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The Journal of biological chemistry, ISSN 1083-351X, 06/2013, Volume 288, Issue 25, pp. 18146 - 18161
Journal Article
American journal of physiology: endocrinology and metabolism, ISSN 0193-1849, 06/2017, Volume 312, Issue 6, pp. E495 - E507
...), which stimulates the adrenal grand to make cortisol. Skeletal muscle wasting occurs in pathophysiological response to Cushing's syndrome. The forkhead box (FOX... 
Skeletal muscle atrophy | Cushing’s syndrome | Forkhead box O3 | Glucocorticoid receptor | Endocrinology & Metabolism | Physiology | Life Sciences & Biomedicine | Science & Technology | Receptors, Glucocorticoid - antagonists & inhibitors | Cushing Syndrome - physiopathology | SKP Cullin F-Box Protein Ligases - genetics | Ubiquitin-Protein Ligases - antagonists & inhibitors | Male | Muscle, Skeletal - metabolism | Receptors, Glucocorticoid - metabolism | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Cushing Syndrome - metabolism | Promoter Regions, Genetic - drug effects | Receptors, Glucocorticoid - agonists | Forkhead Box Protein O3 - genetics | Chromatin Immunoprecipitation | Cushing Syndrome - pathology | RNA Interference | Muscle, Skeletal - drug effects | Muscle Proteins - metabolism | Forkhead Box Protein O3 - antagonists & inhibitors | Muscle Proteins - antagonists & inhibitors | Muscular Atrophy - etiology | Disease Models, Animal | Cell Line | Tripartite Motif Proteins - antagonists & inhibitors | Ubiquitin-Protein Ligases - metabolism | Tripartite Motif Proteins - agonists | Tripartite Motif Proteins - genetics | SKP Cullin F-Box Protein Ligases - metabolism | SKP Cullin F-Box Protein Ligases - antagonists & inhibitors | Rats, Sprague-Dawley | Forkhead Box Protein O3 - metabolism | Hormone Antagonists - pharmacology | Forkhead Box Protein O3 - agonists | Gene Expression Regulation - drug effects | Muscle Proteins - genetics | Response Elements - drug effects | Muscle Proteins - agonists | Animals | Active Transport, Cell Nucleus - drug effects | Muscle Fibers, Skeletal - pathology | Glucocorticoids - pharmacology | Genes, Reporter - drug effects | Tripartite Motif Proteins - metabolism | Muscle, Skeletal - pathology | Ubiquitin-Protein Ligases - genetics | Muscles | Physiological aspects | Atrophy, Muscular | Cushing syndrome | Index Medicus
Journal Article
Journal Article
Journal Article
Molecular cell, ISSN 1097-2765, 08/2017, Volume 67, Issue 4, pp. 685 - 701.e6
...), which regulates GLUT1-mediated glucose transport via stomatin. The loss of PON2 initiates the cellular starvation response and activates AMP-activated protein kinase (AMPK... 
GLUT1 | anoikis | glucose | pancreatic cancer | metabolism | PON2 | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Proto-Oncogene Proteins p21(ras) - genetics | Humans | AMP-Activated Protein Kinases - analysis | Glucose Transporter Type 1 - metabolism | Male | Carcinoma, Pancreatic Ductal - genetics | RNA Interference | Time Factors | Transcription, Genetic | Liver Neoplasms - enzymology | Liver Neoplasms - secondary | Lung Neoplasms - enzymology | Liver Neoplasms - genetics | Signal Transduction | Pancreatic Neoplasms - pathology | Tumor Burden | Aryldialkylphosphatase - genetics | Aryldialkylphosphatase - metabolism | Forkhead Box Protein O3 - metabolism | Apoptosis Regulatory Proteins - metabolism | Cell Movement - drug effects | Lung Neoplasms - prevention & control | Mice, Nude | Cell Line, Tumor | Glucose - metabolism | Mutation | AMP-Activated Protein Kinases - genetics | Energy Metabolism - drug effects | AMP-Activated Protein Kinases - metabolism | Gene Expression Regulation, Neoplastic | Tumor Suppressor Protein p53 - genetics | Forkhead Box Protein O3 - genetics | Pancreatic Neoplasms - drug therapy | Transfection | Lung Neoplasms - secondary | Apoptosis Regulatory Proteins - genetics | Female | Antineoplastic Agents - pharmacology | Liver Neoplasms - prevention & control | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Pancreatic Neoplasms - enzymology | Tumor Suppressor Protein p53 - metabolism | Pancreatic Neoplasms - genetics | Proto-Oncogene Proteins - genetics | Carcinoma, Pancreatic Ductal - pathology | Carcinoma, Pancreatic Ductal - enzymology | Xenograft Model Antitumor Assays | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Glucose Transporter Type 1 - genetics | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Deregulation | Growth | Pancreatic cancer | Physiological aspects | Genetic transcription | Metastasis | Glucose | Tumor proteins | Dextrose | Glutamine | Glucose metabolism | Medical colleges | Protein kinases | Index Medicus
Journal Article
Diabetes (New York, N.Y.), ISSN 1939-327X, 03/2019, Volume 68, Issue 3, pp. 556 - 570
Insulin deficiency and uncontrolled diabetes lead to a catabolic state with decreased muscle strength, contributing to disease-related morbidity. FoxO... 
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Amino Acids - blood | Phosphorylation | Humans | Diabetes Mellitus, Experimental - genetics | Male | Muscle, Skeletal - metabolism | Autophagy - physiology | Insulin - blood | Diabetes Mellitus, Experimental - blood | Forkhead Box Protein O3 - genetics | Lysosomes - metabolism | Forkhead Transcription Factors - metabolism | Proteolysis | Muscle Proteins - metabolism | Female | Diabetes Mellitus, Experimental - metabolism | Diabetes Mellitus, Experimental - physiopathology | Forkhead Box Protein O1 - metabolism | Muscular Atrophy - metabolism | Muscular Atrophy - blood | Muscular Atrophy - genetics | Forkhead Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Forkhead Box Protein O3 - metabolism | Mice, Knockout | Muscle Proteins - genetics | Animals | Mice | Muscle, Skeletal - pathology | DNA, Complementary - metabolism | Forkhead Box Protein O1 - genetics | Prevention | Complications and side effects | Control | Transcription factors | Analysis | Genetic aspects | Diabetes | Atrophy, Muscular | Risk factors | Ubiquitin | Streptozocin | Diabetes mellitus | Diabetes mellitus (insulin dependent) | Autophagy | Insulin | Morbidity | Atrophy | FOXO1 protein | Proteasomes | Forkhead protein | Muscular strength | Phagocytosis | Signal Transduction | 0705
Journal Article
Biochimica et biophysica acta. Molecular cell research, ISSN 0167-4889, 11/2011, Volume 1813, Issue 11, pp. 1938 - 1945
Journal Article