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Scientific Reports, ISSN 2045-2322, 02/2014, Volume 4, Issue 1, pp. 4012 - 4012
The quiescent (G0) phase of the cell cycle is the reversible phase from which the cells exit from the cell cycle. Due to the difficulty of defining the G0... 
PROGENITOR CELLS | SKELETAL-MUSCLE | MUSCLE REGENERATION | CDK INHIBITORS | S-PHASE ENTRY | MULTIDISCIPLINARY SCIENCES | HEMATOPOIETIC STEM-CELLS | NICHE | SATELLITE CELLS | P27(KIP1) | G-G TRANSITION | RNA-Binding Proteins - genetics | Selenium-Binding Proteins - genetics | Tumor Suppressor Protein p53 - biosynthesis | Stem Cells - cytology | Retinoblastoma Protein - biosynthesis | RNA-Binding Proteins - biosynthesis | Selenium-Binding Proteins - biosynthesis | Apoptosis Regulatory Proteins - genetics | Cyclin-Dependent Kinase Inhibitor p16 - biosynthesis | G1 Phase - genetics | Apoptosis Regulatory Proteins - biosynthesis | Recombinant Fusion Proteins - biosynthesis | Cell Line | Cyclin-Dependent Kinase Inhibitor p21 - biosynthesis | Gene Expression | Carrier Proteins - biosynthesis | Bacterial Proteins - genetics | Mice, Transgenic | MicroRNAs - biosynthesis | Carrier Proteins - genetics | Cyclin-Dependent Kinase Inhibitor p57 - biosynthesis | Animals | Resting Phase, Cell Cycle - genetics | Recombinant Fusion Proteins - genetics | Luminescent Proteins - genetics | Mice | 3T3 Cells | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Cyclin-dependent kinase | Rodents | Stem cells | Cell cycle | Defective mutant | Transgenic mice | Stem cell transplantation | Cyclin-dependent kinase inhibitor p27 | Fusion protein | Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 06/2014, Volume 33, Issue 25, pp. 3334 - 3341
Human epidermal growth factor receptor 2 (HER2)-directed treatment using trastuzumab has shown clinical benefit in HER2-positive gastric cancer. Clinical... 
lapatinib resistance | HER2-positive gastric cancer | β-catenin | epithelial-mesenchymal transition | XAV939 | Testican-1 | b-catenin | beta-catenin | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | CELL-LINES | TRASTUZUMAB RESISTANCE | GEFITINIB RESISTANCE | CELL BIOLOGY | BREAST-CANCER | LUNG-CANCER | ONCOLOGY | GROWTH | GENETICS & HEREDITY | TYROSINE KINASE INHIBITOR | HER2 | EXPRESSION | Proto-Oncogene Proteins c-met - metabolism | Transcription, Genetic - drug effects | Receptor, ErbB-2 - genetics | Humans | Receptor, ErbB-2 - metabolism | Stomach Neoplasms - metabolism | Drug Resistance, Neoplasm | Epithelial-Mesenchymal Transition - drug effects | G1 Phase - drug effects | Epithelial-Mesenchymal Transition - genetics | Cell Cycle Checkpoints - genetics | G1 Phase - genetics | Stomach Neoplasms - genetics | Proteoglycans - metabolism | Signal Transduction - genetics | Stomach Neoplasms - drug therapy | beta Catenin - metabolism | beta Catenin - genetics | Proto-Oncogene Proteins c-met - genetics | Up-Regulation - drug effects | Signal Transduction - drug effects | Cell Cycle Checkpoints - drug effects | Cell Line, Tumor | Quinazolines - pharmacology | Proteoglycans - genetics | Oncology, Experimental | Genetic research | Genetic aspects | Research | Properties | Drug resistance | Growth factors | Stomach cancer | Cancer | Signal transduction | Chemotherapy | Epidermal growth factor | Gene expression | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2005, Volume 7, Issue 8, pp. 831 - 836
The cyclin-dependent kinase inhibitor p27Kip1 is known as a negative regulator of cell-cycle progression and as a tumour suppressor. Cdk2 is the main target of... 
