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Cerebral Cortex, ISSN 1047-3211, 01/2014, Volume 24, Issue 1, pp. 186 - 198
Journal Article
Nature Neuroscience, ISSN 1097-6256, 07/2003, Volume 6, Issue 7, pp. 701 - 707
Dorsoventral patterning of the telencephalon is established early in forebrain development and underlies many of the regional subdivisions that are critical to... 
MAMMALIAN TELENCEPHALON | FATE MAP | FOREBRAIN | FLOOR PLATE | SONIC HEDGEHOG | BONE MORPHOGENETIC PROTEINS | GENE-EXPRESSION | CHICK-EMBRYO | CENTRAL-NERVOUS-SYSTEM | DIFFERENTIATION | NEUROSCIENCES | Immunohistochemistry | Bone Morphogenetic Proteins - physiology | Homeodomain Proteins - metabolism | Repressor Proteins | Fibroblast Growth Factors - genetics | Wnt Proteins | Ectoderm - physiology | Stem Cells - metabolism | Aging - genetics | Gene Expression Regulation, Developmental | Cell Differentiation | In Situ Hybridization - methods | Organ Culture Techniques | Basic Helix-Loop-Helix Transcription Factors | Fibroblast Growth Factors - physiology | Frizzled Receptors | Proteins - physiology | Fibroblast Growth Factors - classification | Telencephalon - metabolism | Telencephalon - cytology | Paired Box Transcription Factors | Zebrafish Proteins | Embryonic Induction - physiology | Proto-Oncogene Proteins - genetics | PAX6 Transcription Factor | Signal Transduction - genetics | Telencephalon - embryology | Epithelium | Chick Embryo | Nerve Tissue Proteins - genetics | Homeodomain Proteins - genetics | Nerve Tissue Proteins - metabolism | Ectoderm - cytology | Telencephalon - physiology | Pyrroles - pharmacology | Animals | Proto-Oncogene Proteins - physiology | Signal Transduction - physiology | Mice | Transcription Factors | Receptors, Fibroblast Growth Factor - physiology | Eye Proteins | Aging - metabolism | Telencephalon | Neural transmission | Regulation | Research | Gene expression | Analysis | Index Medicus | Pyrroles/pharmacology | Signal Transduction/genetics/physiology | Bone Morphogenetic Proteins/physiology | Homeodomain Proteins/genetics/metabolism | In Situ Hybridization/methods | Gene Expression Regulation; Developmental | Fibroblast Growth Factors/classification/genetics/physiology | Nerve Tissue Proteins/genetics/metabolism | Aging/genetics/metabolism | Stem Cells/metabolism | Ectoderm/cytology/physiology | Proteins/physiology | Receptors; Fibroblast Growth Factor/physiology | Telencephalon/cytology/embryology/metabolism/physiology | Proto-Oncogene Proteins/genetics/physiology | Embryonic Induction/physiology
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 07/2006, Volume 208, Issue 1, pp. 87 - 96
Canonical Wnt signaling (β‐catenin/TCF) has emerged as a key regulator of skeletogenesis. In this study, chondrogenesis is examined in a mouse model in which... 
