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Seminars in immunopathology, ISSN 1863-2300, 2011, Volume 34, Issue 1, pp. 43 - 62
The interaction of coagulation factors with the perivascular environment affects the development of disease in ways that extend beyond their traditional roles... 
Autoimmunity | Complement receptor 3 | Multiple sclerosis | Stroke | Alzheimer’s disease | Internal Medicine | Macrophages | Blood brain barrier | Inflammatory disease | Microglia | Biomedicine | Immunology | Rheumatoid arthritis | CD11b/CD18 | Atherosclerosis | Plasminogen | Anticoagulant therapy | Alzheimer's disease | Pathology | Life Sciences & Biomedicine | Science & Technology | Colitis - genetics | Humans | Brain Injuries - metabolism | Arthritis, Rheumatoid - metabolism | Bacterial Infections - genetics | Inflammation - metabolism | Blood Coagulation - immunology | Muscular Dystrophy, Duchenne - immunology | Alzheimer Disease - immunology | Fibrinogen - immunology | Thromboplastin - immunology | Pulmonary Fibrosis - immunology | Vascular Diseases - genetics | Brain Injuries - genetics | Thrombin - genetics | Kidney Diseases - immunology | Arthritis, Rheumatoid - genetics | Neoplasms - immunology | Colitis - metabolism | Muscular Dystrophy, Duchenne - genetics | Bacterial Infections - metabolism | Neoplasms - metabolism | Pulmonary Fibrosis - genetics | Spinal Cord Injuries - genetics | Kidney Diseases - genetics | Stroke - genetics | Brain Injuries - immunology | Neoplasms - genetics | Bacterial Infections - immunology | Pulmonary Fibrosis - metabolism | Colitis - immunology | Thrombin - immunology | Stroke - immunology | Kidney Diseases - metabolism | Multiple Sclerosis - metabolism | Vascular Diseases - immunology | Spinal Cord Injuries - metabolism | Multiple Sclerosis - genetics | Inflammation - immunology | Fibrinogen - genetics | Stroke - metabolism | Animals | Thromboplastin - genetics | Alzheimer Disease - metabolism | Fibrinogen - metabolism | Inflammation - genetics | Multiple Sclerosis - immunology | Blood Coagulation - genetics | Spinal Cord Injuries - immunology | Muscular Dystrophy, Duchenne - metabolism | Thrombin - metabolism | Thromboplastin - metabolism | Alzheimer Disease - genetics | Arthritis, Rheumatoid - immunology | Vascular Diseases - metabolism | Nervous system diseases | Bacterial infections | Oncology, Experimental | Thrombin | Inflammation | Anticoagulants (Medicine) | Research | Rheumatoid factor | Fibrin | Fibrinogen | Genetic research | Colitis | Cancer | Index Medicus | Traumatic brain injury | Coagulation | Tissue factor | hemostasis | Spinal cord injury | Inflammatory diseases | Kidney | Signal transduction | Duchenne's muscular dystrophy | Neurodegenerative diseases | Therapeutic applications | Thrombosis | Coagulation factors | Infection | Molecular modelling | Fibrosis | Brain injury
Journal Article
Inflammation research, ISSN 1023-3830, 02/2018, Volume 67, Issue 2, pp. 169 - 177
OBJECTIVE: To investigate the ex vivo pro-inflammatory properties of classical and non-classical monocytes as well as myeloid dendritic cells (mDCs) in... 
Dendrites/metabolism | Humans | Middle Aged | Male | Journal Article | Interleukin-8/metabolism | Immunology | Adult | Female | Classical monocytes | Interferons/metabolism | Pulmonary Fibrosis/metabolism | Myeloid dendritic cells | Inflammation | Sialic Acid Binding Ig-like Lectin 1/metabolism | Pharmacology | Non-classical monocytes | Scleroderma, Systemic/metabolism | Chemokine CCL4/metabolism | Systemic sclerosis | Toll-Like Receptor 4/metabolism | Aged | Myeloid Cells/metabolism | Chemokines | Cytokines/biosynthesis | Chemokine CXCL10/metabolism | Monocytes/metabolism | Dermatology | Rheumatology | Allergology | Neurology | Biomedicine | Pharmacology/Toxicology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Dendrites - metabolism | Scleroderma, Systemic - metabolism | Monocytes - metabolism | Toll-Like Receptor 4 - metabolism | Sialic Acid Binding Ig-like Lectin 1 - metabolism | Interferons - metabolism | Myeloid Cells - metabolism | Chemokine CCL4 - metabolism | Pulmonary Fibrosis - metabolism | Interleukin-8 - metabolism | Chemokine CXCL10 - metabolism | Cytokines - biosynthesis | Interferon | Systemic scleroderma | Dendritic cells | Biological response modifiers | Analysis | Scleroderma (Disease) | Cluster analysis | Flow cytometry | Respiratory function | Carbon tetrachloride | Stimulation | Parameter identification | Interleukin 6 | Proteins | Peripheral blood | Toll-like receptors | Immune system | Cytokines | Lung diseases | Clustering | Patients | Cytometry | Monocytes | Production methods | γ-Interferon | CXCL10 protein | Fibrosis | Pulmonary functions | Index Medicus | Original Research Paper
Journal Article
Journal of gastroenterology and hepatology, ISSN 1440-1746, 09/2008, Volume 23, Issue 9, pp. 1327 - 1338
The renin–angiotensin system (RAS) is a key regulator of vascular resistance, sodium and water homeostasis and the response to tissue injury. Historically,... 
