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2004, 2nd ed., Advanced Texts, ISBN 1859962475, 269
Molecular Biology of Cancer has been extensively revised and covers heredity cancer, microarray technology and increased study of childhood cancers. It... 
Genetic aspects | Molecular diagnosis | Molecular aspects | Cancer | Clinical & internal medicine | Molecular Biology | General Science | Cell Biology
eBook
2003, Cancer drug discovery and development., ISBN 9780896039308, x, 326
Book
2000, Current topics in microbiology and immunology, ISBN 9783540666080, Volume 249., vi, 169
Book
1995, 2nd ed., Jones and Bartlett series in biology, ISBN 9780867209372, xv, 384
Book
Immunology, ISSN 0019-2805, 05/2018, Volume 154, Issue 1, pp. 144 - 155
Summary A recent study indicated that Lectin‐type oxidized LDL receptor‐1 (LOX‐1) was a distinct surface marker for human polymorphisms myeloid‐derived... 
polymorphonuclear myeloid‐derived suppressor cell | lectin‐type oxidized LDL receptor‐1 | prognosis | endoplasmic reticulum stress | hepatocelluar carcinoma patients | polymorphonuclear myeloid-derived suppressor cell | lectin-type oxidized LDL receptor-1 | LYMPHOCYTE RATIO ACTS | PROGNOSTIC-FACTOR | DISEASE | IMMUNOLOGY | Cell Proliferation | Reactive Oxygen Species - metabolism | Coculture Techniques | Humans | Arginase - metabolism | Lewis X Antigen - metabolism | Case-Control Studies | T-Lymphocytes - metabolism | Liver Neoplasms - pathology | Carcinoma, Hepatocellular - immunology | Myeloid-Derived Suppressor Cells - metabolism | Myeloid-Derived Suppressor Cells - pathology | Myeloid-Derived Suppressor Cells - immunology | Signal Transduction | Lymphocyte Activation | Cells, Cultured | Liver Neoplasms - immunology | Fucosyltransferases - metabolism | Scavenger Receptors, Class E - metabolism | Endoplasmic Reticulum Stress | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Interferons - metabolism | T-Lymphocytes - immunology | Carcinoma, Hepatocellular - metabolism | Oxidases | Hepatitis | Liver | Low density lipoproteins | Lectins | Stress (Physiology) | Hepatoma | T cells | Liver cirrhosis | Protein binding | Cell proliferation | Lipoproteins (low density) | Arginase | Hepatocellular carcinoma | Lymphocytes T | Suppressor cells | NAD(P)H oxidase | Liver cancer | Cell growth | Lymphocytes | CYBB protein | Hepatitis B virus | Stresses | Cell survival | Nucleotide sequence | Gene expression | Ribonucleic acid--RNA | Patients | Stress | Cirrhosis | Inhibitors | Liquid oxygen | Medical prognosis | Interferon | Endoplasmic reticulum | Surface markers | Hepatitis B | Original
Journal Article
British Journal of Haematology, ISSN 0007-1048, 07/2017, Volume 178, Issue 2, pp. 292 - 301
Journal Article
Cancer Immunology, Immunotherapy, ISSN 0340-7004, 8/2013, Volume 62, Issue 8, pp. 1421 - 1430
Myeloid-derived suppressor cells (MDSCs) are known as key immune regulators in various human malignancies, and it is reported that CD14+HLA-DR−/low MDSCs are... 
Recurrence | Immunology | Medicine & Public Health | Myeloid-derived suppressor cells | Radiofrequency ablation | Oncology | Cancer Research | Hepatocellular carcinoma | Cancer | IMMUNE CELLS | RISK-FACTORS | DENDRITIC CELLS | PHASE-II | PERIPHERAL-BLOOD | IMMUNOLOGY | CLINICAL CANCER STAGE | ONCOLOGY | INFLAMMATION | REGULATORY T-CELLS | DIFFERENTIATION | CORRELATE | Multivariate Analysis | T-Lymphocytes, Regulatory - metabolism | Prognosis | Cell Count | Humans | Interleukin-10 - blood | Interleukin-13 - immunology | Catheter Ablation | Male | T-Lymphocytes, Regulatory - pathology | Lipopolysaccharide Receptors - metabolism | T-Lymphocytes, Regulatory - immunology | Myeloid Cells - immunology | Female | Liver Neoplasms - pathology | Carcinoma, Hepatocellular - immunology | HLA-DR Antigens - metabolism | Interleukin-13 - blood | Cytokines - blood | Cytokines - immunology | Kaplan-Meier Estimate | Neoplasm Recurrence, Local | Proportional Hazards Models | Vascular Endothelial Growth Factor A - blood | Carcinoma, Hepatocellular - surgery | Lipopolysaccharide Receptors - immunology | Liver Neoplasms - immunology | Liver Neoplasms - surgery | Vascular Endothelial Growth Factor A - immunology | Disease Progression | Carcinoma, Hepatocellular - pathology | Myeloid Cells - metabolism | Aged | HLA-DR Antigens - immunology | Myeloid Cells - pathology | Flow Cytometry - statistics & numerical data | Interleukin-10 - immunology
Journal Article
Oncogene, ISSN 0950-9232, 09/2008, Volume 27, Issue 38, pp. 5115 - 5123
Cancer development results from deregulated control of stem cell populations and alterations in their surrounding environment. Notch signaling is an important... 
Epigenetics | Keratinocytes | Cancer stem cells | p63 | Rho signaling | p53 | SIGNALING PATHWAYS | RBP-J-KAPPA | HAIR FOLLICLE | BIOCHEMISTRY & MOLECULAR BIOLOGY | keratinocytes | BETA-CATENIN | P53 HOMOLOG | epigenetics | CELL BIOLOGY | ONCOLOGY | GROWTH ARREST | CERVICAL-CANCER CELLS | GENETICS & HEREDITY | NEGATIVE TRANSCRIPTIONAL REGULATOR | KERATINOCYTE STEM-CELLS | cancer stem cells | NF-KAPPA-B | Species Specificity | Keratinocytes - radiation effects | Humans | Receptors, Notch - genetics | Tumor Suppressor Protein p53 - physiology | Neoplasms - virology | Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Tumor Virus Infections - physiopathology | Tumor Suppressor Proteins - genetics | Female | Neoplasms - physiopathology | Cell Differentiation - physiology | Genes, Tumor Suppressor | Oncogene Proteins, Viral - physiology | Receptors, Notch - physiology | Uterine Cervical Neoplasms - physiopathology | Uterine Cervical Neoplasms - genetics | Ultraviolet Rays - adverse effects | Tumor Suppressor Proteins - physiology | Keratinocytes - pathology | Animals | Uterine Cervical Neoplasms - virology | Signal Transduction - physiology | Mice | Apoptosis - physiology | DNA Damage | Receptor, Notch1 - genetics | Receptor, Notch1 - physiology | Care and treatment | Tumor suppressor genes | Cellular signal transduction | Genetic aspects | Research | Health aspects | Risk factors | Cancer | Signal transduction | Gene expression | Stem cells | Cell cycle | cancer therapy | carcinogenesis
Journal Article