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PloS one, ISSN 1932-6203, 02/2016, Volume 11, Issue 2, p. e0147535
Journal Article
Journal of hepatology, ISSN 0168-8278, 2012, Volume 56, Issue 1, pp. 184 - 191
Background & Aims Mosaic G-protein alpha-subunit ( GNAS )-activating mutations are responsible for the McCune–Albright (MCA) syndrome... 
Gastroenterology and Hepatology | CRP | interleukine 6 signal transducer | GTP | C-reactive protein | jun N-terminal kinase | serum amyloid A | JNK | macroregenerative nodule | complementary deoxyribonucleic acid | MRN | mitogen activated proteins kinase | cAMP | glycoprotein 130 | neuroendocrine secretory protein 55 | hepatocyte nuclear factor 1A | G-protein alpha stimulatory subunit | cDNA | gp130 | protein kinase A | hepatocellular carcinoma | β catenin | IHCA | McCune-Albright syndrome | HCA | PKA | HCC | MAP kinase | MCA | HNF1A | inflammatory hepatocellular adenoma | SAA | cyclic adenosine monophosphate | hepatocellular adenoma | GNAS | CTNNB1 | NESP55 | guanosine-5′-triphosphate | IL6ST | INACTIVATION | PATHWAY | HEPATOCELLULAR ADENOMA | GS-ALPHA | GROWTH | CLASSIFICATION | HUMAN CANCERS | MCCUNE-ALBRIGHT-SYNDROME | GASTROENTEROLOGY & HEPATOLOGY | G-PROTEIN | BINDING | Liver Neoplasms - classification | Humans | Middle Aged | Adenoma, Liver Cell - pathology | Male | Fibrous Dysplasia, Polyostotic - genetics | GTP-Binding Protein alpha Subunits, Gs - genetics | Chromogranins | DNA Mutational Analysis | Base Sequence | Carcinoma, Hepatocellular - genetics | Adult | Female | Liver Neoplasms - pathology | Adenoma, Liver Cell - genetics | STAT3 Transcription Factor - metabolism | Liver Neoplasms - genetics | Signal Transduction | Adenoma, Liver Cell - metabolism | Models, Biological | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Aged | DNA, Neoplasm - genetics | Mutation | Carcinoma, Hepatocellular - metabolism | Liver cancer | Genetic aspects | Nucleic acids | Adenylic acid | Mitogens | Protein kinases | Index Medicus | GTP-Binding Protein alpha Subunits, Gs | Fibrous Dysplasia, Polyostotic | Liver Neoplasms | Life Sciences | Adenoma, Liver Cell | DNA, Neoplasm | STAT3 Transcription Factor | Carcinoma, Hepatocellular | Cancer
Journal Article
Journal Article
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2017, Volume 152, Issue 4, pp. 880 - 894.e6
Background & Aims Hepatocellular adenomas (HCAs) are benign liver tumors that can be assigned to molecular subtypes based on inactivating mutations in... 
Gastroenterology and Hepatology | HCC | SHH | Tumor Progression | Benign | HEDGEHOG PATHWAY | FOLLOW-UP | CANCER | TUMORS | SOMATIC MUTATIONS | INACTIVATION | INHIBITION | THERAPEUTIC TARGETS | GASTROENTEROLOGY & HEPATOLOGY | LIVER ADENOMATOSIS | ACTIVATING MUTATIONS | Liver Neoplasms - classification | Hepatocyte Nuclear Factor 1-alpha - genetics | Adenoma - genetics | Cytokine Receptor gp130 - genetics | Humans | Middle Aged | Transcriptome | Hedgehog Proteins - metabolism | Male | Liver Neoplasms - chemistry | GTP-Binding Protein alpha Subunits, Gs - genetics | Young Adult | Protein-Tyrosine Kinases - genetics | Telomerase - genetics | Cell Transformation, Neoplastic - genetics | Carcinoma, Hepatocellular - genetics | Hemorrhage - etiology | Aged, 80 and over | Adult | Chromogranins - genetics | Female | Carcinoma, Hepatocellular - chemistry | Inhibin-beta Subunits - genetics | Neoplasm Proteins - genetics | Child | STAT3 Transcription Factor - genetics | Liver Neoplasms - genetics | Signal Transduction | Zinc Finger Protein GLI1 - genetics | Risk Factors | Janus Kinase 2 - genetics | Gene Fusion | Adenoma - chemistry | beta Catenin - genetics | Sequence Analysis, RNA | Adenoma - classification | Adolescent | Carcinoma, Hepatocellular - pathology | Aged | Mutation | Liver cancer | Liver | Risk factors | Adenoma | Life Sciences | Human health and pathology | Santé publique et épidémiologie | Hépatology and Gastroenterology | Tissues and Organs
Journal Article
Scientific reports, ISSN 2045-2322, 2017, Volume 7, Issue 1, pp. 4394 - 14
Journal Article