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Journal Article
European Journal of Nuclear Medicine and Molecular Imaging, ISSN 1619-7070, 4/2013, Volume 40, Issue 4, pp. 524 - 531
Journal Article
Nature communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, p. 7629
...) complex bile acids in the intestinal lumen and decrease intestinal FXR activity. The BAS-BA complex also induces glucagon-like peptide-1 (GLP-1... 
GLP-1 SECRETION | FXR | BILE-ACID RECEPTORS | PROTEIN | GLUCOSE | MULTIDISCIPLINARY SCIENCES | INTESTINE | METABOLIC-RATE | EXPRESSION | Colon - cytology | Intestinal Mucosa - metabolism | Sequestering Agents - pharmacology | Humans | Ileum - metabolism | RNA, Messenger - metabolism | Colesevelam Hydrochloride - pharmacology | Obesity - genetics | Glucagon-Like Peptide 1 - genetics | Jejunum - metabolism | Insulin-Secreting Cells - metabolism | Proglucagon - drug effects | Diet, High-Fat | Jejunum - cytology | Enteroendocrine Cells - metabolism | Ileum - cytology | Proglucagon - metabolism | Insulin Secretion | Glucagon-Like Peptide 1 - metabolism | Signal Transduction | Bile Acids and Salts - metabolism | Nuclear Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Colon - metabolism | Mice, Knockout | Obesity - metabolism | Transcription Factors - metabolism | Insulin - metabolism | Animals | Glycolysis | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice, Obese | Mice | Proglucagon - genetics | Receptors, G-Protein-Coupled - genetics | Blood Glucose - metabolism | Anticholesteremic Agents - pharmacology | Intestines - cytology | Carbohydrates | Glucose | Gene expression | Insulin | Cell and Molecular Biology | RAT SMALL-INTESTINE | GLUCOSE-HOMEOSTASIS | OBESITY | Endokrinologi och diabetes | MICE | Multidisciplinary Sciences | Cell- och molekylärbiologi | TYPE-2 DIABETES-MELLITUS | Endocrinology and Diabetes
Journal Article
Journal Article
Drugs, ISSN 0012-6667, 4/2017, Volume 77, Issue 5, pp. 493 - 503
Journal Article
Diabetes (New York, N.Y.), ISSN 1939-327X, 2009, Volume 58, Issue 9, pp. 2148 - 2161
Glucagon-Like Peptide-1 Receptor Activation Modulates Pancreatitis-Associated Gene Expression But Does Not Modify the Susceptibility to Experimental Pancreatitis in Mice Jacqueline A. Koehler , Laurie L... 
DISPERSED ACINI | INHIBITION | EXENDIN RECEPTORS | ENDOCRINE | EXENATIDE | ENDOCRINOLOGY & METABOLISM | INCRETIN RECEPTORS | SECRETION | IDENTIFICATION | HELODERMA-SUSPECTUM VENOM | EXOCRINE | Gene Expression - drug effects | Male | Pancreas, Exocrine - drug effects | Hypoglycemic Agents - toxicity | Peptides - toxicity | Dietary Fats - pharmacology | Mice, Mutant Strains | Gene Expression - physiology | Early Growth Response Protein 1 - genetics | Pancreatitis - physiopathology | Disease Models, Animal | Venoms - toxicity | Receptors, Glucagon - genetics | Severity of Illness Index | Genes, fos - physiology | Glucagon-Like Peptide 1 - metabolism | Glucagon-Like Peptide 1 - analogs & derivatives | Mice, Inbred C57BL | Ceruletide - toxicity | Receptors, Glucagon - metabolism | Pancreatitis - chemically induced | Pancreatitis-Associated Proteins | Pancreas, Exocrine - physiology | Animals | Glucagon-Like Peptide-1 Receptor | Signal Transduction - drug effects | Signal Transduction - physiology | Mice | Pancreatitis - metabolism | Glucagon-Like Peptide 1 - toxicity | Liraglutide | Complications and side effects | Pancreatic beta cells | Pancreatitis | Development and progression | Hypoglycemic agents | Genetic aspects | Risk factors | Index Medicus | Abridged Index Medicus | Original
Journal Article