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Nature, ISSN 0028-0836, 2012, Volume 490, Issue 7418, pp. 107 - 111
Detection of microbial products by host inflammasomes is an important mechanism of innate immune surveillance. Inflammasomes activate the caspase-1 (CASP1)... 
CYTOSOLIC PHOSPHOLIPASE A | FLAGELLIN | III SECRETION APPARATUS | MECHANISM | MICE DEFICIENT | MULTIDISCIPLINARY SCIENCES | NLRC4 INFLAMMASOME | INFECTION | PYROPTOSIS | CASPASE-1 ACTIVATION | IMMUNE RECOGNITION | Inflammation - pathology | Inflammasomes - metabolism | Hematocrit | Salmonella Infections - immunology | Eicosanoids - biosynthesis | Caspase 1 - metabolism | Male | Inflammation - metabolism | Time Factors | Flagellin - metabolism | Bacterial Toxins - genetics | Antigens, Bacterial - chemistry | Neuronal Apoptosis-Inhibitory Protein - deficiency | Macrophages, Peritoneal - immunology | Calcium-Binding Proteins - deficiency | Interleukin-1beta | Bacterial Toxins - chemistry | Apoptosis Regulatory Proteins - metabolism | Cyclooxygenase 1 - deficiency | Body Fluids - metabolism | Caspase 1 - deficiency | Recombinant Fusion Proteins - genetics | Cytosol - metabolism | Death | Mice | Antigens, Bacterial - metabolism | Capillary Permeability | Peritoneal Cavity | Legionella pneumophila | Intestines - metabolism | Antigens, Bacterial - genetics | Recombinant Fusion Proteins - metabolism | Peritoneal Lavage | Apoptosis Regulatory Proteins - deficiency | Flagellin - genetics | Female | Interleukin-18 | Calcium Signaling | Calcium-Binding Proteins - metabolism | Flagellin - immunology | Salmonella typhimurium - immunology | Mice, Inbred C57BL | Inflammation - immunology | Body Temperature | Neuronal Apoptosis-Inhibitory Protein - metabolism | Immunity, Innate - immunology | Bacterial Toxins - metabolism | Animals | Eicosanoids - metabolism | Fluid Shifts | Cellular signal transduction | Research | Properties | Bacterial proteins | Flagella (Microbiology) | Immune system | Proteins | Bone marrow | Pathology | Infections | Biosynthesis | Rodents | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2007, Volume 282, Issue 26, pp. 18810 - 18818
Interleukin-1β (IL-1β) is a pro-inflammatory cytokine that plays an important role in host defense and inflammatory diseases. The maturation and secretion of... 
LISTERIA-MONOCYTOGENES | IL-1-BETA-CONVERTING ENZYME | IL-1-BETA SECRETION | MICE DEFICIENT | P2X RECEPTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | RELEASE | MATURATION | HUMAN MONOCYTES | ATP | INTERLEUKIN-1-BETA CONVERTING-ENZYME | Potassium - metabolism | Escherichia coli Infections - physiopathology | Lipopolysaccharides - metabolism | Caspase 1 - metabolism | Salmonella Infections - metabolism | Salmonella Infections - physiopathology | Salmonella typhimurium | Adenosine Triphosphate - pharmacology | Flagellin - pharmacology | Extracellular Space - metabolism | Mice, Mutant Strains | Adenosine Triphosphate - metabolism | Receptors, Purinergic P2 - genetics | Flagellin - metabolism | Macrophages - microbiology | Staphylococcus aureus | Staphylococcal Infections - metabolism | Calcium-Binding Proteins - metabolism | Listeriosis - metabolism | Escherichia coli Infections - metabolism | Apoptosis Regulatory Proteins - metabolism | Bacterial Infections - physiopathology | Receptors, Purinergic P2X7 | Macrophages - metabolism | Animals | Extracellular Space - microbiology | Listeriosis - physiopathology | Receptors, Purinergic P2 - metabolism | Lipopolysaccharides - pharmacology | Staphylococcal Infections - physiopathology | Cytosol - metabolism | Macrophages - drug effects | Cytosol - microbiology | Mice | Bacterial Infections - metabolism | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 01/2013, Volume 110, Issue 5, pp. 1851 - 1856
A flagellin-independent caspase-1 activation pathway that does not require NAIP5 or NRLC4 is induced by the intracellular pathogen Legionella pneumophila .... 
