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Hippocampus, ISSN 1050-9631, 08/2017, Volume 27, Issue 8, pp. 890 - 898
Hippocampal rhythms in clock gene expression, enzymatic activity, and long‐term potentiation (LTP) are thought to underlie day–night differences in memory... 
circadian | glycogen synthase kinase‐3 | period2 | bmal1 | long‐term potentiation | glycogen synthase kinase-3 | long-term potentiation | PROTEIN | PHOSPHORYLATION | GLYCOGEN-SYNTHASE KINASE-3 | MOUSE | NEUROSCIENCES | CIRCADIAN CLOCK | INHIBITION | MEMORY | PERIOD | MICE | LITHIUM LENGTHENS | Phosphorylation | Neuronal Plasticity - drug effects | Male | Tubulin - genetics | Hippocampus - drug effects | Neuronal Plasticity - genetics | Tubulin - metabolism | Neuronal Plasticity - physiology | Period Circadian Proteins - genetics | Organ Culture Techniques | ARNTL Transcription Factors - genetics | Mice, Inbred C57BL | Circadian Rhythm - genetics | Enzyme Inhibitors - pharmacology | ARNTL Transcription Factors - metabolism | Gene Expression Regulation - physiology | Mice, Transgenic | Circadian Rhythm - physiology | Pyrimidines - pharmacology | Glycogen Synthase Kinase 3 - metabolism | Patch-Clamp Techniques | Animals | Period Circadian Proteins - metabolism | Glycogen Synthase Kinase 3 - genetics | Mice | Pyridines - pharmacology | Hippocampus - physiology | In Vitro Techniques | Circadian Rhythm - drug effects | Synthesis | Glycogen | Analysis | Genes | Genetic engineering | Gene expression | Neurophysiology | Potentiation | Circadian rhythms | Long-term potentiation | Transgenic mice | Periodicity | Glycogen synthase kinase 3 | Rhythms | CLOCK protein | Circadian rhythm | Plasticity (hippocampal) | Kinases | Light effects | Plasticity (synaptic) | Enzymatic activity | Rodents | BMAL1 protein | Isoforms | Period 2 protein | Hippocampus
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2010, Volume 5, Issue 1, p. e8549
Background: A point mutation in the Drosophila gene technical knockout (tko), encoding mitoribosomal protein S12, was previously shown to cause a phenotype of... 
CONTROLLED CLINICAL-TRIAL | FAT-BODY | OXIDATIVE-PHOSPHORYLATION | ACTIVATED PROTEIN-KINASE | MALE COURTSHIP BEHAVIOR | GLYCOGEN-SYNTHASE KINASE-3 | FEEDING-BEHAVIOR | BIOLOGY | SEX-DETERMINATION | ETHYLMALONIC ENCEPHALOPATHY | DNA PHOSPHODIESTERASE | Mitochondrial Diseases - genetics | Point Mutation | Gene Expression | Animals | Ribosomal Proteins - genetics | Oxidative Phosphorylation | Female | Male | Gene Expression Profiling | Disease Models, Animal | Drosophila - genetics | Enzymes | Biotechnology | Nervous system diseases | Gene mutations | Analysis | Drosophila | Physiological aspects | Dietary fat | Genetic aspects | Gene expression | Heart | Disease | Disorders | Mitochondrial DNA | Biochemistry | Males | Delay | Proteins | Signal transduction | Mitochondria | Biomolecules | Life sciences | Deoxyribonucleic acid--DNA | Starvation | Medical technology | Polypeptides | Nutrient deficiency | Gametogenesis | Abnormalities | Metabolism | Neurophysiology | Hospitals | Insects | Diet | Protein synthesis | Signal processing | Fats | Mutation | Gene therapy | Human behavior | Courtship | Phosphorylation | Genes | Gene regulation | Clinical trials | Absorptivity | Kinases | Adenosine | Congenital diseases | Amyotrophic lateral sclerosis | Lactate dehydrogenase | L-Lactate dehydrogenase | Signaling | Oxidative phosphorylation | Point mutation | Catabolism | Anaplerotic pathways | Lactic acid | Electron transport | Deoxyribonucleic acid | DNA
Journal Article
PLOS GENETICS, ISSN 1553-7404, 12/2012, Volume 8, Issue 12
Understanding the functional relationship between intracellular factors and extracellular signals is required for reconstructing gene regulatory networks (GRN)... 
ANEMONE NEMATOSTELLA-VECTENSIS | EARLY XENOPUS-EMBRYOS | CAENORHABDITIS-ELEGANS | ANTERIOR-POSTERIOR AXIS | GERM-LAYER | GLYCOGEN-SYNTHASE KINASE-3 | ANIMAL-VEGETAL AXIS | MIDBLASTULA TRANSITION | GENETICS & HEREDITY | SEA-URCHIN EMBRYO | MAJOR DEVELOPMENTAL TRANSITION
Journal Article
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 09/2008, Volume 33, Issue 10, pp. 2407 - 2415
The molecular mechanism of action of the mood stabilizer lithium is assumed to involve changes in gene expression leading to neuronal adaptation. The... 
