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Glia, ISSN 0894-1491, 12/2015, Volume 63, Issue 12, pp. 2340 - 2361
Journal Article
Neuroscience Research, ISSN 0168-0102, 01/2018, Volume 126, pp. 39 - 43
After traumatic injuries of the central nervous system (CNS), including spinal cord injury (SCI), astrocytes surrounding the lesion become reactive and... 
Glial scar | Spinal cord injury | Astrocytes | Laser microdissection | Axonal regeneration | FUNCTIONAL RECOVERY | CELLS | REGENERATION | TISSUE | STAT3 | CNS | NEUROSCIENCES | Animals | Spinal Cord Injuries - metabolism | Cicatrix - physiopathology | Gliosis - metabolism | Humans | Nerve Regeneration | Astrocytes - metabolism | Spinal Cord Injuries - pathology
Journal Article
Journal Article
JOURNAL OF NEUROINFLAMMATION, ISSN 1742-2094, 09/2017, Volume 14
Background: Neuroinflammation involves cytokine release, astrocyte reactivity and migration. Neuronal Thy-1 promotes DITNC1 astrocyte migration by engaging... 
MICROGLIAL ACTIVATION | GLIAL SCAR | Reactive astrocytes | Amyotrophic lateral sclerosis | Inflammation | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | NEUROSCIENCES | TNF-ALPHA | ATP RELEASE | UP-REGULATION | EXPRESSION | Cell migration | BETA INTEGRIN | BRAIN | Integrins | TRIGGER MOTONEURON DEATH
Journal Article
Glia, ISSN 0894-1491, 09/2018, Volume 66, Issue 9, pp. 1915 - 1928
The transactivator of transcription protein, HIV‐1 Tat, is linked to neuroAIDS, where degeneration of dopamine neurons occurs. Using a mouse model expressing... 
neuroAIDS | dopamine neurons | HIV‐1 Tat | microglia | microglia‐neuron interactions | HIV-1 Tat | microglia-neuron interactions | TYROSINE-HYDROXYLASE | BASAL GANGLIA | IMMUNE-DEFICIENCY-SYNDROME | SUBSTANTIA-NIGRA | NEUROSCIENCES | NEURONAL DAMAGE | VENTRAL TEGMENTAL AREA | IMMUNODEFICIENCY-VIRUS TYPE-1 | GENE-EXPRESSION | PARS COMPACTA | PARKINSONS-DISEASE | Calcium-Binding Proteins - metabolism | HIV-1 | Microglia - metabolism | Tyrosine 3-Monooxygenase - metabolism | Synaptic Transmission - physiology | Mice, Inbred C57BL | Cells, Cultured | Dopaminergic Neurons - virology | Brain - virology | Male | Mice, Transgenic | Nuclear Proteins - metabolism | RNA, Messenger - metabolism | Brain - metabolism | Nerve Tissue Proteins - metabolism | Animals | Dopaminergic Neurons - metabolism | Microglia - virology | Brain - pathology | Microglia - pathology | Microfilament Proteins - metabolism | Dopamine - metabolism | tat Gene Products, Human Immunodeficiency Virus - metabolism | Tyrosine | Brain | Nervous system diseases | Neurons | Analysis | Phenols | HIV (Viruses) | Immunohistochemistry | Ventral tegmentum | Phenotypes | Firing pattern | Dopamine | Transcription | Substantia nigra | Glial fibrillary acidic protein | Hydroxylase | Activation | Exposure | Tyrosine 3-monooxygenase | Microglia | Proteins | Tat protein | Side effects | Neurodegeneration | Human immunodeficiency virus--HIV | Doxycycline | Degeneration | Dopamine receptors
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 3/2018, Volume 55, Issue 3, pp. 1966 - 1976
Journal Article