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Journal Article
Journal Article
Experimental Neurology, ISSN 0014-4886, 2005, Volume 194, Issue 1, pp. 161 - 174
Approximately 50% of human spinal cord injuries (SCI) are at the cervical level, resulting in impairments in motor function of the upper extremity. Even modest... 
Cervical injury | Forelimb behavior | Corticospinal tract | Digital flexors | Grip strength | PATHWAYS | RED NUCLEUS | MOVEMENTS | forelimb behavior | digital flexors | cervical injury | OVERGROUND LOCOMOTION | NEUROSCIENCES | LESIONS | ORGANIZATION | RECOVERY | PROMOTE AXONAL REGENERATION | corticospinal tract | grip strength | DORSAL COLUMN | PROJECTIONS | Somatosensory Disorders - etiology | Spinal Cord Injuries - complications | Movement Disorders - diagnosis | Muscle, Skeletal - innervation | Forelimb - innervation | Forelimb - physiopathology | Pyramidal Tracts - physiopathology | Dextrans | Neurologic Examination - instrumentation | Somatosensory Disorders - diagnosis | Somatosensory Disorders - physiopathology | Spinal Cord - pathology | Gait Disorders, Neurologic - physiopathology | Pyramidal Tracts - injuries | Recovery of Function - physiology | Female | Neck Injuries - physiopathology | Paresis - etiology | Disease Models, Animal | Disability Evaluation | Motor Skills - physiology | Rats | Gait Disorders, Neurologic - diagnosis | Rats, Sprague-Dawley | Animals | Movement Disorders - physiopathology | Movement Disorders - etiology | Muscle, Skeletal - physiopathology | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Neurologic Examination - methods | Hand Strength - physiology | Gait Disorders, Neurologic - etiology | Paresis - diagnosis | Spinal Cord Injuries - diagnosis | Biotin - analogs & derivatives | Neck Injuries - diagnosis | Paresis - physiopathology | Spinal cord injuries | Index Medicus
Journal Article
Experimental Neurology, ISSN 0014-4886, 2009, Volume 216, Issue 1, pp. 193 - 206
The purpose of this research was to investigate whether pathways in the dorsal part of the lateral spinal funiculus (DLF) can compensate for loss of... 
Dorsolateral funiculus | Locomotion | Ground reaction forces | Skilled reaching | Horizontal ladder | Rubrospinal tract | Behaviour | Compensation | Kinematics | Corticospinal tract | Kinetics | RED NUCLEUS | SENSORIMOTOR BEHAVIOR | ADULT RATS | OPERANTLY CONDITIONED RATS | DORSAL FUNICULAR LESIONS | OVERGROUND LOCOMOTION | MOTOR-PERFORMANCE | FUNCTIONAL ORGANIZATION | NEUROSCIENCES | CORD-INJURY | Rats, Long-Evans | Muscle, Skeletal - innervation | Forelimb - innervation | Forelimb - physiopathology | Pyramidal Tracts - physiopathology | Neuronal Plasticity - physiology | Spinal Cord - pathology | Gait Disorders, Neurologic - physiopathology | Pyramidal Tracts - injuries | Gait Disorders, Neurologic - rehabilitation | Recovery of Function - physiology | Female | Pyramidal Tracts - pathology | Motor Skills - physiology | Adaptation, Physiological - physiology | Rats | Efferent Pathways - pathology | Spinal Cord - anatomy & histology | Efferent Pathways - physiopathology | Animals | Movement Disorders - physiopathology | Efferent Pathways - injuries | Muscle, Skeletal - physiopathology | Red Nucleus - physiopathology | Extremities - innervation | Spinal Cord Injuries - rehabilitation | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Movement Disorders - rehabilitation | Locomotion - physiology | Extremities - physiopathology | Index Medicus
Journal Article
Journal Article
Experimental Neurology, ISSN 0014-4886, 2007, Volume 206, Issue 2, pp. 318 - 331
There is increasing interest in developing rodent models for cervical spinal cord injury (SCI) and techniques to assess forelimb motor function. Previously, we... 
