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The American journal of clinical nutrition, ISSN 0002-9165, 03/2015, Volume 101, Issue 3, pp. 646 - 658
Methylenetetrahydrofolate reductase | Lipid metabolism | Liver | Choline metabolism | Folic acid | Life Sciences & Biomedicine | Nutrition & Dietetics | Science & Technology | Liver - pathology | Enzyme Inhibitors - poisoning | Folic Acid - poisoning | Non-alcoholic Fatty Liver Disease - etiology | Homocystinuria - etiology | Male | Methylenetetrahydrofolate Reductase (NADPH2) - metabolism | Homocystinuria - metabolism | Liver - physiopathology | Homocystinuria - physiopathology | Psychotic Disorders - physiopathology | Methylenetetrahydrofolate Reductase (NADPH2) - genetics | Mice, Mutant Strains | Psychotic Disorders - pathology | Lipogenesis | Methylenetetrahydrofolate Reductase (NADPH2) - deficiency | Dietary Supplements - poisoning | Methylenetetrahydrofolate Reductase (NADPH2) - antagonists & inhibitors | Specific Pathogen-Free Organisms | Liver - metabolism | Gene Expression Regulation | Organ Size | Lipid Metabolism | Muscle Spasticity - metabolism | Muscle Spasticity - pathology | Homocystinuria - pathology | Muscle Spasticity - etiology | Psychotic Disorders - metabolism | Animals | Heterozygote | Mice, Inbred BALB C | Muscle Spasticity - physiopathology | Mutation | Methylation | Psychotic Disorders - etiology | Liver diseases | Metabolism | Health aspects | Risk factors | Lipids | Vitamin B | Rodents | Index Medicus | Abridged Index Medicus
Journal Article
The Journal of nutritional biochemistry, ISSN 0955-2863, 12/2016, Volume 38, pp. 41 - 49
Oxidative stress | Folic acid deficiency | Autophagy | Cerebral ischemia–reperfusion | Cortex | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Nutrition & Dietetics | Science & Technology | Folic Acid Deficiency - metabolism | Neurons - pathology | Cell Body - drug effects | Microtubule-Associated Proteins - metabolism | Cerebral Cortex - pathology | Brain Ischemia - metabolism | Male | Cell Body - ultrastructure | Autophagy - drug effects | Cerebral Cortex - metabolism | Cerebral Cortex - ultrastructure | Autophagosomes - drug effects | Neurons - ultrastructure | Adenine - therapeutic use | Neurons - metabolism | Cerebral Cortex - drug effects | Neurons - drug effects | Biomarkers - metabolism | Brain Ischemia - complications | DNA Damage - drug effects | Microscopy, Electron, Transmission | Adenine - analogs & derivatives | Specific Pathogen-Free Organisms | Cell Body - pathology | Cell Body - metabolism | Autophagosomes - metabolism | Random Allocation | Rats, Sprague-Dawley | Autophagosomes - pathology | Autophagosomes - ultrastructure | Nerve Tissue Proteins - metabolism | Up-Regulation - drug effects | Animals | Folic Acid Deficiency - complications | Reperfusion Injury - prevention & control | Folic Acid Deficiency - pathology | Brain Ischemia - pathology | Oxidative Stress - drug effects | Beclin-1 - metabolism | Stroke (Disease) | Brain | Cerebral ischemia | Neurons | Cell death | Brain damage | Injuries | Folic acid | Safety and security measures | Food | Index Medicus
Journal Article
American journal of medical genetics. Part A, ISSN 1552-4825, 11/2017, Volume 173, Issue 11, pp. 3042 - 3057
folate one‐carbon metabolism | genetic variants | genetic association | human neural tube defects | methylation | folate one-carbon metabolism | Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | United States | Humans | Neural Tube Defects - genetics | Folic Acid Deficiency - epidemiology | Neural Tube Defects - epidemiology | Neural Tube Defects - pathology | Socioeconomic Factors | Neural Tube - pathology | Pregnancy | Folic Acid Deficiency - pathology | Neural Tube - metabolism | Folic Acid - metabolism | Female | Folic Acid - genetics | Folic Acid Deficiency - genetics | Neural Tube - growth & development | Genetic research | Folic acid | Grain | Neural tube defects | Vitamin B | Socio-economic aspects | DNA methylation | Vitamin deficiency | Genomes | FDA approval | Gestation | Gene expression | Index Medicus
Journal Article
Neurology, ISSN 0028-3878, 03/2015, Volume 84, Issue 10, pp. 1026 - 1033
Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Folic Acid Deficiency - diet therapy | Folic Acid - pharmacology | Humans | Middle Aged | Male | Treatment Outcome | Vitamin B 12 Deficiency - physiopathology | Polyneuropathies - physiopathology | Vitamin B Complex - pharmacology | Folic Acid - administration & dosage | Beriberi - physiopathology | Disease Progression | Vitamin B Complex - administration & dosage | Polyneuropathies - chemically induced | Folic Acid Deficiency - complications | Sural Nerve - pathology | Polyneuropathies - pathology | Aged, 80 and over | Adult | Female | Aged | Alcoholism - complications | Folic Acid Deficiency - blood | Folic Acid Deficiency - drug therapy | Index Medicus | Abridged Index Medicus
Journal Article
Blood, ISSN 0006-4971, 05/2017, Volume 129, Issue 19, pp. 2603 - 2611
Life Sciences & Biomedicine | Hematology | Science & Technology | Humans | Vitamin B 12 Deficiency - pathology | Vitamin B 12 Deficiency - therapy | Vitamin B 12 - blood | Anemia, Megaloblastic - etiology | Intestinal Absorption | Anemia, Megaloblastic - metabolism | Animals | Anemia, Pernicious - metabolism | Vitamin B 12 Deficiency - complications | Folic Acid - metabolism | Anemia, Pernicious - etiology | Vitamin B 12 Deficiency - metabolism | Anemia, Megaloblastic - pathology | Vitamin B 12 - metabolism | Anemia, Pernicious - pathology
Journal Article
Behavioural brain research, ISSN 0166-4328, 04/2015, Volume 283, pp. 215 - 226
Chronic hypoperfusion | Matrix metalloproteinases-9 | Uracil-DNA glycosylase | Homocysteine | Morris watermaze | Folate | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Behavioral Sciences | Science & Technology | Learning Disorders - physiopathology | Male | Gliosis - physiopathology | Glial Fibrillary Acidic Protein | Gliosis - pathology | Learning Disorders - pathology | Brain - blood supply | Matrix Metalloproteinase 9 - metabolism | Basilar Artery - physiopathology | Female | Disease Models, Animal | Uracil-DNA Glycosidase - deficiency | Cerebrovascular Disorders - physiopathology | Cognition Disorders - physiopathology | Maze Learning - physiology | Brain - physiopathology | Mice, Inbred C57BL | Cognition Disorders - pathology | Homocysteine - blood | Random Allocation | Folic Acid Deficiency - physiopathology | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Diet | Folic Acid Deficiency - pathology | Carotid Artery Diseases | Basilar Artery - pathology | Brain - pathology | Cerebrovascular Disorders - pathology | Chronic Disease | Uracil-DNA Glycosidase - genetics | Pyrimidines | DNA | Folic acid | Index Medicus
Journal Article
The Journal of pathology, ISSN 0022-3417, 09/2019, Volume 249, Issue 1, pp. 65 - 78
acute kidney injury | cell death | proximal tubule | inflammation | mitochondria | PGC‐1α | PGC-1α | Pathology | Oncology | Life Sciences & Biomedicine | Science & Technology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - genetics | Cell Proliferation | Kidney - pathology | Humans | Acute Kidney Injury - genetics | Organelle Biogenesis | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - deficiency | Kidney - metabolism | Nephritis, Interstitial - metabolism | Nephritis, Interstitial - pathology | Mitochondria - genetics | Cell Death | Inflammation Mediators - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Female | Acute Kidney Injury - chemically induced | Lipocalin-2 - genetics | Kidney - physiopathology | Disease Models, Animal | Folic Acid | Severity of Illness Index | Cell Line | Cytokines - metabolism | Signal Transduction | Acute Kidney Injury - pathology | Mice, Inbred C57BL | Nephritis, Interstitial - physiopathology | Mitochondria - metabolism | Mitochondria - pathology | Mice, Knockout | Animals | Lipocalin-2 - metabolism | Nephritis, Interstitial - genetics | Acute Kidney Injury - metabolism | Kidneys | Renal function | Deactivation | Cytokines | Genes | Mortality | Homeostasis | Inflammation | Gene expression | Inactivation | Folic acid | Energy balance | Reduction | Mitochondria | Cell activation | Injury prevention | Cell death | Mice | Clear cell-type renal cell carcinoma | Injury analysis | Apoptosis | Index Medicus
Journal Article