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Investigative ophthalmology & visual science, ISSN 0146-0404, 10/2018, Volume 59, Issue 12, pp. 5060 - 5066
PURPOSE. The aim of the study was to investigate the signaling of growth hormone-releasing hormone receptor (GHRH-R) in the pathogenesis of pterygium and... 
GHRH-R signaling | GHRH-R antagonist | histopathology | TUMOR | apoptosis | CANCER | PATHOGENESIS | pterygium | RETINOBLASTOMA | INVASION | EPITHELIAL-CELLS | INFLAMMATION | GHRH | MDM2 | OPHTHALMOLOGY | EXPRESSION
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2018, Volume 115, Issue 47, pp. 12028 - 12033
The effects of the growth hormone-releasing hormone (GHRH) agonist MR409 on various human cancer cells were investigated. In H446 small cell lung cancer (SCLC)... 
Signaling pathways | GHRH receptors | GHRH antagonists | GHRH agonists | Lung cancers | VITRO | MULTIDISCIPLINARY SCIENCES | lung cancers | PROLIFERATION | SPLICE VARIANTS | MESENCHYMAL STEM-CELLS | CHEMOTHERAPY | signaling pathways | PANCREATIC-ISLETS | BIOLOGICAL EVALUATION | TUMOR-GROWTH | EXPRESSION | ANTAGONISTIC ANALOGS | Biological Sciences
Journal Article
Oncotarget, ISSN 1949-2553, 09/2018, Volume 9, Issue 69, pp. 33054 - 33055
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2016, Volume 113, Issue 51, pp. 14745 - 14750
Journal Article
POLISH ARCHIVES OF INTERNAL MEDICINE-POLSKIE ARCHIWUM MEDYCYNY WEWNETRZNEJ, ISSN 0032-3772, 03/2019, Volume 129, Issue 3, pp. 208 - 210
Journal Article
Experimental Eye Research, ISSN 0014-4835, 04/2019, Volume 181, pp. 277 - 284
The receptor for growth hormone-releasing hormone (GHRH-R) has been shown to upregulate specifically in the ciliary and iris epithelial cells and infiltrating... 
Eye | Ciliary body | Iris | GHRH-R | Leukocyte | Uveitis | RATS | ENDOTOXIN-INDUCED UVEITIS | INTERLEUKIN-6 | ADHESION | GHRH | OPHTHALMOLOGY | ANTAGONISTS | EXPRESSION | BODY | Immunohistochemistry | Somatotropin | Cytokines | Somatotropin releasing hormone | Analysis | Inflammation | Mitogens
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2016, Volume 113, Issue 50, pp. 14396 - 14401
Journal Article
APPLIED IMMUNOHISTOCHEMISTRY & MOLECULAR MORPHOLOGY, ISSN 1541-2016, 01/2018, Volume 26, Issue 1, pp. 1 - 5
Purpose:Growth hormone-releasing hormone (GHRH) has been shown to modify the growth behavior of many cancers, including breast. GHRH is produced by tumor... 
triple-negative breast cancer | SUBTYPES | MEDULLARY CARCINOMA | ANATOMY & MORPHOLOGY | RECEPTOR | CLINICAL-FEATURES | PATHOLOGY | BASAL-LIKE | HORMONE-RELEASING-HORMONE | THERAPY | GHRH | GROWTH | histologic types of triple-negative breast cancer | GHRH-R | GHRH-R antagonists | MEDICAL LABORATORY TECHNOLOGY | ANTAGONISTS | METAPLASTIC CARCINOMAS
Journal Article
Journal Article
International Journal of Cancer, ISSN 0020-7136, 06/2018, Volume 142, Issue 11, pp. 2394 - 2404
We investigated the effects of novel antagonists of growth hormone releasing hormone (GHRH)—MIA602 and MIA690—on three human small cell lung cancer (SCLC)... 
