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Journal of Clinical Investigation, ISSN 0021-9738, 11/2016, Volume 126, Issue 11, pp. 4372 - 4386
Obese, insulin-resistant states are characterized by a paradoxical pathogenic condition in which the liver appears to be selectively insulin resistant.... 
RAT-LIVER | MEDICINE, RESEARCH & EXPERIMENTAL | METABOLIC SYNDROME | DE-NOVO LIPOGENESIS | TRANSCRIPTION FACTOR FOXO1 | FATTY LIVER-DISEASE | OB/OB MICE | ELEMENT-BINDING PROTEIN | HEPATIC STEATOSIS | ADIPOSE-TISSUE | PLASMA TRIGLYCERIDES | Glucose Intolerance - metabolism | Glucose-6-Phosphatase - genetics | Fatty Liver - pathology | Humans | Male | Glucose - biosynthesis | Glucose Intolerance - pathology | Fatty Liver - chemically induced | Glycolysis - drug effects | Glycolysis - genetics | Lipogenesis - genetics | Female | Glucose Intolerance - chemically induced | Insulin - genetics | Nuclear Proteins - genetics | Fatty Liver - genetics | Forkhead Box Protein O1 - metabolism | Glucose Intolerance - genetics | Fatty Liver - metabolism | Fructose - toxicity | Insulin Resistance | Glucose - genetics | Nuclear Proteins - metabolism | Signal Transduction - genetics | Transcription Factors - genetics | Glucose-6-Phosphatase - metabolism | Mice, Knockout | Transcription Factors - metabolism | Insulin - metabolism | Animals | Signal Transduction - drug effects | Lipogenesis - drug effects | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Forkhead Box Protein O1 - genetics | Obesity | Analysis | Insulin resistance | Genetic aspects | Genetic transcription | Research | Risk factors | Protein binding | Glucose | Metabolites | Rodents | Liver | Index Medicus | Abridged Index Medicus
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 05/2016, Volume 22, Issue 9, pp. 2290 - 2300
Journal Article
British Journal of Nutrition, ISSN 0007-1145, 04/2018, Volume 119, Issue 7, pp. 727 - 733
Journal Article
Immunity, ISSN 1074-7613, 04/2017, Volume 46, Issue 4, pp. 596 - 608
Understanding immunological memory formation depends on elucidating how multipotent memory precursor (MP) cells maintain developmental plasticity and longevity... 
PRC2 | Polycomb repressive complex 2 | CD8+ T cell differentiation | H3K27ac | plasticity | terminal differentiation | epigenetics | FOXO1 | H3K27me3 | EZH2 | T cell differentiation | CD8 | BET | IDENTITY | DNA METHYLATION | MEMORY | H3 LYSINE 27 | IMMUNOLOGY | TRANSCRIPTION FACTOR | EXPRESSION | VIRAL-INFECTION | DIVISION | Chromatin - metabolism | Polycomb Repressive Complex 2 - genetics | Multipotent Stem Cells - metabolism | Immunoblotting | Multipotent Stem Cells - immunology | Cell Differentiation - genetics | Flow Cytometry | Forkhead Box Protein O1 - immunology | Chromatin - immunology | Gene Expression - immunology | Polycomb Repressive Complex 2 - immunology | CD8-Positive T-Lymphocytes - metabolism | Lysine - metabolism | Immunologic Memory - genetics | Immunologic Memory - immunology | Forkhead Box Protein O1 - metabolism | Enhancer of Zeste Homolog 2 Protein - genetics | Enhancer of Zeste Homolog 2 Protein - metabolism | Histones - immunology | Mice, Inbred C57BL | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Cell Differentiation - immunology | Models, Immunological | Animals | Enhancer of Zeste Homolog 2 Protein - immunology | Histones - metabolism | CD8-Positive T-Lymphocytes - immunology | Methylation | Polycomb Repressive Complex 2 - metabolism | Chromatin - genetics | Forkhead Box Protein O1 - genetics | Lysine - immunology | Chromatin | Transcription factors | Profiling | CD8 antigen | Genes | Differentiation (biology) | Effector cells | Cytotoxicity | Infections | Lymphocytes T | Genomes | Immunity | Immunology | FOXO1 protein | Lymphocytes | DNA methylation | Plasticity (developmental) | Deposition | Antigens | Immunological memory | Memory cells | Cell survival | Cytokines | Maturation | Cell division | T cell receptors | Longevity | Cell differentiation | Gene expression | Survival | Polycomb group proteins | Gene silencing | Stem cells | Epigenetics | Viral infections | Index Medicus | CD8+ T cell differentiation
Journal Article
Journal Article
International journal of oncology, ISSN 1019-6439, 12/2018, Volume 53, Issue 6, pp. 2503 - 2517
Prostate cancer (PCa) is a malignant tumor with a high incidence in males. Localized tumors can be treated via surgery or radiation; however, it remains... 
