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Journal of Biological Chemistry, ISSN 0021-9258, 08/2013, Volume 288, Issue 32, pp. 22942 - 22960
TGR5 is a G protein-coupled receptor that mediates bile acid (BA) effects on energy balance, inflammation, digestion, and sensation. The mechanisms and... 
GROWTH-FACTOR RECEPTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | GPBAR1 TGR5 | DEPENDENT ENDOCYTOSIS | PROTEIN-COUPLED RECEPTORS | RESONANCE ENERGY-TRANSFER | KINASE ACTIVATION | MU-OPIOID RECEPTOR | BETA-ADRENERGIC RECEPTOR | ACTIVATED RECEPTOR-2 | EGF RECEPTOR | Cholagogues and Choleretics - pharmacology | Phenylalanine - analogs & derivatives | Receptors, G-Protein-Coupled - metabolism | Membrane Microdomains - metabolism | Phenylalanine - pharmacology | Humans | ErbB Receptors - genetics | Oleanolic Acid - pharmacology | Arrestins - genetics | Protein Transport - physiology | Protein Transport - drug effects | G-Protein-Coupled Receptor Kinase 2 - genetics | G-Protein-Coupled Receptor Kinase 2 - metabolism | Arrestins - metabolism | beta-Cyclodextrins - pharmacology | Endosomes - metabolism | Mitogen-Activated Protein Kinase 1 - genetics | HEK293 Cells | Antineoplastic Agents - pharmacology | Cyclic AMP - genetics | Cyclic AMP - metabolism | ErbB Receptors - antagonists & inhibitors | ErbB Receptors - metabolism | Endosomes - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Endocytosis - drug effects | Enzyme Inhibitors - pharmacology | Thiophenes - pharmacology | Tyrphostins - pharmacology | Endocytosis - physiology | Arrestins - antagonists & inhibitors | G-Protein-Coupled Receptor Kinase 5 - metabolism | Deoxycholic Acid - pharmacology | Mitogen-Activated Protein Kinase 3 - metabolism | beta-Arrestin 2 | beta-Arrestin 1 | beta-Arrestins | Receptors, G-Protein-Coupled - genetics | Quinazolines - pharmacology | G-Protein-Coupled Receptor Kinase 5 - genetics | Membrane Microdomains - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | Lipid Raft | Bile Acid | Endocytosis | G Protein-coupled Receptors (GPCR) | Arrestin | Signal Transduction
Journal Article
Circulation Research, ISSN 0009-7330, 04/2013, Volume 112, Issue 8, pp. 1121 - 1134
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2013, Volume 123, Issue 11, pp. 4714 - 4730
Journal Article
Circulation, ISSN 0009-7322, 05/2011, Volume 123, Issue 18, pp. 1953 - 1962
Journal Article
Cancer Letters, ISSN 0304-3835, 11/2017, Volume 408, pp. 10 - 21
Increasing evidence demonstrates that long noncoding RNAs (lncRNAs) regulate gene and protein expression by exerting an influence on transcriptional and... 
UCA1 | Ubiquitination | lncRNA | Metastasis | Gastric cancer | ACTIVATION | MECHANISMS | CBL | INVASION | ONCOLOGY | PROTEIN-COUPLED RECEPTOR | RING | DISEASE | EXPRESSION | PROGRESSION | KINASES | Cell Proliferation | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Stomach Neoplasms - metabolism | Male | Stomach Neoplasms - pathology | G-Protein-Coupled Receptor Kinase 2 - genetics | G-Protein-Coupled Receptor Kinase 2 - metabolism | Matrix Metalloproteinase 9 - metabolism | Lung Neoplasms - secondary | Matrix Metalloproteinase 9 - genetics | Proteolysis | Mitogen-Activated Protein Kinase 1 - genetics | Tumor Cells, Cultured | Lung Neoplasms - genetics | Stomach Neoplasms - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | RNA, Long Noncoding - genetics | Xenograft Model Antitumor Assays | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Mice, Nude | Mice | Mice, Inbred BALB C | Apoptosis | Cell Movement | Mitogen-Activated Protein Kinase 1 - metabolism | Ubiquitin | Antisense RNA | Stomach cancer | Cancer | Medical colleges | Genetic transcription | Post-transcription | Transcription | b-Adrenergic-receptor kinase | Kinases | Experiments | Metastases | Degradation | Proteins | Ethics | Signal transduction | Rodents | Cbl protein | Wound healing | Mortality | Extracellular signal-regulated kinase | Gene expression | Ribonucleic acid--RNA | Gelatinase B | Signaling | Tumors
Journal Article
PLoS Pathogens, ISSN 1553-7366, 2015, Volume 11, Issue 9, p. e1005171
Kaposi's sarcoma (KS) is a highly disseminated angiogenic tumor of endothelial cells linked to infection by Kaposi's sarcoma-associated herpesvirus (KSHV).... 
