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Nature immunology, ISSN 1529-2916, 2012, Volume 13, Issue 5, pp. 449 - 456
Intestinal phagocytes transport oral antigens and promote immune tolerance, but their role in innate immune responses remains unclear. Here we found that... 
FLAGELLIN | PATTERN-RECOGNITION | NOD-LIKE RECEPTORS | ACTIVATES CASPASE-1 | MACROPHAGES | INTERLEUKIN-1-BETA | INFLAMMASOME RECEPTORS | INFECTION | NLRP3 INFLAMMASOME | SECRETION | IMMUNOLOGY | Inflammasomes - metabolism | Humans | Receptors, Interleukin-1 - genetics | Salmonella Infections - genetics | Salmonella Infections - immunology | Clonal Anergy | Caspase 1 - metabolism | Monocytes - metabolism | Monocytes - immunology | Intestines - immunology | Host-Pathogen Interactions - immunology | Signal Transduction - immunology | Calcium-Binding Proteins - immunology | Interleukin-1beta - metabolism | Tumor Necrosis Factor-alpha - immunology | Apoptosis Regulatory Proteins - genetics | Pseudomonas - immunology | Toll-Like Receptors - metabolism | Neutrophil Infiltration - immunology | Neutrophils - pathology | Apoptosis Regulatory Proteins - immunology | Flagellin - immunology | Receptors, Interleukin-1 - immunology | Mice, Inbred C57BL | Toll-Like Receptors - immunology | Neutrophils - immunology | Salmonella - genetics | Signal Transduction - genetics | Phagocytes - immunology | Neutrophil Infiltration - genetics | Mice, Knockout | Pseudomonas Infections - immunology | Salmonella - immunology | Animals | Intestines - microbiology | Phagocytes - microbiology | Inflammasomes - immunology | Interleukin-6 - immunology | Interleukin-6 - biosynthesis | Mice | Mice, Inbred BALB C | Tumor Necrosis Factor-alpha - biosynthesis | Calcium-Binding Proteins - genetics
Journal Article
PLoS pathogens, ISSN 1553-7374, 2013, Volume 9, Issue 6, p. e1003400
Inflammasome activation is important for antimicrobial defense because it induces cell death and regulates the secretion of IL-1 family cytokines, which play a... 
LEGIONELLA-PNEUMOPHILA | FLAGELLIN | PROTEIN | DISTINCT PATHWAYS | RECOGNITION | INFLAMMASOME ACTIVATION | INTERLEUKIN-1-BETA | INNATE | LEGIONNAIRES DISEASE | MICROBIOLOGY | DIFFERENTIAL REQUIREMENT | VIROLOGY | PARASITOLOGY | Interleukin-1alpha - immunology | Caspases - immunology | NLR Family, Pyrin Domain-Containing 3 Protein | Cytosol - immunology | Enzyme Activation - immunology | Legionnaires' Disease - immunology | Calcium-Binding Proteins - immunology | Legionnaires' Disease - microbiology | Legionella pneumophila - pathogenicity | Bacterial Secretion Systems - immunology | Carrier Proteins - immunology | Macrophages - immunology | Macrophages - microbiology | Cell Line | Legionella pneumophila - immunology | Apoptosis Regulatory Proteins - immunology | Macrophages - pathology | Caspases - genetics | Interleukin-1beta - immunology | Inflammasomes - genetics | Mice, Knockout | Immunity, Innate - immunology | Animals | Inflammasomes - immunology | Legionnaires' Disease - pathology | Cytosol - microbiology | Mice | Host-parasite relationships | Physiological aspects | Research | Interleukins | Cytosol | Health aspects | Bacterial infections | Microbiology | Cytokines | Infections | Experiments | Bacteriology | Recruitment | Scholarships & fellowships | E coli | Bone marrow | Bacteria | Interferon | Gram-negative bacteria | Adapter proteins | Apoptosis
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2014, Volume 41, Issue 3, pp. 478 - 492
Journal Article
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2013, Volume 341, Issue 6151, pp. 1246 - 1249
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 9/2005, Volume 102, Issue 38, pp. 13610 - 13615
Commensal and enteroinvasive microbes in the human gut release bacterial flagellin, a specific microbial ligand of Toll-like receptor 5 (TLR5). However, the... 
Biological Sciences | Inflammatory bowel diseases | Commensals | Cytokines | Mucosa | Antibodies | Inflammation | Colitis | Colon | Epithelium | Apoptosis | colitis | PROTEIN | RECOGNITION | INDUCE | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | INTESTINAL EPITHELIAL-CELLS | BOWEL-DISEASE | IN-VITRO | innate immunity | TOLL-LIKE RECEPTORS | GENE-EXPRESSION | commensal bacteria | TLR5 | Inflammation - pathology | Toll-Like Receptor 2 | Apoptosis - drug effects | Humans | Colitis - pathology | Male | Peptidoglycan - immunology | Toll-Like Receptor 5 | Lipopolysaccharides - immunology | Flagellin - pharmacology | Signal Transduction - immunology | Toll-Like Receptors | Intestinal Mucosa - immunology | Teichoic Acids - pharmacology | Colitis - chemically induced | MAP Kinase Kinase 2 - immunology | Tumor Necrosis Factor-alpha - immunology | Colon - physiopathology | MAP Kinase Kinase 1 - immunology | Membrane Glycoproteins - immunology | Colitis - immunology | Chemokines - immunology | Flagellin - immunology | Colon - pathology | Tissue Culture Techniques | Membrane Glycoproteins - agonists | Receptors, Cell Surface - agonists | Inflammation - immunology | Teichoic Acids - immunology | Receptors, Cell Surface - immunology | Keratinocytes - immunology | Keratinocytes - pathology | Animals | Apoptosis - immunology | Signal Transduction - drug effects | Interleukin-6 - immunology | Lipopolysaccharides - pharmacology | Mice | Peptidoglycan - pharmacology | Intestinal Mucosa - pathology | Gastric mucosa | Research
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 10/2009, Volume 86, Issue 4, pp. 863 - 875
Endotoxin tolerance interferes with TLR4 signalosome assembly, kinase/transcription factor activation, and increases negative TLR pathway regulators. Endotoxin... 
