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1. Full Text A multiply redundant genetic switch 'locks in' the transcriptional signature of regulatory T cells
by Fu, Wenxian and Ergun, Ayla and Lu, Ting and Hill, Jonathan A and Haxhinasto, Sokol and Fassett, Marlys S and Gazit, Roi and Adoro, Stanley and Glimcher, Laurie and Chan, Susan and Kastner, Philippe and Rossi, Derrick and Collins, James J and Mathis, Diane and Benoist, Christophe
Nature Immunology, ISSN 1529-2908, 10/2012, Volume 13, Issue 10, pp. 972 - 980
The transcription factor Foxp3 participates dominantly in the specification and function of Foxp3(+)CD4(+) regulatory T cells (T-reg cells) but is neither... 
POPULATION | RESPONSES | LINEAGE IN-VIVO | SUPPRESSIVE FUNCTION | T(H)17 | FACTOR FOXP3 | GENERATION | DIFFERENTIATION | SPECIFICATION | IMMUNOLOGY | EXPRESSION | T-Lymphocytes, Regulatory - metabolism | Humans | Lymphoid Enhancer-Binding Factor 1 - genetics | Ikaros Transcription Factor - biosynthesis | X-Box Binding Protein 1 | T-Lymphocytes, Regulatory - immunology | Ikaros Transcription Factor - genetics | Serine Endopeptidases - genetics | T-Lymphocytes, Regulatory - cytology | Transcription, Genetic | Cell Differentiation | Lymphocyte Activation | Mice, Inbred C57BL | Computational Biology | Interferon Regulatory Factors - genetics | Transcription Factors - biosynthesis | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Forkhead Transcription Factors - genetics | Regulatory Factor X Transcription Factors | Transcription Factors - metabolism | Animals | Matrix Attachment Region Binding Proteins - genetics | GATA1 Transcription Factor - genetics | Mice | CD4 Antigens - biosynthesis | DNA-Binding Proteins - biosynthesis | Physiological aspects | Transcription factors | Genetic aspects | Research | Genetic transcription | T cells | Index Medicus | Cell culture
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2. Full Text A global role for KLF1 in erythropoiesis revealed by ChIP-seq in primary erythroid cells
by Tallack, Michael R and Whitington, Tom and Yuen, Wai Shan and Wainwright, Elanor N and Keys, Janelle R and Gardiner, Brooke B and Nourbakhsh, Ehsan and Cloonan, Nicole and Grimmond, Sean M and Bailey, Timothy L and Perkins, Andrew C
Genome Research, ISSN 1088-9051, 08/2010, Volume 20, Issue 8, pp. 1052 - 1063
KLF1 regulates a diverse suite of genes to direct erythroid cell differentiation from bipotent progenitors. To determine the local cis-regulatory contexts and... 
PRIMITIVE ERYTHROPOIESIS | HEMOGLOBIN-STABILIZING PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR-BINDING-SITES | MEMBRANE-PROTEIN | EKLF-DEFICIENT MICE | CHROMATIN OCCUPANCY | TRANSCRIPTION FACTOR GATA-1 | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | KRUPPEL-LIKE FACTOR | BETA-THALASSEMIA | Promoter Regions, Genetic | Gene Expression | Oligonucleotide Array Sequence Analysis | Cytoskeleton - genetics | Erythroid Cells - metabolism | Molecular Sequence Data | Apoptosis - genetics | Globins - biosynthesis | GATA1 Transcription Factor - metabolism | Globins - genetics | Animals | Heme - genetics | Erythrocyte Membrane - genetics | Heme - biosynthesis | Base Sequence | GATA1 Transcription Factor - genetics | Mice | Kruppel-Like Transcription Factors - genetics | Erythropoiesis - genetics | Erythropoiesis | Transcription factors | Analysis | Liver | Physiological aspects | Genetic aspects | Research | Cell differentiation | Index Medicus
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3. Full Text The BET Protein BRD2 Cooperates with CTCF to Enforce Transcriptional and Architectural Boundaries
by Hsu, Sarah C and Gilgenast, Thomas G and Bartman, Caroline R and Edwards, Christopher R and Stonestrom, Aaron J and Huang, Peng and Emerson, Daniel J and Evans, Perry and Werner, Michael T and Keller, Cheryl A and Giardine, Belinda and Hardison, Ross C and Raj, Arjun and Phillips-Cremins, Jennifer E and Blobel, Gerd A
Molecular Cell, ISSN 1097-2765, 04/2017, Volume 66, Issue 1, pp. 102 - 116.e7
Bromodomain and extraterminal motif (BET) proteins are pharmacologic targets for the treatment of diverse diseases, yet the roles of individual BET family... 
