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PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, p. e35506
Background: The inflammatory response plays a critical role in hypertension-induced cardiac remodeling. We aimed to study how interaction among inflammatory... 
FIBROBLAST PRECURSORS | SYSTEM | CYTOKINE PROFILES | INTERFERON-GAMMA | MULTIDISCIPLINARY SCIENCES | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | ANGIOTENSIN-II | MYOCARDITIS | HYPERTENSION | NF-KAPPA-B | EXPRESSION | Inflammation - chemically induced | Angiotensin II - pharmacology | Myocardium - immunology | Interferon-gamma - metabolism | Fibrosis - metabolism | Mice, Knockout | Inflammation - metabolism | Animals | T-Lymphocytes - metabolism | Myocardium - metabolism | Fibrosis - immunology | Chemokine CCL2 - metabolism | Mice | Interferon-gamma - genetics | Body Weight - physiology | Heart | Cell culture | Collagen (type I) | Transcription factors | Disease | Leukocyte migration | Smooth muscle | Nervous system | Leukocytes | Recruitment | Metastases | Proteins | Ischemia | Education | Fibroblasts | Blood pressure | Cytokines | Cardiac muscle | Tumor necrosis factor-α | Low level | Hospitals | Monocyte chemoattractant protein | Nitric oxide | γ-Interferon | Fibrosis | Infiltration | Cell migration | Monocyte chemoattractant protein 1 | Flow cytometry | Laboratories | Inflammatory response | Lymphocytes T | Insulin-like growth factors | Macrophages | Remodeling | Infusion | Lymphocytes | Actin | Rodents | Tumor necrosis factor-TNF | Angiotensin II | Heart diseases | Hypertension | Kidneys | Muscles | Inflammation | White blood cells | Angiotensin | Interferon | gamma -Interferon | Tumor necrosis factor- alpha
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 09/2013, Volume 133, Issue 9, pp. 2170 - 2179
Journal Article
Helicobacter, ISSN 1083-4389, 02/2013, Volume 18, Issue 1, pp. 13 - 21
Journal Article
Journal Article
Neurogastroenterology & Motility, ISSN 1350-1925, 09/2011, Volume 23, Issue 9, pp. e372 - e382
Background  Enteric glial cells (EGCs) have been recently indicated as key regulators of intestinal inflammation in animals. Whether or not this is true and... 
enteric glial cells | nitric oxide | intestinal inflammation | S100B | glial fibrillary acidic protein | Intestinal inflammation | Enteric glial cells | Glial fibrillary acidic protein | Nitric oxide | NERVOUS-SYSTEM | CROHNS-DISEASE | PEROXYNITRITE | ULCERATIVE-COLITIS | S100B PROTEIN | ASTROCYTES | NEUROSCIENCES | CLINICAL NEUROLOGY | SYNTHASE | INFLAMMATORY-BOWEL-DISEASE | ENTERIC GLIAL-CELLS | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Enteric Nervous System - cytology | Genes, MHC Class II | Cell Proliferation | Humans | Middle Aged | Male | Nerve Growth Factors - metabolism | Glial Fibrillary Acidic Protein - metabolism | Neuroglia - drug effects | Inflammation - metabolism | Neuroglia - cytology | Female | Biomarkers - metabolism | Nitric Oxide - biosynthesis | S100 Calcium Binding Protein beta Subunit | Autocrine Communication - physiology | Cells, Cultured | Proto-Oncogene Proteins c-fos - metabolism | S100 Proteins - metabolism | Animals | Nitric Oxide Synthase Type II - genetics | Lipopolysaccharides - pharmacology | Neuroglia - metabolism | Interferon-gamma - pharmacology | Nitric Oxide Synthase Type II - metabolism | Inflammation | Intermediate filament proteins | Mitogens | Cell proliferation | Advanced glycosylation end products | Data processing | Glial cells | Nitric-oxide synthase | Lipopolysaccharides | S100 beta protein | Major histocompatibility complex | Intestine | gamma -Interferon | Autocrine signalling | c-Fos protein
Journal Article
Journal Article