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Journal of Biological Chemistry, ISSN 0021-9258, 11/2010, Volume 285, Issue 45, pp. 35123 - 35132
The evolutionary loss of hepatic urate oxidase (uricase) has resulted in humans with elevated serum uric acid (urate). Uricase loss may have been beneficial to... 
HYPERURICEMIA | ORGANIC ANION TRANSPORT | GOUT | BIOCHEMISTRY & MOLECULAR BIOLOGY | IDENTIFICATION | EXCRETION | APICAL MEMBRANE | URIC-ACID | HOMINOID EVOLUTION | FAMILY | EXCHANGER | Glucose Transport Proteins, Facilitative - metabolism | Organic Anion Transporters, Sodium-Independent - genetics | Organic Anion Transport Protein 1 - metabolism | Diuretics - pharmacokinetics | Sodium-Phosphate Cotransporter Proteins, Type I - genetics | Humans | Hyperuricemia - metabolism | Mutation, Missense | Organic Anion Transporters - metabolism | Urate Oxidase - metabolism | Organic Anion Transporters - genetics | Urate Oxidase - genetics | Furosemide - adverse effects | Organic Anion Transporters, Sodium-Independent - metabolism | Kidney Tubules, Proximal - secretion | Ion Transport - drug effects | Glucose Transport Proteins, Facilitative - genetics | Bumetanide - adverse effects | Genetic Predisposition to Disease | Uric Acid - metabolism | Organic Cation Transport Proteins - metabolism | Diuretics - pharmacology | Furosemide - pharmacokinetics | Liver - metabolism | Xenopus laevis | Organic Anion Transport Protein 1 - genetics | Bumetanide - pharmacokinetics | Bumetanide - pharmacology | Hyperuricemia - genetics | Gout - metabolism | Animals | Models, Biological | Gout - genetics | Furosemide - pharmacology | Organic Cation Transport Proteins - genetics | Sodium-Phosphate Cotransporter Proteins, Type I - metabolism | Diuretics - adverse effects | Hyperuricemia - chemically induced | Ion Transport - genetics | Index Medicus | Transport Drugs | Membrane Biology | Gout | Epithelial Cell | Uric Acid | Diuretics | Kidney
Journal Article
Clinical Pharmacology & Therapeutics, ISSN 0009-9236, 09/2016, Volume 100, Issue 3, pp. 259 - 267
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 06/2013, Volume 110, Issue 25, pp. E2308 - E2316
Alveolar fluid clearance driven by active epithelial Na+ and secondary Cl- absorption counteracts edema formation in the intact lung. Recently, we showed that... 
Epithelial Cl | Pulmonary edema | transport | pulmonary edema | ION-TRANSPORT | MULTIDISCIPLINARY SCIENCES | VOLUME | EPITHELIAL ION | NA+ ABSORPTION | RESPIRATORY-DISTRESS-SYNDROME | epithelial Cl- transport | ACUTE PULMONARY-EDEMA | FUROSEMIDE | II CELLS | CHANNELS | CFTR | Male | Sodium-Potassium-Chloride Symporters - metabolism | Pulmonary Edema - etiology | Chlorides - metabolism | Pulmonary Alveoli - metabolism | Pulmonary Edema - metabolism | Cystic Fibrosis Transmembrane Conductance Regulator - antagonists & inhibitors | Heart Failure - complications | Mice, Inbred CFTR | Rabbits | Diuretics - pharmacology | Mice, Inbred C57BL | Rats | Sodium-Potassium-Chloride Symporters - genetics | Body Fluids - secretion | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Heart Failure - metabolism | Hydrostatic Pressure | Pulmonary Edema - drug therapy | Rats, Sprague-Dawley | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Solute Carrier Family 12, Member 2 | Body Fluids - metabolism | Amiloride - pharmacology | Cystic Fibrosis Transmembrane Conductance Regulator - genetics | Mice | Respiratory Mucosa - metabolism | Furosemide - pharmacology | Cystic fibrosis | Lung diseases | Rodents | Adenosine triphosphatase | Index Medicus | Biological Sciences | epithelial Cl− transport | PNAS Plus
Journal Article
Pflügers Archiv - European Journal of Physiology, ISSN 0031-6768, 12/2015, Volume 467, Issue 12, pp. 2413 - 2421
Journal Article
Journal Article
International Journal of Pharmaceutics, ISSN 0378-5173, 2009, Volume 381, Issue 2, pp. 199 - 204
Intestinal efflux transporters can significantly reduce the absorption of the drug after peroral application. In this work we studied secretion of glutathione... 
