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Journal Article
Journal Article
Anti-Cancer Agents in Medicinal Chemistry, ISSN 1871-5206, 2018, Volume 18, Issue 13, pp. 1885 - 1891
Background: Recently, we have reported that Fisetin, a natural flavonol, is able to regress AflatoxinB1 (AFB1) induced hepatocellular carcinoma (HCC) by... 
Fisetin | pAkt | p-GSK3β | AflatoxinB1 (AFB1) | Hepatocellular carcinoma (HCC) | PDK1 | TARGET | ACTIVATION | CHEMISTRY, MEDICINAL | CYCLIN D1 | PHOSPHORYLATION | PROTEIN-KINASE | pAkt(Th308) | CELL-PROLIFERATION | CANCER | ONCOLOGY | p-GSK3 beta(Ser9) | PROGRESSION
Journal Article
Journal Article
Brain Research, ISSN 0006-8993, 2014, Volume 1581, pp. 129 - 140
Abstract The multifaceted pathogenesis of temporal lobe epilepsy (TLE) offers a number of adjunctive therapeutic prospects. One such therapeutic strategy could... 
Neurology | Kainic acid (KA) | cAMP response element binding protein (CREB) | 4-(2-{2-[(2R)-2-methylpyrrolidinyl]ethyl}-benzofuran-5-yl)benzonitrile (ABT-239) | Phospho-GSK3β (Ser9, pGSK3βser9) | Neurodegeneration | Histamine H3 receptor (H3R) | Phospho-GSK3β (Ser9, pGSK3β | ABT-239 | 4-(2-{2-[(2R)-2-methylpyrrolidinyl]ethyl}-benzofuran-5-yl)benzonitrile | Neurons - pathology | Gene Expression - drug effects | Seizures - drug therapy | Benzofurans - pharmacology | Glycogen Synthase Kinase 3 beta | Male | Hippocampus - drug effects | Neurodegenerative Diseases - drug therapy | Anticonvulsants - pharmacology | Valproic Acid - pharmacology | Kainic Acid | Pyrrolidines - pharmacology | Dose-Response Relationship, Drug | Seizures - physiopathology | Neuroprotective Agents - pharmacology | Seizures - pathology | Neurons - physiology | Drug Therapy, Combination | Neurons - drug effects | Thiadiazoles - pharmacology | Neurodegenerative Diseases - pathology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Hippocampus - pathology | Random Allocation | Glycogen Synthase Kinase 3 - metabolism | Histamine H3 Antagonists - pharmacology | Animals | Neurodegenerative Diseases - physiopathology | Mice | Protein Kinase Inhibitors - pharmacology | Hippocampus - physiopathology | Histamine | Glucose metabolism | Synthesis | Glycogen | Analysis | Epilepsy | Seizures (Medicine) | Protein binding | Index Medicus
Journal Article
by Gao, XJ and He, YJ and Gao, LM and Feng, JX and Xie, YY and Liu, XH and Liu, LX
BIOCHEMISTRY AND CELL BIOLOGY-BIOCHIMIE ET BIOLOGIE CELLULAIRE, ISSN 0829-8211, 10/2014, Volume 92, Issue 5, pp. 349 - 356
The activity of glycogen synthase kinase beta (GSK3 beta) is mainly regulated by its Ser9 phosphorylation. It has been believed for a long time that Ser9... 
CANCER-CELLS | SURVIVAL | ACTIVATION | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | Ser9 phosphorylation | apoptosis | BETA-CATENIN | FATE | TAU-PHOSPHORYLATION | KINASE 3-BETA | CELL BIOLOGY | GLYCOGEN-SYNTHASE KINASE-3-BETA | INHIBITION | interaction | 14-3-3 zeta | GSK3 beta
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 12/2006, Volume 26, Issue 12, pp. 1479 - 1489
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 10/2017, Volume 56, Issue 10, pp. 2301 - 2316
Although GSK3 has been reported to have contrasting effects on the progression of different tumors, it's possible functions in esophageal squamous cell... 
GSK3β | PI3‐kinase | esophageal cancer | STAT3 | PI3-kinase | CANCER-CELLS | SURVIVAL | ACTIVATION | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLIFERATION | BREAST-CANCER | ONCOLOGY | GSK3 | SIGNALING PATHWAY | GSK3-BETA | EXPRESSION | EPIDEMIOLOGY | Up-Regulation | Phosphorylation | Prognosis | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Gene Expression Regulation, Neoplastic | Male | Esophageal Neoplasms - pathology | Lithium Chloride - pharmacology | Esophageal Neoplasms - metabolism | Female | STAT3 Transcription Factor - metabolism | Esophageal Squamous Cell Carcinoma | Cell Survival - drug effects | Tyrphostins - pharmacology | Glycogen Synthase Kinase 3 beta - metabolism | Disease Progression | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Survival Analysis | Cell Line, Tumor | Mice | Pyridines - pharmacology | Development and progression | Squamous cell carcinoma | Synthesis | Glycogen | Analysis | Esophageal cancer | Leukocyte migration | Lithium chloride | Kinases | Tissues | Metastases | Cell adhesion & migration | Xenografts | Inhibition | Data analysis | Stat3 protein | Glycogen synthase kinase 3 | Data processing | Pharmacology | siRNA | Esophagus | Molecular modelling | Inhibitors | Cell lines | Differentiation | Viability | In vitro methods and tests | Cell migration | Tumors | Cancer | Esophageal Cancer | PI3-Kinase
Journal Article
Journal of Hepatology, ISSN 0168-8278, 07/2018, Volume 69, Issue 1, pp. 99 - 109
Journal Article
Journal Article
Oncotarget, ISSN 1949-2553, 2017, Volume 8, Issue 8, pp. 13632 - 13651
We previously reported the role of cancer/testis antigen CAGE in the response to anti-cancer drugs. CAGE increased the expression of cyclinD1, and pGSK3... 
GSK3β | CyclinD1 | CAGE | Peptides | Anti-cancer drug-resistance | CANCER/TESTIS ANTIGEN CAGE | PHOSPHORYLATION | cyclinD1 | peptides | BETA-CATENIN | CANCER | anti-cancer drug-resistance | PEPTIDE | FEEDBACK LOOP | CELL BIOLOGY | TUMOR-CELL GROWTH | GENE CAGE | IN-VIVO | GSK3 beta
Journal Article
Behavioural Brain Research, ISSN 0166-4328, 10/2019, Volume 372, p. 112033
Binge drinking is a pattern of alcohol intake characterized by excessive and intermittent alcohol consumption over a very short period of time that is more... 
NMDA | Adolescence | GSK3β | Intermittent ethanol drinking | AMPA | Spatial memory | BINGE DRINKING | METABOTROPIC GLUTAMATE RECEPTORS | SYNAPTIC PLASTICITY | NEUROSCIENCES | ADOLESCENT | GLYCOGEN-SYNTHASE KINASE-3-BETA | MESSENGER-RNA | BEHAVIORAL SCIENCES | GSK3 beta | LONG-TERM POTENTIATION | DENTATE GYRUS | ALCOHOL EXPOSURE | WORKING-MEMORY
Journal Article