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Pain, ISSN 0304-3959, 12/2014, Volume 155, Issue 12, pp. 2618 - 2629
Journal Article
GLIA, ISSN 0894-1491, 08/2017, Volume 65, Issue 8, p. 1251
Activation of microglia resulting in exacerbated inflammation expression plays an important role in degeneration of dopaminergic (DA) neurons in the... 
Inflammation | Tumor necrosis factor | Neurons | Analysis | Parkinson's disease | Pathogenesis | Oct-4 protein | Activation | MPP | Neuronal-glial interactions | Neurodegeneration | Rodents | Tumor necrosis factor-TNF | Degeneration | Damage | Movement disorders | Dopamine receptors | Organic cation transporter | Dopamine | Neurodegenerative diseases | MiRNA | Microglia | Mice | Cations | MPTP | Transporter
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 01/2016, Volume 13, Issue 1, p. 19
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 04/2010, Volume 31, Issue 4, pp. 665 - 677
Extracellular S100B is known to affect astrocytic, neuronal and microglial activities, with different effects depending on its concentration. Whereas at... 
NF-κB | COX-2 | TNF-α | RAGE | IL-1β | S100B | AP-1 | Microglia | GLIAL ACTIVATION | TNF-alpha | ALZHEIMERS-DISEASE | P38 MAPK | IL-1 beta | NEUROSCIENCES | GERIATRICS & GERONTOLOGY | RAGE ACTIVATION | NF-kappa B | NEURITE OUTGROWTH | CYCLOOXYGENASE-2 EXPRESSION | CULTURED ASTROCYTES | GROWTH-FACTOR | GLYCATION END-PRODUCTS | MYOBLAST PROLIFERATION
Journal Article
Journal Article
Molecular Pain, ISSN 1744-8069, 5/2011, Volume 7, Issue 1, pp. 40 - 40
Background: HIV-associated sensory neuropathy (HIV-SN) is one of the most common forms of peripheral neuropathy, affecting about 30% of people with acquired... 
Glia | HIV pain | TNFα | ACQUIRED-IMMUNODEFICIENCY-SYNDROME | MECHANICAL ALLODYNIA | NERVOUS-SYSTEM | TNF alpha | ENVELOPE GLYCOPROTEIN | NECROSIS-FACTOR-ALPHA | glia | CYTOKINE EXPRESSION | NEUROSCIENCES | SENSORY NEURONS | IMMUNE-RESPONSES | INFLAMMATORY PAIN | TRANSGENIC RAT | Immunohistochemistry | Tumor Necrosis Factor-alpha - metabolism | Glial Fibrillary Acidic Protein - genetics | Posterior Horn Cells - metabolism | Spinal Cord - metabolism | Neuroglia - pathology | Tumor Necrosis Factor-alpha - genetics | Neuralgia - metabolism | Male | Neuralgia - pathology | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Gene Knockdown Techniques | Hyperalgesia - pathology | Time Factors | Spinal Cord - pathology | Microfilament Proteins - metabolism | Microfilament Proteins - genetics | Calcium-Binding Proteins - metabolism | Receptors, Tumor Necrosis Factor - metabolism | Hyperalgesia - metabolism | RNA, Messenger - genetics | Solubility | Rats | Up-Regulation - genetics | HIV Envelope Protein gp120 - administration & dosage | Rats, Sprague-Dawley | Protein Transport | Animals | Ganglia, Spinal - pathology | Posterior Horn Cells - pathology | Neuroglia - metabolism | Nociceptors - metabolism | Nociceptors - drug effects | Calcium-Binding Proteins - genetics | Ganglia, Spinal - metabolism | RNA, Small Interfering - metabolism
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 02/2011, Volume 36, Issue 3, pp. 664 - 676
Tolerance and dependence are common complications of long-term treatment of pain with opioids, which substantially limit the long-term use of these drugs. The... 