ENDOCHONDRAL OSSIFICATION | IN-VITRO | PHYSIOLOGY | TGF-BETA | BONE-FORMATION | GENE-EXPRESSION | GROWTH-PLATE | INDIAN HEDGEHOG | BETA-CATENIN | CARTILAGE DEVELOPMENT | N-CADHERIN | CELL BIOLOGY | Growth Plate - pathology | Immunohistochemistry | Intercellular Signaling Peptides and Proteins - analysis | Chondrocytes - chemistry | Osteogenesis - physiology | Bone Morphogenetic Proteins - physiology | Fibroblasts - physiology | Collagen Type II - analysis | Core Binding Factor Alpha 1 Subunit - analysis | Gene Expression Profiling | Hypertrophy - pathology | Membrane Proteins - analysis | Trans-Activators - physiology | Hedgehog Proteins | Membrane Proteins - physiology | beta Catenin - physiology | Gene Expression Regulation, Developmental - physiology | Chondrocytes - pathology | Membrane Proteins - genetics | High Mobility Group Proteins - analysis | Core Binding Factor Alpha 1 Subunit - physiology | Signal Transduction - genetics | Fibroblasts - pathology | Reverse Transcriptase Polymerase Chain Reaction | Wnt Proteins - analysis | Blotting, Western | Mice, Knockout | Mice | Transcription Factors - analysis | Wnt Proteins - physiology | Growth Plate - physiopathology | SOX9 Transcription Factor | Collagen Type X - analysis | Collagen Type X - physiology | beta Catenin - analysis | Gene Expression Regulation, Developmental - genetics | RNA, Messenger - analysis | Trans-Activators - analysis | Hypertrophy - physiopathology | Intercellular Signaling Peptides and Proteins - physiology | Cell Differentiation - genetics | Transforming Growth Factor beta - analysis | Wnt Proteins - genetics | Chondrocytes - physiology | Trans-Activators - genetics | Bone Morphogenetic Proteins - genetics | Cell Differentiation - physiology | Osteogenesis - genetics | Fibroblasts - chemistry | Bone Morphogenetic Proteins - analysis | Collagen Type II - physiology | Transcription Factors - physiology | Bone Morphogenetic Protein 2 | RNA, Messenger - genetics | Cells, Cultured | Intercellular Signaling Peptides and Proteins - genetics | Transforming Growth Factor beta - physiology | Collagen Type X - genetics | Transcription Factors - genetics | Collagen Type II - genetics | beta Catenin - genetics | Animals | Transforming Growth Factor beta - genetics | Signal Transduction - physiology | High Mobility Group Proteins - physiology | Core Binding Factor Alpha 1 Subunit - genetics | Growth Plate - chemistry | High Mobility Group Proteins - genetics | Index Medicus
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 1/2005, Volume 168, Issue 3, pp. 477 - 488
Simian Virus 40 (SV40) has been shown to enter host cells by caveolar endocytosis followed by transport via caveosomes to the endoplasmic reticulum (ER). Using... 
Endocytosis | Caveolae | Microscopy | Internalization | Cell lines | Viruses | Infections | Cell membranes | Organelles | Animal cells | TRANSPORT | INTERNALIZATION | SMOOTH ENDOPLASMIC-RETICULUM | PATHWAY | LIPID RAFTS | RECEPTOR | DYNAMIN | PLASMA-MEMBRANE | GOLGI-COMPLEX | GPI-ANCHORED PROTEINS | CELL BIOLOGY | Dynamin II - physiology | Detergents - chemistry | Cholesterol - physiology | Humans | Nocodazole - pharmacology | Brefeldin A - pharmacology | Caveolins - genetics | Fibroblasts - ultrastructure | Caveolae - physiology | Caveolin 2 | Caveolin 1 | Thiazolidines | Actin Cytoskeleton - drug effects | Caveolins - physiology | Gene Expression | Cholesterol - deficiency | Clathrin - physiology | Endocytosis - drug effects | Caveolins - analysis | Microtubules - physiology | Membrane Microdomains - chemistry | Mice, Knockout | Antigens, Viral, Tumor - metabolism | Actin Cytoskeleton - physiology | Adaptor Proteins, Signal Transducing | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Embryo, Mammalian - cytology | Fibroblasts - drug effects | Cell Line, Tumor | Membrane Microdomains - physiology | Mice | Simian virus 40 - metabolism | Transferrin - metabolism | Calcium-Binding Proteins - genetics | Endoplasmic Reticulum, Smooth - physiology | Semliki forest virus - physiology | Tubulin - genetics | Microtubules - drug effects | ADP-Ribosylation Factors - physiology | Membrane Proteins - metabolism | ADP-Ribosylation Factors - genetics | Vesicular Transport Proteins | Fibroblasts - virology | Cell Line | Microscopy, Electron, Transmission | Transport Vesicles - physiology | Dynamin II - genetics | Endoplasmic Reticulum, Smooth - chemistry | Intracellular Signaling Peptides and Proteins | Phosphoproteins - genetics | Endocytosis - physiology | Animals | Genistein - pharmacology | Thiazoles - pharmacology | Microscopy, Fluorescence | Transport Vesicles - ultrastructure | Virus diseases | SV40 (Virus) | Research | Cytology | Cellular biology | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 04/2014, Volume 129, Issue 15, pp. 1586 - 1597
BACKGROUND—Pericytes and their crosstalk with endothelial cells are critical for the development of a functional microvasculature and vascular remodeling. It... 