hepatic fibrosis | angiotensin converting enzyme 2 | renin–angiotensin system | angiotensin (Ang)‐(1–7) | Hepatic fibrosis | Angiotensin converting enzyme 2 | Angiotensin (Ang)-(1-7) | Renin-angiotensin system | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Proto-Oncogene Proteins - metabolism | Kinins - metabolism | Peptide Fragments - metabolism | Liver - enzymology | Angiotensin II - metabolism | Receptors, G-Protein-Coupled - metabolism | Angiotensin I | Humans | Liver - metabolism | Liver - physiopathology | Angiotensin II Type 1 Receptor Blockers - pharmacology | Animals | Hypertension, Portal - metabolism | Liver Cirrhosis - metabolism | Peptidyl-Dipeptidase A - metabolism | Angiotensins - metabolism | Renin-Angiotensin System - drug effects | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Liver Diseases - physiopathology | Liver Diseases - metabolism | Kallikreins - metabolism | Renin-Angiotensin System, drug effects | Proto-Oncogene Proteins, metabolism | Angiotensin II Type 1 Receptor Blockers, pharmacology | Kallikreins, metabolism | Liver Cirrhosis, metabolism | Kinins, metabolism | Liver Diseases, physiopathology | Liver, metabolism | Liver Diseases, metabolism | Renin-Angiotensin System | Hypertension, Portal, metabolism | Liver, enzymology | Liver, physiopathology | Receptors, G-Protein-Coupled, metabolism | Angiotensin-Converting Enzyme Inhibitors, pharmacology | Angiotensin II, metabolism | Angiotensins, metabolism | Peptide Fragments, metabolism | Peptidyl-Dipeptidase A, metabolism | Liver diseases | Peptide hormones | Angiotensin converting enzyme | Angiotensin | Index Medicus | Basic Science of Gastroenterology and Hepatology
Journal Article
PloS one, ISSN 1932-6203, 10/2017, Volume 12, Issue 10, pp. e0186615 - e0186615
Background Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive and fatal disease. Histone deacetylase 6 (HDAC6) alters function and fate of various... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Idiopathic Pulmonary Fibrosis - genetics | Ribosomal Protein S6 Kinases - metabolism | TOR Serine-Threonine Kinases - metabolism | Humans | Middle Aged | Phosphoprotein Phosphatases - metabolism | Male | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Autophagy - drug effects | Idiopathic Pulmonary Fibrosis - metabolism | Mechanistic Target of Rapamycin Complex 1 | Autophagosomes - drug effects | Multiprotein Complexes - metabolism | Tubulin - metabolism | Bleomycin | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Aged, 80 and over | Female | Indoles - pharmacology | Lung - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Hydroxamic Acids - pharmacology | Fibroblasts - metabolism | Lung - pathology | Collagen Type I - metabolism | Histone Deacetylases - genetics | Histone Deacetylase 6 | RNA, Messenger - genetics | Histone Deacetylases - metabolism | Nuclear Proteins - metabolism | Autophagosomes - metabolism | Mice, Knockout | Transforming Growth Factor beta - pharmacology | Animals | Signal Transduction - drug effects | Fibroblasts - drug effects | Idiopathic Pulmonary Fibrosis - pathology | Hydroxamic Acids - therapeutic use | Indoles - therapeutic use | Aged | Transforming Growth Factor beta - metabolism | Idiopathic Pulmonary Fibrosis - drug therapy | Histone deacetylase | Deregulation | Collagen (type I) | Phosphorylation | Disease | Mesenchyme | Pathogenesis | Critical care | AKT protein | Leucine | Kinases | Autophagy | Proteins | Fibroblasts | Vascular endothelial growth factor | Internal medicine | Lung diseases | Environmental health | 1-Phosphatidylinositol 3-kinase | Medicine | Pulmonary fibrosis | Inhibitors | Lungs | Lysine | Deacetylation | Collagen | Fibrosis | Mice | Protein phosphatase | Phagocytosis | Index Medicus
Journal Article