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Journal of Biological Chemistry, ISSN 0021-9258, 06/2011, Volume 286, Issue 23, pp. 20217 - 20227
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2008, Volume 118, Issue 5, pp. 1739 - 1749
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, p. e63187
Journal Article
Oncogene, ISSN 0950-9232, 04/2008, Volume 27, Issue 18, pp. 2583 - 2593
When normal cells come under stress, the wild-type (WT) p53 level increases resulting in the regulation of gene expression responsible for growth arrest or... 
DNA replication | Wild-type p53 | Cell cycle | Microarray analysis | Transcriptional repression | P53-MEDIATED TRANSCRIPTIONAL REPRESSION | microarray analysis | PROTEIN | VIRAL PROMOTERS | cell cycle | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | G/M PROMOTERS | CELL-CYCLE ARREST | IDENTIFICATION | CELL BIOLOGY | transcriptional repression | ONCOLOGY | GENETICS & HEREDITY | TUMOR-SUPPRESSOR | wild-type p53 | MODULATION | E2F5 Transcription Factor - biosynthesis | Base Sequence - genetics | Tumor Suppressor Proteins - metabolism | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Humans | Tumor Suppressor Protein p53 - metabolism | Gene Expression Regulation - physiology | Nuclear Proteins - metabolism | Cell Cycle Proteins - biosynthesis | Gene Expression Profiling | DNA-Binding Proteins - genetics | Promoter Regions, Genetic - genetics | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Tumor Protein p73 | DNA Damage - physiology | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Cell Line, Tumor | Apoptosis - physiology | Nuclear Proteins - genetics | E2F5 Transcription Factor - genetics | Sequence Deletion - genetics | Usage | DNA microarrays | Tumor suppressor genes | Cellular signal transduction | Genetic aspects | Research | Health aspects | Oncology | Genetics | Gene expression | Lung cancer
Journal Article
BBA - Gene Regulatory Mechanisms, ISSN 1874-9399, 08/2012, Volume 1819, Issue 8, pp. 855 - 862
The promoters of genes which regulate entry into and progress through mitosis are typically induced maximally in G2 by transcription factors that include B-Myb... 
Mitosis | FoxM1 | Pioneer transcription factor | Cell cycle | B-Myb (MybL2) | ACTIVATION | PHOSPHORYLATION | STABILITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTIONAL NETWORK | MITOTIC PROGRESSION | CELL-CYCLE PROGRESSION | HUMAN BREAST-CANCER | REPRESSOR COMPLEX | BIOPHYSICS | G/M GENES | EXPRESSION | Genetic research | Genetic transcription | RNA | Phosphotransferases | Genes
Journal Article
Journal Article
CELL CYCLE, ISSN 1538-4101, 08/2007, Volume 6, Issue 15, pp. 1903 - 1913
Here we report the identification of the LIN complex ( LINC), a human multiprotein complex that is required for transcriptional activation of G2/ M genes. LINC... 
ACTIVATION | PROTEIN | G/M | E2F FAMILY | pocket-protein | chromatin | CELL-CYCLE CONTROL | DROSOPHILA | NEGATIVE REGULATOR | DNA-REPLICATION | CELL BIOLOGY | ELEGANS | E2F | B-MYB | TRANSCRIPTION FACTOR
Journal Article
JOURNAL OF VIROLOGY, ISSN 0022-538X, 02/2019, Volume 93, Issue 4
Vesicular stomatitis virus (VSV) (a rhabdovirus) and its variant VSV-Delta M51 are widely used model systems to study mechanisms of virus-host interactions.... 
paclitaxel | APOPTOSIS | G/M | CANCER CELLS | Sendai virus | cell cycle | vesicular stomatitis virus | MATRIX PROTEIN | ONCOLYTIC VIROTHERAPY | MITOTIC INHIBITION | TRANSCRIPTION | colchicine | INDUCTION | type I interferon | G2/M ARREST | nonsegmented negative-strand RNA virus | VIROLOGY | SIGNALING PATHWAY | mitotic inhibition of transcription | VESICULAR STOMATITIS-VIRUS
Journal Article