CYCLIN-E | ENTRY | CDK2 | GROWTH | MOUSE | DEPENDENT-KINASE INHIBITOR | HYPERPLASIA | MAMMALIAN-CELLS | MICE LACKING | P27(KIP1) | CELL BIOLOGY | Cell Proliferation | Cyclin-Dependent Kinases - metabolism | Ovarian Neoplasms - pathology | Gonads - pathology | Male | Mitosis - genetics | Body Weight - genetics | CDC2 Protein Kinase - metabolism | Ovarian Neoplasms - genetics | Cyclin-Dependent Kinase 2 | Cyclins - metabolism | 2-Aminopurine - analogs & derivatives | Cyclin-Dependent Kinases - antagonists & inhibitors | Thymus Gland - metabolism | Fibroblasts - metabolism | Tumor Suppressor Proteins - metabolism | Pituitary Neoplasms - genetics | Cell Cycle Proteins - metabolism | Enzyme Inhibitors - pharmacology | CDC2-CDC28 Kinases - metabolism | Mice, Knockout | Fibroblasts - drug effects | Fibroblasts - cytology | Mice | Interphase - genetics | Interphase - physiology | Infertility - genetics | Transfection | Tumor Suppressor Proteins - genetics | Thymus Gland - pathology | Cell Cycle Proteins - genetics | Female | G1 Phase - genetics | CDC2 Protein Kinase - genetics | Mice, Inbred C57BL | Pituitary Neoplasms - pathology | 2-Aminopurine - pharmacology | G1 Phase - physiology | S Phase - genetics | Cyclin-Dependent Kinase Inhibitor p27 | Animals | Spleen - metabolism | RNA, Double-Stranded - genetics | Sex Factors | Protein Binding | S Phase - physiology | S Phase - drug effects | Cyclin E - metabolism | CDC2-CDC28 Kinases - genetics | Interphase - drug effects | Crosses, Genetic | Physiological aspects | Tumor suppressor genes | Control | Cellular signal transduction | Research | Cell cycle | Index Medicus
Journal Article
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 09/2014, Volume 452, Issue 3, pp. 746 - 752
Tumor metastasis is the leading cause of mortality and morbidity of prostate cancer (PCa) patients. Epithelial–mesenchymal transition (EMT) plays a critical... 
Epithelial–mesenchymal transition | miR30a | Metformin | Prostate cancer | SOX4 | Epithelial-mesenchymal transition | IN-VITRO | ONCOGENE | BIOPHYSICS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | EXPRESSION | EMT | Prostatic Neoplasms - metabolism | Luciferases - metabolism | Cadherins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Epithelial-Mesenchymal Transition - drug effects | Male | MicroRNAs - metabolism | RNA, Messenger - metabolism | Antigens, CD - genetics | Prostate - metabolism | Antigens, CD - metabolism | Luciferases - genetics | Prostate - pathology | Prostatic Neoplasms - genetics | Cadherins - genetics | Genes, Reporter | Prostatic Neoplasms - pathology | SOXC Transcription Factors - genetics | Signal Transduction | Metformin - pharmacology | RNA, Messenger - genetics | SOXC Transcription Factors - metabolism | beta Catenin - metabolism | G1 Phase Cell Cycle Checkpoints - genetics | beta Catenin - genetics | Hypoglycemic Agents - pharmacology | Cell Movement - drug effects | Transforming Growth Factor beta - pharmacology | Cell Line, Tumor | Prostatectomy | MicroRNAs - genetics | G1 Phase Cell Cycle Checkpoints - drug effects | Genes | Stem cells | Genetic aspects | Cancer | Tumors | Index Medicus | MORTALITY | PROSTATE | PATIENTS | DISEASE INCIDENCE | NEOPLASMS | METASTASES | CELL PROLIFERATION | TUMOR CELLS | 60 APPLIED LIFE SCIENCES | MESSENGER-RNA | GENES | CELL CYCLE | RADICALS | PROTEINS
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 05/2019, Volume 234, Issue 5, pp. 7257 - 7265
Journal Article
BioMed Research International, ISSN 2314-6133, 2014, Volume 2014, pp. 546353 - 12
Journal Article