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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2010, Volume 107, Issue 42, pp. 18143 - 18148
Effective treatment of brain neurological disorders such as Alzheimer’s disease, multiple sclerosis, or tumors should be possible with drug delivery through... 
Glioma | Antineoplastics | Brain neoplasms | Cell membranes | Drug design | Liposomes | Polymers | Blood brain barrier | Tumors | Endosomes | Glioma treatment | Blood-brain barrier | Laminin-411 | Endosomal escape | Polymalic acid | blood-brain barrier | laminin-411 | GENE DELIVERY | ANGIOGENESIS | PERMEABILITY | MULTIDISCIPLINARY SCIENCES | BARRIER | endosomal escape | glioma treatment | PEPTIDE | CHEMOTHERAPY | LAMININ-8 | polymalic acid | IN-VIVO | MALIGNANT GLIOMAS | HPMA COPOLYMER | Polymers - administration & dosage | Brain Neoplasms - pathology | Brain Neoplasms - drug therapy | Endosomes - metabolism | Nanoparticles | Polymers - therapeutic use | Animals | Mice, Nude | Malates - pharmacokinetics | Polymers - pharmacokinetics | Mice | Infusions, Intravenous | Malates - administration & dosage | Malates - therapeutic use | Blood-Brain Barrier | Hydrogen-Ion Concentration | Drugs | Care and treatment | Drug delivery systems | Brain tumors | Malic acid | Dosage and administration | Research | Vehicles | Multiple sclerosis | Intravenous administration | Neurodegenerative diseases | Biodegradability | Tumor cells | Antisense oligonucleotides | Esters | endosomes | Drug delivery | Leucine | Drug development | pH effects | Neurological diseases | Angiogenesis | Glioma cells | Monoclonal antibodies | nanotechnology | Alzheimer's disease | Cytoplasm | Biological Sciences | Physical Sciences | blood–brain barrier
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e85311
As neovascularization is essential for tumor growth and metastasis, controlling angiogenesis is a promising tactic in limiting cancer progression. Melatonin... 
IN-VITRO | SIGNALING PATHWAYS | STEM-CELLS | RAT GLIOMA | INHIBITION | MULTIDISCIPLINARY SCIENCES | PROLIFERATION | VASCULAR-PERMEABILITY | VEIN ENDOTHELIAL-CELLS | EXPRESSION | CARCINOMA | Neoplasm Transplantation | Receptor, Epidermal Growth Factor - genetics | Humans | Neovascularization, Pathologic | Ki-67 Antigen - metabolism | Transplantation, Heterologous | Insulin-Like Growth Factor I - genetics | Vascular Endothelial Growth Factor A - metabolism | von Willebrand Factor - genetics | Vascular Endothelial Growth Factor A - genetics | Vascular Endothelial Growth Factor Receptor-2 - genetics | Receptor, Epidermal Growth Factor - metabolism | Female | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | von Willebrand Factor - metabolism | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Antioxidants - pharmacology | Breast Neoplasms - blood supply | Breast Neoplasms - drug therapy | Animals | Breast Neoplasms - genetics | Ki-67 Antigen - genetics | Tumor Burden - drug effects | Breast Neoplasms - pathology | Mice, Nude | Mice | Melatonin - pharmacology | Insulin-Like Growth Factor I - metabolism | Immunohistochemistry | Melatonin | Growth | Von Willebrand factor | Breast cancer | SPECT imaging | Neovascularization | Vascular endothelial growth factor | Cell proliferation | Cell culture | Surgical implants | Laboratories | Technetium | Metastasis | Kinases | Vascularization | Metastases | Angiogenesis | Densitometers | Cell growth | Epidermal growth factor | Computed tomography | Rodents | Animal tissues | Xenografts | Densitometry | Growth factors | Photon emission | Insulin | Single photon emission computed tomography | Medical prognosis | Breast | Hypoxia | In vivo methods and tests | Cell size | Molecular biology | Viability | Tumors | Cancer
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2013, Volume 123, Issue 6, pp. 2475 - 2487
Glioblastomas (GBMs) are very aggressive tumors that are resistant to conventional chemo- and radiotherapy. New molecular therapeutic strategies are required... 
