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Nature, ISSN 0028-0836, 05/2012, Volume 484, Issue 7397, pp. 246 - 250
Journal Article
Immunity, ISSN 1074-7613, 03/2015, Volume 42, Issue 3, pp. 443 - 456
The mechanisms underlying human natural killer (NK) cell phenotypic and functional heterogeneity are unknown. Here, we describe the emergence of diverse... 
NATURAL-KILLER-CELLS | ACTIVATION | CMV REACTIVATION | INNATE | RECEPTOR | FCR-GAMMA | DIFFERENTIATION | IMMUNOLOGY | PLZF CONTROLS | EXPRESSION | T-CELLS | Cell Proliferation | Humans | Transcription Factors - deficiency | NK Cell Lectin-Like Receptor Subfamily C - genetics | Receptors, IgG - deficiency | Intracellular Signaling Peptides and Proteins - immunology | Cytomegalovirus Infections - immunology | Gene Expression Profiling | Killer Cells, Natural - pathology | Cytomegalovirus Infections - genetics | Protein-Tyrosine Kinases - deficiency | Protein-Tyrosine Kinases - immunology | T-Lymphocytes, Cytotoxic - pathology | Intracellular Signaling Peptides and Proteins - deficiency | NK Cell Lectin-Like Receptor Subfamily C - immunology | DNA Methylation | Protein-Tyrosine Kinases - genetics | Receptors, IgG - genetics | Microarray Analysis | Antibodies - immunology | Cytomegalovirus Infections - virology | Killer Cells, Natural - immunology | Cytomegalovirus Infections - pathology | Kruppel-Like Transcription Factors - deficiency | Transcription Factors - immunology | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Adaptive Immunity | T-Lymphocytes, Cytotoxic - immunology | Promoter Regions, Genetic | GPI-Linked Proteins - immunology | Signal Transduction | Killer Cells, Natural - virology | T-Lymphocytes, Cytotoxic - virology | NK Cell Lectin-Like Receptor Subfamily C - deficiency | Immunophenotyping | Receptors, IgG - immunology | Transcription Factors - genetics | Killer Cells, Natural - classification | Promyelocytic Leukemia Zinc Finger Protein | Cytomegalovirus - immunology | Epigenesis, Genetic - immunology | Kruppel-Like Transcription Factors - genetics | Kruppel-Like Transcription Factors - immunology | GPI-Linked Proteins - genetics | Virus diseases | Proteins | Epigenetic inheritance | Killer cells | Immunotherapy | Genomics | Medical genetics | Cytomegalovirus infections | T cells | Methylation | Health aspects | Antigens | Flow cytometry | Cytomegalovirus | Data analysis | Biomedical research | Transplants & implants | Cytokines | Leukemia | Cytotoxicity | Infections | Kinases | Diversification | Gene expression | Experiments | Scholarships & fellowships | Lymphocytes | DNA methylation | Epigenetics | Viral infections | Immune system | Cancer | Index Medicus
Journal Article
Immunity, ISSN 1074-7613, 03/2015, Volume 42, Issue 3, pp. 431 - 442
Long-lived “memory-like” NK cells have been identified in individuals infected by human cytomegalovirus (HCMV), but little is known about how the memory-like... 
ADAPTIVE IMMUNITY | CD94/NKG2C RECEPTOR | REPERTOIRE | NATURAL-KILLER-CELLS | ACTIVATION | TYROSINE KINASE | FCR-GAMMA | UMBILICAL-CORD BLOOD | DIFFERENTIATION | IMMUNOLOGY | CHILDREN | Cell Proliferation | Humans | NK Cell Lectin-Like Receptor Subfamily C - genetics | Receptors, IgG - deficiency | Intracellular Signaling Peptides and Proteins - immunology | Cytomegalovirus Infections - immunology | Gene Expression Profiling | Killer Cells, Natural - pathology | Cytomegalovirus Infections - genetics | Protein-Tyrosine Kinases - deficiency | Protein-Tyrosine Kinases - immunology | Intracellular Signaling Peptides and Proteins - deficiency | NK Cell Lectin-Like Receptor Subfamily C - immunology | DNA Methylation | Protein-Tyrosine Kinases - genetics | Receptors, IgG - genetics | Microarray Analysis | Antibodies - immunology | Cytomegalovirus Infections - virology | Killer Cells, Natural - immunology | Cytomegalovirus Infections - pathology | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Promoter Regions, Genetic | GPI-Linked Proteins - immunology | Signal Transduction | Killer Cells, Natural - virology | NK Cell Lectin-Like Receptor Subfamily C - deficiency | Immunophenotyping | Receptors, IgG - immunology | Killer Cells, Natural - classification | Cytomegalovirus - immunology | Epigenesis, Genetic - immunology | Immunologic Memory | GPI-Linked Proteins - genetics | Proteins | Viral antibodies | Epigenetic inheritance | Killer cells | Molecular genetics | Antibodies | Gene expression | Antigens | Cytomegalovirus | Cloning | Colleges & universities | Infections | Experiments | Long term | DNA methylation | Epigenetics | Expansion | Viral infections | Deoxyribonucleic acid--DNA | Immune system | Index Medicus
Journal Article
Journal of Dental Research, ISSN 0022-0345, 9/2014, Volume 93, Issue 9, pp. 882 - 890
Recent genome-wide association studies (GWAS) of chronic periodontitis (CP) offer rich data sources for the investigation of candidate genes, functional... 
