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Nature, ISSN 0028-0836, 01/2008, Volume 451, Issue 7177, pp. 425 - 430
Journal Article
Journal Article
eLife, ISSN 2050-084X, 06/2015, Volume 4, Issue JUNE, pp. 1 - 21
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2012, Volume 109, Issue 47, pp. 19071 - 19078
Neurons within each layer in the mammalian cortex have Stereotypic projections. Four genes—Fezf2, Ctip2, Tbr1, and Satb2—regulate these projection identities.... 
Axons | Brain | Mesencephalon | Neurons | Thalamus | Genetic loci | Gene expression regulation | Pyramidal tracts | Repression | Identity | Cerebral cortex | Cell fate | Axon guidance | Transcription factor | transcription factor | CALLOSAL | cell fate | MULTIDISCIPLINARY SCIENCES | AUTISM SPECTRUM DISORDER | TBR1 | GUIDANCE | SPECIFICATION | DEVELOPING CEREBRAL-CORTEX | cerebral cortex | NEURON IDENTITY | CORTICOSPINAL TRACT | NETRIN-1 | DIFFERENTIATION | axon guidance | Axons - enzymology | Thalamus - metabolism | Alkaline Phosphatase - metabolism | Genetic Loci - genetics | Receptor, EphA4 - metabolism | Gene Regulatory Networks | Cerebral Cortex - metabolism | DNA-Binding Proteins - metabolism | Neocortex - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Gene Expression Regulation, Developmental | Isoenzymes - metabolism | Cell Adhesion Molecules, Neuronal - metabolism | Neurons - metabolism | Netrin Receptors | Repressor Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Neocortex - growth & development | Receptors, Cell Surface - metabolism | Repressor Proteins - genetics | Nuclear Proteins - metabolism | DNA-Binding Proteins - genetics | Mutation - genetics | GPI-Linked Proteins - metabolism | Nerve Tissue Proteins - metabolism | Animals | Protein Binding | Mice | Physiological aspects | Genetic aspects | Gene mutations | Neocortex | Mutation | Gene expression | Medical disorders | Genes | Index Medicus | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2011, Volume 108, Issue 25, pp. 10337 - 10342
Spinal muscular atrophy (SMA), caused by the deletion of the SMN1 gene, is the leading genetic cause of infant mortality. SMN protein is present at high levels... 
Axons | Motor neurons | Growth cones | Neuroscience | Messenger RNA | RNA | Neurons | Antibodies | Gene expression regulation | SMN complex proteins | Neuritin | Local protein synthesis | Embryonic lethal abnormal vision drosophila-like 4 (ELAV-L4) | LOCALIZATION | RIBONUCLEOPROTEIN | MULTIDISCIPLINARY SCIENCES | ACTIVITY-DEPENDENT EXPRESSION | IDENTIFICATION | neuritin | GROWTH CONES | TRANSPORT | DETERMINING GENE-PRODUCT | SPINAL MUSCULAR-ATROPHY | IN-VIVO | ASSOCIATION | embryonic lethal abnormal vision Drosophila-like 4 (ELAV-L4) | local protein synthesis | ELAV-Like Protein 4 | Humans | RNA, Messenger - metabolism | Zebrafish - embryology | Recombinant Fusion Proteins - metabolism | Embryo, Mammalian - anatomy & histology | Motor Neurons - cytology | Survival of Motor Neuron 1 Protein - genetics | Neuropeptides - genetics | Animals, Genetically Modified | RNA, Messenger - genetics | Cells, Cultured | ELAV Proteins - metabolism | Axons - metabolism | Neuropeptides - metabolism | Survival of Motor Neuron 1 Protein - metabolism | Nerve Tissue Proteins - genetics | GPI-Linked Proteins - metabolism | Motor Neurons - metabolism | Nerve Tissue Proteins - metabolism | Embryo, Mammalian - physiology | Animals | Axons - pathology | Recombinant Fusion Proteins - genetics | Zebrafish - physiology | Mice | ELAV Proteins - genetics | GPI-Linked Proteins - genetics | Gene mutations | Physiological aspects | Genetic aspects | Research | Health aspects | Risk factors | Spinal muscular atrophy | Proteins | Genotype & phenotype | Neuromuscular diseases | Zebrafish | Infant mortality | Ribonucleic acid--RNA | Index Medicus | Spinal cord | Translation | Cell survival | Mental disorders | Axonogenesis | Mortality | SMN protein | survival | Mass spectroscopy | Infants | Gene deletion | infant mortality | HuD protein | Pathology | RNA-binding protein | Actin | Neuromuscular junctions | spinal muscular atrophy | Axon guidance | Mass spectrometry | Biological Sciences
Journal Article
Nature Neuroscience, ISSN 1097-6256, 05/2012, Volume 15, Issue 5, pp. 703 - 712
In the adult mammalian CNS, chondroitin sulfate proteoglycans (CSPGs) and myelin-associated inhibitors (MAIs) stabilize neuronal structure and restrict... 