chromatin architecture | BRD2 | BET proteins | CTCF | transcription | SELECTIVE-INHIBITION | ORGANIZATION | 1ST BROMODOMAIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR GATA-1 | ERYTHROID GENE-EXPRESSION | COHESIN | CHROMATIN INTERACTIONS | DOMAINS | BINDING | GENOME | CELL BIOLOGY | Chromatin - metabolism | Embryonic Stem Cells - metabolism | Receptors, Estrogen - metabolism | RNA, Messenger - metabolism | Recombinant Fusion Proteins - metabolism | Transfection | Membrane Transport Proteins - genetics | Membrane Transport Proteins - metabolism | Transcription, Genetic | Nuclear Proteins - genetics | Binding Sites | Repressor Proteins - metabolism | Cell Line | Receptors, Estrogen - genetics | Chromosomal Proteins, Non-Histone - metabolism | RNA, Messenger - genetics | Chromatin Assembly and Disassembly | Repressor Proteins - genetics | In Situ Hybridization, Fluorescence | Nuclear Proteins - metabolism | Single Molecule Imaging - methods | Transcription Factors - genetics | GATA1 Transcription Factor - metabolism | Chromosomal Proteins, Non-Histone - genetics | Transcription Factors - metabolism | Animals | Enhancer Elements, Genetic | CCCTC-Binding Factor | CRISPR-Cas Systems | GATA1 Transcription Factor - genetics | Protein Binding | Recombinant Fusion Proteins - genetics | Gene Editing - methods | Mice | Chromatin - genetics | Proteins | Medical colleges | RNA | Genes | Genomics | Fluorescence | Genetic aspects | Genetic transcription | Protein binding | Anopheles | Chromatin | Genomes | Index Medicus
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4. Full Text Systematic mapping of functional enhancer-promoter connections with CRISPR interference
by Fulco, Charles P and Munschauer, Mathias and Anyoha, Rockwell and Munson, Glen and Grossman, Sharon R and Perez, Elizabeth M and Kane, Michael and Cleary, Brian and Lander, Eric S and Engreitz, Jesse M
Science, ISSN 0036-8075, 11/2016, Volume 354, Issue 6313, pp. 769 - 773
Gene expression in mammals is regulated by noncoding elements that can affect physiology and disease, yet the functions and target genes of most noncoding... 
ELEMENTS | CELLS | HUMAN GENOME | GENE | DNA | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | DIFFERENTIATION | BINDING | REVEALS | Cell Proliferation - genetics | Disease - genetics | Humans | Gene Expression Regulation | Promoter Regions, Genetic - genetics | Promoter Regions, Genetic - physiology | Chromosome Mapping - methods | CRISPR-Cas Systems | Clustered Regularly Interspaced Short Palindromic Repeats | GATA1 Transcription Factor - genetics | K562 Cells | Enhancer Elements, Genetic - genetics | Enhancer Elements, Genetic - physiology | Proto-Oncogene Proteins c-myc - genetics | High-Throughput Nucleotide Sequencing - methods | Real-Time Polymerase Chain Reaction | Physiological aspects | Gene expression | Proteins | Index Medicus | Genes | Interference | Genetics | Strategy | Mammals | Screens | Joints
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5. Full Text Ribosome Levels Selectively Regulate Translation and Lineage Commitment in Human Hematopoiesis
by Khajuria, Rajiv K and Munschauer, Mathias and Ulirsch, Jacob C and Fiorini, Claudia and Ludwig, Leif S and McFarland, Sean K and Abdulhay, Nour J and Specht, Harrison and Keshishian, Hasmik and Mani, D.R and Jovanovic, Marko and Ellis, Steven R and Fulco, Charles P and Engreitz, Jesse M and Schütz, Sabina and Lian, John and Gripp, Karen W and Weinberg, Olga K and Pinkus, Geraldine S and Gehrke, Lee and Regev, Aviv and Lander, Eric S and Gazda, Hanna T and Lee, Winston Y and Panse, Vikram G and Carr, Steven A and Sankaran, Vijay G
Cell, ISSN 0092-8674, 03/2018, Volume 173, Issue 1, pp. 90 - 103.e19
Blood cell formation is classically thought to occur through a hierarchical differentiation process, although recent studies have shown that lineage commitment... 
