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The Journal of clinical investigation, ISSN 0021-9738, 09/2008, Volume 118, Issue 9, pp. 3038 - 3050
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Protein Kinases - metabolism | Cell Proliferation | Antigens, CD34 - biosynthesis | Humans | Phosphatidylinositol 3-Kinases - metabolism | Gene Expression Regulation, Leukemic | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Animals | Antigens, CD19 - biosynthesis | Cell Transformation, Neoplastic | Models, Biological | Burkitt Lymphoma - genetics | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Mice | TOR Serine-Threonine Kinases | Fusion Proteins, bcr-abl - metabolism | Cell proliferation | Control | Translocation (Genetics) | Physiological aspects | Genetic aspects | Research | Chronic myeloid leukemia | Risk factors | Prevention | Leukemia | Index Medicus | Abridged Index Medicus
Journal Article
Clinical cancer research, ISSN 1078-0432, 02/2012, Volume 18, Issue 4, pp. 930 - 937
Life Sciences & Biomedicine | Oncology | Science & Technology | Biological and medical sciences | Medical sciences | Antineoplastic agents | Chromosome aberrations | Pharmacology. Drug treatments | Medical genetics | Humans | Translational Medical Research | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Antineoplastic Agents - therapeutic use | Animals | Signal Transduction - drug effects | Protein Kinase Inhibitors - therapeutic use | Fusion Proteins, bcr-abl - antagonists & inhibitors | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Antineoplastic Agents - pharmacology | Mice | Protein Kinase Inhibitors - pharmacology | Fusion Proteins, bcr-abl - metabolism | Index Medicus
Journal Article
Angewandte Chemie (International ed.), ISSN 1433-7851, 01/2016, Volume 55, Issue 2, pp. 807 - 810
inhibitors | cancer | drug design | E3 ubiquitin ligases | protein degradation | Cell Line, Tumor | Cell Line | Proteolysis | Humans | Fusion Proteins, bcr-abl - metabolism | Ubiquitin | Developmental biology | Ligases | Binding | BCR protein | Modular design | Abl protein | Drug development | Recruitment | Proteins | Degradation | Ubiquitination | Inhibitors | Technology | Strategy | Fusion protein | Ubiquitin-protein ligase | Index Medicus | Drug Design
Journal Article
Nature reviews. Molecular cell biology, ISSN 1471-0072, 01/2004, Volume 5, Issue 1, pp. 33 - 44
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Amino Acid Sequence | Phosphorylation | Proto-Oncogene Proteins c-abl - genetics | src-Family Kinases | Gene Expression Regulation - genetics | Protein-Tyrosine Kinases - metabolism | Humans | Proto-Oncogene Proteins c-abl - chemistry | Molecular Sequence Data | src Homology Domains | Sequence Homology, Amino Acid | Point Mutation | Fusion Proteins, bcr-abl - genetics | Protein-Tyrosine Kinases - genetics | Sequence Alignment | Animals | Protein-Tyrosine Kinases - chemistry | Proto-Oncogene Proteins c-abl - metabolism | Protein Conformation | Catalysis | Binding Sites | Fusion Proteins, bcr-abl - metabolism | Leukemia - genetics | Index Medicus
Journal Article
Leukemia, ISSN 0887-6924, 2019, Volume 33, Issue 7, pp. 1583 - 1597
Oncology | Life Sciences & Biomedicine | Hematology | Science & Technology | Leukemia, Large Granular Lymphocytic - drug therapy | Cell Proliferation | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | STAT5 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Tumor Suppressor Proteins - genetics | Antineoplastic Agents - pharmacology | Leukemia, Large Granular Lymphocytic - pathology | Tumor Suppressor Proteins - metabolism | Interferons - pharmacology | Survival Rate | Mice, SCID | Cell Transformation, Neoplastic - metabolism | Mice, Knockout | Xenograft Model Antitumor Assays | Fusion Proteins, bcr-abl - genetics | Animals | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Leukemia, Large Granular Lymphocytic - metabolism | Mice, Inbred NOD | Mice | Cell Transformation, Neoplastic - drug effects | Mutation | Cell Transformation, Neoplastic - pathology | Fusion Proteins, bcr-abl - metabolism | RNA sequencing | Usage | Gene mutations | Leukemia | Development and progression | Genetic aspects | Cellular signal transduction | Research | BCR protein | Biotechnology | Transformation | Deregulation | Transcription factors | Profiling | Transcription | Abl gene | BCR gene | Kinases | Gene sequencing | α-Interferon | Signal transduction | Janus kinase | Transducers | Cell survival | Colonies | Inhibition (psychology) | Stat5 protein | Ribonucleic acid--RNA | Disease control | Survival | Hemopoiesis | Lymphocytes B | γ-Interferon | Leukemogenesis | Interferon | Tumors | Index Medicus | Cell signalling | Acute lymphocytic leukaemia
Journal Article
Blood, ISSN 0006-4971, 12/2007, Volume 110, Issue 12, pp. 4055 - 4063