RETINAL GANGLION-CELLS | NEURITE GROWTH | AXON REGENERATION | OPTIC-NERVE REGENERATION | SPINAL-CORD-INJURY | TYROSINE-PHOSPHATASE-SIGMA | CORTICOSPINAL TRACT REGENERATION | NOGO RECEPTOR | OLIGODENDROCYTE-MYELIN GLYCOPROTEIN | FUNCTIONAL RECEPTOR | NEUROSCIENCES | Protein Binding - genetics | Embryo, Mammalian | Humans | Myelin Proteins - deficiency | Nerve Regeneration - physiology | Myelin-Associated Glycoprotein - genetics | Myelin-Associated Glycoprotein - metabolism | Ganglia, Spinal - cytology | Dose-Response Relationship, Drug | Tubulin - metabolism | Transfection | GPI-Linked Proteins - deficiency | Receptor-Like Protein Tyrosine Phosphatases, Class 4 - pharmacology | Protein Binding - drug effects | Optic Nerve Injuries - metabolism | Neurons - metabolism | Nogo Receptor 1 | Neurons - drug effects | Receptors, Tumor Necrosis Factor - genetics | Animals, Newborn | Receptors, Tumor Necrosis Factor - metabolism | Gene Expression Regulation - genetics | Cells, Cultured | Chondroitin Sulfate Proteoglycans - metabolism | Gene Expression Regulation - physiology | Rats | Receptors, Cell Surface - metabolism | Mutation - genetics | Myelin Proteins - genetics | GPI-Linked Proteins - metabolism | Mice, Knockout | Gene Expression Regulation - drug effects | Animals | Analysis of Variance | Central Nervous System - cytology | Receptors, Cell Surface - deficiency | Mice | Myelin Proteins - metabolism | GPI-Linked Proteins - genetics | Receptors, Cell Surface - genetics | Neuroplasticity | Physiological aspects | Proteoglycans | Research | Chondroitin | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 08/2014, Volume 111, Issue 32, pp. E3297 - E3305
Journal Article
Brain, ISSN 0006-8950, 2012, Volume 135, Issue 6, pp. 1794 - 1818
Multiple sclerosis involves demyelination and axonal degeneration of the central nervous system. The molecular mechanisms of axonal degeneration are relatively... 
collapsin response mediator protein 2 | Nogo receptor | axonal degeneration | experimental autoimmune encephalomyelitis | Nogo-A | ENCEPHALOMYELITIS | FUNCTIONAL RECOVERY | RESPONSE MEDIATOR PROTEIN-2 | ALZHEIMERS-DISEASE | AXONAL REGENERATION | RHO-KINASE | NEUROSCIENCES | LESIONS | CLINICAL NEUROLOGY | MYELIN OLIGODENDROCYTE GLYCOPROTEIN | HYPERPHOSPHORYLATION | INHIBITOR | Humans | Middle Aged | Myelin Proteins - deficiency | Encephalomyelitis, Autoimmune, Experimental - immunology | tau Proteins - metabolism | Male | Green Fluorescent Proteins - genetics | Receptors, Cell Surface - antagonists & inhibitors | Nerve Degeneration - metabolism | Retinal Ganglion Cells - pathology | Time Factors | Nogo Receptor 1 | GPI-Linked Proteins - antagonists & inhibitors | Disease Models, Animal | Transduction, Genetic | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Myelin Proteins - antagonists & inhibitors | Silver Staining | Myelin-Oligodendrocyte Glycoprotein | Glycoproteins - adverse effects | Mutation - genetics | CD3 Complex - metabolism | Receptors, Cell Surface - immunology | Mice, Knockout | Analysis of Variance | Axons - pathology | Encephalomyelitis, Autoimmune, Experimental - complications | Receptors, Cell Surface - deficiency | Cell Line, Tumor | Mice | Peptide Fragments - adverse effects | Nerve Degeneration - etiology | Neurofilament Proteins - metabolism | Optic Nerve - metabolism | Optic Nerve - pathology | Phosphorylation | Immunoprecipitation | Spinal Cord - metabolism | Demyelinating Diseases - metabolism | Retinal Ganglion Cells - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Tubulin - metabolism | GPI-Linked Proteins - deficiency | Spinal Cord - pathology | Axons - ultrastructure | Myelin Proteins - immunology | Adult | Female | Demyelinating Diseases - etiology | Demyelinating Diseases - pathology | Neuroblastoma - pathology | Severity of Illness Index | Antibodies - therapeutic use | Green Fluorescent Proteins - metabolism | GPI-Linked Proteins - immunology | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Intercellular Signaling Peptides and Proteins - genetics | Axons - metabolism | Nerve Tissue Proteins - genetics | Multiple Sclerosis - complications | Nerve Tissue Proteins - metabolism | Animals | Multiple Sclerosis - pathology | Index Medicus | Abridged Index Medicus | Original
Journal Article