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PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e94298
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2017, Volume 127, Issue 5, pp. 1651 - 1663
Mature B cell pools retain a substantial proportion of polyreactive and self-reactive clonotypes, suggesting that activation checkpoints exist to reduce the... 
MEDICINE, RESEARCH & EXPERIMENTAL | SYSTEMIC-LUPUS-ERYTHEMATOSUS | INTERFERON-GAMMA | TOLL-LIKE RECEPTOR-9 | MARGINAL ZONE | ESCAPE TOLERANCE | LYMPHOCYTE STIMULATOR | SOMATIC MUTATION | AUTOANTIBODY PRODUCTION | T-CELLS | CUTTING EDGE | Toll-Like Receptor 9 - genetics | Male | T-Box Domain Proteins - immunology | G1 Phase Cell Cycle Checkpoints - immunology | MAP Kinase Signaling System - immunology | Toll-Like Receptor 9 - immunology | Cell Differentiation - genetics | Interleukins - genetics | MAP Kinase Signaling System - genetics | Interleukins - immunology | B-Cell Activating Factor - immunology | Female | CD40 Antigens - genetics | Interferon-gamma - genetics | Cell Line | DNA - immunology | p38 Mitogen-Activated Protein Kinases - genetics | p38 Mitogen-Activated Protein Kinases - immunology | T-Box Domain Proteins - genetics | G1 Phase Cell Cycle Checkpoints - genetics | Receptors, Antigen, B-Cell - immunology | Mice, Knockout | Cell Differentiation - immunology | Animals | B-Lymphocytes - immunology | B-Cell Activating Factor - genetics | Interferon-gamma - immunology | Antigens - immunology | Receptors, Antigen, B-Cell - genetics | Mice | CD40 Antigens - immunology | Antigens | DNA | Cytokine receptors | TLR9 | Cellular signal transduction | Research | B cells | CD27 antigen | CD40 antigen | Laboratories | B-cell receptor | Kinases | Genotype & phenotype | Mitochondria | Cell activation | Lymphocytes | Cell cycle | Interleukin 21 | Toll-like receptors | Deoxyribonucleic acid--DNA | Immunization | Cell survival | Cytokines | Secretion | Cell division | T cell receptors | Survival | TLR9 protein | Immune response (humoral) | Medicine | Lymphocytes B | BLyS protein | Ligands | Interferon | Regulation | Autoimmune diseases | Apoptosis
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2013, Volume 288, Issue 42, pp. 30320 - 30329
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2015, Volume 17, Issue 8, pp. 1036 - 1048
Robust mechanisms to control cell proliferation have evolved to maintain the integrity of organ architecture. Here, we investigated how two critical... 
RNA-POLYMERASE-I | TRANSCRIPTIONAL NETWORK | C-MYC | GENE-EXPRESSION | MURINE DEVELOPMENT | INTESTINAL CRYPTS | RIBOSOMAL DNA | CONDITIONAL DELETION | BETA-CATENIN | DNA-REPLICATION | CELL BIOLOGY | E2F Transcription Factors - deficiency | E2F2 Transcription Factor - genetics | Cell Proliferation | Epithelial Cells - metabolism | Intestine, Small - pathology | Male | Mice, 129 Strain | E2F2 Transcription Factor - metabolism | E2F Transcription Factors - metabolism | S Phase Cell Cycle Checkpoints | Time Factors | G1 Phase Cell Cycle Checkpoints | Female | Transcription, Genetic | Binding Sites | Promoter Regions, Genetic | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Chromatin Assembly and Disassembly | E2F3 Transcription Factor - genetics | Epithelial Cells - pathology | Genotype | Retinoblastoma Protein - deficiency | E2F1 Transcription Factor - metabolism | Proto-Oncogene Proteins c-myc - metabolism | G2 Phase Cell Cycle Checkpoints | Mice, Knockout | Cell Cycle Checkpoints | Phenotype | Animals | Retinoblastoma Protein - genetics | Proto-Oncogene Proteins c-myc - deficiency | E2F3 Transcription Factor - metabolism | Proto-Oncogene Proteins c-myc - genetics | Intestine, Small - metabolism | E2F1 Transcription Factor - genetics | E2F Transcription Factors - genetics | Cell proliferation | Transcription factors | Genetic aspects | Genetic transcription | Properties | Health aspects | Life Sciences | Animal genetics | Genetics | Molecular biology | Biochemistry, Molecular Biology | Cancer
Journal Article
Cell Stem Cell, ISSN 1934-5909, 07/2012, Volume 11, Issue 1, pp. 36 - 49
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e98207
Recent studies have demonstrated that the anti-diabetic drug, metformin, can exhibit direct antitumoral effects, or can indirectly decrease tumor proliferation... 
PROSTATE | OBESITY | ACTIVATED PROTEIN-KINASE | INHIBITION | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | GROWTH | AKT | AUTOPHAGY | TUMOR SUPPRESSORS | AMP-Activated Protein Kinases - metabolism | Oxidants - pharmacology | Hydrogen - pharmacology | Apoptosis - drug effects | Humans | Metformin - pharmacology | Enzyme Activation - drug effects | Neoplasm Proteins - metabolism | Breast Neoplasms - metabolism | Hypoglycemic Agents - pharmacology | Breast Neoplasms - pathology | Forkhead Transcription Factors - metabolism | Cell Line, Tumor | Female | Resting Phase, Cell Cycle - drug effects | Oxidative Stress - drug effects | Forkhead Box Protein O3 | G1 Phase Cell Cycle Checkpoints - drug effects | AMP-Activated Protein Kinases - genetics | Oxidative stress | Cancer cells | Cell cycle | Breast cancer | Metformin | Diabetes therapy | Apoptosis | Flow cytometry | Cell culture | Phosphorylation | Hydrogen peroxide | Bax protein | Bcl-2 protein | AKT protein | Superoxide dismutase | Biology | Activation | Kinases | Caspase-3 | Cancer therapies | Western blotting | Necrosis | Proteins | Signal transduction | Cell growth | Catalase | Penicillin | FOXO3 protein | G1 phase | Diabetes mellitus | Extracellular signal-regulated kinase | Caspase | MAP kinase | Pharmacology | Gene expression | Insulin | Zinc | Polymerase chain reaction | Cytometry | Molecular modelling | Cell number | Diabetes | Viability | Prostate | Cancer
Journal Article