genomics | genetics | bacteria | pathway analysis | inflammation | periodontal disease | PATHWAYS | POPULATION | SUSCEPTIBILITY | ADULTS | RECEPTOR | ATHEROSCLEROSIS RISK | CIRCADIAN CLOCK | DENTISTRY, ORAL SURGERY & MEDICINE | DISEASE | HEALTH | Prospective Studies | Tumor Necrosis Factor Decoy Receptors - genetics | Microtubule-Associated Proteins - genetics | Cytoskeletal Proteins - genetics | Atherosclerosis - genetics | Humans | Middle Aged | Apoptosis - genetics | Male | Monoacylglycerol Lipases - genetics | Receptors, Peptide - genetics | Receptors, Tumor Necrosis Factor, Member 10c | ras GTPase-Activating Proteins - genetics | Chronic Periodontitis - microbiology | Chronic Periodontitis - genetics | Adult | Female | Atherosclerosis - microbiology | Adaptor Protein Complex beta Subunits - genetics | Nuclear Proteins - genetics | Porphyromonas gingivalis - genetics | Genome-Wide Association Study | Genetic Association Studies | Membrane Proteins - genetics | Risk Factors | Linkage Disequilibrium - genetics | Potassium Channels, Tandem Pore Domain - genetics | Chromosome Mapping | Membrane Glycoproteins - genetics | Aggregatibacter actinomycetemcomitans - genetics | Adaptor Protein Complex 3 - genetics | Polymorphism, Single Nucleotide - genetics | Aged | Receptors, G-Protein-Coupled - genetics | Mucins - genetics | Cohort Studies | GPI-Linked Proteins - genetics | Index Medicus | Dentistry | Research Reports
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2014, Volume 111, Issue 38, pp. 13924 - 13929
Genetic variation can modulate gene expression, and thereby phenotypic variation and susceptibility to complex diseases such as type 2 diabetes (T2D). Here we... 
Beta cells | Databases | Allelic imbalance | Genes | Genetic loci | P values | Type 2 diabetes mellitus | Islets of Langerhans | Sequencing | Type 1 diabetes mellitus | PROVIDES INSIGHTS | VARIANTS | ALIGNMENT | RNA-SEQ | BETA-CELL TRANSCRIPTOME | MULTIDISCIPLINARY SCIENCES | INSULIN-SECRETION | GLYCEMIC TRAITS | DIABETES SUSCEPTIBILITY LOCI | EXPRESSION | ASSOCIATION | 5'-Nucleotidase - genetics | Cell Line | p21-Activated Kinases - genetics | Diabetes Mellitus, Type 2 - genetics | Genomics | Humans | Vesicular Transport Proteins - genetics | GPI-Linked Proteins - biosynthesis | Vesicular Transport Proteins - biosynthesis | Glucose - genetics | Male | RNA, Long Noncoding - genetics | Diabetes Mellitus, Type 2 - metabolism | Tetraspanins - biosynthesis | p21-Activated Kinases - biosynthesis | RNA, Long Noncoding - biosynthesis | Glucose - metabolism | Female | RNA Editing - physiology | Tetraspanins - genetics | Transcriptome - physiology | 5'-Nucleotidase - biosynthesis | GPI-Linked Proteins - genetics | Glucose metabolism | Pancreatic beta cells | Cell research | Genetic research | Genetic aspects | Genetic transcription | Research | Transcription factors | Pathogenesis | Glucose | Pancreas | Metabolism | Index Medicus | Biological Sciences | Clinical Medicine | Endokrinologi och diabetes | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Endocrinology and Diabetes
Journal Article
Nature, ISSN 0028-0836, 07/2010, Volume 466, Issue 7302, pp. 113 - 117
Alopecia areata (AA) is among the most highly prevalent human autoimmune diseases, leading to disfiguring hair loss due to the collapse of immune privilege of... 