SCLC | GHRH receptors | signaling pathways | NSCLC | GHRH antagonists | RECEPTOR | PROLIFERATION | SUPPRESSION | BOMBESIN | ONCOLOGY | GHRH | IN-VIVO | PROSTATE-CANCER | TUMOR-GROWTH | CARCINOMA | EXPRESSION | Apoptosis - drug effects | Humans | Small Cell Lung Carcinoma - drug therapy | Small Cell Lung Carcinoma - metabolism | Sermorelin - pharmacology | Sermorelin - analogs & derivatives | Female | Antineoplastic Agents - pharmacology | Cyclic AMP - metabolism | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Cell Survival - drug effects | Gene Expression | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Small Cell Lung Carcinoma - genetics | Growth Hormone-Releasing Hormone - genetics | Xenograft Model Antitumor Assays | Growth Hormone-Releasing Hormone - metabolism | Animals | Signal Transduction - drug effects | Mice, Nude | Cyclic AMP Response Element-Binding Protein - metabolism | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Carcinoma, Non-Small-Cell Lung - drug therapy | Cell Cycle - drug effects | Growth Hormone-Releasing Hormone - antagonists & inhibitors | Cell Movement | Somatotropin | Somatotropin releasing hormone | Lung cancer, Non-small cell | Analysis | Protein binding | Phosphorylation | Animal models | Transcription | Lung cancer | Cyclic AMP | Non-small cell lung carcinoma | Antagonists | Kinases | E-cadherin | Cell adhesion & migration | Signaling | Growth hormone-releasing hormone | Lungs | Pituitary | Animal tissues | Cyclin-dependent kinase inhibitor p27 | Growth hormones | Inhibition | Growth hormone | Viability | Cell migration | Tumors | Cancer | Apoptosis
Journal Article
The Prostate, ISSN 0270-4137, 09/2018, Volume 78, Issue 12, pp. 915 - 926
Background Therapeutic strategies should be designed to transform aggressive prostate cancer phenotypes to a chronic situation. To evaluate the effects of the... 
antagonists | GHRH‐R | prostate cancer | cell proliferation | angiogenesis | cell adhesion | GHRH-R | HUMAN BREAST | CELL-ADHESION | E-CADHERIN | MESENCHYMAL TRANSITION | BETA-CATENIN | SPLICE VARIANTS | P21 | INHIBITION | GHRH | ENDOCRINOLOGY & METABOLISM | UROLOGY & NEPHROLOGY | EXPRESSION | Cell Survival - drug effects | Prostatic Neoplasms - pathology | PC-3 Cells | Apoptosis - drug effects | beta Catenin - analysis | Humans | Receptors, Neuropeptide - antagonists & inhibitors | Male | Vascular Endothelial Growth Factor A - analysis | Resting Phase, Cell Cycle | Vascular Endothelial Growth Factor A - metabolism | Cell Adhesion - drug effects | Sermorelin - pharmacology | Cell Line, Tumor | Sermorelin - analogs & derivatives | Cell Proliferation - drug effects | Cell Cycle - drug effects | Receptors, Pituitary Hormone-Regulating Hormone - antagonists & inhibitors | Wound Healing - drug effects | Prostatic Neoplasms - drug therapy | Cell proliferation | Phenotypes | Bax protein | p53 Protein | Malignancy | E-cadherin | Gelatinase B | Cell adhesion & migration | Gelatinase A | Angiogenesis | Growth hormone-releasing hormone | Cell lines | Cell adhesion | Proliferating cell nuclear antigen | Growth hormones | Growth hormone | Vascular endothelial growth factor | Prostate cancer | Prostate | Cell migration | Apoptosis
Journal Article
JOURNAL OF PHYSICAL CHEMISTRY B, ISSN 1520-6106, 07/2019, Volume 123, Issue 29, pp. 6169 - 6177
Recently, we proposed a high-throughput screening workflow for the elucidation of agonistic or antagonistic growth hormone-releasing hormone (GHRH) potencies... 
SEPARATIONS | TEMPERATURE | GHRH | GAS | DYNAMICS | CHEMISTRY, PHYSICAL | SHIFT-REAGENTS | PROLIFERATION | ANTAGONISTS | EXCHANGE | SOLVENT
Journal Article
Journal Article