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PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, pp. 315 - 326
Secondary damage following primary spinal cord injury extends pathology beyond the site of initial trauma, and effective management is imperative for... 
SURVIVAL | PROTEIN-KINASE-B | TYROSINE-PHOSPHATASE INHIBITION | MULTIDISCIPLINARY SCIENCES | CONTUSION INJURY | PTEN | AKT | UP-REGULATION | EXPRESSION | BRAIN | CELL-DEATH | Neuroprotective Agents - therapeutic use | Spinal Cord Injuries - drug therapy | Recovery of Function - drug effects | TOR Serine-Threonine Kinases - metabolism | Microtubule-Associated Proteins - metabolism | Cervical Vertebrae - blood supply | Protein Transport - drug effects | Autophagy - drug effects | Motor Neurons - pathology | Spinal Cord Injuries - pathology | Forelimb - physiopathology | Neuroprotective Agents - pharmacology | Spinal Cord Injuries - enzymology | Vanadium Compounds - pharmacology | Female | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Forelimb - drug effects | Motor Neurons - drug effects | Contusions - complications | Phagosomes - drug effects | Cervical Vertebrae - drug effects | Phagosomes - metabolism | Contusions - physiopathology | PTEN Phosphohydrolase - metabolism | Rats | Vanadium Compounds - therapeutic use | Enzyme Activation - drug effects | Rats, Sprague-Dawley | Contusions - drug therapy | Animals | Signal Transduction - drug effects | Models, Biological | Cervical Vertebrae - pathology | Spinal Cord Injuries - physiopathology | Spinal cord injuries | Phosphatases | TOR protein | Neuroprotection | Neurosciences | Spinal cord | Vanadium compounds | Traumatic brain injury | Central nervous system | Vanadium | Recovery of function | Homology | AKT protein | Spinal cord injury | Kinases | Phosphatase | Autophagy | Western blotting | Proteins | Rodents | Cell cycle | Bisperoxovanadium | Tumorigenesis | Tensin | Hematology | Neurons | Rapamycin | Pharmacology | Functional analysis | Trauma | Medicine | Signaling | Injury prevention | Brain research | Insects | Immunofluorescence | Potassium | Histochemistry | Phagocytosis | Apoptosis
Journal Article
Biomaterials, ISSN 0142-9612, 2013, Volume 35, Issue 9, pp. 2617 - 2629
Abstract While neural stem/progenitor cells (NPCs) show promise for traumatic spinal cord injury (SCI), their efficacy in cervical SCI remains to be... 
Advanced Basic Science | Dentistry | Cervical cord injury | Functional recovery | Tissue preservation | Neural precursor cell | Self-assembling nanofiber | CHONDROITINASE ABC | AXONS | MATERIALS SCIENCE, BIOMATERIALS | NEUROLOGICAL RECOVERY | ENGINEERING, BIOMEDICAL | GLIAL SCAR | REGENERATION | TRANSPLANTATION | NANOFIBER SCAFFOLD | INFLAMMATION | STEM-CELL | DYSFUNCTION | Inflammation - pathology | Spinal Cord Injuries - drug therapy | Rats, Wistar | Recovery of Function - drug effects | Motor Activity - drug effects | Gliosis - physiopathology | Neural Stem Cells - cytology | Motor Neurons - pathology | Spinal Cord Injuries - pathology | Gliosis - pathology | Forelimb - physiopathology | Forelimb - pathology | Microglia - pathology | Neural Stem Cells - transplantation | Female | Cervical Vertebrae - physiopathology | Spinal Cord Injuries - therapy | Forelimb - drug effects | Wound Healing - drug effects | Motor Neurons - drug effects | Cell Survival - drug effects | Microglia - drug effects | Cervical Vertebrae - drug effects | Rats | Peptides - pharmacology | Cell Movement - drug effects | Gait - drug effects | Animals | Cervical Vertebrae - pathology | Gliosis - drug therapy | Spinal Cord Injuries - physiopathology | Peptides - therapeutic use | Neurosciences | Spinal cord injuries | Peptides | Analysis | Earthquakes | Neurons
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 04/2011, Volume 31, Issue 16, pp. 5977 - 5988
Journal Article
Journal Article
Neuroscience, ISSN 0306-4522, 2008, Volume 152, Issue 4, pp. 959 - 969
Journal Article
Experimental Neurology, ISSN 0014-4886, 03/2013, Volume 241, Issue 1, pp. 45 - 55
T-lymphocytes promote cerebral inflammation, thus aggravating neuronal injury after stroke. Fingolimod, a sphingosine 1-phosphate receptor analog, prevents the... 
