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Journal Article
WORLD JOURNAL OF GASTROENTEROLOGY, ISSN 1007-9327, 02/2015, Volume 21, Issue 8, pp. 2323 - 2335
AIM: To investigate the role of profilin-1 (PFN1) in gastric cancer and the underlying mechanisms. METHODS: Immunohistochemical analysis, quantitative... 
MIGRATION | APOPTOSIS | PROTEIN | INVOLVEMENT | DOWN-REGULATION | CELL-PROLIFERATION | Fibronectin | FAK | INVASION | Focal adhesion kinase | PROTEOMIC ANALYSIS | GASTROENTEROLOGY & HEPATOLOGY | Gastric cancer | Integrin beta 1 | EXPRESSION | Profilin-1
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 2015, Volume 210, Issue 3, pp. 503 - 515
The Hippo pathway is involved in the regulation of contact inhibition of proliferation and responses to various physical and chemical stimuli. Recently,... 
YES-ASSOCIATED PROTEIN | GROWTH-CONTROL | ACTIVATION | FOCAL ADHESION | TYROSINE PHOSPHORYLATION | CONTACT INHIBITION | CELL-PROLIFERATION | SRC FAMILY KINASES | YAP PATHWAY | PHOSPHOINOSITIDE 3-KINASE | CELL BIOLOGY | Cell Line | Focal Adhesion Kinase 1 - genetics | Signal Transduction | Humans | src-Family Kinases - antagonists & inhibitors | Cell Proliferation - physiology | Protein-Serine-Threonine Kinases - genetics | Phosphatidylinositol 3-Kinases - metabolism | Phosphoproteins - antagonists & inhibitors | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Fibronectins - metabolism | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Contact Inhibition - physiology | Polylysine - metabolism | Phosphotransferases (Alcohol Group Acceptor) - antagonists & inhibitors | RNA Interference | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | src-Family Kinases - metabolism | RNA, Small Interfering | Laminin - metabolism | src-Family Kinases - genetics | Focal Adhesion Kinase 1 - metabolism | Protein-Serine-Threonine Kinases - metabolism | Fibronectins | Cellular signal transduction | Health aspects | Identification and classification | Phosphotransferases | Cell adhesion | Proteins | Signal transduction | Ligands | Epidermal growth factor | Kinases | Cell adhesion & migration | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 05/2016, Volume 534, Issue 7605, pp. 55 - 62
Somatic mutations have been extensively characterized in breast cancer, but the effects of these genetic alterations on the proteomic landscape remain poorly... 
PATHWAYS | HETEROGENEITY | PIK3CA MUTATIONS | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | GENES | BIOLOGY | RECEPTOR | EXPRESSION | SIGNATURE | REVEALS | Protein Kinases - metabolism | Focal Adhesion Kinase 1 - genetics | Receptor, Epidermal Growth Factor - genetics | Protein Kinases - genetics | Cyclin-Dependent Kinases - metabolism | Receptor, ErbB-2 - genetics | Receptors, G-Protein-Coupled - metabolism | Genomics | Humans | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Phosphoproteins - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - genetics | Tumor Suppressor Protein p53 - genetics | Breast Neoplasms - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - metabolism | Breast Neoplasms - enzymology | Receptor, Epidermal Growth Factor - metabolism | Phosphoproteins - analysis | Mass Spectrometry | src-Family Kinases - metabolism | Female | Cyclin-Dependent Kinases - genetics | Focal Adhesion Kinase 1 - metabolism | Chromosomes, Human, Pair 5 - genetics | Breast Neoplasms - classification | Chromosome Deletion | p21-Activated Kinases - genetics | Signal Transduction | Molecular Sequence Annotation | Calcium-Binding Proteins - deficiency | Phosphoproteins - genetics | Mutation - genetics | S-Phase Kinase-Associated Proteins - metabolism | p21-Activated Kinases - metabolism | Phosphatidylinositol 3-Kinases - genetics | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | Proteomics | S-Phase Kinase-Associated Proteins - genetics | Receptors, G-Protein-Coupled - genetics | src-Family Kinases - genetics | Calcium-Binding Proteins - genetics | Breast cancer | Genetic aspects | Research | Oncology, Experimental | Cancer | Physiological aspects | Methods | Mutation (Biology) | Proteins | Gene amplification | Peptides | Protein expression | Genomes | Mutation | Kinases | Deoxyribonucleic acid--DNA | Tumors | Index Medicus
Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 02/2004, Volume 6, Issue 2, pp. 154 - 161
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 10/2013, Volume 105, Issue 19, pp. 1485 - 1495
Journal Article
Journal Article
Journal Article