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Journal Article
Experimental and Molecular Medicine, ISSN 1226-3613, 10/2008, Volume 40, Issue 5, pp. 495 - 504
Journal Article
Journal Article
Nature, ISSN 0028-0836, 05/2016, Volume 534, Issue 7605, pp. 55 - 62
Somatic mutations have been extensively characterized in breast cancer, but the effects of these genetic alterations on the proteomic landscape remain poorly... 
PATHWAYS | HETEROGENEITY | PIK3CA MUTATIONS | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | GENES | BIOLOGY | RECEPTOR | EXPRESSION | SIGNATURE | REVEALS | Protein Kinases - metabolism | Focal Adhesion Kinase 1 - genetics | Receptor, Epidermal Growth Factor - genetics | Protein Kinases - genetics | Cyclin-Dependent Kinases - metabolism | Receptor, ErbB-2 - genetics | Receptors, G-Protein-Coupled - metabolism | Genomics | Humans | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Phosphoproteins - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - genetics | Tumor Suppressor Protein p53 - genetics | Breast Neoplasms - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - metabolism | Breast Neoplasms - enzymology | Receptor, Epidermal Growth Factor - metabolism | Phosphoproteins - analysis | Mass Spectrometry | src-Family Kinases - metabolism | Female | Cyclin-Dependent Kinases - genetics | Focal Adhesion Kinase 1 - metabolism | Chromosomes, Human, Pair 5 - genetics | Breast Neoplasms - classification | Chromosome Deletion | p21-Activated Kinases - genetics | Signal Transduction | Molecular Sequence Annotation | Calcium-Binding Proteins - deficiency | Phosphoproteins - genetics | Mutation - genetics | S-Phase Kinase-Associated Proteins - metabolism | p21-Activated Kinases - metabolism | Phosphatidylinositol 3-Kinases - genetics | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | Proteomics | S-Phase Kinase-Associated Proteins - genetics | Receptors, G-Protein-Coupled - genetics | src-Family Kinases - genetics | Calcium-Binding Proteins - genetics | Breast cancer | Genetic aspects | Research | Oncology, Experimental | Cancer | Physiological aspects | Methods | Mutation (Biology) | Proteins | Gene amplification | Peptides | Protein expression | Genomes | Mutation | Kinases | Deoxyribonucleic acid--DNA | Tumors
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2013, Volume 288, Issue 32, pp. 22942 - 22960
TGR5 is a G protein-coupled receptor that mediates bile acid (BA) effects on energy balance, inflammation, digestion, and sensation. The mechanisms and... 
GROWTH-FACTOR RECEPTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | GPBAR1 TGR5 | DEPENDENT ENDOCYTOSIS | PROTEIN-COUPLED RECEPTORS | RESONANCE ENERGY-TRANSFER | KINASE ACTIVATION | MU-OPIOID RECEPTOR | BETA-ADRENERGIC RECEPTOR | ACTIVATED RECEPTOR-2 | EGF RECEPTOR | Cholagogues and Choleretics - pharmacology | Phenylalanine - analogs & derivatives | Receptors, G-Protein-Coupled - metabolism | Membrane Microdomains - metabolism | Phenylalanine - pharmacology | Humans | ErbB Receptors - genetics | Oleanolic Acid - pharmacology | Arrestins - genetics | Protein Transport - physiology | Protein Transport - drug effects | G-Protein-Coupled Receptor Kinase 2 - genetics | G-Protein-Coupled Receptor Kinase 2 - metabolism | Arrestins - metabolism | beta-Cyclodextrins - pharmacology | Endosomes - metabolism | Mitogen-Activated Protein Kinase 1 - genetics | HEK293 Cells | Antineoplastic Agents - pharmacology | Cyclic AMP - genetics | Cyclic AMP - metabolism | ErbB Receptors - antagonists & inhibitors | ErbB Receptors - metabolism | Endosomes - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Endocytosis - drug effects | Enzyme Inhibitors - pharmacology | Thiophenes - pharmacology | Tyrphostins - pharmacology | Endocytosis - physiology | Arrestins - antagonists & inhibitors | G-Protein-Coupled Receptor Kinase 5 - metabolism | Deoxycholic Acid - pharmacology | Mitogen-Activated Protein Kinase 3 - metabolism | beta-Arrestin 2 | beta-Arrestin 1 | beta-Arrestins | Receptors, G-Protein-Coupled - genetics | Quinazolines - pharmacology | G-Protein-Coupled Receptor Kinase 5 - genetics | Membrane Microdomains - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | Lipid Raft | Bile Acid | Endocytosis | G Protein-coupled Receptors (GPCR) | Arrestin | Signal Transduction
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 01/2017, Volume 118, Issue 1, pp. 66 - 73
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2011, Volume 286, Issue 22, pp. 19880 - 19891
The angiotensin II peptide analog [Sar(1), Ile(4), Ile(8)] AngII (SII) is a biased AT(1A) receptor agonist that stimulates receptor phosphorylation,... 