Neuroprotection | Fingolimod | Inflammation | Brain edema | Behavior | Lymphocyte | Intracerebral hemorrhage | ARTERY OCCLUSION | BASAL GANGLIA | NEUROSCIENCES | RAT MODEL | CELL-DEATH | STROKE | SPHINGOSINE 1-PHOSPHATE RECEPTOR-1 | INTERFERON-GAMMA | MOUSE MODEL | SUBARACHNOID HEMORRHAGE | ISCHEMIC BRAIN-INJURY | Cell Count | Immunosuppressive Agents - therapeutic use | Cerebral Cortex - pathology | Male | Interferon-gamma - metabolism | Cerebral Hemorrhage - immunology | Collagenases - toxicity | Brain Edema - prevention & control | Forelimb - physiopathology | Time Factors | Space Perception - drug effects | Propylene Glycols - therapeutic use | Functional Laterality - drug effects | Behavior, Animal - drug effects | Cerebral Hemorrhage - etiology | Disease Models, Animal | Cerebral Hemorrhage - chemically induced | Lymphocytes - metabolism | Propylene Glycols - pharmacology | Blood Transfusion, Autologous - adverse effects | Cerebral Hemorrhage - drug therapy | Brain Edema - etiology | Fingolimod Hydrochloride | Rats | Behavior, Animal - physiology | Basal Ganglia - pathology | Maze Learning - drug effects | CD3 Complex - metabolism | Rats, Sprague-Dawley | Psychomotor Disorders - drug therapy | Psychomotor Disorders - etiology | Sphingosine - pharmacology | Interleukin-17 - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Nervous System Diseases - prevention & control | Sphingosine - analogs & derivatives | Animals | Analysis of Variance | Lymphocytes - drug effects | Nervous System Diseases - etiology | Leukocytes - drug effects | Sphingosine - therapeutic use | Mice | Cerebral Hemorrhage - complications | Cerebral Hemorrhage - pathology | Brain Edema
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 6, p. e21402
Androgens are important regulators of bone mass but the relative importance of testosterone (T) versus dihydrotestosterone (DHT) for the activation of the... 
