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Oncogene, ISSN 0950-9232, 01/2019, Volume 38, Issue 1, pp. 33 - 46
Fibroblast growth factors (FGFs) and their receptors are significant components during fundamental cellular processes. FGF18 plays a distinctive role in... 
MIGRATION | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MIRDB | CANCER | CELL BIOLOGY | INVASION | ONCOLOGY | GROWTH | GENETICS & HEREDITY | GENE AMPLIFICATION | MICRORNA TARGET PREDICTION | INHIBITOR | EXPRESSION | Up-Regulation | MicroRNAs - antagonists & inhibitors | Prognosis | Humans | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Fibroblast Growth Factors - genetics | Stomach Neoplasms - metabolism | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - pharmacology | Gene Expression Profiling | Gene Knockdown Techniques | Heterografts | RNA Interference | RNA, Neoplasm - antagonists & inhibitors | Neoplasm Proteins - genetics | Stomach Neoplasms - etiology | Chromosomal Instability | Fibroblast Growth Factors - physiology | Stomach Neoplasms - genetics | Autocrine Communication | Fibroblast Growth Factors - pharmacology | Signal Transduction - genetics | Recombinant Proteins - pharmacology | Animals | Signal Transduction - drug effects | Cell Transformation, Neoplastic | Cell Line, Tumor | RNA, Neoplasm - genetics | Mice | MicroRNAs - genetics | Fibroblast Growth Factors - antagonists & inhibitors | Care and treatment | MicroRNA | Development and progression | Genetic aspects | Fibroblast growth factors | Gene expression | Health aspects | Stomach cancer | Fibroblast growth factor | Cell survival | Tumor cells | Therapeutic applications | Extracellular signal-regulated kinase | MAP kinase | Genomic instability | Signal transduction | Cell lines | Cell cycle | Tumor suppressor genes | Tumorigenesis | Fibroblast growth factor 18 | Autocrine signalling | Immunofluorescence | Growth factors | Gastric cancer | Fibroblast growth factor receptors | G1 phase
Journal Article
by Speliotes, Elizabeth and Willer, Cristen and Berndt, Sonja and Monda, Keri and Thorleifsson, Gudmar and Jackson, Anne and Allen, H.L and Lindgren, Cecilia and Luan, J and Mägi, Reedik and Randall, Joshua and Vedantam, Sailaja and Winkler, Thomas and Qi, Lu and Workalemahu, Tsegaselassie and Heid, Iris and Steinthorsdottir, Valgerdur and Stringham, Heather and Wheeler, Eleanor and Wood, Anew and Ferreira, Teresa and Weyant, Robert and Segrè, Ayellet and Eestrada, Karol and Liang, Liming and Nemesh, James and Park, J.H and Gustafsson, Stefan and Kilpeläinen, Tuomas and Yang, Joanna and Bouatia-Naji, Nabila and Eesko, Tõnu and Feitosa, Mary Furlan and Kutalik, Zoltán and Mangino, Massimo and Raychaudhuri, Soumya and Scherag, Ane and Smith, Albert Vernon and Welch, Ryan and Zhao, Jing Hua and Aben, Katja and Absher, Devin and Amin, Najaf and Dixon, Anna and Fisher, Eeva and Glazer, Nicole and Goddard, Michael and Heard-Costa, Nancy and Hoesel, Volker and Hottenga, Jouke Jan and Johansson, Åsa and Johnson, Toby and Ketkar, Shamika and Lamina, Claudia and Li, Shengxu and Moffatt, Miriam and Myers, Richard and Narisu, Narisu and Perry, John and Peters, Marjolein and Preuss, Michael and Ripatti, Samuli and Rivadeneira Ramirez, Fernando and Sandholt, Camilla and Scott, Laura and Timpson, Nicholas and Tyrer, Jonathan and Wingerden, Sophie and White, Charles and Wiklund, Freik and Barlassina, Christina and Chasman, Daniel and Cooper, Matthew and Jansson, J.O and Lawrence, Robert and Pellikka, Niina and Prokopenko, Inga and Shi, Jianxin and Thiering, Eelisabeth and Alavere, Helene