Diamond-Blackfan anemia | hematopoiesis | genetics | translation | ribosome | lineage commitment | erythropoiesis | GATA1 | GENETIC-VARIATION | MESSENGER-RNAS | STEM-CELLS | ERYTHROPOIESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DIAMOND-BLACKFAN ANEMIA | TRANSCRIPTOME ANALYSES | DIFFERENTIATION | IDENTIFICATION | GENOME | CELL BIOLOGY | Humans | Ribosomes - metabolism | Male | Mutation, Missense | Ribosomal Proteins - metabolism | RNA Interference | Apoptosis Regulatory Proteins - genetics | Female | Anemia, Diamond-Blackfan - pathology | Ribosomal Proteins - antagonists & inhibitors | Ribosomal Proteins - genetics | Cells, Cultured | Hematopoietic Stem Cells - metabolism | Transcription Factors - genetics | GATA1 Transcription Factor - metabolism | Apoptosis Regulatory Proteins - metabolism | Transcription Factors - metabolism | Ribosomes - genetics | 5' Untranslated Regions | Anemia, Diamond-Blackfan - genetics | Hematopoietic Stem Cells - cytology | Apoptosis Regulatory Proteins - antagonists & inhibitors | GATA1 Transcription Factor - genetics | Bone Marrow Cells - metabolism | RNA, Small Interfering - metabolism | Proteins | Medical research | Anemia | Analysis | Medical genetics | Medicine, Experimental | Cell differentiation | Genetic translation | Hematopoietic stem cells | Index Medicus
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6. Full Text Computational identification of gene—social environment interaction at the human IL6 locus
by Steven W. Cole and Jesusa M. G. Arevalo and Rie Takahashi and Erica K. Sloan and Susan K. Lutgendorf and Anil K. Sood and John F. Sheridan and Teresa E. Seeman and Bruce S. McEwen
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2010, Volume 107, Issue 12, pp. 5681 - 5686
To identify genetic factors that interact with social environments to impact human health, we used a bioinformatic strategy that couples expression array—based... 
Social interaction | B lymphocytes | Genetic variation | Genes | Mortality | Alleles | Environmental disorders | Norepinephrine | Bioinformatics | Depressive disorders | Inflammation | Transcription | Social epidemiology | Stress | Gene-environment interaction | C-REACTIVE PROTEIN | REGULATORY SNPS | YOUNG-ADULTS | social epidemiology | stress | TH2 CELLS | transcription | MULTIDISCIPLINARY SCIENCES | gene-environment interaction | RAT HEARTS | inflammation | LIFE STRESS | ARTERY RISK DEVELOPMENT | EXPRESSION | SOCIOECONOMIC-STATUS | Promoter Regions, Genetic | RNA, Small Interfering - genetics | Interleukin-6 - genetics | Oligonucleotide Array Sequence Analysis | Humans | Transcriptional Activation | Computational Biology | Social Environment | Transcription Factors - genetics | DNA - genetics | Sympathetic Nervous System - physiology | Molecular Epidemiology | Base Sequence | GATA1 Transcription Factor - genetics | Stress, Psychological | GATA1 Transcription Factor - antagonists & inhibitors | Models, Genetic | Polymorphism, Single Nucleotide | Usage | Transcription factors | Physiological aspects | Genetic aspects | Research | Risk factors | Computational biology | Interleukin-6 | Proteins | Gene loci | Gene expression | Polymorphism | Index Medicus | Biological Sciences | gene–environment interaction
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7. Full Text Exome sequencing identifies GATA1 mutations resulting in Diamond-Blackfan anemia
by Sankaran, Vijay G and Ghazvinian, Roxanne and Do, Ron and Thiru, Prathapan and Vergilio, Jo-Anne and Beggs, Alan H and Sieff, Colin A and Orkin, Stuart H and Nathan, David G and Lander, Eric S and Gazda, Hanna T
Journal of Clinical Investigation, ISSN 0021-9738, 07/2012, Volume 122, Issue 7, pp. 2439 - 2443
Diamond-Blackfan anemia (DBA) is a hypoplastic anemia characterized by impaired production of red blood cells, with approximately half of all cases attributed... 
