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European Journal of Endocrinology, ISSN 0804-4643, 03/2018, Volume 178, Issue 3, pp. R101 - R111
Aldosterone is the main mineralocorticoid hormone in humans and plays a key role in maintaining water and electrolyte homeostasis. Primary aldosteronism (PA),... 
HYPERALDOSTERONISM TYPE-II | GLUCOCORTICOID-REMEDIABLE ALDOSTERONISM | K+ CHANNEL MUTATIONS | ENDOCRINOLOGY & METABOLISM | KCNJ5 MUTATIONS | BETA-CATENIN | FAMILIAL HYPERALDOSTERONISM | SOMATIC MUTATIONS | ADRENAL-HYPERPLASIA | ADENOMAS | POTASSIUM CHANNEL
Journal Article
Journal Article
Endocrine practice : official journal of the American College of Endocrinology and the American Association of Clinical Endocrinologists, ISSN 1530-891X, 04/2015, Volume 21, Issue 4, pp. 400 - 405
Objective: The American Association of Clinical Endocrinologists Adrenal Scientific Committee has developed a series of articles to update members on the... 
GLUCOCORTICOID-REMEDIABLE ALDOSTERONISM | ENDOCRINOLOGY & METABOLISM | CHANNEL MUTATIONS | ATP1A1 | HYPERTENSION | SOMATIC MUTATIONS | ADENOMAS | Hyperaldosteronism - diagnosis | Genetic Testing | Humans | Hyperaldosteronism - genetics | Adult | Male | Hyperaldosteronism - therapy
Journal Article
Journal of Molecular Endocrinology, ISSN 0952-5041, 07/2017, Volume 59, Issue 1, pp. R47 - R63
Primary aldosteronism (PA), the most common form of secondary hypertension, is caused in the majority of cases by unilateral aldosterone-producing adenoma... 
Calcium channels | Familial hyperaldosteronism | Primary aldosteronism | Somatic mutations | Aldosterone-producing adenoma | ATPase | Germline mutations | Potassium channels | Wnt/β-catenin pathway | aldosterone-producing adenoma | HYPERALDOSTERONISM TYPE-II | MEMBRANE CA2+ ATPASE | familial hyperaldosteronism | calcium channels | Wnt/beta-catenin pathway | germline mutations | PCR-SSCP ANALYSIS | somatic mutations | MOUSE ADRENAL-CORTEX | GLUCOCORTICOID-REMEDIABLE ALDOSTERONISM | potassium channels | K+ CHANNEL MUTATIONS | primary aldosteronism | ENDOCRINOLOGY & METABOLISM | CLINICAL-PRACTICE GUIDELINE | KCNJ5 MUTATIONS | POTASSIUM CHANNEL | Adrenal Glands - pathology | Sodium-Potassium-Exchanging ATPase - genetics | Plasma Membrane Calcium-Transporting ATPases - genetics | Adenoma - genetics | Humans | Hyperaldosteronism - pathology | Hyperaldosteronism - metabolism | Adrenal Hyperplasia, Congenital - genetics | Adenoma - metabolism | Plasma Membrane Calcium-Transporting ATPases - metabolism | Hyperaldosteronism - diagnosis | Adrenal Hyperplasia, Congenital - diagnosis | Hypokalemia - metabolism | Adrenal Hyperplasia, Congenital - pathology | G Protein-Coupled Inwardly-Rectifying Potassium Channels - metabolism | Hypertension - genetics | Hypertension - diagnosis | Hypokalemia - pathology | Adenoma - diagnosis | Gene Expression | Genetic Predisposition to Disease | Adrenal Hyperplasia, Congenital - metabolism | Hyperaldosteronism - genetics | G Protein-Coupled Inwardly-Rectifying Potassium Channels - genetics | beta Catenin - metabolism | Hypertension - pathology | beta Catenin - genetics | Hypertension - metabolism | Hypokalemia - diagnosis | Sodium-Potassium-Exchanging ATPase - metabolism | Calcium Channels, L-Type - genetics | Adrenal Glands - metabolism | Adenoma - pathology | Calcium Channels, L-Type - metabolism | Mutation | Hypokalemia - genetics
Journal Article
Experimental and Clinical Endocrinology & Diabetes, ISSN 0947-7349, 06/2016, Volume 124, Issue 6, pp. 335 - 341
Journal Article