Bromodomain containing protein 4 | Bromodomain inhibitor | c-Myc | Forkhead box protein O1 | Prostate cancer | p21 | APOPTOSIS | CELL-PROLIFERATION | bromodomain inhibitor | bromodomain containing protein 4 | forkhead box protein O1 | LYMPHOMAGENESIS | prostate cancer | THERAPY | ONCOLOGY | CLIP-SEQ | STARBASE | BET BROMODOMAIN PROTEINS | CARCINOMA | Neoplasm Transplantation | Prostatic Neoplasms - metabolism | Humans | Middle Aged | Male | Neoplasm Metastasis | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Prostatic Neoplasms - genetics | Aged, 80 and over | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Nuclear Proteins - genetics | Cell Survival - drug effects | Forkhead Box Protein O1 - metabolism | Prostatic Neoplasms - pathology | RNA, Small Interfering - pharmacology | Nuclear Proteins - metabolism | Transcription Factors - genetics | Proto-Oncogene Proteins c-myc - metabolism | Azepines - pharmacology | Transcription Factors - metabolism | Triazoles - pharmacology | Up-Regulation - drug effects | Cell Movement - drug effects | Animals | Cell Line, Tumor | Aged | Cell Proliferation - drug effects | Mice | Proto-Oncogene Proteins c-myc - genetics | Cell Cycle - drug effects | Neoplasm Staging | Forkhead Box Protein O1 - genetics | Antigens | Pathogenesis | RNA polymerase | Metastasis | Gene expression | Kinases | Cancer therapies | Studies | Proteins | Androgens | Cell cycle | Tumors | Apoptosis | Index Medicus
Journal Article
Aging Cell, ISSN 1474-9718, 10/2017, Volume 16, Issue 5, pp. 976 - 987
Aging is accompanied with unfavorable geometric and functional changes in the heart involving dysregulation of Akt and autophagy. This study examined the... 
cardiac geometry | contractile function | autophagy | aging | Akt | RAPAMYCIN | PROTEIN-KINASE | DIASTOLIC DYSFUNCTION | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | HYPERTROPHY | LONGEVITY | OVEREXPRESSION | INSULIN-RESISTANCE | CALORIC RESTRICTION | SENESCENCE | MITOCHONDRIAL INJURY | Adaptation, Physiological | Protein Kinases - metabolism | Sirtuin 1 - metabolism | Phosphorylation | Protein Kinases - genetics | Microtubule-Associated Proteins - genetics | Calcium - metabolism | Microtubule-Associated Proteins - metabolism | Autophagy-Related Protein 7 - metabolism | Proto-Oncogene Proteins c-akt - deficiency | Atrial Remodeling - drug effects | Myocardial Contraction - drug effects | Cardiomegaly - pathology | Male | Mitochondrial Proteins - genetics | Sirtuin 1 - genetics | Autophagy - drug effects | Heterocyclic Compounds, 4 or More Rings - pharmacology | Proto-Oncogene Proteins c-akt - genetics | Mitochondrial Proteins - metabolism | Myocardium - metabolism | Membrane Proteins - metabolism | Autophagy - genetics | Forkhead Box Protein O1 - metabolism | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Signal Transduction | Membrane Proteins - genetics | Gene Expression Regulation | Myocardium - pathology | Mitochondria - metabolism | Longevity - genetics | Mitochondria - pathology | Autophagy-Related Protein 7 - genetics | Sirolimus - pharmacology | Mice, Knockout | Animals | Cardiomegaly - prevention & control | Beclin-1 - genetics | Mice | Beclin-1 - metabolism | Cardiomegaly - genetics | Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics | Forkhead Box Protein O1 - genetics | Cardiomegaly - metabolism | Heart | Laws, regulations and rules | Ablation (Surgery) | AKT protein | Autophagy | Ca2+-transporting ATPase | Mitochondria | FOXO1 protein | Aging | Oxidation | Fura-2 | Acetylation | Heart diseases | Calcium (intracellular) | Echocardiography | Contractility | AKT2 protein | Cardiomyocytes | Rapamycin | Insulin | Muscle contraction | BNIP3 protein | Life span | PTEN-induced putative kinase | Parkin protein | Intracellular | PTEN protein | Phagocytosis | Hypertrophy | Index Medicus | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, pp. e0171004 - e0171004
Insulin resistance and compensatory hyperinsulinemia are characteristic features of obesity and polycystic ovary syndrome, and both are associated with reduced... 