Glycogen synthase kinase 3 | Transgenic luciferase reporter gene mice | Lithium | Phosphorylation | cAMP-responsive element (CRE) binding protein (CREB) | Psychosocial stress | SYSTEM | psychosocial stress | MESSENGER-RNA EXPRESSION | PSYCHIATRY | GLYCOGEN-SYNTHASE KINASE-3 | BIPOLAR DISORDER | NEUROSCIENCES | lithium | CREB | MOOD DISORDERS | IN-VIVO | PHARMACOLOGY & PHARMACY | RAT-BRAIN | MYOINOSITOL | PROTEIN-PHOSPHORYLATION | phosphorylation | transgenic luciferase reporter gene mice | glycogen synthase kinase 3 | Glycogen Synthase Kinase 3 - drug effects | Transcriptional Activation - genetics | Brain - anatomy & histology | Transcriptional Activation - drug effects | Bipolar Disorder - physiopathology | Male | Adenylyl Cyclases - drug effects | Cyclic AMP Response Element-Binding Protein - drug effects | Brain - metabolism | Social Behavior | Lithium Compounds - pharmacology | Behavior, Animal - drug effects | Phosphorylation - drug effects | Genes, Reporter | Chronic Disease - drug therapy | Disease Models, Animal | Antimanic Agents - pharmacology | Drug Administration Schedule | Gene Expression Regulation - genetics | Stress, Psychological - drug therapy | Behavior, Animal - physiology | Mice, Transgenic | Up-Regulation - genetics | Signal Transduction - genetics | Adenylyl Cyclases - metabolism | Down-Regulation - drug effects | Glycogen Synthase Kinase 3 - metabolism | Bipolar Disorder - drug therapy | Bipolar Disorder - genetics | Down-Regulation - genetics | Brain - drug effects | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Stress, Psychological - genetics | Animals | Signal Transduction - drug effects | Cyclic AMP Response Element-Binding Protein - metabolism | Mice | Stress, Psychological - physiopathology
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 12/2013, Volume 1832, Issue 12, pp. 1969 - 1979
A growing body of evidence suggests that the circadian molecular system is involved in the pathogenic and therapeutic mechanisms underlying bipolar disorders.... 
Per2 | Egr1 | Bipolar disorder | Lithium | Circadian gene | Mood stabilizer | CLOCK GENES | IMMEDIATE-EARLY GENE | ACTIVATED PROTEIN-KINASE | C-FOS | BIOCHEMISTRY & MOLECULAR BIOLOGY | SCHIZOAFFECTIVE DISORDER | GLYCOGEN-SYNTHASE KINASE-3-BETA | NGFI-A | BIOPHYSICS | MESSENGER-RNA | CIRCADIAN GENES | BIPOLAR-I-DISORDER | Egrl | Genetic research | Genetic transcription | Analysis | Genes
Journal Article
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2012, Volume 58, Issue 1, pp. 65 - 72
Background & Aims The tumor fate derives from cell autonomous properties and niche microenvironmental cues. The transforming growth factor β (TGFβ) is a major... 
Gastroenterology and Hepatology | TGFβ | GSK3β | HCC | Gene therapy | EMT | HNF4α | TRANSCRIPTIONAL ACTIVITY | DNA-BINDING | PHOSPHORYLATION | PROTEIN-KINASE | NUCLEAR FACTOR 4-ALPHA | HNF4 alpha | EPITHELIAL-MESENCHYMAL TRANSITION | GLYCOGEN-SYNTHASE KINASE-3-BETA | GROWTH-FACTOR-BETA | PLASMA TRANSFORMING GROWTH-FACTOR-BETA-1 | LIVER | GSK3 beta | TGF beta | GASTROENTEROLOGY & HEPATOLOGY | Genetic Therapy | Epithelial-Mesenchymal Transition - physiology | Humans | Glycogen Synthase Kinase 3 beta | Carcinoma, Hepatocellular - secondary | Liver Neoplasms - therapy | Tumor Microenvironment - physiology | Epithelial-Mesenchymal Transition - genetics | Cell Movement - physiology | Hepatocytes - cytology | Tumor Microenvironment - genetics | Carcinoma, Hepatocellular - genetics | Genes, Tumor Suppressor - physiology | Liver Neoplasms - pathology | Gene Expression Regulation, Neoplastic - physiology | Hepatocyte Nuclear Factor 4 - metabolism | Liver Neoplasms - genetics | Hepatocyte Nuclear Factor 4 - genetics | Glycogen Synthase Kinase 3 - metabolism | Hep G2 Cells | Animals | Transforming Growth Factor beta - genetics | Cell Line, Tumor | Carcinoma, Hepatocellular - therapy | Mice | Transforming Growth Factor beta - metabolism | Cell Line, Transformed | Hépatology and Gastroenterology | Gene Expression Regulation, Neoplastic | Tumor Microenvironment | Glycogen Synthase Kinase 3 | Liver Neoplasms | Life Sciences | Microbiology and Parasitology | Human health and pathology | Hepatocytes | Epithelial-Mesenchymal Transition | Carcinoma, Hepatocellular | Hepatocyte Nuclear Factor 4 | Genes, Tumor Suppressor | Transforming Growth Factor beta | Cell Movement | Cancer
Journal Article