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Journal of Dental Research, ISSN 0022-0345, 9/2014, Volume 93, Issue 9, pp. 882 - 890
..., osteoporosis, and vitamin D deficiency (Genco and Borgnakke, 2013). The investigation of genetic factors in CP has been based on early twin and family studies (Michalowicz... 
genomics | genetics | bacteria | pathway analysis | inflammation | periodontal disease | Life Sciences & Biomedicine | Dentistry, Oral Surgery & Medicine | Science & Technology | Prospective Studies | Tumor Necrosis Factor Decoy Receptors - genetics | Microtubule-Associated Proteins - genetics | Cytoskeletal Proteins - genetics | Atherosclerosis - genetics | Humans | Middle Aged | Apoptosis - genetics | Male | Monoacylglycerol Lipases - genetics | Receptors, Peptide - genetics | Receptors, Tumor Necrosis Factor, Member 10c | ras GTPase-Activating Proteins - genetics | Chronic Periodontitis - microbiology | Chronic Periodontitis - genetics | Adult | Female | Atherosclerosis - microbiology | Adaptor Protein Complex beta Subunits - genetics | Nuclear Proteins - genetics | Porphyromonas gingivalis - genetics | Genome-Wide Association Study | Genetic Association Studies | Membrane Proteins - genetics | Risk Factors | Linkage Disequilibrium - genetics | Potassium Channels, Tandem Pore Domain - genetics | Chromosome Mapping | Membrane Glycoproteins - genetics | Aggregatibacter actinomycetemcomitans - genetics | Adaptor Protein Complex 3 - genetics | Polymorphism, Single Nucleotide - genetics | Aged | Receptors, G-Protein-Coupled - genetics | Mucins - genetics | Cohort Studies | GPI-Linked Proteins - genetics | Index Medicus | Dentistry | Research Reports
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 03/2015, Volume 42, Issue 3, pp. 431 - 442
... cells that are often deficient for multiple transcription factors and signaling proteins, including... 
Life Sciences & Biomedicine | Immunology | Science & Technology | Cell Proliferation | Humans | NK Cell Lectin-Like Receptor Subfamily C - genetics | Receptors, IgG - deficiency | Intracellular Signaling Peptides and Proteins - immunology | Cytomegalovirus Infections - immunology | Gene Expression Profiling | Killer Cells, Natural - pathology | Cytomegalovirus Infections - genetics | Protein-Tyrosine Kinases - deficiency | Protein-Tyrosine Kinases - immunology | Intracellular Signaling Peptides and Proteins - deficiency | NK Cell Lectin-Like Receptor Subfamily C - immunology | DNA Methylation | Protein-Tyrosine Kinases - genetics | Receptors, IgG - genetics | Microarray Analysis | Antibodies - immunology | Cytomegalovirus Infections - virology | Killer Cells, Natural - immunology | Cytomegalovirus Infections - pathology | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Promoter Regions, Genetic | GPI-Linked Proteins - immunology | Signal Transduction | Killer Cells, Natural - virology | NK Cell Lectin-Like Receptor Subfamily C - deficiency | Immunophenotyping | Receptors, IgG - immunology | Killer Cells, Natural - classification | Cytomegalovirus - immunology | Epigenesis, Genetic - immunology | Immunologic Memory | GPI-Linked Proteins - genetics | Proteins | Viral antibodies | Epigenetic inheritance | Killer cells | Molecular genetics | Antibodies | Gene expression | Antigens | Cytomegalovirus | Cloning | Colleges & universities | Infections | Experiments | Long term | DNA methylation | Epigenetics | Expansion | Viral infections | Deoxyribonucleic acid--DNA | Immune system | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, pp. e46307 - e46307