RISK LOCUS | CELLS | HAIR FOLLICLE | MULTIDISCIPLINARY SCIENCES | GENES | SUSCEPTIBILITY | NKG2D LIGAND | PEROXIREDOXIN-5 | AUTOIMMUNE-DISEASE | EXPRESSION | CANCER | Humans | Middle Aged | GPI-Linked Proteins | Immunity, Innate - genetics | NK Cell Lectin-Like Receptor Subfamily K - immunology | Male | Alopecia Areata - genetics | Antigens, CD - genetics | Case-Control Studies | Adaptive Immunity - genetics | T-Lymphocytes, Regulatory - immunology | Autoimmune Diseases - genetics | Ikaros Transcription Factor - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Interleukin-2 Receptor alpha Subunit - genetics | Peroxiredoxins - genetics | T-Lymphocytes, Regulatory - cytology | Qa-SNARE Proteins - genetics | Adult | Female | Hair Follicle - immunology | Adaptive Immunity - immunology | Alopecia Areata - immunology | Genetic Predisposition to Disease | Genome-Wide Association Study | Autoimmune Diseases - immunology | Intercellular Signaling Peptides and Proteins - genetics | CTLA-4 Antigen | Immunity, Innate - immunology | Hair Follicle - cytology | Hair Follicle - metabolism | Alleles | Polymorphism, Single Nucleotide - genetics | Aged | Alopecia | Immune response | Baldness | Genetic aspects | Research | Health aspects | Risk factors | Haplotypes | Review boards | Principal components analysis | Celiac disease | Rheumatoid arthritis | Genetics | Autoimmune diseases | Health risk assessment | Chromosomes | Immune system | Validation studies | Hair | Antigens | Receptors | Pathogenesis | Genes | Zinc | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, pp. e63812 - e63812
Environmental factors such as tobacco smoking may have long-lasting effects on DNA methylation patterns, which might lead to changes in gene expression and in... 
POPULATION | MICROARRAY | PROMOTER METHYLATION | LYMPHOBLASTS | SMOKERS | MULTIDISCIPLINARY SCIENCES | AH DIOXIN RECEPTOR | CANCER | EXPRESSION | PLACENTAL ALKALINE-PHOSPHATASE | HYDROCARBON RECEPTOR REPRESSOR | Smoking - adverse effects | Oligonucleotide Array Sequence Analysis | Humans | Middle Aged | Male | Genome, Human - drug effects | Epigenomics - methods | Time Factors | Adult | Female | Electrophoretic Mobility Shift Assay | Alkaline Phosphatase - genetics | Basic Helix-Loop-Helix Transcription Factors - genetics | Gene Expression Regulation - genetics | Isoenzymes - genetics | Repressor Proteins - genetics | Linear Models | Smoking Cessation - statistics & numerical data | Genome, Human - genetics | Gene Expression Regulation - drug effects | Receptors, Thrombin - genetics | Analysis of Variance | Sex Factors | CpG Islands - genetics | Aged | DNA Methylation - drug effects | GPI-Linked Proteins - genetics | Medical research | Smokers | Genes | Genomics | DNA | Genetic research | Medicine, Experimental | Development and progression | Methylation | Gene expression | Smoking | Protein binding | Dermatology | Change detection | Environmental health | Tobacco smoking | Environmental changes | Cardiovascular disease | Genomes | Environmental factors | Phosphatase | Epidemiology | Studies | Tobacco | DNA methylation | Epigenetics | Population | Genetics | Bioinformatics | Deoxyribonucleic acid--DNA | Cancer | Index Medicus | Deoxyribonucleic acid
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2011, Volume 108, Issue 25, pp. 10337 - 10342
Spinal muscular atrophy (SMA), caused by the deletion of the SMN1 gene, is the leading genetic cause of infant mortality. SMN protein is present at high levels... 
