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Nature Chemical Biology, ISSN 1552-4450, 2012, Volume 8, Issue 3, pp. 301 - 310
Guanine-rich DNA sequences that can adopt non-Watson-Crick structures in vitro are prevalent in the human genome. Whether such structures normally exist in... 
TELOMERE | HUMAN GENOME | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | TRANSCRIPTION | DOUBLE-STRAND BREAKS | INHIBITOR | G-QUADRUPLEX | HELICASE | CANCER | Genetics | Oncology | Pharmacology | Molecular biology | DNA damage | DNA structure | Chromatin | Immunoprecipitation | Motility | Nucleotide sequence | Transcription | Breast cancer | Genomes | Src protein | Mammalian cells | genomics | Proto-oncogenes
Journal Article
Chemical Reviews, ISSN 0009-2665, 06/2017, Volume 117, Issue 12, pp. 8272 - 8325
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2014, Volume 289, Issue 36, pp. 25112 - 25136
Journal Article
CURRENT MEDICINAL CHEMISTRY, ISSN 0929-8673, 2017, Volume 24, Issue 15, pp. 1488 - 1503
DNA-damaging drugs in cancer present two main problems: therapeutic resistance and side effects and both can associate with DNA repair, which can be targeted... 
bleomycin | mitomycin C | CHEMISTRY, MEDICINAL | TOPOISOMERASE-II | DNA quadruplex | SMALL-MOLECULE | BIOCHEMISTRY & MOLECULAR BIOLOGY | synthetic lethality | DEPENDENT PROTEIN-KINASE | DOUBLE-STRAND BREAKS | DNA repair | FANCONI-ANEMIA | TUMOR-INITIATING CELLS | CANCER STEM-CELLS | DNA-damaging drugs | PHARMACOLOGY & PHARMACY | DNA topoisomerase 1 and 2 inhibitors | HUMAN RECQ HELICASES | HIGH-RISK NEUROBLASTOMA | RNA G-QUADRUPLEX | DNA Damage - drug effects | Antibiotics, Antineoplastic - toxicity | G-Quadruplexes - drug effects | Phosphoric Diester Hydrolases - metabolism | Topoisomerase Inhibitors - toxicity | Humans | DNA Topoisomerases - metabolism | DNA Topoisomerases - chemistry | Neoplasms - drug therapy | Phosphoric Diester Hydrolases - chemistry | Topoisomerase Inhibitors - therapeutic use | Antibiotics, Antineoplastic - therapeutic use | Bleomycin - therapeutic use | DNA Repair | Topoisomerase Inhibitors - chemistry | Neoplasms - pathology | Bleomycin - toxicity | Cell proliferation | Drugs | Therapy | Protein kinase C | Toxicity | Stabilization | DNA damage | Poly(ADP-ribose) | Xeroderma pigmentosum | Lethality | Kinases | ADP | Guanine | Proteins | Antitumor agents | Bleomycin | Pathways | Ribose | Mitomycin C | Catalysis | Repair | Telomerase | Deoxyribonucleic acid--DNA | Adducts | Phosphodiesterase | Mitomycin | Poly(ADP-ribose) polymerase | Nucleotide excision repair | Crosslinking | Substrates | Polymerase | Telomeres | Poly(ADP-ribose) Polymerase 1 | Side effects | DNA-dependent protein kinase | DNA adducts | Cancer
Journal Article
Dalton Transactions, ISSN 1477-9226, 2/2015, Volume 44, Issue 8, pp. 355 - 3526
Journal Article
Dalton transactions (Cambridge, England : 2003), ISSN 1477-9226, 02/2015, Volume 44, Issue 8, pp. 3505 - 3526
Increasing numbers of DNA structures are being revealed using biophysical, spectroscopic and genomic methods. The diversity of transition metal complexes is... 
Transition Elements - chemistry | DNA - chemistry | Molecular Docking Simulation | Nucleic Acid Conformation | Binding Sites | Coordination Complexes - chemistry | Platinum - chemistry | Ruthenium - chemistry | Intercalating Agents - chemistry
Journal Article