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Anticancer Research, ISSN 0250-7005, 08/2018, Volume 38, Issue 8, pp. 4535 - 4542
Background: Chemoresistance of cancer stem cells (CSCs) is considered a major cause of post-treatment recurrence that negatively impacts the prognosis of... 
Drug repositioning | Repurposing | Drug resistance | Apoptosis | H3K27 DEMETHYLASE INHIBITOR | ONCOLOGY | GSKJ4 | apoptosis | repurposing | drug resistance | DRUG-RESISTANCE | SIRNA
Journal Article
by Yan, NN and Xu, L and Wu, XB and Zhang, L and Fei, XC and Cao, YL and Zhang, FC
EXPERIMENTAL CELL RESEARCH, ISSN 0014-4827, 10/2017, Volume 359, Issue 2, pp. 405 - 414
Recently, studies have been suggested that H3K27me3 is implicated with maintenance of cancer stem cells (CSCs), however, the roles of H3K27me3 in Breast cancer... 
JMJD3 | LYSINE-27 | GSKJ4 | SELF-RENEWAL | TUMORIGENICITY | CELL BIOLOGY | THERAPY | ONCOLOGY | Epigenetic modification | HISTONE H3 | OVARIAN | DIFFERENTIATION | Breast cancer stem cells | EXPRESSION | H3K27me3 | CHROMATIN STATE
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 2018, Volume 9, p. 2272
Sepsis, defined as life-threatening organ dysfunction, is one of the most common causes of mortality in intensive care units with limited therapeutic options.... 
JMJD3 | Sepsis | MiR-146a | Inflammation | GSKJ4 | RESPONSES | INHIBITION | inflammation | DEMETHYLASE JMJD3 | miR-146a | INDUCTION | IMMUNOLOGY | EXPRESSION | sepsis | MESENCHYMAL STEM-CELLS | Epigenetic inheritance | Genetic aspects | MicroRNA | Research | Genetic transcription
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 05/2018, Volume 17, Issue 5, pp. 1070 - 1078
Journal Article
International Journal of Cancer, ISSN 0020-7136, 12/2018, Volume 143, Issue 12, pp. 3106 - 3119
Ovarian high‐grade serous carcinoma (HGSC) is the most lethal gynecological malignancy. Prevailing evidences suggest that drug resistance and recurrence of... 
UTX | cancer stem cells | GATA3 | ovarian cancer | GSKJ4 | MOLECULAR SUBTYPES | DEMETHYLASE INHIBITOR | RECEPTOR | PROLIFERATION | IDENTIFICATION | BREAST-CANCER | GENE | ONCOLOGY | SUPPRESSES | DIFFERENTIATION | EXPRESSION | Prognosis | Cadherins - metabolism | Humans | Alkaline Phosphatase - metabolism | GATA3 Transcription Factor - physiology | Ovarian Neoplasms - pathology | Spheroids, Cellular - enzymology | Antigens, CD - metabolism | Antineoplastic Combined Chemotherapy Protocols - pharmacology | GATA3 Transcription Factor - drug effects | Neoplastic Stem Cells - metabolism | Biomarkers, Tumor - metabolism | Neoplastic Stem Cells - pathology | Female | Cell Differentiation | Ovarian Neoplasms - metabolism | Paclitaxel - administration & dosage | GATA3 Transcription Factor - metabolism | Ovarian Neoplasms - drug therapy | Deoxycytidine - administration & dosage | Spheroids, Cellular - metabolism | Nuclear Proteins - metabolism | Cell Lineage | Histone Demethylases - metabolism | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Cell Line, Tumor | Protein Binding | Deoxycytidine - analogs & derivatives | Epigenetic inheritance | Carcinoma | Lysine | Analysis | Stem cells | Drug resistance | Health aspects | Ovarian cancer | Cancer | Phenotypes | Gemcitabine | Therapeutic applications | Cytotoxicity | Malignancy | Progenitor cells | Cells (biology) | Cell lines | Paclitaxel | GATA-3 protein | Biomarkers | Bioindicators | Histone H3 | Apoptosis
Journal Article
Experimental Cell Research, ISSN 0014-4827, 10/2017, Volume 359, Issue 2, pp. 405 - 414
Recently, studies have been suggested that H3K27me3 is implicated with maintenance of cancer stem cells (CSCs), however, the roles of H3K27me3 in Breast cancer... 