Cell death | Innate immunity | Inflammasome | IMMUNITY | PATTERN-RECOGNITION | innate immunity | cell death | inflammasome | MULTIDISCIPLINARY SCIENCES | NLRP3 INFLAMMASOME ACTIVATION | INFECTION | RECEPTORS | IPAF | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Adaptor Proteins, Vesicular Transport - genetics | Caspase 1 - metabolism | Immunoblotting | Legionella pneumophila - genetics | Adaptor Proteins, Vesicular Transport - metabolism | Necrosis | Caspases - metabolism | Flagellin - genetics | Flagellin - metabolism | Apoptosis Regulatory Proteins - genetics | Cytoskeletal Proteins - metabolism | Receptor, Interferon alpha-beta - genetics | Macrophages - microbiology | Receptor, Interferon alpha-beta - metabolism | Calcium-Binding Proteins - metabolism | Cytokines - metabolism | Caspases - genetics | Mice, Inbred C57BL | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Macrophages - cytology | Legionella pneumophila - metabolism | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Host-Pathogen Interactions | Carrier Proteins - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Caspase 1 - genetics | CARD Signaling Adaptor Proteins | Bone Marrow Cells - microbiology | Legionella pneumophila - physiology | Mice | Enzyme Activation | Mutation | Myeloid Differentiation Factor 88 - metabolism | Bone Marrow Cells - metabolism | Apoptosis | Calcium-Binding Proteins - genetics | Legionella pneumophila | Genetic aspects | Research | Macrophages | Properties | Immunological research | Proteins | Bacteria | Medical treatment | Cells | Index Medicus | Biological Sciences
Journal Article
International Immunology, ISSN 0953-8178, 4/2007, Volume 19, Issue 4, pp. 345 - 354
Forkhead box P3 (FOXP3) is currently thought to be the most specific marker for naturally occurring CD4+CD25+ T regulatory cells (nTregs). In mice, expression... 
INTERLEUKIN-2 | TGF-BETA | T cell activation | transcription factors | INDUCTION | IMMUNOLOGY | T cells | REGULATORY-CELLS | TRANSCRIPTION FACTOR FOXP3 | COSTIMULATION | GENERATION | FLOW-CYTOMETRY | DIFFERENTIATION | human | EXPRESSION | tolerance | T-Lymphocyte Subsets - immunology | Cysteine - analogs & derivatives | T-Lymphocytes, Regulatory - metabolism | Cell Proliferation | Coculture Techniques | Humans | Clonal Anergy | Receptors, Nerve Growth Factor - metabolism | Interferon-gamma - metabolism | Antigens, CD - metabolism | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | Flagellin - pharmacology | Lymphocyte Activation - immunology | Forkhead Transcription Factors - metabolism | T-Lymphocyte Subsets - drug effects | Cysteine - pharmacology | Antigens, Differentiation - metabolism | Interleukin-2 - metabolism | Receptors, Tumor Necrosis Factor - metabolism | Cell Line | Cytokines - metabolism | Lectins, C-Type | Toll-Like Receptors - immunology | CD4-Positive T-Lymphocytes - metabolism | Glucocorticoid-Induced TNFR-Related Protein | Antigens, Differentiation, T-Lymphocyte - metabolism | CD28 Antigens - immunology | Antibodies - pharmacology | CTLA-4 Antigen | T-Lymphocytes, Regulatory - drug effects | Interleukin-2 Receptor alpha Subunit - metabolism | Lipoproteins - pharmacology | T-Lymphocyte Subsets - metabolism | Interleukin-7 Receptor alpha Subunit - metabolism | CD3 Complex - immunology | Interleukin-2 - pharmacology | CD4-Positive T-Lymphocytes - drug effects | Index Medicus
Journal Article
Molecular Human Reproduction, ISSN 1360-9947, 09/2017, Volume 23, Issue 9, pp. 628 - 645
STUDY QUESTION: Does A20 regulate mediators involved in the terminal processes of human labour in primary myometrial and amnion cells? SUMMARY ANSWER: A20 is a... 