morphine withdrawal | gene therapy | TNFa | PAG | soluble TNF receptor | TNF alpha | PSYCHIATRY | PHYSICAL-DEPENDENCE | SYNAPTIC-TRANSMISSION | ELEMENT-BINDING PROTEIN | MEDIATED GENE-TRANSFER | OPIATE WITHDRAWAL | FOS-LIKE IMMUNOREACTIVITY | NEUROSCIENCES | NUCLEUS-ACCUMBENS | NEUROPATHIC PAIN | GLUTAMATE TRANSPORTERS | PHARMACOLOGY & PHARMACY | OPIOID WITHDRAWAL | Enzyme-Linked Immunosorbent Assay - methods | Oncogene Proteins v-fos - metabolism | Tumor Necrosis Factor-alpha - metabolism | Transduction, Genetic | Substance Withdrawal Syndrome - etiology | Microinjections - methods | Periaqueductal Gray - drug effects | Rats | Substance Withdrawal Syndrome - drug therapy | Male | Naloxone - therapeutic use | Green Fluorescent Proteins - genetics | Extracellular Signal-Regulated MAP Kinases - metabolism | Glial Fibrillary Acidic Protein - metabolism | Rats, Sprague-Dawley | Periaqueductal Gray - metabolism | Substance Withdrawal Syndrome - pathology | Behavior, Animal | Gene Expression Regulation - drug effects | CREB-Binding Protein - metabolism | Animals | Narcotic Antagonists - therapeutic use | Phosphorylation - drug effects | Morphine - adverse effects | Tumor necrosis factor receptors | Phosphorylation | Cytokines | Mesencephalon | Astrocytes | Glial fibrillary acidic protein | Periaqueductal gray area | Extracellular signal-regulated kinase | Morphine | Inflammation | Regulatory sequences | Cyclic AMP response element-binding protein | Microinjection | Naloxone | Fos protein | Drug tolerance | Opioids | Immunoreactivity | Tumor necrosis factor- alpha | TNFα | behavioral science | withdrawal | TNF | morphine | addiction & substance abuse | neuropharmacology | Original | opioids
Journal Article
Journal of Neuroimmunology, ISSN 0165-5728, 2014, Volume 276, Issue 1, pp. 104 - 111
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 1, p. e16100
There is increasing evidence that soluble factors in inflammatory central nervous system diseases not only regulate the inflammatory process but also directly... 
NERVOUS-SYSTEM | TUMOR-NECROSIS-FACTOR | ACTIVATED T-CELLS | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION FACTOR NFAT1 | CALCINEURIN | DEATH | NUCLEAR FACTOR | UP-REGULATION | EXPRESSION | ASTROCYTES | Tumor Necrosis Factor-alpha - pharmacology | Apoptosis - drug effects | Microarray Analysis | Humans | NFATC Transcription Factors - metabolism | Caspase 3 - metabolism | Cell Line, Tumor | Transcription, Genetic | Active Transport, Cell Nucleus | Fas Ligand Protein - genetics | Calcineurin - metabolism | Neuroblastoma - pathology | Cytochrome | Multiple sclerosis | Transcription factors | Transcription | Calcium | Calcineurin | Central nervous system | Medical services | Nervous system | Activation | Neuroblastoma | Caspase-3 | Neuronal-glial interactions | Proteins | Cell activation | Acquired immune deficiency syndrome--AIDS | Lymphocytes | Dementia disorders | Tumor necrosis factor-TNF | Physiology | Alzheimer's disease | Vector-borne diseases | Translocation | Nervous system diseases | Stroke | Malaria | Neurodegenerative diseases | Neurons | Astrocytes | Mortality | Central nervous system diseases | Caspase | Gene expression | Apolipoproteins | Diseases | Neurological diseases | Cytochrome c | Lipofectin | DNA microarrays | FasL protein | Cell death | Caspase-9 | Ligands | Viability | Apoptosis | Acquired immune deficiency syndrome | AIDS
Journal Article