cell communication | transforming growth factor beta | fibroblast growth factor 2 | pericytes | endothelilal cells | pulmonary arterial hypertension | interleukin-6 | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | BONE MORPHOGENETIC PROTEIN | MICROVASCULAR PERICYTES | INHIBITION | ARTERIAL-HYPERTENSION | ADAMS-OLIVER SYNDROME | PERIPHERAL VASCULAR DISEASE | MICE | DIFFERENTIATION | CONTRIBUTES | MOLECULAR-MECHANISMS | Pericytes - drug effects | Rats, Wistar | Humans | Middle Aged | Pericytes - cytology | Fibroblast Growth Factor 2 - pharmacology | Hypertension, Pulmonary - physiopathology | Cell Communication - physiology | Male | Pericytes - physiology | Retinal Vessels - physiology | Microcirculation - physiology | Pulmonary Circulation - physiology | Interleukin-6 - physiology | Retinal Vessels - cytology | Adult | Female | Fibroblast Growth Factor 2 - physiology | Cell Differentiation - physiology | Endothelial Cells - physiology | Muscle, Smooth, Vascular - physiology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Cells, Cultured | Rats | Mice, Transgenic | Muscle, Smooth, Vascular - cytology | Neovascularization, Physiologic - physiology | Animals | Interleukin-6 - pharmacology | Cell Differentiation - drug effects | Endothelial Cells - cytology | Cell Communication - drug effects | Mice | Hypertension, Pulmonary - pathology | Endothelial Cells - drug effects | Physiological aspects | Smooth muscle | Fibroblast growth factors | Research | Pulmonary hypertension | Interleukin-6 | Index Medicus | Abridged Index Medicus
Journal Article
Nature Biotechnology, ISSN 1087-0156, 06/2004, Volume 22, Issue 6, pp. 707 - 716
Human embryonic stem (hES) cells hold promise for generating an unlimited supply of cells for replacement therapies. To characterize hES cells at the molecular... 
JAK-STAT | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | EMBRYONIC STEM-CELLS | GENE-EXPRESSION | ENHANCER | PATTERNS | LEUKEMIA INHIBITORY FACTOR | GENERATION | HUMAN BLASTOCYSTS | IDENTIFICATION | LINES | Humans | Fibroblast Growth Factors - genetics | Dimethyl Sulfoxide - pharmacology | Gene Expression Profiling | Wnt Proteins | Antigens, CD - genetics | Membrane Glycoproteins - physiology | Expressed Sequence Tags | Fibroblast Growth Factors - physiology | Receptors, G-Protein-Coupled - physiology | Tretinoin - pharmacology | Cytokine Receptor gp130 | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Sequence Analysis, DNA | Cell Division - drug effects | Down-Regulation - genetics | Cell Division - physiology | Embryo, Mammalian - cytology | Growth Substances - pharmacology | Antigens, CD - physiology | Receptors, G-Protein-Coupled - genetics | Transcription, Genetic - genetics | Nodal Protein | Gene Expression - drug effects | Stem Cells - cytology | Stem Cells - metabolism | Intercellular Signaling Peptides and Proteins - physiology | Interleukin-6 | RNA - genetics | Cell Differentiation - genetics | RNA - isolation & purification | Receptors, Fibroblast Growth Factor - genetics | Cell Differentiation - physiology | Leukemia Inhibitory Factor | Cell Division - genetics | Cell Line | Proteins - physiology | Transcription Factors - physiology | Gene Library | Intercellular Signaling Peptides and Proteins - genetics | Transforming Growth Factor beta - physiology | Proto-Oncogene Proteins - genetics | Up-Regulation - genetics | Transcription Factors - genetics | Down-Regulation - drug effects | Membrane Glycoproteins - genetics | Proteins - genetics | Up-Regulation - drug effects | Transforming Growth Factor beta - genetics | Cell Differentiation - drug effects | Proto-Oncogene Proteins - physiology | Signal Transduction - physiology | Receptors, Fibroblast Growth Factor - physiology | Index Medicus
Journal Article
Immunity, ISSN 1074-7613, 06/2012, Volume 36, Issue 6, pp. 933 - 946
The mitochondrial protein MAVS (also known as IPS-1, VISA, and CARDIF) interacts with RIG-I-like receptors (RLRs) to induce type I interferon (IFN-I). NLRX1 is... 