Life Sciences & Biomedicine | Hematology | Science & Technology | Hematologic and hematopoietic diseases | Chemotherapy | Pharmacology. Drug treatments | Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis | Biological and medical sciences | Medical sciences | Antineoplastic agents | Protein-Tyrosine Kinases - metabolism | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Drug Resistance, Neoplasm | Neoplasm Proteins - antagonists & inhibitors | Piperazines - chemistry | Thiazoles - therapeutic use | Neoplasm Proteins - metabolism | Pyrimidines - chemistry | Dose-Response Relationship, Drug | Protein Kinase Inhibitors - chemistry | Fusion Proteins, bcr-abl | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Protein-Serine-Threonine Kinases - metabolism | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - enzymology | Dasatinib | Quinone Reductases - metabolism | Piperazines - therapeutic use | Discoidin Domain Receptor 1 | Pyrimidines - pharmacology | Receptor Protein-Tyrosine Kinases - metabolism | Imatinib Mesylate | Piperazines - pharmacology | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Quinone Reductases - antagonists & inhibitors | K562 Cells | Proteomics | Thiazoles - chemistry | Protein Kinase Inhibitors - pharmacology | Thiazoles - pharmacology | Benzamides | Drug Screening Assays, Antitumor | Protein-Tyrosine Kinases - antagonists & inhibitors | Index Medicus | Abridged Index Medicus
Journal Article
Leukemia, ISSN 0887-6924, 07/2008, Volume 22, Issue 7, pp. 1402 - 1409
Oncology | Life Sciences & Biomedicine | Hematology | Science & Technology | Hematologic and hematopoietic diseases | Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis | Biological and medical sciences | Medical sciences | Chromosome aberrations | Medical genetics | Blast Crisis - drug therapy | Caspase 9 - metabolism | Immunoprecipitation | Leukemia - pathology | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Leukemia - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Novobiocin - pharmacology | Cytochromes c - metabolism | Leukemia - drug therapy | Pyrimidines - pharmacology | Lactams, Macrocyclic - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Proteasome Endopeptidase Complex - physiology | Benzoquinones - pharmacology | Fusion Proteins, bcr-abl - analysis | HSP90 Heat-Shock Proteins - antagonists & inhibitors | K562 Cells | HL-60 Cells | HSP90 Heat-Shock Proteins - metabolism | Cell Proliferation - drug effects | Benzamides | Fusion Proteins, bcr-abl - metabolism | Care and treatment | Heat shock proteins | Genetic aspects | Acute lymphocytic leukemia | Diagnosis | Health aspects | Philadelphia chromosome | Index Medicus
Journal Article
Oncotarget, ISSN 1949-2553, 2017, Volume 8, Issue 27, pp. 43662 - 43677
BCR-ABL+ human leukemia | Icaritin (SNG162 and SNG1153) | ERa36 | Leukemic stem cells | Tyrosine kinase inhibitors | Oncology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Cell Membrane | Phosphorylation | Neoplastic Stem Cells - drug effects | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Chromans - pharmacology | Neoplastic Stem Cells - metabolism | Estrogen Receptor alpha - metabolism | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - mortality | Gene Expression | Protein Transport | Fusion Proteins, bcr-abl - genetics | Estrogen Receptor alpha - genetics | Models, Biological | Fusion Proteins, bcr-abl - antagonists & inhibitors | Cell Line, Tumor | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Protein Binding | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Mutation | GRB2 Adaptor Protein - metabolism | Fusion Proteins, bcr-abl - metabolism | Amino Acid Substitution | Index Medicus
Journal Article
Nature (London), ISSN 0028-0836, 01/2010, Volume 463, Issue 7280, pp. 501 - 506
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Molecular and cellular biology | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Male | Transplantation, Heterologous | Piperazines - chemistry | Pyrimidines - chemistry | Pyrimidines - metabolism | Antineoplastic Agents - metabolism | Mass Spectrometry | Bone Marrow Transplantation | Inhibitory Concentration 50 | Female | Antineoplastic Agents - pharmacology | Binding Sites | Disease Models, Animal | Fusion Proteins, bcr-abl - chemistry | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - enzymology | Protein Structure, Tertiary | Crystallization | Antineoplastic Combined Chemotherapy Protocols | Models, Molecular | Pyrimidines - pharmacology | Antineoplastic Agents - chemistry | Mutation - genetics | Imatinib Mesylate | Piperazines - pharmacology | Fusion Proteins, bcr-abl - genetics | Animals | Cell Line, Tumor | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Mice | Benzamides | Fusion Proteins, bcr-abl - metabolism | Drug Resistance, Neoplasm - drug effects | Nilotinib | Dasatinib | Drug resistance in microorganisms | Usage | Nuclear magnetic resonance spectroscopy | Research | Chronic myeloid leukemia | X-ray crystallography | Chromosome abnormalities | Gene mutations | Analysis | Genetic aspects | Diagnosis | Binding proteins | Drug therapy | Health aspects | Proteins | Studies | Competition | Enzymes | E coli | Kinases | Drug resistance | Crystal structure | Index Medicus
Journal Article