Axons | Motor neurons | Growth cones | Neuroscience | Messenger RNA | RNA | Neurons | Antibodies | Gene expression regulation | SMN complex proteins | Neuritin | Local protein synthesis | Embryonic lethal abnormal vision drosophila-like 4 (ELAV-L4) | LOCALIZATION | RIBONUCLEOPROTEIN | MULTIDISCIPLINARY SCIENCES | ACTIVITY-DEPENDENT EXPRESSION | IDENTIFICATION | neuritin | GROWTH CONES | TRANSPORT | DETERMINING GENE-PRODUCT | SPINAL MUSCULAR-ATROPHY | IN-VIVO | ASSOCIATION | embryonic lethal abnormal vision Drosophila-like 4 (ELAV-L4) | local protein synthesis | ELAV-Like Protein 4 | Humans | RNA, Messenger - metabolism | Zebrafish - embryology | Recombinant Fusion Proteins - metabolism | Embryo, Mammalian - anatomy & histology | Motor Neurons - cytology | Survival of Motor Neuron 1 Protein - genetics | Neuropeptides - genetics | Animals, Genetically Modified | RNA, Messenger - genetics | Cells, Cultured | ELAV Proteins - metabolism | Axons - metabolism | Neuropeptides - metabolism | Survival of Motor Neuron 1 Protein - metabolism | Nerve Tissue Proteins - genetics | GPI-Linked Proteins - metabolism | Motor Neurons - metabolism | Nerve Tissue Proteins - metabolism | Embryo, Mammalian - physiology | Animals | Axons - pathology | Recombinant Fusion Proteins - genetics | Zebrafish - physiology | Mice | ELAV Proteins - genetics | GPI-Linked Proteins - genetics | Gene mutations | Physiological aspects | Genetic aspects | Research | Health aspects | Risk factors | Spinal muscular atrophy | Proteins | Genotype & phenotype | Neuromuscular diseases | Zebrafish | Infant mortality | Ribonucleic acid--RNA | Index Medicus | Spinal cord | Translation | Cell survival | Mental disorders | Axonogenesis | Mortality | SMN protein | survival | Mass spectroscopy | Infants | Gene deletion | infant mortality | HuD protein | Pathology | RNA-binding protein | Actin | Neuromuscular junctions | spinal muscular atrophy | Axon guidance | Mass spectrometry | Biological Sciences
Journal Article
Nature Neuroscience, ISSN 1097-6256, 03/2004, Volume 7, Issue 3, pp. 221 - 228
Axon regeneration in the adult CNS is prevented by inhibitors in myelin. These inhibitors seem to modulate RhoA activity by binding to a receptor complex... 
NEURITE OUTGROWTH | GROWTH CONE GUIDANCE | AXONAL REGENERATION | RHO GTPASES | INHIBITOR | PROTEINS | PROMOTES | BINDING | NEUROSCIENCES | MYELIN-ASSOCIATED GLYCOPROTEIN | P75 | Humans | Nerve Regeneration - physiology | DNA, Complementary - genetics | GPI-Linked Proteins | Myelin-Associated Glycoprotein - genetics | Myelin-Associated Glycoprotein - metabolism | Molecular Sequence Data | rhoA GTP-Binding Protein - metabolism | Receptors, Nerve Growth Factor - metabolism | Amino Acid Sequence - genetics | Recombinant Fusion Proteins - metabolism | Myelin Sheath - metabolism | Fetus | Membrane Proteins - metabolism | Nogo Receptor 1 | Receptors, Cell Surface - isolation & purification | Animals, Newborn | DNA, Complementary - analysis | Base Sequence - genetics | Membrane Proteins - isolation & purification | Membrane Proteins - genetics | Cells, Cultured | Axons - metabolism | Myelin-Oligodendrocyte Glycoprotein | Rats | Receptors, Cell Surface - metabolism | Myelin-Associated Glycoprotein - isolation & purification | Nerve Tissue Proteins | Protein Structure, Tertiary - genetics | Signal Transduction - genetics | Mutation - genetics | Receptor, Nerve Growth Factor | Macromolecular Substances | Animals | Recombinant Fusion Proteins - genetics | Myelin Proteins - metabolism | Astrocytes - metabolism | Receptors, Cell Surface - genetics | Pharmaceutical industry | RhoA protein | LINGO-1 protein | Nogo receptors | Index Medicus
Journal Article