Epigenetic modification | Breast cancer stem cells | GSKJ4 | H3K27me3 | RNA, Small Interfering - genetics | Histone Demethylases - antagonists & inhibitors | Neoplastic Stem Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | Spheroids, Cellular - pathology | Histone Demethylases - genetics | SOXB1 Transcription Factors - metabolism | Octamer Transcription Factor-3 - genetics | MCF-7 Cells | Neoplastic Stem Cells - metabolism | SOXB1 Transcription Factors - genetics | Jumonji Domain-Containing Histone Demethylases - antagonists & inhibitors | Neoplastic Stem Cells - pathology | Female | Antineoplastic Agents - pharmacology | Spheroids, Cellular - drug effects | Nuclear Proteins - genetics | Nanog Homeobox Protein - genetics | Signal Transduction | Spheroids, Cellular - metabolism | Enzyme Inhibitors - pharmacology | Nuclear Proteins - metabolism | Benzazepines - pharmacology | Pyrimidines - pharmacology | Jumonji Domain-Containing Histone Demethylases - genetics | Xenograft Model Antitumor Assays | Histone Demethylases - metabolism | Animals | Tumor Burden - drug effects | Mice, Nude | Nuclear Proteins - antagonists & inhibitors | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Mice | Jumonji Domain-Containing Histone Demethylases - metabolism | Nanog Homeobox Protein - metabolism | RNA, Small Interfering - metabolism | Analysis | Breast cancer | Stem cells | Epigenetic inheritance | Medical colleges
Journal Article
Clinical Epigenetics, ISSN 1868-7075, 01/2019, Volume 11, Issue 1, pp. 8 - 14
BackgroundIn osteosarcoma (OS), chemotherapy resistance has become one of the greatest issues leading to high mortality among patients. However, the mechanisms... 
KDM6B | H3 lysine 27 trimethylation (H3K27me3) | Osteosarcoma | Chemoresistance | Apoptosis | KDM6A | JMJD3 | SURVIVAL | STEM-CELLS | ACTIVATION | GSKJ4 | DEMETHYLASE INHIBITOR | PKC-ALPHA | KINASE-C-ALPHA | ONCOLOGY | PROGRESS | UTX | Osteosarcoma - drug therapy | Up-Regulation | Epigenesis, Genetic | Humans | Protein Kinase C-alpha - genetics | Gene Expression Regulation, Neoplastic | Drug Resistance, Neoplasm | Male | Histone Demethylases - genetics | Bone Neoplasms - metabolism | Cisplatin - administration & dosage | Female | Bone Neoplasms - genetics | Bone Neoplasms - drug therapy | Nuclear Proteins - genetics | Osteosarcoma - metabolism | Cell Survival - drug effects | Enhancer of Zeste Homolog 2 Protein - genetics | Enhancer of Zeste Homolog 2 Protein - metabolism | Nuclear Proteins - metabolism | Cisplatin - pharmacology | Jumonji Domain-Containing Histone Demethylases - genetics | Myeloid Cell Leukemia Sequence 1 Protein - genetics | Drug Synergism | Xenograft Model Antitumor Assays | Histone Demethylases - metabolism | Animals | Sequence Analysis, RNA | Cell Line, Tumor | Mice | Histones - metabolism | Osteosarcoma - genetics | Jumonji Domain-Containing Histone Demethylases - metabolism | Treatment outcome | Usage | Care and treatment | Analysis | Research | Drug resistance | Cisplatin | Phosphorylation | Kinases | Cancer therapies | Proteins | Demethylation | Histone methyltransferase | Bones | Tumorigenesis | Bioinformatics | Extracellular signal-regulated kinase | MAP kinase | Ribonucleic acid--RNA | Mcl-1 protein | Bone cancer | Chemotherapy | Lysine | Medical prognosis | Stem cells | Epigenetics | Methylation | Histone H3 | Tumors
Journal Article
by Sui, AX and Xu, YB and Li, YT and Hu, QL and Wang, ZY and Zhang, HT and Yang, JJ and Guo, XQ and Zhao, WQ
ONCOTARGET, 09/2017, Volume 8, Issue 40, pp. 68591 - 68598
Glioma is regarded as the most prevalent malignant carcinoma of the central nervous system, and lack of effective treatment. Thus, the development of new... 
JMJD3 | KDM6B | GSK-J4 | GSKJ4 | histone demethylase | CELLULAR SENESCENCE | CANCER | CELL BIOLOGY | EPIGENETIC THERAPY | inhibitor | ACUTE LYMPHOBLASTIC-LEUKEMIA | UTX | glioma | INTRINSIC PONTINE GLIOMAS | MUTATIONS
Journal Article
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