ESTROGEN-RECEPTOR-BETA | ANDROGEN RECEPTOR | TRABECULAR BONE | CORTICAL BONE | TESTOSTERONE-RECEPTOR | MULTIDISCIPLINARY SCIENCES | SRD5A3 | ALPHA | GROWTH-PLATE | STEROID 5-ALPHA-REDUCTASE | EXPRESSION | Bone and Bones - pathology | Bone Density - physiology | Forelimb - physiology | Receptors, Prolactin - blood | Body Weight - drug effects | Male | Gene Expression Profiling | RNA, Messenger - metabolism | Testosterone - pharmacology | Membrane Proteins - deficiency | Isoenzymes - metabolism | 3-Oxo-5-alpha-Steroid 4-Dehydrogenase - metabolism | Female | Ovary - drug effects | Membrane Proteins - metabolism | Forelimb - drug effects | Ovary - pathology | Viscera - metabolism | Isoenzymes - genetics | Membrane Proteins - genetics | RNA, Messenger - genetics | Receptors, Prolactin - genetics | Viscera - drug effects | Enzyme Activation - drug effects | 3-Oxo-5-alpha-Steroid 4-Dehydrogenase - deficiency | Mice, Knockout | Orchiectomy | Bone Density - drug effects | Bone and Bones - physiopathology | Organ Size - drug effects | Animals | 3-Oxo-5-alpha-Steroid 4-Dehydrogenase - genetics | Mice | Muscle Strength - drug effects | Androgens - metabolism | Body Weight - physiology | Hand Strength - physiology | Muscle Strength - physiology | Regulators | Liver | Estrogen | Arthritis | Insulin-like growth factors | Bone (trabecular) | Males | Dihydrotestosterone | Coding | Rodents | Animal tissues | Grip strength | Muscular strength | Bone (cortical) | Drug dosages | Polycystic ovary syndrome | Enzymes | Isoenzymes | Metabolism | Bone mineral content | Medicine | Testosterone | Androgens | Bone mass | Nitric oxide | Bone mineral density | Clinical medicine | Females | Prostate | Reductase | Bone and Bones | Body Weight | pathology | RNA | Ovary | Receptors | genetics | Endokrinologi och diabetes | Prolactin | Viscera | pharmacology | Muscle Strength | Forelimb | Hand Strength | Endocrinology and Diabetes | Bone Density | physiology | Organ Size | drug effects | Messenger | blood | Knockout | 3-Oxo-5-alpha-Steroid 4-Dehydrogenase | Membrane Proteins | physiopathology | deficiency | metabolism | Enzyme Activation
Journal Article
Journal Article
Brain, ISSN 0006-8950, 2012, Volume 135, Issue 4, pp. 1210 - 1223
Stroke is the dominant cause of sensorimotor disability that primarily affects the elderly. We now show that neuroplasticity and functional recovery after... 
plasticity | ageing | chondroitin sulphate proteoglycans | stroke | perineuronal nets | FUNCTIONAL RECOVERY | INDUSTRY-ROUND-TABLE | FOCAL CEREBRAL-ISCHEMIA | ADULT-RATS | CORTEX LESIONS | PREDICTS MOTOR IMPAIRMENT | NEUROSCIENCES | CLINICAL NEUROLOGY | LESS-AFFECTED FORELIMB | BRAIN REORGANIZATION | TACTILE EXTINCTION | SPINAL-CORD-INJURY | Recovery of Function - drug effects | Rats, Long-Evans | Chondroitin ABC Lyase - administration & dosage | Neuronal Plasticity - drug effects | Sensation Disorders - etiology | Stroke - complications | Male | Stroke - pathology | Forelimb - physiopathology | Plant Lectins | Time Factors | Acoustic Stimulation - adverse effects | Aging | Functional Laterality - drug effects | Biotin - metabolism | Female | Neurons - metabolism | Neurons - drug effects | Brain Infarction - drug therapy | Pyramidal Tracts - pathology | Disease Models, Animal | Dextrans - metabolism | Double-Blind Method | Chondroitin Sulfate Proteoglycans - metabolism | Rats | Amidines | Psychomotor Performance - drug effects | Stroke - drug therapy | Brain Infarction - etiology | Animals | Analysis of Variance | Movement Disorders - etiology | Gait Disorders, Neurologic - drug therapy | Injections, Spinal - methods | Receptors, N-Acetylglucosamine | Gait Disorders, Neurologic - etiology | Biotin - analogs & derivatives | Spinal cord | Stroke | Brain architecture | sensorimotor system | Clinical trials | Recovery of function | Nervous system | Pyramidal tracts | Retrograde transport | Anterograde transport | Plasticity (functional) | Chondroitin ABC lyase | Plasticity (spinal) | Functional anatomy | Extracellular matrix | Geriatrics | aging
Journal Article
Psychopharmacology, ISSN 0033-3158, 2014, Volume 231, Issue 12, pp. 2405 - 2415
Journal Article
Tissue Engineering Part A, ISSN 1937-3341, 12/2011, Volume 17, Issue 23-24, pp. 333 - 3044
Journal Article