MEDICINE, RESEARCH & EXPERIMENTAL | PROTEIN | GENE DELETIONS | INHERITED MUTATION | ERYTHROPOIESIS | DOWN-SYNDROME | LEUKEMIA | EXPRESSION | Sequence Deletion | Genetic Association Studies | Humans | Male | GATA1 Transcription Factor - metabolism | Case-Control Studies | Exome | Young Adult | Anemia, Diamond-Blackfan - genetics | DNA Mutational Analysis | Hematopoiesis | Base Sequence | GATA1 Transcription Factor - genetics | Real-Time Polymerase Chain Reaction | Causes of | Genetic aspects | Research | Genetic transcription | Gene mutations | Anemia | Index Medicus | Abridged Index Medicus | Brief Report
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8. Full Text Downregulation of GATA1 drives impaired hematopoiesis in primary myelofibrosis
by Gilles, Laure and Arslan, Ahmet Dirim and Marinaccio, Christian and Wen, Qiang Jeremy and Arya, Priyanka and McNulty, Maureen and Yang, Qiong and Zhao, Jonathan C and Konstantinoff, Katerina and Lasho, Terra and Pardanani, Animesh and Stein, Brady and Plo, Isabelle and Sundaravel, Sriram and Wickrema, Amittha and Migliaccio, Annarita and Gurbuxani, Sandeep and Vainchenker, William and Platanias, Leonidas C and Tefferi, Ayalew and Crispino, John D
Journal of Clinical Investigation, ISSN 0021-9738, 04/2017, Volume 127, Issue 4, pp. 1316 - 1320
Primary myelofibrosis (PMF) is a clonal hematologic malignancy characterized by BM fibrosis, extramedullary hematopoiesis, circulating CD34+ cells,... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | RIBOSOMOPATHIES | MULTICENTER | GENE | IDIOPATHIC MYELOFIBROSIS | QUIESCENCE | MEGAKARYOCYTES | LENALIDOMIDE | THROMBOPOIETIN | STEM | Chromosome Deletion | Thrombopoiesis | Ribosomal Proteins - genetics | Down-Regulation | Humans | Ribosomal Proteins - biosynthesis | Chromosomes, Human, Pair 5 - metabolism | Primary Myelofibrosis - pathology | Megakaryocytes - pathology | Primary Myelofibrosis - genetics | Animals | GATA1 Transcription Factor - biosynthesis | GATA1 Transcription Factor - genetics | Mice | Megakaryocytes - metabolism | Primary Myelofibrosis - metabolism | Chromosomes, Human, Pair 5 - genetics | Care and treatment | Transcription factors | Development and progression | Genetic aspects | Gene expression | Health aspects | Myeloproliferative disorders | Proteins | Medical research | Morphology | Cell cycle | Writing | Mutation | Kinases | Patients | Defects | Index Medicus | Abridged Index Medicus
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9. Full Text The landscape of somatic mutations in Down syndrome-related myeloid disorders
by Yoshida, Kenichi and Toki, Tsutomu and Okuno, Yusuke and Kanezaki, Rika and Shiraishi, Yuichi and Sato-Otsubo, Aiko and Sanada, Masashi and Park, Myoung-Ja and Terui, Kiminori and Suzuki, Hiromichi and Kon, Ayana and Nagata, Yasunobu and Sato, Yusuke and Wang, RuNan and Shiba, Norio and Chiba, Kenichi and Tanaka, Hiroko and Hama, Asahito and Muramatsu, Hideki and Hasegawa, Daisuke and Nakamura, Kazuhiro and Kanegane, Hirokazu and Tsukamoto, Keiko and Adachi, Souichi and Kawakami, Kiyoshi and Kato, Koji and Nishimura, Ryosei and Izraeli, Shai and Hayashi, Yasuhide and Miyano, Satoru and Kojima, Seiji and Ito, Etsuro and Ogawa, Seishi
Nature Genetics, ISSN 1061-4036, 11/2013, Volume 45, Issue 11, pp. 1293 - 1301
Transient abnormal myelopoiesis (TAM) is a myeloid proliferation resembling acute megakaryoblastic leukemia (AMKL), mostly affecting perinatal infants with... 