INITIATING CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | PHASE-II TRIAL | SIGNALING PATHWAYS | IN-VITRO | GLIOMAGENESIS | HUMAN BRAIN | TUMOR STEM-CELLS | GLIOMA | FACTOR RECEPTOR EXPRESSION | PROTEIN-KINASE DYRK1A | RNA, Small Interfering - genetics | Cell Proliferation | Protein-Tyrosine Kinases - metabolism | Humans | Brain Neoplasms - pathology | Spheroids, Cellular - pathology | Brain Neoplasms - metabolism | Gene Knockdown Techniques | Protein-Tyrosine Kinases - genetics | Receptor, Epidermal Growth Factor - metabolism | Neoplastic Stem Cells - metabolism | Proteolysis | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Neoplastic Stem Cells - pathology | Glioblastoma - metabolism | Antineoplastic Agents - pharmacology | Protein Stability | Protein-Serine-Threonine Kinases - metabolism | Cell Survival - drug effects | Gene Expression | Signal Transduction | Harmine - pharmacology | Neural Stem Cells - drug effects | Protein-Serine-Threonine Kinases - genetics | Neural Stem Cells - physiology | Tumor Burden | Brain Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Animals | Mice, Nude | Glioblastoma - pathology | Cell Line, Tumor | Mice | Glioblastoma - drug therapy | Protein-Tyrosine Kinases - antagonists & inhibitors | Physiological aspects | Development and progression | Genetic aspects | Research | Glioblastoma multiforme | Protein kinases | Studies | Ethics | Phosphorylation | Hypotheses | Epidermal growth factor | Rodents | Stem cells | Radiation therapy | Kinases | Cancer therapies | Tumors | Cancer
Journal Article
Brain, ISSN 0006-8950, 2013, Volume 136, Issue 2, pp. 564 - 576
Journal Article
Journal Article
Journal Article
Cancer Science, ISSN 1347-9032, 12/2018, Volume 109, Issue 12, pp. 3874 - 3882
Autocrine and paracrine factors, including glutamate and epidermal growth factor (EGF), are potent inducers of brain tumor cell invasion, a pathological... 
xCT | glioma | epidermal growth factor receptor | GluN2B | glutamate | MIGRATION | CELLS | TYROSINE PHOSPHORYLATION | NMDA RECEPTORS | EGFR | INVASION | ONCOLOGY | BIOLOGY | DEFICIENT | EXPRESSION | MODULATION | Neoplasm Transplantation | Phosphorylation | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | Sulfasalazine - pharmacology | Brain Neoplasms - metabolism | Glioma - metabolism | Amino Acid Transport System y+ - metabolism | Receptors, N-Methyl-D-Aspartate - chemistry | Dizocilpine Maleate - administration & dosage | Protein Domains | Cell Survival - drug effects | ErbB Receptors - metabolism | Brain Neoplasms - drug therapy | Disease Progression | Drug Synergism | Sulfasalazine - administration & dosage | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Glutamic Acid - metabolism | Mice | Epidermal Growth Factor - pharmacology | Dizocilpine Maleate - pharmacology | Glioma - drug therapy | Methyl aspartate | Epidermal growth factor | Gliomas | Brain tumors | Development and progression | Aspartate | Glutamate | Immunoglobulins | Cell survival | Epidermal growth factor receptors | Paracrine signalling | N-Methyl-D-aspartic acid receptors | Glutamic acid receptors | Kinases | Cell adhesion & migration | Glioma cells | Sulfasalazine | Autocrine signalling | Cell migration | Tumors | Original
Journal Article
Cell, ISSN 0092-8674, 01/2018, Volume 172, Issue 3, pp. 534 - 548.e19
Journal Article
Science, ISSN 0036-8075, 5/2013, Volume 340, Issue 6132, pp. 626 - 630
The recent discovery of mutations in metabolic enzymes has rekindled interest in harnessing the altered metabolism of cancer cells for cancer therapy. One... 
Enzymes | Cell growth | Glioma | RNA | Neurons | REPORTS | Methylation | Cellular differentiation | Genetic mutation | Heterologous transplantation | Tumors | TRANSFORMATION | GLIOBLASTOMA | MULTIDISCIPLINARY SCIENCES | INTEGRATED GENOMIC ANALYSIS | PHENOTYPE | ACUTE MYELOID-LEUKEMIA | MUTATIONS | ONCOMETABOLITE 2-HYDROXYGLUTARATE | BRAIN | Benzeneacetamides - toxicity | Protein Multimerization | Imidazoles - administration & dosage | Gene Expression Profiling | Isocitrate Dehydrogenase - antagonists & inhibitors | Glioma - genetics | RNA Interference | Enzyme Inhibitors - toxicity | Glioma - pathology | Imidazoles - toxicity | Gene Expression Regulation, Neoplastic - drug effects | Benzeneacetamides - pharmacology | Mutant Proteins - antagonists & inhibitors | Glioma - enzymology | Enzyme Inhibitors - pharmacology | Isocitrate Dehydrogenase - genetics | Mutant Proteins - metabolism | Imidazoles - pharmacology | Mice, SCID | Benzeneacetamides - administration & dosage | Xenograft Model Antitumor Assays | Animals | Cell Differentiation - drug effects | Cell Transformation, Neoplastic | Isocitrate Dehydrogenase - chemistry | Mutant Proteins - chemistry | Isocitrate Dehydrogenase - metabolism | Glutarates - metabolism | Mice | Histones - metabolism | Glioma - drug therapy | Cell proliferation | Gene mutations | Gliomas | Growth | Cancer cells | Physiological aspects | Genetic aspects | Research | Cancer | Genes | Cells | Mutation | Drugs | Binding | Mutations | Inhibitors | Online | Delay
Journal Article