Cervical injury | Corticospinal tract | Digital flexors | Grip strength | Rat | Forelimb | MOVEMENTS | rat | digital flexors | STABILITY | cervical injury | NEUROSCIENCES | LESIONS | CORD CONTUSION INJURY | RECOVERY | corticospinal tract | QUANTITATIVE ASSESSMENT | grip strength | MICE | forelimb | DORSAL COLUMN | Muscle Strength Dynamometer | Wallerian Degeneration - pathology | Muscle, Skeletal - innervation | Gliosis - physiopathology | Glial Fibrillary Acidic Protein - metabolism | Forelimb - innervation | Spinal Cord Injuries - pathology | Gliosis - pathology | Forelimb - physiopathology | Pyramidal Tracts - physiopathology | Dextrans | Movement - physiology | Neuronal Plasticity - physiology | Recovery of Function - physiology | Female | Pyramidal Tracts - physiology | Disease Models, Animal | Muscle Weakness - etiology | Motor Skills - physiology | Movement Disorders - pathology | Gliosis - metabolism | Wallerian Degeneration - physiopathology | Adaptation, Physiological - physiology | Rats | Rats, Sprague-Dawley | Efferent Pathways - physiopathology | Animals | Movement Disorders - physiopathology | Movement Disorders - etiology | Muscle, Skeletal - physiopathology | Spinal Cord Injuries - physiopathology | Muscle Weakness - physiopathology | Hand Strength - physiology | Biotin - analogs & derivatives | Muscle Strength - physiology | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 8, pp. e23083 - e23083
Inflammation is known to be responsible for the sensitization of peripheral sensory neurons, leading to spontaneous pain and invalidating pain... 
TETRODOTOXIN-RESISTANT | RESISTANT SODIUM-CHANNEL | PERSISTENT NA+ CURRENT | RAT | NA(V)1.9 KNOCKOUT MICE | DRG NEURONS | MOUSE | BIOLOGY | PRIMARY SENSORY NEURONS | ROOT GANGLIA NEURONS | EXPRESSION | Immunohistochemistry | Sodium Channels - physiology | Gene Knockdown Techniques | Forelimb - physiopathology | Hindlimb - physiopathology | Time Factors | Sodium Channels - metabolism | Sensory Receptor Cells - metabolism | Forelimb - drug effects | Gene Expression | Arthritis, Experimental - physiopathology | Mice, Inbred C57BL | Rats | Forelimb - metabolism | NAV1.9 Voltage-Gated Sodium Channel | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | Hyperalgesia - physiopathology | Mice, Knockout | Animals | Pain - physiopathology | Hindlimb - drug effects | Edema - physiopathology | Mice | Sodium Channels - genetics | Carrageenan | Edema - chemically induced | Chronic Disease | Hindlimb - metabolism | Ganglia, Spinal - metabolism | Inflammation - physiopathology | Allergy | Care and treatment | Carrageenin | Pain | Analysis | Genetic engineering | Inflammation | Allergic reaction | Animal models | Antisense | Arthritis | mRNA | Kinases | Dorsal root ganglia | Analgesics | Rodents | Animal tissues | Sensory neurons | Sodium channels (voltage-gated) | Localization | Pain perception | Neurons | Hypersensitivity | Excitability | Ganglia | Inflammatory bowel disease | Ankle | Gene silencing | Heat | Sodium | Freund's adjuvant | Weight reduction | Immunoreactivity | Axonal transport | Index Medicus | Life Sciences | Biomolecules | Biochemistry, Molecular Biology
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 01/2006, Volume 23, Issue 1, pp. 36 - 54
Journal Article
Brain, ISSN 0006-8950, 4/2007, Volume 130, Issue 4, pp. 926 - 939
Journal Article
Spine, ISSN 0362-2436, 08/2013, Volume 38, Issue 17, pp. 1459 - 1465
Journal Article
Journal Article
Journal of Neuroscience Methods, ISSN 0165-0270, 03/2013, Volume 214, Issue 1, pp. 52 - 61
Journal Article