proteins | Angiotensin II | MECHANISMS | MYELOID-RELATED PROTEIN-14 | RAGE | MAPK | MYELOID-RELATED-PROTEIN-8/14 | INHIBITION | inflammation | angiotensin II | PERIPHERAL VASCULAR DISEASE | TNF-ALPHA | DYSFUNCTION | hypertension | NF-KAPPA-B | EXPRESSION | Immunohistochemistry | Calgranulin B - metabolism | Oligonucleotide Array Sequence Analysis | Myocytes, Cardiac - immunology | Calgranulin A - genetics | Inflammation - metabolism | Myocardium - metabolism | Antibodies - immunology | Antigens, Ly - metabolism | Calgranulin A - immunology | Hypertension - genetics | Cytokines - genetics | Neutrophils - metabolism | Receptor for Advanced Glycation End Products | Fibroblasts - metabolism | Animals, Newborn | Angiotensin II - pharmacology | Neutrophils - drug effects | Toll-Like Receptor 4 - genetics | Hypertension - immunology | Toll-Like Receptor 4 - immunology | Reverse Transcriptase Polymerase Chain Reaction | Toll-Like Receptor 4 - metabolism | Myocytes, Cardiac - drug effects | Fibroblasts - drug effects | Myocytes, Cardiac - metabolism | Fibroblasts - immunology | Mice | Receptors, Immunologic - genetics | Myocardium - immunology | Calgranulin A - metabolism | Transcriptome - immunology | Calgranulin B - immunology | Hypertension - prevention & control | Calgranulin B - genetics | Receptors, Immunologic - immunology | Cytokines - immunology | CD11b Antigen - immunology | Cytokines - metabolism | Mice, Inbred C57BL | Cells, Cultured | Neutrophils - immunology | Myocardium - pathology | Inflammation - immunology | Transcriptome - drug effects | Angiotensin II - immunology | Antibodies - pharmacology | Animals | Antigens, Ly - immunology | Inflammation - genetics | CD11b Antigen - metabolism | Receptors, Immunologic - metabolism
Journal Article
Journal of investigative dermatology, ISSN 0022-202X, 01/2012, Volume 132, Issue 1, pp. 216 - 225
Journal Article