and Alibrandi, Maria and Almgren, Peter and Arnold, Alice and Aspelund, Thor and Atwood, Larry and Balkau, Beverley and Balmforth, Anthony and Bennett, Amanda and Ben-Shlomo, Y and Bergman, Richard and Bergmann, Sven and Biebermann, Heike and Blakemore, Alexana and Boes, Tanja and Bonnycastle, Lori and Bornstein, Stefan and Brown, Morris and Buchanan, Thomas and Busonero, F and Campbell, Harry and ... and MAGIC and Procardis Consortium and on behalf of Procardis Consortium and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Institutionen för genetik och patologi and Uppsala universitet
Nature Genetics, ISSN 1061-4036, 11/2010, Volume 42, Issue 11, pp. 937 - 948
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by Liu, Jia and Liu, Yang and Liu, Xiuping and Liu, Wenbin and Liu, Yuexin and Sethi, Nilay S and Hinoue, Toshinori and Schneider, Barbara G and Cherniack, Andrew D and Sanchez-Vega, Francisco and Seoane, Jose A and Farshidfar, Farshad and Bowlby, Reanne and Islam, Mirazul and Kim, Jaegil and Chatila, Walid K and Chatila, Walid and Akbani, Rehan and Kanchi, Rupa S and Rabkin, Charles S and Willis, Joseph E and Wang, Jing and Wang, Linghua and Wang, Timothy and Wang, Jioajiao and Wang, Min and Wang, Kenneth K and Wang, Zhining and McCall, Shannon J and McCall, Shannon and Mishra, Lopa and Ojesina, Akinyemi I and Bullman, Susan and Pedamallu, Chandra Sekhar and Lazar, Alexander J and Sakai, Ryo and Caesar-Johnson, Samantha J and Demchok, John A and Felau, Ina and Kasapi, Melpomeni and Ferguson, Martin L and Hutter, Carolyn M and Sofia, Heidi J and Tarnuzzer, Roy and Yang, Ian and Yang, Ju Dong and Yang, Hannah and Yang, Liming and Zenklusen, Jean C and Zhang, Hongzheng and Zhang, Wei and Zhang, Hongxin and Zhang, Jiexin and Zhang, Hailei and Zhang, Jiashan (Julia) and Zhang, Lizhi and Chudamani, Sudha and Lolla, Laxmi and Naresh, Rashi and Pihl, Todd and Sun, Qiang and Sun, Yichao and Wan, Yunhu and Wu, Ye and Cho, Juok and DeFreitas, Timothy and Frazer, Scott and Gehlenborg, Nils and Getz, Gad and Heiman, David I and Lawrence, Michael S and Lin, Pei and Meier, Sam and Noble, Michael S and Saksena, Gordon and Voet, Doug and Bernard, Brady and Chambwe, Nyasha and Dhankani, Varsha and Knijnenburg, Theo and Kramer, Roger and Leinonen, Kalle and Miller, Judy and Miller, Michael and Miller, Christopher A and Reynolds, Sheila and Shmulevich, Ilya and Thorsson, Vésteinn and Thorsson, Vesteinn and Broom, Bradley M and Hegde, Apurva M and Ju, Zhenlin and Korkut, Anil and Li, Jun and Li, Wei and Liang, Han and Ling, Shiyun and Lu, Yiling and Mills, Gordon B and Ng, Kwok-Shing and ... and Canc Genome Atlas Res Network and Cancer Genome Atlas Research Network
Cancer Cell, ISSN 1535-6108, 04/2018, Volume 33, Issue 4, pp. 721 - 735.e8
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 09/2018, Volume 503, Issue 4, pp. 2393 - 2399
-asparagine essentially regulates growth and proliferation of cancer cells. -asparaginase is an anti-cancer enzyme that deprives the cancer cells of... 
l-asparagine | Gastric adenocarcinoma | Intrinsic apoptosis | Tumor progression | l-asparaginase | L-asparaginase | L-asparagine | ACTIVATION | ASSAY | ACID | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLIFERATION | P53 | IN-VITRO | BIOPHYSICS | METABOLISM | GROWTH | Adenocarcinoma | Enzymes | Analysis | Asparagine | Development and progression | Nuclear nonproliferation | Permeability | Tumor proteins | Stomach cancer | Apoptosis
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 06/2017, Volume 376, Issue 22, pp. 2147 - 2159
Journal Article