monocytes/macrophages | lipopolysaccharide | signal transduction | suppression/anergy | cell activation | Lipopolysaccharide | Signal transduction | Cell activation | Monocytes/macrophages | Suppression/anergy | STRANDED-RNA | ACTIVATION | DOWN-REGULATION | NECROSIS-FACTOR-ALPHA | BACTERIAL FLAGELLIN | IMMUNOLOGY | CUTTING EDGE | CELL BIOLOGY | RECEPTOR-ASSOCIATED KINASE | TOLL-LIKE RECEPTORS | HEMATOLOGY | NF-KAPPA-B | INNATE IMMUNITY | Protein Kinase C - genetics | Cytoskeletal Proteins - genetics | Humans | Adaptor Proteins, Vesicular Transport - genetics | Suppressor of Cytokine Signaling 1 Protein | Monocytes - metabolism | Monocytes - immunology | Armadillo Domain Proteins - genetics | Adaptor Proteins, Vesicular Transport - metabolism | Interferon Regulatory Factor-3 - genetics | Lipopolysaccharides - immunology | Signal Transduction - immunology | Protein Kinase C - metabolism | Myeloid Differentiation Factor 88 - immunology | Cytoskeletal Proteins - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Cytokines - genetics | Protein-Serine-Threonine Kinases - metabolism | Cytokines - immunology | Interferon Regulatory Factor-3 - immunology | Cell Line | Cytokines - metabolism | Gene Expression Regulation - immunology | Suppressor of Cytokine Signaling Proteins - immunology | Myeloid Differentiation Factor 88 - genetics | Protein-Serine-Threonine Kinases - genetics | Suppressor of Cytokine Signaling Proteins - genetics | Toll-Like Receptor 4 - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Protein Kinase C - immunology | Toll-Like Receptor 4 - immunology | p38 Mitogen-Activated Protein Kinases - immunology | Adaptor Proteins, Vesicular Transport - immunology | Armadillo Domain Proteins - immunology | Toll-Like Receptor 4 - metabolism | Gene Expression Regulation - drug effects | Cytoskeletal Proteins - immunology | Signal Transduction - drug effects | Armadillo Domain Proteins - metabolism | Lipopolysaccharides - pharmacology | Interferon Regulatory Factor-3 - metabolism | Protein-Serine-Threonine Kinases - immunology | Suppressor of Cytokine Signaling Proteins - metabolism | Myeloid Differentiation Factor 88 - metabolism | monocytes | macrophages | suppression | Receptors, Signal Transduction, & Genes | anergy
Journal Article
PLoS genetics, ISSN 1553-7404, 2017, Volume 13, Issue 5, p. e1006639
Plant immunity protects plants from numerous potentially pathogenic microbes. The biological network that controls plant inducible immunity must function... 
REGRESSION | RESPONSES | ETHYLENE | SALICYLIC-ACID | GENE | GENETICS & HEREDITY | PLANTS | MECHANISMS | SELECTION | JASMONIC ACID | EXPRESSION | Carboxylic Ester Hydrolases - immunology | Salicylic Acid - metabolism | Oxylipins - immunology | Plant Diseases - immunology | Arabidopsis - immunology | Transcriptome - immunology | Ethylenes - metabolism | Host-Pathogen Interactions - immunology | Ethylenes - immunology | Salicylic Acid - immunology | Cyclopentanes - immunology | Flagellin - genetics | Gene Expression Regulation, Plant | Plant Diseases - genetics | Carboxylic Ester Hydrolases - genetics | Gene Regulatory Networks - immunology | Arabidopsis Proteins - genetics | Flagellin - immunology | Plant Immunity - genetics | Signal Transduction | Transcriptome - genetics | Arabidopsis Proteins - immunology | Arabidopsis - genetics | Oxylipins - metabolism | Host-Pathogen Interactions - genetics | Cyclopentanes - metabolism | Arabidopsis | Phytoalexin | Analysis | Ethylene | Genetic aspects | Research | Jasmonates | Networks | Ontology | Transcription | Laboratories | Genomics | Colleges & universities | Flagellin | Genomes | Biology | Plants (organisms) | Immunity | Consortia | Signal transduction | Biological effects | Scholarships & fellowships | Charitable foundations | Buffers | Bioinformatics | Jasmonic acid | Salicylic acid | Pathogens | Plant protection | Gene expression | Resilience | Computer science | Effectors | Regulatory mechanisms (biology) | Genetic engineering | Internet | Mutation | Dismantling
Journal Article
Nature immunology, ISSN 1529-2916, 2008, Volume 9, Issue 10, pp. 1171 - 1178
Journal Article
PLoS pathogens, ISSN 1553-7374, 2009, Volume 5, Issue 4, pp. e1000361 - e1000361
Journal Article