Bone morphogenetic proteins (BMPs) are members of the transforming growth factor beta superfamily that exert their effects via type I and type II serine threonine kinase receptors and the SMAD intracellular signaling pathway... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Bone Morphogenetic Protein 5 - chemistry | Bone Morphogenetic Protein 6 - chemistry | Bone Morphogenetic Protein 7 - chemistry | Hemochromatosis Protein | Bone Morphogenetic Protein 7 - genetics | Humans | Nerve Tissue Proteins - chemistry | Transfection | Bone Morphogenetic Protein 6 - genetics | Solutions | Growth Differentiation Factors - genetics | Genes, Reporter | Cell Line | Cell Adhesion Molecules, Neuronal - chemistry | Signal Transduction | Membrane Proteins - genetics | Bone Morphogenetic Protein 5 - genetics | Recombinant Proteins - chemistry | Histocompatibility Antigens Class I - genetics | Recombinant Proteins - genetics | Histocompatibility Antigens Class I - chemistry | Nerve Tissue Proteins - genetics | Cell Adhesion Molecules, Neuronal - genetics | GPI-Linked Proteins - chemistry | Membrane Proteins - chemistry | Protein Binding | Ligands | Kinetics | GPI-Linked Proteins - genetics | Growth Differentiation Factors - chemistry | Physiological aspects | Bone morphogenetic proteins | Genetic aspects | Research | Protein binding | Smad protein | Phosphorylation | Nephrology | Transforming growth factor-b | Homeostasis | Intracellular signalling | Iron | Biology | Kinases | Medical schools | Proteins | Signal transduction | Receptors | Rodents | Growth factors | Binding | Bone morphogenetic protein 6 | Bone morphogenetic protein 5 | Bone morphogenetic protein 4 | Bone morphogenetic protein 2 | Threonine | Research & development--R&D | Anemia | Affinity | Protein-serine/threonine kinase | Surface plasmon resonance | Mutation | Receptor mechanisms | Bone morphogenetic protein 9 | Bone morphogenetic protein 7 | Index Medicus | Research & development | R&D
Journal Article
Virus research, ISSN 0168-1702, 2011, Volume 162, Issue 1, pp. 12 - 18
Influenza A and B viruses are a major cause of respiratory disease in humans. In addition, influenza A viruses continuously re-emerge from animal reservoirs... 
Influenza A | Review | Type 1 interferon | Influenza B | Life Sciences & Biomedicine | Science & Technology | Virology | Antigens, CD - immunology | Ubiquitins - genetics | Virus Replication - genetics | Humans | Viral Proteins - immunology | eIF-2 Kinase - immunology | Interferon Type I - immunology | Antigens, CD - genetics | GTP-Binding Proteins - genetics | Viral Proteins - metabolism | Host-Pathogen Interactions - immunology | Orthomyxoviridae - immunology | Interferon Type I - biosynthesis | Myxovirus Resistance Proteins | Influenza, Human - virology | Cytokines - genetics | Cytokines - immunology | eIF-2 Kinase - genetics | DEAD Box Protein 58 | GPI-Linked Proteins - immunology | Ubiquitins - immunology | GTP-Binding Proteins - immunology | Toll-Like Receptors - immunology | Orthomyxoviridae - metabolism | Viral Proteins - genetics | Virus Replication - immunology | Immunity, Innate | Proteins - immunology | 2',5'-Oligoadenylate Synthetase - genetics | Proteins - genetics | DEAD-box RNA Helicases - genetics | Animals | Immune Evasion | 2',5'-Oligoadenylate Synthetase - immunology | Toll-Like Receptors - genetics | DEAD-box RNA Helicases - immunology | Antigens, Differentiation - genetics | Mice | Antigens, Differentiation - immunology | GPI-Linked Proteins - genetics | Influenza, Human - immunology | Influenza viruses | Interferon | Biological response modifiers | Health aspects | Influenza | Index Medicus
Journal Article
PLoS genetics, ISSN 1553-7404, 2006, Volume 2, Issue 8, pp. e131 - 1206
The transcription factor signal transducer and activator of transcription-1 (STAT1) plays a key role in immunity against mycobacterial and viral infections.... 