TRANSIENT MYELOPROLIFERATIVE DISORDER | 17Q21.31 MICRODELETION SYNDROME | ACUTE LYMPHOBLASTIC-LEUKEMIA | DOWN-SYNDROME | GENETICS & HEREDITY | ACUTE MEGAKARYOBLASTIC LEUKEMIA | GENE-EXPRESSION | COHESIN | KANSL1 CAUSE | SEQUENCING DATA | HUMAN CANCER | Cell Proliferation | Polycomb Repressive Complex 2 - genetics | Humans | Repressor Proteins - genetics | Gene Expression Profiling | Chromosomes, Human, Pair 21 - genetics | Myeloproliferative Disorders - genetics | Sequence Analysis, DNA | Enhancer of Zeste Homolog 2 Protein | Chromosomal Proteins, Non-Histone - genetics | Myeloid Cells | Down Syndrome - immunology | Leukemoid Reaction - genetics | Base Sequence | CCCTC-Binding Factor | Down Syndrome - genetics | GATA1 Transcription Factor - genetics | Cell Cycle Proteins - genetics | Leukemia, Megakaryoblastic, Acute - genetics | Nuclear Proteins - genetics | Development and progression | Genetic aspects | Down syndrome | Gene mutations | Identification and classification | Myelodysplastic syndromes | Genes | Technological change | Science | Epigenetics | Genomes | Mutation | Cancer | Index Medicus
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10. Full Text C/EBPβ enhances platinum resistance of ovarian cancer cells by reprogramming H3K79 methylation
by Liu, Dan and Zhang, Xiao-Xue and Li, Meng-Chen and Cao, Can-Hui and Wan, Dong-Yi and Xi, Bi-Xin and Tan, Jia-Hong and Wang, Ji and Yang, Zong-Yuan and Feng, Xin-Xia and Ye, Fei and Chen, Gang and Wu, Peng and Xi, Ling and Wang, Hui and Zhou, Jian-Feng and Feng, Zuo-Hua and Ma, Ding and Gao, Qing-Lei
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1739 - 13
Chemoresistance is a major unmet clinical obstacle in ovarian cancer treatment. Epigenetics plays a pivotal role in regulating the malignant phenotype, and has... 
HISTONE LYSINE METHYLATION | DIVERSE FUNCTIONS | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | GENE-EXPRESSION | TUMOR | DRUG-RESISTANCE | ACQUIRED CISPLATIN RESISTANCE | CARCINOMA | CHEMOTHERAPY | MLL-REARRANGED LEUKEMIA | Chromatin - metabolism | Epithelial Cells - metabolism | Prognosis | Epigenesis, Genetic | Epithelial Cells - drug effects | Humans | Methyltransferases - metabolism | Methyltransferases - genetics | Ovarian Neoplasms - pathology | Ovarian Neoplasms - mortality | Interferon Regulatory Factor-3 - genetics | Ovarian Neoplasms - genetics | Biomarkers, Tumor - metabolism | Female | Antineoplastic Agents - pharmacology | Nuclear Proteins - genetics | Ovarian Neoplasms - drug therapy | Chromatin - chemistry | Databases, Factual | GATA4 Transcription Factor - metabolism | CCAAT-Enhancer-Binding Protein-beta - genetics | Epithelial Cells - pathology | GATA4 Transcription Factor - genetics | Nuclear Proteins - metabolism | Cisplatin - pharmacology | Transcription Factors - genetics | GATA1 Transcription Factor - metabolism | CCAAT-Enhancer-Binding Protein-beta - metabolism | Transcription Factors - metabolism | Drug Resistance, Neoplasm - genetics | Histones - genetics | GATA1 Transcription Factor - genetics | Survival Analysis | Cell Line, Tumor | Interferon Regulatory Factor-3 - metabolism | Biomarkers, Tumor - genetics | Histones - metabolism | Primary Cell Culture | Methylation | Regulators | Phenotypes | Chromatin | Transcription factors | Ovarian carcinoma | Tumor cells | Genes | Methyltransferase | Chemoresistance | Drug resistance | Modulators | Ovarian cancer | GATA-1 protein | Platinum | Epigenetics | DNA methylation | Cancer | Tumors | Index Medicus