Myometrium | Pro-labour mediators | Inflammation | Preterm birth | Foetal membranes | A20 | preterm birth | TOLL-LIKE RECEPTOR | foetal membranes | NECROSIS-FACTOR-ALPHA | DEVELOPMENTAL BIOLOGY | PRETERM LABOR | TERM LABOR | OBSTETRICS & GYNECOLOGY | myometrium | PRO-LABOR MEDIATORS | IN-VITRO | REPRODUCTIVE BIOLOGY | EPITHELIAL-CELLS | inflammation | pro-labour mediators | SMOOTH-MUSCLE-CELLS | NF-KAPPA-B | EXPRESSION | Interleukin-8 - genetics | Myometrium - drug effects | RNA, Small Interfering - genetics | Interleukin-1beta - pharmacology | Premature Birth - genetics | Humans | NF-kappa B - metabolism | Tumor Necrosis Factor alpha-Induced Protein 3 - metabolism | Chemokine CXCL1 - genetics | Dinoprost - metabolism | Flagellin - pharmacology | Matrix Metalloproteinase 9 - metabolism | Term Birth - metabolism | Premature Birth - physiopathology | Cyclooxygenase 2 - genetics | Matrix Metalloproteinase 9 - genetics | Myometrium - metabolism | Interleukin-1alpha - genetics | Interleukin-8 - metabolism | Chemokine CXCL1 - metabolism | Interleukin-6 - metabolism | Poly I-C - pharmacology | Term Birth - genetics | Interleukin-6 - genetics | Signal Transduction | Amnion - cytology | Labor, Obstetric - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Dinoprost - genetics | Intercellular Adhesion Molecule-1 - metabolism | Intercellular Adhesion Molecule-1 - genetics | Vascular Cell Adhesion Molecule-1 - metabolism | RNA, Small Interfering - metabolism | Interleukin-1alpha - metabolism | Receptors, Prostaglandin - metabolism | Amnion - metabolism | Receptors, Prostaglandin - genetics | Gene Expression Regulation, Developmental | Female | Vascular Cell Adhesion Molecule-1 - genetics | Amnion - drug effects | Chemokine CCL2 - metabolism | Premature Birth - metabolism | Chemokine CCL2 - genetics | Myometrium - cytology | Pregnancy | Tumor Necrosis Factor alpha-Induced Protein 3 - genetics | NF-kappa B - genetics | Cyclooxygenase 2 - metabolism | Labor, Obstetric - genetics | Primary Cell Culture | Tumor Necrosis Factor alpha-Induced Protein 3 - antagonists & inhibitors | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2004, Volume 279, Issue 41, pp. 42984 - 42992
The capacity of non-pathogenic enteric bacteria to induce a pro-inflammatory response is under debate in terms of its effect on the symbiosis between the... 
TOLL-LIKE RECEPTOR-4 | BIOCHEMISTRY & MOLECULAR BIOLOGY | DISEASE | INNATE IMMUNE-RESPONSE | LIPOPOLYSACCHARIDE | BACTERIAL FLAGELLIN | PROINFLAMMATORY GENE-EXPRESSION | PROTEINS | NF-KAPPA-B | REGULATED EXPRESSION | INTERLEUKIN-8 | Ileum - pathology | Intestinal Mucosa - metabolism | Membrane Glycoproteins - metabolism | Enterocytes - metabolism | Humans | Membrane Glycoproteins - chemistry | Lipopolysaccharides - metabolism | Culture Media, Conditioned - pharmacology | NF-kappa B - metabolism | Chemokine CCL20 | RNA, Messenger - metabolism | Toll-Like Receptor 5 | Toll-Like Receptors | Cell Nucleus - metabolism | Transfection | Time Factors | Escherichia coli - metabolism | Flagellin - chemistry | Flagellin - metabolism | Antigens, Differentiation - metabolism | Receptors, Cell Surface - chemistry | Transcription, Genetic | Interleukin-8 - metabolism | Epithelial Cells - cytology | Caco-2 Cells | Cell Line | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Signal Transduction | Macrophage Inflammatory Proteins - metabolism | Cells, Cultured | Receptors, Cell Surface - metabolism | Inflammation | Myeloid Differentiation Factor 88 | Reverse Transcriptase Polymerase Chain Reaction | Plasmids - metabolism | Mice, Inbred C3H | Adaptor Proteins, Signal Transducing | Phenotype | Animals | Biopsy | Mice | Mutation | Receptors, Immunologic - metabolism | Chemokines, CC - metabolism | Index Medicus | Biological Sciences | Naturvetenskap | Immunology | Immunologi | Medical and Health Sciences | Microbiology | Medicin och hälsovetenskap | Biologiska vetenskaper | Mikrobiologi | Natural Sciences
Journal Article
Science, ISSN 0036-8075, 1/2009, Volume 323, Issue 5910, pp. 95 - 101
The perception of pathogen or microbe-associated molecular pattern molecules by plants triggers a basal defense response analogous to animal innate immunity... 