SYNTHASE | ACTIVATION | SET | STIMULATION | PHOSPHORYLATION | BETA-ARRESTIN-2 | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | COMPLEXES | SMOOTH-MUSCLE-CELLS | CYCLOOXYGENASE-1 | Phosphorylation - physiology | Dinoprostone - biosynthesis | Histone Chaperones - genetics | Humans | Histone Chaperones - metabolism | Arrestins - genetics | Glycogen Synthase Kinase 3 beta | Arrestins - metabolism | GTP-Binding Proteins - genetics | Receptor, Angiotensin, Type 1 - genetics | Mitogen-Activated Protein Kinase 1 - genetics | HEK293 Cells | Phosphorylation - drug effects | Vasoconstrictor Agents - pharmacology | Angiotensin II - pharmacology | Cyclooxygenase 1 - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Dinoprostone - genetics | Protein Phosphatase 2 - genetics | Angiotensin II - analogs & derivatives | Transcription Factors - genetics | Glycogen Synthase Kinase 3 - metabolism | Transcription Factors - metabolism | Glycogen Synthase Kinase 3 - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | beta-Arrestin 2 | beta-Arrestin 1 | beta-Arrestins | Receptor, Angiotensin, Type 1 - metabolism | Protein Phosphatase 2 - metabolism | Signal Transduction - physiology | Cyclooxygenase 1 - metabolism | GTP-Binding Proteins - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Prostaglandins | Signal Transduction | AT1 Receptor | PGES3 | G-protein Coupled Receptors (GPCR) | PP2A | Arrestin | Akt PKB | Biased Agonism | I2PP2A
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2015, Volume 290, Issue 25, pp. 15799 - 15811
Background: MicroRNA-21 (miR-21) is an oncomiR in human hepatocellular carcinoma and is highly expressed in liver, but its regulation is uncharacterized.... 
METHYL-BETA-CYCLODEXTRIN | MIGRATION | gene transcription | VARIANT ER-ALPHA-36 | microRNA biogenesis | microRNA (miRNA) | estrogen receptor | hormones | BIOCHEMISTRY & MOLECULAR BIOLOGY | GPR30 | PROLIFERATION | G-protein-coupled receptor (GPCR) | BREAST-CANCER CELLS | EPIDERMAL-GROWTH-FACTOR | PROSTATE-CANCER | GENE-EXPRESSION | UP-REGULATION | Transcription, Genetic - drug effects | Receptors, Estrogen - metabolism | Adjuvants, Immunologic - pharmacology | Receptors, G-Protein-Coupled - metabolism | Transcription Factor AP-1 - genetics | Humans | Receptors, Androgen - metabolism | Male | Phosphatidylinositol 3-Kinases - metabolism | Transcription Factor AP-1 - metabolism | MAP Kinase Signaling System - genetics | Carcinoma, Hepatocellular - genetics | Mitogen-Activated Protein Kinase 1 - genetics | src-Family Kinases - metabolism | Estrogen Receptor alpha - metabolism | Female | Liver Neoplasms - pathology | Gene Expression Regulation, Neoplastic - drug effects | Gene Expression Regulation, Neoplastic - genetics | Receptors, Estrogen - genetics | Liver Neoplasms - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Response Elements | Dehydroepiandrosterone - pharmacology | Proto-Oncogene Proteins c-jun - genetics | Proto-Oncogene Proteins c-fos - metabolism | MicroRNAs - biosynthesis | Hep G2 Cells | Phosphatidylinositol 3-Kinases - genetics | Estrogen Receptor alpha - genetics | MAP Kinase Signaling System - drug effects | RNA, Neoplasm - biosynthesis | Receptors, Androgen - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Proto-Oncogene Proteins c-jun - metabolism | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | RNA, Neoplasm - genetics | Proto-Oncogene Proteins c-fos - genetics | MicroRNAs - genetics | Receptors, G-Protein-Coupled - genetics | Transcription, Genetic - genetics | src-Family Kinases - genetics | Carcinoma, Hepatocellular - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus | Signal Transduction
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2011, Volume 286, Issue 39, pp. 33832 - 33840
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2017, Volume 292, Issue 24, pp. 9932 - 9943
G protein-coupled receptor 30 (GPR30), also called G protein-coupled estrogen receptor 1 (GPER1), is thought to play important roles in breast cancer and... 
ACTIVATION | COMPLEX | 30 GPR30 | CANCER CELLS | KINASE-A | BIOCHEMISTRY & MOLECULAR BIOLOGY | CALCINEURIN | SCAFFOLDS | ENDOCYTOSIS | PLASMA-MEMBRANE | ANCHORING PROTEIN | GTP-Binding Protein alpha Subunits, Gi-Go - agonists | GTP-Binding Protein alpha Subunits, Gi-Go - genetics | Receptors, Estrogen - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Receptors, G-Protein-Coupled - agonists | Recombinant Fusion Proteins - metabolism | GTP-Binding Protein alpha Subunits, Gi-Go - chemistry | Quinolines - pharmacology | Mitogen-Activated Protein Kinase 1 - chemistry | A Kinase Anchor Proteins - genetics | Protein Processing, Post-Translational - drug effects | RNA Interference | Mitogen-Activated Protein Kinase 1 - genetics | HEK293 Cells | Phosphorylation - drug effects | Radioligand Assay | Mitogen-Activated Protein Kinase 3 - chemistry | Phosphatidylinositol 3-Kinase - metabolism | A Kinase Anchor Proteins - metabolism | A Kinase Anchor Proteins - antagonists & inhibitors | Receptors, Estrogen - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Enzyme Inhibitors - pharmacology | Models, Molecular | Cyclopentanes - pharmacology | Recombinant Fusion Proteins - chemistry | Enzyme Activation - drug effects | Phosphatidylinositol 3-Kinase - chemistry | Up-Regulation - drug effects | MAP Kinase Signaling System - drug effects | Receptors, Estrogen - chemistry | Mitogen-Activated Protein Kinase 3 - metabolism | Phosphatidylinositol 3-Kinase - genetics | PDZ Domains | Receptors, G-Protein-Coupled - genetics | Mutation | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism | Receptors, G-Protein-Coupled - chemistry | Amino Acid Substitution | Mitogen-Activated Protein Kinase 1 - metabolism | Signal Transduction | GPER1 | A-kinase-anchoring protein (AKAP) | G protein-coupled receptor (GPCR) | GPR30 | PDZ domain | calcineurin | extracellular signal-regulated kinase (ERK) | G protein
Journal Article