SGLT | MRP2 | Glutathione conjugates | Glucose | Hypoosmolarity | CELLS | GLUCURONIDE | TRANSPORTERS | MECHANISMS | EXCRETION | MERCAPTURIC ACID | EFFLUX | PHARMACOLOGY & PHARMACY | ABSORPTION | MULTIDRUG-RESISTANCE PROTEIN-2 | EXPRESSION | Succinimides - metabolism | Cell Polarity | Rats, Wistar | Enterocytes - metabolism | Glutathione - metabolism | Male | ATP-Binding Cassette Transporters - agonists | Glucose - administration & dosage | Furosemide - metabolism | Sulfanilamides - metabolism | Methylglucosides - administration & dosage | Osmolar Concentration | Sodium-Glucose Transport Proteins - metabolism | Membrane Transport Proteins - metabolism | Glutathione - secretion | Glutathione - analogs & derivatives | Benzbromarone - metabolism | Glutathione Transferase - metabolism | Monosaccharides - administration & dosage | Rats | Multidrug Resistance-Associated Proteins - antagonists & inhibitors | Permeability | Intestine, Small - secretion | Membrane Transport Modulators - metabolism | Secretory Pathway - drug effects | Secretory Pathway - physiology | Animals | Glucose - metabolism | Dinitrochlorobenzene - metabolism | Multidrug Resistance-Associated Proteins - agonists | Glutathione - chemistry | Intestine, Small - metabolism | Methylglucosides - metabolism | In Vitro Techniques | Monosaccharides - metabolism | ATP-Binding Cassette Transporters - antagonists & inhibitors | Thiols | Galactose | Furosemide | Fructose | Dextrose | Glutathione | Index Medicus
Journal Article
Journal Article
PLoS Pathogens, ISSN 1553-7366, 12/2017, Volume 13, Issue 12, pp. e1006744 - e1006744
Nuclear factor of activated T cells 5 (NFAT5)/Tonicity enhancer binding protein (TonEBP) is a transcription factor induced by hypertonic stress in the kidney.... 
HIGH-DOSE FUROSEMIDE | POLY(A)-BINDING PROTEIN | STIMULATES TRANSCRIPTION | MICROBIOLOGY | FACTOR-KAPPA-B | SALINE SOLUTION | BINDING PROTEIN | IN-VITRO | VIROLOGY | HYPERTONIC STRESS-RESPONSE | NITRIC-OXIDE | CONGESTIVE-HEART-FAILURE | PARASITOLOGY | Transcription Factors - chemistry | Mice, Inbred A | Myocytes, Cardiac - immunology | Humans | Nitric Oxide Synthase Type II - chemistry | Substrate Specificity | Male | Enterovirus B, Human - physiology | Recombinant Fusion Proteins - metabolism | Viral Proteins - metabolism | Coxsackievirus Infections - virology | Cysteine Endopeptidases - metabolism | RNA Interference | Nitric Oxide Synthase Type II - antagonists & inhibitors | Proteolysis | Myocarditis - immunology | Peptide Fragments - genetics | Cell Line | Peptide Fragments - metabolism | Coxsackievirus Infections - metabolism | Gene Expression Regulation | Transcription Factors - antagonists & inhibitors | Recombinant Fusion Proteins - chemistry | Transcription Factors - genetics | Enterovirus B, Human - enzymology | Coxsackievirus Infections - pathology | Transcription Factors - metabolism | Myocarditis - virology | Myocytes, Cardiac - pathology | Myocytes, Cardiac - virology | Peptide Fragments - chemistry | Animals | Nitric Oxide Synthase Type II - genetics | Virus Replication | Myocarditis - metabolism | Enterovirus B, Human - immunology | Myocytes, Cardiac - metabolism | Mutation | Myocarditis - pathology | Coxsackievirus Infections - immunology | Amino Acid Substitution | Nitric Oxide Synthase Type II - metabolism | Physiological aspects | Transcription factors | Coxsackieviruses | Research | Proteases | Heart | Animal models | Laboratories | Syngeneic grafts | Funding | Proteinase | Cardiovascular disease | Infections | NF-AT protein | Drug development | Kinases | Glycine | Inactivation | Proteins | Lymphocytes | Protease | Rodents | Mannitol | Cleavage | Inhibition | Heart diseases | Deactivation | Kidneys | Organs | Multiplication & division | Gene expression | Coronary artery disease | Nitric-oxide synthase | Virology | Medicine | Myocarditis | Pathology | Hospitals | Mutagenesis | Nitric oxide | Site-directed mutagenesis | Predictions | Replication | Cardiovascular diseases | Viral infections | Index Medicus
Journal Article
Development, ISSN 0950-1991, 08/2009, Volume 136, Issue 16, pp. 2837 - 2848
Journal Article
Drug metabolism and disposition: the biological fate of chemicals, ISSN 0090-9556, 11/2018, Volume 46, Issue 11, pp. 1497 - 1506
Journal Article