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Neuro-oncology (Charlottesville, Va.), ISSN 1523-5866, 2017, Volume 19, Issue 11, pp. 1460 - 1468
Journal Article
International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 10/2017, Volume 99, Issue 2, pp. E65 - E65
Journal Article
International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 10/2017, Volume 99, Issue 2, pp. E617 - E617
Journal Article
Biophysical Journal, ISSN 0006-3495, 02/2017, Volume 112, Issue 3, pp. 337a - 338a
Journal Article
Journal Article
Brain pathology (Zurich, Switzerland), ISSN 1015-6305, 2016, Volume 26, Issue 5, pp. 569 - 580
Somatic mutations of the H3F3A and HIST1H3B genes encoding the histone H3 variants, H3.3 and H3.1, were recently identified in high‐grade gliomas arising in... 
glioblastoma | astrocytoma | diffuse midline glioma | H3F3A | K27M mutation | histone H3.3 | DIPG | histone H3.1 | HIST1H3B | diffuse intrinsic pontine glioma | Diffuse intrinsic pontine glioma | Glioblastoma | Histone H3.1 | Diffuse midline glioma | Histone H3.3 | Astrocytoma | histone H3 | INTRINSIC PONTINE GLIOMA | ACTIVATING ACVR1 MUTATIONS | MOLECULAR CHARACTERISTICS | SUBGROUPS | PATHOLOGY | BRAIN-STEM GLIOMAS | PEDIATRIC GLIOBLASTOMA | NEUROSCIENCES | CLINICAL NEUROLOGY | GLIONEURONAL TUMORS | H3F3A K27M MUTATIONS | HIGH-FREQUENCY | HIGH-GRADE ASTROCYTOMAS | Receptor, Epidermal Growth Factor - genetics | Humans | Middle Aged | Brain Neoplasms - pathology | Child, Preschool | Male | X-linked Nuclear Protein - metabolism | Brain Neoplasms - metabolism | Tumor Suppressor Protein p53 - genetics | Glioma - metabolism | Glioma - genetics | Young Adult | Receptor, Epidermal Growth Factor - metabolism | Glioma - pathology | Adult | Female | Child | Gene Expression Regulation, Neoplastic - genetics | Glycine - genetics | PTEN Phosphohydrolase - genetics | Genetic Association Studies | Tumor Suppressor Protein p53 - metabolism | Brain Neoplasms - genetics | PTEN Phosphohydrolase - metabolism | Isocitrate Dehydrogenase - genetics | Mutation - genetics | Histones - genetics | Adolescent | Isocitrate Dehydrogenase - metabolism | Aged | Methionine - genetics | Tumor proteins | Gliomas | Genetic aspects | Spinal cord | Mutation | Rodents | Tumors
Journal Article
Cancer cell, ISSN 1535-6108, 2018, Volume 33, Issue 5, pp. 829 - 842.e5
Journal Article
Acta neuropathologica, ISSN 1432-0533, 2018, Volume 135, Issue 4, pp. 601 - 615
textabstractThe optimal treatment for patients with low-grade glioma (LGG) WHO grade II remains controversial. Overall survival ranges from 2 to over 15 years... 
TMZ | DNA methylation | DDR genes | MGMT-STP27 | Randomized trial | Low-grade glioma | Pathology | Neurosciences | Medicine & Public Health | CELLS | METHYLATION | MGMT | SENSITIVITY | MISMATCH REPAIR | PATHOLOGY | TUMORS | NEUROSCIENCES | CLINICAL NEUROLOGY | TEMOZOLOMIDE | GLIOBLASTOMA | RADIOTHERAPY | EXPRESSION | Epigenesis, Genetic | Humans | Brain Neoplasms - pathology | DNA Repair Enzymes - genetics | Male | Glioma - genetics | Temozolomide - therapeutic use | Neoplasm Grading | DNA Methylation - radiation effects | Glioma - pathology | Tumor Suppressor Proteins - genetics | Glioma - therapy | Adult | Female | Promoter Regions, Genetic | Discoidin Domain Receptors - genetics | Brain Neoplasms - genetics | Isocitrate Dehydrogenase - genetics | Treatment Outcome | Antineoplastic Agents, Alkylating - therapeutic use | DNA Modification Methylases - genetics | DNA | Progression-Free Survival | Brain Neoplasms - therapy | CpG Islands | DNA Methylation - drug effects | Medical colleges | Medical research | Care and treatment | DNA damage | Genes | Medicine, Experimental | Genetic aspects | Radiotherapy | Methylation | Cancer | Preservation | Brain tumors | Genotoxicity | Cognitive ability | Genomes | Radiation therapy | Survival | Quality of life | Glioma | Temozolomide | Isocitrate dehydrogenase | Deoxyribonucleic acid--DNA | CpG islands | Original Paper
Journal Article