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1. Full Text High folic acid consumption leads to pseudo-MTHFR deficiency, altered lipid metabolism, and liver injury in mice
by Christensen, Karen E and Mikael, Leonie G and Leung, Kit-Yi and Lévesque, Nancy and Deng, Liyuan and Wu, Qing and Malysheva, Olga V and Best, Ana and Caudill, Marie A and Greene, Nicholas D.E and Rozen, Rima
The American journal of clinical nutrition, ISSN 0002-9165, 03/2015, Volume 101, Issue 3, pp. 646 - 658
Methylenetetrahydrofolate reductase | Lipid metabolism | Liver | Choline metabolism | Folic acid | Life Sciences & Biomedicine | Nutrition & Dietetics | Science & Technology | Liver - pathology | Enzyme Inhibitors - poisoning | Folic Acid - poisoning | Non-alcoholic Fatty Liver Disease - etiology | Homocystinuria - etiology | Male | Methylenetetrahydrofolate Reductase (NADPH2) - metabolism | Homocystinuria - metabolism | Liver - physiopathology | Homocystinuria - physiopathology | Psychotic Disorders - physiopathology | Methylenetetrahydrofolate Reductase (NADPH2) - genetics | Mice, Mutant Strains | Psychotic Disorders - pathology | Lipogenesis | Methylenetetrahydrofolate Reductase (NADPH2) - deficiency | Dietary Supplements - poisoning | Methylenetetrahydrofolate Reductase (NADPH2) - antagonists & inhibitors | Specific Pathogen-Free Organisms | Liver - metabolism | Gene Expression Regulation | Organ Size | Lipid Metabolism | Muscle Spasticity - metabolism | Muscle Spasticity - pathology | Homocystinuria - pathology | Muscle Spasticity - etiology | Psychotic Disorders - metabolism | Animals | Heterozygote | Mice, Inbred BALB C | Muscle Spasticity - physiopathology | Mutation | Methylation | Psychotic Disorders - etiology | Liver diseases | Metabolism | Health aspects | Risk factors | Lipids | Vitamin B | Rodents | Index Medicus | Abridged Index Medicus
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2. Full Text Folic acid deficiency increases brain cell injury via autophagy enhancement after focal cerebral ischemia
by Zhao, Yaqian and Huang, Guowei and Chen, Shuang and Gou, Yun and Dong, Zhiping and Zhang, Xumei
The Journal of nutritional biochemistry, ISSN 0955-2863, 12/2016, Volume 38, pp. 41 - 49
Oxidative stress | Folic acid deficiency | Autophagy | Cerebral ischemia–reperfusion | Cortex | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Nutrition & Dietetics | Science & Technology | Folic Acid Deficiency - metabolism | Neurons - pathology | Cell Body - drug effects | Microtubule-Associated Proteins - metabolism | Cerebral Cortex - pathology | Brain Ischemia - metabolism | Male | Cell Body - ultrastructure | Autophagy - drug effects | Cerebral Cortex - metabolism | Cerebral Cortex - ultrastructure | Autophagosomes - drug effects | Neurons - ultrastructure | Adenine - therapeutic use | Neurons - metabolism | Cerebral Cortex - drug effects | Neurons - drug effects | Biomarkers - metabolism | Brain Ischemia - complications | DNA Damage - drug effects | Microscopy, Electron, Transmission | Adenine - analogs & derivatives | Specific Pathogen-Free Organisms | Cell Body - pathology | Cell Body - metabolism | Autophagosomes - metabolism | Random Allocation | Rats, Sprague-Dawley | Autophagosomes - pathology | Autophagosomes - ultrastructure | Nerve Tissue Proteins - metabolism | Up-Regulation - drug effects | Animals | Folic Acid Deficiency - complications | Reperfusion Injury - prevention & control | Folic Acid Deficiency - pathology | Brain Ischemia - pathology | Oxidative Stress - drug effects | Beclin-1 - metabolism | Stroke (Disease) | Brain | Cerebral ischemia | Neurons | Cell death | Brain damage | Injuries | Folic acid | Safety and security measures | Food | Index Medicus
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3. Full Text Finding the genetic mechanisms of folate deficiency and neural tube defects—Leaving no stone unturned
by Au, Kit Sing and Findley, Tina O and Northrup, Hope
American journal of medical genetics. Part A, ISSN 1552-4825, 11/2017, Volume 173, Issue 11, pp. 3042 - 3057
folate one‐carbon metabolism | genetic variants | genetic association | human neural tube defects | methylation | folate one-carbon metabolism | Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | United States | Humans | Neural Tube Defects - genetics | Folic Acid Deficiency - epidemiology | Neural Tube Defects - epidemiology | Neural Tube Defects - pathology | Socioeconomic Factors | Neural Tube - pathology | Pregnancy | Folic Acid Deficiency - pathology | Neural Tube - metabolism | Folic Acid - metabolism | Female | Folic Acid - genetics | Folic Acid Deficiency - genetics | Neural Tube - growth & development | Genetic research | Folic acid | Grain | Neural tube defects | Vitamin B | Socio-economic aspects | DNA methylation | Vitamin deficiency | Genomes | FDA approval | Gestation | Gene expression | Index Medicus
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4. Full Text Clinicopathologic features of folate-deficiency neuropathy
by Koike, Haruki and Takahashi, Mie and Ohyama, Ken and Hashimoto, Rina and Kawagashira, Yuichi and Iijima, Masahiro and Katsuno, Masahisa and Doi, Hiroshi and Tanaka, Fumiaki and Sobue, Gen
Neurology, ISSN 0028-3878, 03/2015, Volume 84, Issue 10, pp. 1026 - 1033
Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Folic Acid Deficiency - diet therapy | Folic Acid - pharmacology | Humans | Middle Aged | Male | Treatment Outcome | Vitamin B 12 Deficiency - physiopathology | Polyneuropathies - physiopathology | Vitamin B Complex - pharmacology | Folic Acid - administration & dosage | Beriberi - physiopathology | Disease Progression | Vitamin B Complex - administration & dosage | Polyneuropathies - chemically induced | Folic Acid Deficiency - complications | Sural Nerve - pathology | Polyneuropathies - pathology | Aged, 80 and over | Adult | Female | Aged | Alcoholism - complications | Folic Acid Deficiency - blood | Folic Acid Deficiency - drug therapy | Index Medicus | Abridged Index Medicus
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5. Full Text Vitamin B-12 deficiency from the perspective of a practicing hematologist
by Green, Ralph
Blood, ISSN 0006-4971, 05/2017, Volume 129, Issue 19, pp. 2603 - 2611
Life Sciences & Biomedicine | Hematology | Science & Technology | Humans | Vitamin B 12 Deficiency - pathology | Vitamin B 12 Deficiency - therapy | Vitamin B 12 - blood | Anemia, Megaloblastic - etiology | Intestinal Absorption | Anemia, Megaloblastic - metabolism | Animals | Anemia, Pernicious - metabolism | Vitamin B 12 Deficiency - complications | Folic Acid - metabolism | Anemia, Pernicious - etiology | Vitamin B 12 Deficiency - metabolism | Anemia, Megaloblastic - pathology | Vitamin B 12 - metabolism | Anemia, Pernicious - pathology
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6. Full Text Elevated levels of plasma homocysteine, deficiencies in dietary folic acid and uracil–DNA glycosylase impair learning in a mouse model of vascular cognitive impairment
by Jadavji, Nafisa M and Farr, Tracy D and Lips, Janet and Khalil, Ahmed A and Boehm-Sturm, Philipp and Foddis, Marco and Harms, Christoph and Füchtemeier, Martina and Dirnagl, Ulrich
Behavioural brain research, ISSN 0166-4328, 04/2015, Volume 283, pp. 215 - 226
Chronic hypoperfusion | Matrix metalloproteinases-9 | Uracil-DNA glycosylase | Homocysteine | Morris watermaze | Folate | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Behavioral Sciences | Science & Technology | Learning Disorders - physiopathology | Male | Gliosis - physiopathology | Glial Fibrillary Acidic Protein | Gliosis - pathology | Learning Disorders - pathology | Brain - blood supply | Matrix Metalloproteinase 9 - metabolism | Basilar Artery - physiopathology | Female | Disease Models, Animal | Uracil-DNA Glycosidase - deficiency | Cerebrovascular Disorders - physiopathology | Cognition Disorders - physiopathology | Maze Learning - physiology | Brain - physiopathology | Mice, Inbred C57BL | Cognition Disorders - pathology | Homocysteine - blood | Random Allocation | Folic Acid Deficiency - physiopathology | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Diet | Folic Acid Deficiency - pathology | Carotid Artery Diseases | Basilar Artery - pathology | Brain - pathology | Cerebrovascular Disorders - pathology | Chronic Disease | Uracil-DNA Glycosidase - genetics | Pyrimidines | DNA | Folic acid | Index Medicus
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7. Full Text PGC‐1α deficiency causes spontaneous kidney inflammation and increases the severity of nephrotoxic AKI
by Fontecha‐Barriuso, Miguel and Martín‐Sánchez, Diego and Martinez‐Moreno, Julio M and Carrasco, Susana and Ruiz‐Andrés, Olga and Monsalve, Maria and Sanchez‐Ramos, Cristina and Gómez, Manuel J and Ruiz‐Ortega, Marta and Sánchez‐Niño, Maria D and Cannata‐Ortiz, Pablo and Cabello, Ramiro and Gonzalez‐Enguita, Carmen and Ortiz, Alberto and Sanz, Ana B
The Journal of pathology, ISSN 0022-3417, 09/2019, Volume 249, Issue 1, pp. 65 - 78
acute kidney injury | cell death | proximal tubule | inflammation | mitochondria | PGC‐1α | PGC-1α | Pathology | Oncology | Life Sciences & Biomedicine | Science & Technology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - genetics | Cell Proliferation | Kidney - pathology | Humans | Acute Kidney Injury - genetics | Organelle Biogenesis | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - deficiency | Kidney - metabolism | Nephritis, Interstitial - metabolism | Nephritis, Interstitial - pathology | Mitochondria - genetics | Cell Death | Inflammation Mediators - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Female | Acute Kidney Injury - chemically induced | Lipocalin-2 - genetics | Kidney - physiopathology | Disease Models, Animal | Folic Acid | Severity of Illness Index | Cell Line | Cytokines - metabolism | Signal Transduction | Acute Kidney Injury - pathology | Mice, Inbred C57BL | Nephritis, Interstitial - physiopathology | Mitochondria - metabolism | Mitochondria - pathology | Mice, Knockout | Animals | Lipocalin-2 - metabolism | Nephritis, Interstitial - genetics | Acute Kidney Injury - metabolism | Kidneys | Renal function | Deactivation | Cytokines | Genes | Mortality | Homeostasis | Inflammation | Gene expression | Inactivation | Folic acid | Energy balance | Reduction | Mitochondria | Cell activation | Injury prevention | Cell death | Mice | Clear cell-type renal cell carcinoma | Injury analysis | Apoptosis | Index Medicus
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