POLYCYSTIC-OVARY-SYNDROME | MENSTRUAL-CYCLE | NONPREGNANT HUMAN ENDOMETRIUM | SIGNALING PATHWAYS | FACTOR AFX | EARLY-PREGNANCY | MESSENGER-RNA | MULTIDISCIPLINARY SCIENCES | FACTOR-BINDING PROTEIN-1 | KINASE-B | PROGESTERONE-RECEPTOR | Forkhead Box Protein O1 - metabolism | Insulin - pharmacology | Transcription, Genetic - drug effects | Humans | Stromal Cells - metabolism | Endometrium - drug effects | Medroxyprogesterone Acetate - pharmacology | Down-Regulation - drug effects | Down-Regulation - genetics | Decidua - metabolism | Young Adult | Adolescent | Stromal Cells - drug effects | Adult | Female | Bucladesine - pharmacology | Decidua - drug effects | Endometrium - cytology | Forkhead Box Protein O1 - genetics | Endometrium - metabolism | Complications and side effects | Transcription factors | Genetic aspects | Insulin | Health aspects | Fertility, Human | Hyperinsulinemia | Transformation | Genes | Insulin-like growth factor-binding protein 1 | Reproductive failure | Insulin-like growth factors | Kinases | Inactivation | FOXO1 protein | Prolactin | Fertility | Forkhead protein | Inhibition | Localization | Endometrium | Polycystic ovary syndrome | Deactivation | Implantation | Connective tissue growth factor | Gene expression | Nuclear transport | 1-Phosphatidylinositol 3-kinase | Stromal cells | Infertility | Insulin resistance | Immunofluorescence | Index Medicus
Journal Article
Oncology Reports, ISSN 1021-335X, 03/2018, Volume 39, Issue 3, pp. 1034 - 1042
Gonadotropins, including luteinizing hormone (LH) and follicle stimulating hormone (FSH), are conducive to the growth of ovarian cancer based on the... 
Forkhead box O1 | Epithelial ovarian cancer | PI3K/AKT signaling pathway | Goserelin | Gonadotropin-releasing hormone agonist | Apoptosis | TRANSCRIPTION FACTORS | SURVIVAL | ACTIVATION | goserelin | GONADOTROPIN-RELEASING-HORMONE | apoptosis | PROLIFERATION | FOXO1 | BREAST-CANCER | OVEREXPRESSION | ARREST | ONCOLOGY | PROSTATE-CANCER | gonadotropin-releasing hormone agonist | epithelial ovarian cancer | forkhead box O1 | Up-Regulation | Phosphorylation | Apoptosis - drug effects | Humans | Ovarian Neoplasms - pathology | Neoplasms, Glandular and Epithelial - metabolism | Proto-Oncogene Proteins c-akt - genetics | Biomarkers, Tumor - metabolism | Female | Ovarian Neoplasms - metabolism | Tumor Cells, Cultured | Proto-Oncogene Proteins c-akt - metabolism | Ovarian Neoplasms - drug therapy | Phosphatidylinositol 3-Kinase - metabolism | Forkhead Box Protein O1 - metabolism | Neoplasms, Glandular and Epithelial - pathology | Antineoplastic Agents, Hormonal - pharmacology | Xenograft Model Antitumor Assays | Animals | Goserelin - pharmacology | Phosphatidylinositol 3-Kinase - genetics | Signal Transduction - drug effects | Mice, Nude | Neoplasms, Glandular and Epithelial - drug therapy | Carcinoma, Ovarian Epithelial | Biomarkers, Tumor - genetics | Cell Proliferation - drug effects | Mice | Forkhead Box Protein O1 - genetics | Care and treatment | Oncology, Experimental | Development and progression | Gonadotropin | Research | Carcinogenesis | Ovarian cancer | Physiological aspects | Dosage and administration | Pharmacokinetics | Health aspects | Cancer | Proteins | Flow cytometry | Hypotheses | Medical prognosis | Genes | Cell cycle | Ligands | Tumor necrosis factor-TNF | Breast cancer | Polyclonal antibodies | Index Medicus | PI3K | AKT signaling pathway
Journal Article