ELONGATION-FACTOR TU | HEPATITIS-C VIRUS | RNA | RIG-I | LEUCINE-RICH REPEAT | NUCLEOTIDE-BINDING | ANTIVIRAL IMMUNE-RESPONSES | IMMUNOLOGY | NF-KAPPA-B | ADAPTER PROTEIN | INNATE IMMUNITY | Humans | Recombinant Fusion Proteins - physiology | Molecular Sequence Data | Autophagy - physiology | Mitochondrial Proteins - genetics | Macrophages, Peritoneal - cytology | Interferon Type I - biosynthesis | DEAD-box RNA Helicases - physiology | HEK293 Cells | Microtubule-Associated Proteins - deficiency | Mitochondrial Proteins - deficiency | Cytokines - genetics | Fibroblasts - metabolism | Autophagy-Related Protein 12 | Carrier Proteins - physiology | Amino Acid Sequence | DEAD Box Protein 58 | Proteins - physiology | Peptide Elongation Factor Tu - physiology | Specific Pathogen-Free Organisms | Gene Expression Regulation - immunology | Macrophages, Peritoneal - immunology | Mitochondrial Proteins - physiology | Peptide Elongation Factor Tu - chemistry | Recombinant Fusion Proteins - chemistry | Microtubule-Associated Proteins - physiology | Multiprotein Complexes - physiology | Protein Interaction Mapping | Sequence Homology, Amino Acid | Adaptor Proteins, Signal Transducing - physiology | Sequence Alignment | Animals | Autophagy-Related Protein 5 | Mitochondrial Proteins - chemistry | Interferon Type I - genetics | Mice | Vesiculovirus - physiology | Cytokines - biosynthesis | Virus diseases | Proteins | Magneto-electric machines | Physiological aspects | Interferon | Biological response modifiers | Health aspects | Machinery | Mass spectrometry | Cells | Infections | Plasmids | Autophagy | Rodents | Viral infections | Index Medicus
Journal Article
Journal Article
The Journal of Physiology, ISSN 0022-3751, 08/2017, Volume 595, Issue 15, pp. 5115 - 5127
Accumulation of skeletal muscle extracellular matrix is an unfavourable characteristic of many muscle diseases, muscle injury and sarcopenia. The extent of... 
skeletal muscle regeneration | skeletal muscle derived fibroblasts | co‐culture | myogenesis | satellite cells | co-culture | PHYSIOLOGY | CONNECTIVE-TISSUE FIBROBLASTS | EXTRACELLULAR-MATRIX | FATE | NEUROSCIENCES | Antigens, Differentiation, Myelomonocytic - physiology | Macrophages - physiology | Electric Stimulation | Fibroblasts - physiology | Coculture Techniques | Humans | Muscle Development - physiology | Cells, Cultured | Male | Muscle, Skeletal - injuries | Muscle, Skeletal - physiology | Regeneration - physiology | Human Umbilical Vein Endothelial Cells - physiology | Muscle Contraction | Leukocyte Common Antigens - physiology | Transcription Factor 7-Like 2 Protein - physiology | Adult | Antigens, CD - physiology | Myoblasts - physiology | Muscles | Physiological aspects | Muscle proteins | Cell differentiation | Myosin | Cell proliferation | Neonates | Cell culture | Differentiation (biology) | Stimulation | Satellite cells | Cell fusion | Remodeling | Fibers | Sarcopenia | Rodents | Fibroblasts | Extracellular matrix | Physiology | Matrix protein | Cross sections | Injuries | Myogenin | Embryos | Desmin | Myogenesis | Skeletal muscle | Diseases | Regeneration | Musculoskeletal system | Biopsy | Cell number | Men | Cells (biology) | In vivo methods and tests | In vitro methods and tests | Index Medicus | Research Paper | Molecular and cellular
Journal Article