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Humans | GPI-Linked Proteins | Child, Preschool | Infant | Male | Interferon-gamma - metabolism | DNA-Binding Proteins - metabolism | STAT1 Transcription Factor - metabolism | Transfection - methods | Genes, Dominant | DNA Mutational Analysis | Mycobacterium Infections - genetics | Adult | Female | Transcription, Genetic | Membrane Proteins - metabolism | Child | Interferon-alpha - metabolism | Mycobacterium Infections - etiology | Cell Cycle Proteins - metabolism | Cells, Cultured | Gene Expression Regulation | Models, Molecular | STAT1 Transcription Factor - genetics | Genes, Recessive | Interferon-Stimulated Gene Factor 3, gamma Subunit - metabolism | Immunity, Active - genetics | Models, Biological | Pedigree | Mutant Proteins - chemistry | Adolescent | Alleles | Protein Binding | Heterozygote | Mutation | Allelomorphism | Mycobacterial infections | Genetic aspects | Research | Health aspects | Proteins | Phosphorylation | Disease | Respiratory distress syndrome | Cloning | Genetics | Kinases | Experiments | Viral infections | Index Medicus | Interferon-Stimulated Gene Factor 3, gamma Subunit | Adaptive immunology | Life Sciences | Immunology | Interferon Type II | DNA-Binding Proteins | Immunity, Active | STAT1 Tra | Interferon-alpha | Mycobacterium Infections | Membrane Proteins | Mutant Proteins | Cell Cycle Proteins | Infectious Diseases | Homo (Human) | Genetics of Disease
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 09/2013, Volume 288, Issue 36, pp. 25938 - 25949
NAD(+) is mainly synthesized in human cells via the "salvage" pathways starting from nicotinamide, nicotinic acid, or nicotinamide riboside (NR). The... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | 5'-Nucleotidase - genetics | Gene Expression Regulation, Enzymologic - drug effects | Humans | Membrane Glycoproteins - biosynthesis | GPI-Linked Proteins - biosynthesis | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Nicotinamide Phosphoribosyltransferase - antagonists & inhibitors | Neoplasms - genetics | Nicotinamide Phosphoribosyltransferase - metabolism | Piperidines - pharmacology | NAD - biosynthesis | Cell Death - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Acrylamides - pharmacology | Cytokines - genetics | Gene Expression Regulation, Neoplastic - genetics | 5'-Nucleotidase - biosynthesis | NAD - genetics | ADP-ribosyl Cyclase 1 - genetics | Cytokines - metabolism | Gene Silencing | Neoplasms - enzymology | Down-Regulation - drug effects | Down-Regulation - genetics | Membrane Glycoproteins - genetics | Neoplasms - drug therapy | Nicotinamide Phosphoribosyltransferase - genetics | Gene Expression Regulation, Enzymologic - genetics | ADP-ribosyl Cyclase 1 - biosynthesis | Cell Line, Tumor | Cytokines - antagonists & inhibitors | Nicotinamide Mononucleotide - biosynthesis | Nicotinamide Mononucleotide - genetics | GPI-Linked Proteins - genetics | Index Medicus | NAD | Cancer Therapy | CD73 | Nicotinamide Riboside | NAD Biosynthesis | Signal Transduction | Cell Death | Nicotinamide Mononucleotide | Nicotinamide
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 06/2011, Volume 108, Issue 25, pp. 10337 - 10342
Spinal muscular atrophy (SMA), caused by the deletion of the SMN1 gene, is the leading genetic cause of infant mortality... 
Axons | Motor neurons | Growth cones | Neuroscience | Messenger RNA | RNA | Neurons | Antibodies | Gene expression regulation | SMN complex proteins | Neuritin | Local protein synthesis | Embryonic lethal abnormal vision drosophila-like 4 (ELAV-L4) | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | ELAV-Like Protein 4 | Humans | RNA, Messenger - metabolism | Zebrafish - embryology | Recombinant Fusion Proteins - metabolism | Embryo, Mammalian - anatomy & histology | Motor Neurons - cytology | Survival of Motor Neuron 1 Protein - genetics | Neuropeptides - genetics | Animals, Genetically Modified | RNA, Messenger - genetics | Cells, Cultured | ELAV Proteins - metabolism | Axons - metabolism | Neuropeptides - metabolism | Survival of Motor Neuron 1 Protein - metabolism | Nerve Tissue Proteins - genetics | GPI-Linked Proteins - metabolism | Motor Neurons - metabolism | Nerve Tissue Proteins - metabolism | Embryo, Mammalian - physiology | Animals | Axons - pathology | Recombinant Fusion Proteins - genetics | Zebrafish - physiology | Mice | ELAV Proteins - genetics | GPI-Linked Proteins - genetics | Gene mutations | Physiological aspects | Genetic aspects | Research | Health aspects | Risk factors | Spinal muscular atrophy | Index Medicus | Spinal cord | Translation | Cell survival | Mental disorders | Axonogenesis | Mortality | SMN protein | survival | Mass spectroscopy | Infants | Gene deletion | infant mortality | HuD protein | Proteins | Pathology | RNA-binding protein | Actin | Neuromuscular junctions | spinal muscular atrophy | Axon guidance | Mass spectrometry | Biological Sciences | neuritin | embryonic lethal abnormal vision Drosophila-like 4 (ELAV-L4) | local protein synthesis
Journal Article
Gastroenterology, ISSN 0016-5085, 2016, Volume 152, Issue 1, pp. 206 - 217.e2
Journal Article