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11. Full Text The transcription factor Ehf is involved in TGF-β-induced suppression of FcεRI and c-kit expression and FcεRI-mediated activation in mast cells
by Yamazaki, Susumu and Nakano, Nobuhiro and Honjo, Asuka and Hara, Mutsuko and Maeda, Keiko and Nishiyama, Chiharu and Kitaura, Jiro and Ohtsuka, Yoshikazu and Okumura, Ko and Ogawa, Hideoki and Shimizu, Toshiaki
Journal of Immunology, ISSN 0022-1767, 10/2015, Volume 195, Issue 7, pp. 3427 - 3435
Fc epsilon RI, which is composed of alpha, beta, and gamma subunits, plays an important role in IgE-mediated allergic responses. TGF-beta 1 has been reported... 
DENDRITIC CELLS | MECHANISM | FACTOR ESE-3 | BONE-MARROW | DIFFERENTIATION | IMMUNOLOGY | PROMOTER | TRANSFORMING GROWTH FACTOR-BETA | UP-REGULATION | CHAIN GENE-EXPRESSION | TGF-BETA-1 | STAT5 Transcription Factor - biosynthesis | Transcriptional Activation | Transforming Growth Factor beta1 - metabolism | Proto-Oncogene Proteins c-kit - biosynthesis | RNA, Messenger - metabolism | Promoter Regions, Genetic - genetics | STAT5 Transcription Factor - genetics | Signal Transduction - immunology | GATA2 Transcription Factor - biosynthesis | Immunoglobulin E - immunology | RNA Interference | Proto-Oncogene Proteins c-kit - genetics | Receptors, IgE - immunology | Transcription Factors - immunology | GATA2 Transcription Factor - genetics | Mast Cells - immunology | Receptors, IgE - biosynthesis | Cells, Cultured | Transcription Factors - biosynthesis | Cell Degranulation - immunology | Animals | Bone Marrow Cells | GATA1 Transcription Factor - biosynthesis | GATA1 Transcription Factor - genetics | Mice | Mice, Inbred BALB C | RNA, Small Interfering | Transcription, Genetic - genetics | Smad Proteins - metabolism | Cytokines - biosynthesis | Index Medicus | Abridged Index Medicus
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12. Full Text Exchange of GATA Factors Mediates Transitions in Looped Chromatin Organization at a Developmentally Regulated Gene Locus
by Jing, Huie and Vakoc, Christopher R and Ying, Lei and Mandat, Sean and Wang, Hongxin and Zheng, Xingwu and Blobel, Gerd A
Molecular Cell, ISSN 1097-2765, 2008, Volume 29, Issue 2, pp. 232 - 242
Enhancers can regulate designate promoters over long distances by forming chromatin loops. Whether chromatin loops are lost or reconfigured during gene... 
DNA | CONTROL REGION | TRANSCRIPTION FACTOR GATA-1 | BIOCHEMISTRY & MOLECULAR BIOLOGY | ZINC-FINGER PROTEIN | C-KIT | CHROMOSOME CONFORMATION | CELL-PROLIFERATION | DIFFERENTIATION | ERYTHROID MATURATION | FOG-1 | KIT EXPRESSION | CELL BIOLOGY | Cell Line | Chromatin - metabolism | Quantitative Trait Loci - physiology | Nuclear Proteins - metabolism | Transcription Factors - genetics | Proto-Oncogene Proteins c-kit - biosynthesis | GATA1 Transcription Factor - metabolism |