Hydrolysis | Pathogens | Molecules | Genes | Glucosinolates | Biosynthesis | Research Articles | Plants | Pretreatment | Seedlings | Plant cells | NONHOST RESISTANCE | ABC TRANSPORTER | PHYTOCHELATIN SYNTHASE | THALIANA | BIOSYNTHESIS | MULTIDISCIPLINARY SCIENCES | GLUTATHIONE-DEFICIENT MUTANT | CELL WALL | ABSCISIC-ACID | METAL-BINDING PEPTIDES | DISEASE RESISTANCE | Genes, Plant | Glucans - biosynthesis | Salicylic Acid - metabolism | Aminoacyltransferases - genetics | Arabidopsis - immunology | Glucosinolates - metabolism | Ethylenes - metabolism | Arabidopsis Proteins - metabolism | Indoles - metabolism | Salicylic Acid - pharmacology | ATP-Binding Cassette Transporters - genetics | Peptide Fragments - immunology | Gene Expression Regulation, Plant | ATP-Binding Cassette Transporters - metabolism | Indoles - pharmacology | Arabidopsis Proteins - genetics | Flagellin - immunology | Signal Transduction | N-Glycosyl Hydrolases - genetics | Transcription Factors - genetics | Immunity, Innate | Arabidopsis - metabolism | N-Glycosyl Hydrolases - metabolism | Arabidopsis - genetics | Transcription Factors - metabolism | Arabidopsis - microbiology | Aminoacyltransferases - metabolism | Glycoside Hydrolases - metabolism | Mutation | Immune response | Arabidopsis | Plant immunology | Physiological aspects | Research | Properties | Plant metabolites | Genetics | Molecular biology | Metabolism | Botany | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2015, Volume 5, Issue 1, pp. 17935 - 17935
Inflammasomes are cytoplasmic, multiprotein complexes that trigger caspase-1 activation and IL-1 beta maturation in response to diverse stimuli. Although... 
ASC | OXYGEN SPECIES PRODUCTION | STIMULATION | CASPASE-1 | MULTIDISCIPLINARY SCIENCES | SALMONELLA | IL-1-BETA | UNCOUPLING PROTEIN-2 | NALP3 INFLAMMASOME | CELL-DEATH | BETA | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Ion Channels - genetics | Caspase 1 - metabolism | Mitochondrial Proteins - genetics | Lipopolysaccharides - immunology | Autophagy - drug effects | Mitochondrial Proteins - metabolism | Flagellin - metabolism | Macrophages - immunology | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Peritonitis - etiology | Flagellin - immunology | Pneumonia - etiology | Cytokines - metabolism | Macrophage Activation - immunology | Apoptosis Regulatory Proteins - chemistry | Peritonitis - metabolism | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Carrier Proteins - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Ion Channels - metabolism | Iridoids - pharmacology | CARD Signaling Adaptor Proteins | Chemokines - metabolism | Mice | Uncoupling Protein 2 | Pneumonia - metabolism | Protein Multimerization - drug effects | Mitochondrial uncoupling protein 2 | Reactive oxygen species | Oligomerization | Lung | Neutrophils | Aluminum sulfate | Caspase | Flagellin | IL-1β | Autophagy | Inflammatory diseases | Caspase-1 | Lipopolysaccharides | Genipin | Peritonitis | Phagocytosis | Index Medicus
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 02/2007, Volume 127, Issue 2, pp. 331 - 341
Keratinocytes are continuously in contact with external stimuli and have the capacity to produce several soluble mediators. Pathogen-associated molecular... 
