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gadd153/chop (17) 17
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Journal Article
Biological Trace Element Research, ISSN 0163-4984, 4/2017, Volume 176, Issue 2, pp. 278 - 293
Journal Article
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 12/2012, Volume 265, Issue 3, pp. 325 - 334
Prodigiosin is a bacterial tripyrrole pigment with potent cytotoxicity against diverse human cancer cell lines. Endoplasmic reticulum (ER) stress is initiated... 
ER stress | BCL2 | Breast cancer | CHOP/GADD153 | Prodigiosin | Apoptosis | ELEVATED GADD153/CHOP EXPRESSION | CANCER CELLS | JNK | FAMILY | ANTICANCER AGENT PRODIGIOSIN | BCL-2 | PHARMACOLOGY & PHARMACY | TOXICOLOGY | MELANOMA-CELLS | UP-REGULATION | Transcription Factor CHOP - genetics | Endoribonucleases - genetics | eIF-2 Kinase - metabolism | Humans | Immunoblotting | Activating Transcription Factor 6 - genetics | Breast Neoplasms - metabolism | RNA - genetics | Heat-Shock Proteins - genetics | MCF-7 Cells | Female | Antineoplastic Agents - pharmacology | Cell Death - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Protein-Serine-Threonine Kinases - metabolism | eIF-2 Kinase - genetics | Prodigiosin - pharmacology | Endoplasmic Reticulum Stress - drug effects | Endoribonucleases - metabolism | Heat-Shock Proteins - metabolism | Protein-Serine-Threonine Kinases - genetics | RNA - chemistry | Reverse Transcriptase Polymerase Chain Reaction | Unfolded Protein Response | Breast Neoplasms - drug therapy | Up-Regulation - drug effects | Activating Transcription Factor 6 - metabolism | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Cell Line, Tumor | Transcription Factor CHOP - metabolism | Endoplasmic Reticulum Stress - physiology | Carcinoma | Cell death | Stress (Physiology) | Biochemistry | Cells | Cancer | Index Medicus | Phosphorylation | Cell survival | Transcription | Colonies | Mortality | Poly(ADP-ribose) polymerase | Cytotoxicity | Tumor cell lines | prodigiosin | Survival | Promoters | Stress | Proteins | Breast carcinoma | Signal transduction | c-Jun amino-terminal kinase | Protein folding | Pigments | Initiation factor eIF-2 | Endoplasmic reticulum | thapsigargin | APOPTOSIS | MAMMARY GLANDS | PHOSPHORYLATION | COLONY FORMATION | TOXICITY | CLEAVAGE | 60 APPLIED LIFE SCIENCES | PIGMENTS | BIOLOGICAL STRESS | BIOLOGICAL RECOVERY | ENDOPLASMIC RETICULUM | INHIBITION | PROMOTERS | CARCINOMAS | PROTEINS
Journal Article
Oncogene, ISSN 0950-9232, 04/2001, Volume 20, Issue 17, pp. 2178 - 2185
Journal Article
Journal Article
Molecular and Cellular Neuroscience, ISSN 1044-7431, 07/2006, Volume 32, Issue 3, pp. 242 - 253
Loss of trophic or activity-dependent survival signals is commonly recognized as a stimulus for neuronal apoptosis and may play a significant role in... 
Inositol 1,4,5-trisphosphate receptor | Bcl-2 | Glycogen synthase kinase-3β | Trophic factor withdrawal | Gadd153/CHOP | Cerebellar granule neurons | Endoplasmic reticulum stress | Apoptosis | trophic factor withdrawal | glycogen synthase kinase-3 beta | ER STRESS | ALZHEIMERS-DISEASE | GSK-3-BETA | apoptosis | NEUROSCIENCES | endoplasmic reticulum stress | CELL-DEATH | KINASE 3-BETA | cerebellar granule neurons | GLYCOGEN-SYNTHASE KINASE-3-BETA | UNFOLDED PROTEIN RESPONSE | CALCIUM-RELEASE | inositol 1,4,5-trisphosphate receptor | GROWTH-FACTOR-I | Nerve Growth Factors - deficiency | Caspases - physiology | Caspase 9 | Apoptosis - drug effects | Cerebellum - drug effects | Apoptosis - genetics | Glycogen Synthase Kinase 3 beta | Cerebellum - enzymology | Stress, Physiological - chemically induced | Endoplasmic Reticulum - drug effects | Caspases - biosynthesis | Nerve Growth Factors - physiology | Neurons - drug effects | Endoplasmic Reticulum - enzymology | Cricetinae | Endoplasmic Reticulum - genetics | Glycogen Synthase Kinase 3 - biosynthesis | Caspases - genetics | Stress, Physiological - genetics | Cells, Cultured | Rats | Rats, Sprague-Dawley | Stress, Physiological - enzymology | Animals | Glycogen Synthase Kinase 3 - genetics | Signal Transduction - drug effects | Nerve Growth Factors - genetics | Neurons - enzymology | Signal Transduction - physiology | Apoptosis - physiology
Journal Article
BRAIN RESEARCH BULLETIN, ISSN 0361-9230, 02/2005, Volume 65, Issue 1, pp. 87 - 95
Expression of CCAAT/enhancer-binding protein beta (C/EBPbeta) and growth-arrest DNA damage-inducible t53/C/EBPbeta homology protein (GADD153/CHOP) increased... 
C/EBP beta | ACTIVATED PROTEIN-KINASE | dopamine toxicity | RAT HEPATOCYTES | GADD153/CHOP | DEATH | ENDOPLASMIC-RETICULUM STRESS | alpha-synuclein | NEUROSCIENCES | MOLECULAR PATHWAYS | TRANSCRIPTION FACTOR CHOP | SH-SY5Y cells | PC12 CELLS | UP-REGULATION | PARKINSONS-DISEASE
Journal Article
Neuroscience Research, ISSN 0168-0102, 2008, Volume 60, Issue 1, pp. 29 - 39
Journal Article
Journal Article
Brain Research Bulletin, ISSN 0361-9230, 2005, Volume 65, Issue 1, pp. 87 - 95
Expression of CCAAT/enhancer-binding protein β (C/EBPβ) and growth-arrest DNA damage-inducible 153/C/EBPβ homology protein (GADD153/CHOP) increased after... 
C/EBPβ | Dopamine toxicity | α-Synuclein | SH-SY5Y cells | GADD153/CHOP
Journal Article