DNA-SEQUENCES | VIRUS | CHEMOTAXIS | SARCOMA-ASSOCIATED HERPESVIRUS | MICROBIOLOGY | ENCODED MICRORNAS | HIV-1 NEF | REPLICATION | VIROLOGY | KAPOSIS-SARCOMA | NEUTROPHIL MIGRATION | EXPRESSION | PARASITOLOGY | Human Umbilical Vein Endothelial Cells - metabolism | Sarcoma, Kaposi - metabolism | Humans | Human Umbilical Vein Endothelial Cells - immunology | MicroRNAs - metabolism | G-Protein-Coupled Receptor Kinase 2 - genetics | G-Protein-Coupled Receptor Kinase 2 - metabolism | Human Umbilical Vein Endothelial Cells - virology | Proto-Oncogene Proteins c-akt - genetics | Virus Internalization | Herpesvirus 8, Human - immunology | G-Protein-Coupled Receptor Kinase 2 - antagonists & inhibitors | RNA Interference | Gene Deletion | RNA, Viral - metabolism | Sarcoma, Kaposi - immunology | Proto-Oncogene Proteins c-akt - metabolism | Herpesvirus 8, Human - physiology | Endothelium, Vascular - immunology | RNA - metabolism | Recombinant Proteins - metabolism | Signal Transduction | Neoplasm Invasiveness | Cells, Cultured | Recombinant Proteins - chemistry | Sarcoma, Kaposi - virology | Sarcoma, Kaposi - pathology | Receptors, Interleukin-8B - agonists | Host-Pathogen Interactions | Endothelium, Vascular - metabolism | Endothelium, Vascular - virology | Proto-Oncogene Proteins c-akt - agonists | Receptors, Interleukin-8B - metabolism | Receptors, Interleukin-8B - genetics | Endothelium, Vascular - pathology | Human Umbilical Vein Endothelial Cells - pathology | Mutation | Enzyme Repression | Cell Movement | Human herpesvirus 8 | MicroRNA | Genetic aspects | Host-virus relationships | G proteins | Health aspects | Protein kinases | Kinases | Genes | Rodents | Kaposis sarcoma | Cell adhesion & migration
Journal Article
Circulation, ISSN 0009-7322, 04/2015, Volume 131, Issue 16, pp. 1392 - 1404
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2014, Volume 289, Issue 36, pp. 25262 - 25275
Journal Article
Circulation Research, ISSN 0009-7330, 08/2008, Volume 103, Issue 4, pp. 413 - 422
Journal Article
Annals of Neurology, ISSN 0364-5134, 05/2013, Volume 73, Issue 5, pp. 667 - 678
Objective The concept of inflammation‐induced sensitization is emerging in the field of perinatal brain injury, stroke, Alzheimer disease, and multiple... 
SYSTEMIC INFLAMMATION | RESPONSES | INTERLEUKIN-1 | ACTIVATION | HYPOXIA-ISCHEMIA | NMDA | IMMATURE BRAIN | ISCHEMIC BRAIN-INJURY | TNF-ALPHA | NEUROSCIENCES | CLINICAL NEUROLOGY | BACTERIAL-ENDOTOXIN SENSITIZES | Inflammation - chemically induced | Glial Fibrillary Acidic Protein - genetics | Ibotenic Acid - toxicity | Calcium - metabolism | Neurodegenerative Diseases - etiology | Humans | Cerebral Cortex - pathology | Male | Receptors, Metabotropic Glutamate - metabolism | G-Protein-Coupled Receptor Kinase 2 - genetics | G-Protein-Coupled Receptor Kinase 2 - metabolism | Glial Fibrillary Acidic Protein - metabolism | Cerebral Cortex - metabolism | Inflammation - complications | Neurotoxicity Syndromes - pathology | Receptors, Metabotropic Glutamate - genetics | Interleukin-1beta - toxicity | Female | Neurons - metabolism | Cerebral Cortex - drug effects | Neurons - drug effects | Cytokines - genetics | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Disease Models, Animal | Animals, Newborn | Phospholipase C beta - metabolism | Lipopolysaccharides - toxicity | Cytokines - metabolism | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Rats | Mice, Transgenic | Neurodegenerative Diseases - metabolism | Prenatal Exposure Delayed Effects - physiopathology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Gene Expression Regulation - drug effects | Neurotoxicity Syndromes - etiology | Pregnancy | Animals | Excitatory Amino Acid Agonists - toxicity | Analysis of Variance | Neurotoxicity Syndromes - metabolism | Mice | Prenatal Exposure Delayed Effects - chemically induced | Neurons | Neurodegeneration | Rodents
Journal Article
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, ISSN 0363-6119, 04/2018, Volume 314, Issue 4, pp. R574 - R583
Journal Article