TOLL-LIKE-RECEPTORS | DOUBLE-STRANDED-RNA | DENDRITIC CELLS | SKIN-ASSOCIATED CHEMOKINE | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INNATE IMMUNE-RESPONSE | INFLAMMATORY PROTEIN 3-ALPHA | NECROSIS-FACTOR-ALPHA | FACTOR-KAPPA-B | HUMAN EPIDERMAL-KERATINOCYTES | DERMATOLOGY | Oligonucleotides - genetics | Phosphorylation | Toll-Like Receptor 5 - genetics | Humans | Toll-Like Receptor 9 - genetics | I-kappa B Proteins - metabolism | RNA, Messenger - metabolism | Flagellin - pharmacology | Tissue Distribution | Cell Nucleus - metabolism | Protein Isoforms - metabolism | Biological Transport | Oligonucleotides - pharmacology | Toll-Like Receptor 3 - genetics | Toll-Like Receptors - metabolism | Chemokine CXCL9 | Toll-Like Receptor 5 - metabolism | Poly I-C - pharmacology | Cytokines - metabolism | Cells, Cultured | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Chemokine CXCL10 | Transcription Factor RelA - metabolism | Keratinocytes - metabolism | Toll-Like Receptors - genetics | Chemokines, CXC - metabolism | Lipopolysaccharides - pharmacology | CpG Islands | Ligands | Chemokines - metabolism | Toll-Like Receptor 9 - metabolism | CD40 antigen | Oligonucleotides | Flagellin | mRNA | intercellular adhesion molecule 1 | Immunity | Lipopolysaccharides | CXC chemokines | CCL20 protein | Toll-like receptors | CpG islands | TLR7 protein | Dermatology | Keratinocytes | Nuclear transport | Stress | TLR3 protein | TLR9 protein | TLR1 protein | External stimuli | CXCL10 protein | Histocompatibility antigen HLA | Interferon | Internet | Monocyte chemoattractant protein 1 | Tumor necrosis factor- alpha | Index Medicus
Journal Article
Journal Article
The Journal of Nutritional Biochemistry, ISSN 0955-2863, 09/2013, Volume 24, Issue 9, pp. 1609 - 1615
The therapeutic effects of probiotic treatment in alcoholic liver disease (ALD) have been studied in both patients and experimental animal models. Although the... 
Probiotics | Alcohol | Inflammation | TNF | Liver | TRANSCRIPTION FACTORS | OXIDATIVE STRESS | CYTOKINE RESPONSES | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CHRONIC ETHANOL | NECROSIS-FACTOR-ALPHA | BLOOD MONONUCLEAR-CELLS | CHROMOGENIC ASSAY | NUTRITION & DIETETICS | MESSENGER-RNA | TOLL-LIKE RECEPTORS | Probiotics - administration & dosage | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Leukocytes, Mononuclear - metabolism | Intestinal Mucosa - metabolism | Liver - pathology | Phosphorylation | Toll-Like Receptor 5 - genetics | Humans | Lipopolysaccharides - metabolism | Male | Cytochrome P-450 CYP2E1 - genetics | Cytochrome P-450 CYP2E1 - metabolism | Gram-Negative Bacteria - metabolism | RNA, Messenger - metabolism | Lipopolysaccharides - adverse effects | Liver Diseases, Alcoholic - therapy | Flagellin - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Flagellin - adverse effects | Lactobacillus rhamnosus | Endotoxins - metabolism | Liver Diseases, Alcoholic - pathology | Toll-Like Receptor 5 - metabolism | Ethanol - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - metabolism | Inflammation - therapy | Macrophages - metabolism | Animals | Intestines - microbiology | Probiotics - therapeutic use | Mice | Endotoxins - adverse effects | Index Medicus
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 05/2004, Volume 30, Issue 5, pp. 627 - 634
Journal Article