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Journal of Pathology, ISSN 0022-3417, 2005, Volume 206, Issue 3, pp. 291 - 304
Journal Article
Circulation-Heart failure, ISSN 1941-3289, 01/2013, Volume 6, Issue 1, pp. 107 - 17
Journal Article
Science, ISSN 0036-8075, 10/2013, Volume 342, Issue 6157, pp. 447 - 453
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2014, Volume 289, Issue 48, pp. 33311 - 33319
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, pp. e82478 - e82478
Multidrug resistance (MDR), an unfavorable factor compromising the treatment efficacy of anticancer drugs, involves the upregulation of ATP binding cassette... 
CYCLIN D1 EXPRESSION | MULTIDISCIPLINARY SCIENCES | CANCER DRUG-RESISTANCE | GENE-EXPRESSION | MULTIDRUG-RESISTANCE | BETA-CATENIN | CELL-SURVIVAL | DECREASED EXPRESSION | COLON-CARCINOMA | CACO-2 CELLS | P-GLYCOPROTEIN | Cell Cycle - genetics | Cyclin D1 - metabolism | RNA, Small Interfering - genetics | Apoptosis - drug effects | Colorectal Neoplasms - genetics | Humans | ATP-Binding Cassette, Sub-Family B, Member 1 - metabolism | Galectin 3 - metabolism | Apoptosis - genetics | Glycogen Synthase Kinase 3 beta | Proto-Oncogene Proteins c-bcl-2 - metabolism | Caspases - metabolism | ATP-Binding Cassette Transporters - genetics | Mitochondria - genetics | ATP-Binding Cassette Transporters - metabolism | Multidrug Resistance-Associated Proteins - genetics | Colorectal Neoplasms - metabolism | bcl-2-Associated X Protein - genetics | Epirubicin - toxicity | Caco-2 Cells | Signal Transduction | Caspases - genetics | RNA, Messenger - genetics | Gene Silencing | bcl-2-Associated X Protein - metabolism | Mitochondria - metabolism | Galectin 3 - genetics | Glycogen Synthase Kinase 3 - metabolism | Proto-Oncogene Proteins c-myc - metabolism | beta Catenin - metabolism | beta Catenin - genetics | Cyclin D1 - genetics | Glycogen Synthase Kinase 3 - genetics | ATP-Binding Cassette, Sub-Family B, Member 1 - genetics | Cell Line, Tumor | Proto-Oncogene Proteins c-myc - genetics | Cell Cycle - drug effects | Multidrug Resistance-Associated Proteins - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Epirubicin - pharmacology | RNA, Small Interfering - metabolism | Adenocarcinoma | Bcl-2 protein | Material requirements planning | c-Myc protein | Metastasis | Galectin-3 | Cyclin D1 | Drug resistance | Caspase-3 | Cancer therapies | Anticancer properties | Proteins | Mitochondria | Antitumor agents | Inhibition | P-Glycoprotein | Binding | RNA-mediated interference | Glycoprotein | Gene expression | Chemotherapy | Pancreatic cancer | Caspase-9 | Drugs | Chromatin | Bax protein | Colorectal carcinoma | Leukemia | Colorectal cancer | Science | Myc protein | Kinases | β-catenin | Colon cancer | Medical laboratories | Modulation | Colon | Epirubicin | G1 phase | Multidrug resistance | Caspase | Ribonucleic acid--RNA | Resistance factors | Medicine | Signaling | Medical prognosis | ATP | Apoptosis | Cancer | Index Medicus | RNA | Ribonucleic acid
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e57915 - e57915
Obesity and type 2 diabetes are associated with increased production of Galectin-3 (Gal-3), a protein that modulates inflammation and clearance of glucose... 
INDUCED GLOMERULAR INJURY | RECEPTOR-MEDIATED MECHANISMS | ACTIVATION | GALECTIN-3/AGE-RECEPTOR-3 KNOCKOUT MICE | LIPOXIDATION | MULTIDISCIPLINARY SCIENCES | FATTY LIVER | AGE-RECEPTOR | DIET-INDUCED OBESITY | INSULIN SENSITIVITY | EXPRESSION | Galectin 3 - metabolism | Glucose Tolerance Test - methods | Fibroblast Growth Factors - genetics | Monocytes - metabolism | PPAR gamma - metabolism | Obesity - genetics | Galectin 3 - deficiency | Fibroblast Growth Factors - metabolism | Inflammation - metabolism | Adiponectin - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Adiponectin - metabolism | Insulin - genetics | PPAR gamma - genetics | Liver - metabolism | Glucose - genetics | Lipase - metabolism | Transcription Factors - genetics | Galectin 3 - genetics | Adiposity - genetics | Mice, Knockout | Obesity - metabolism | Transcription Factors - metabolism | Insulin - metabolism | Animals | Diet | Insulin Resistance - genetics | Glucose - metabolism | Inflammation - genetics | Mice, Obese | Adiposity - physiology | Mice | Lipase - genetics | Type 2 diabetes | Obesity | Glucose metabolism | Blood sugar monitoring | Liver | Physiological aspects | Inflammation | Glucose | Glucose tolerance tests | Insulin | Statistics | Dextrose | Fibroblast growth factor | Oxidative stress | Adipose tissue | Microflora | Adipocytes | Galectin-3 | Adiponectin | Proteins | Microbiota | Hyperglycemia | Rodents | Thrombocytosis | Age | Adducts | Heart failure | Medical research | Fasting | Cytokines | Anemia | Diabetes mellitus | Metabolism | Glucose tolerance | Nutrition research | Pathology | Monocytes | Acute phase substances | Antibiotics | Neutrophilia | Diabetes | Kinesiology | Index Medicus
Journal Article
PLoS Genetics, ISSN 1553-7390, 07/2016, Volume 12, Issue 7, pp. e1006038 - e1006038
Journal Article
MOLECULAR PHARMACOLOGY, ISSN 0026-895X, 07/2013, Volume 84, Issue 1, pp. 25 - 40
Cyclooxygenase (COX)-2-derived prostanoids can influence several processes that are linked to carcinogenesis. We aimed to address the hypothesis that platelets... 
EPITHELIAL-MESENCHYMAL TRANSITION | RANDOMIZED CONTROLLED-TRIALS | LOCALIZATION | GLYCOPROTEIN-VI | METASTASIS | LOW-DOSE ASPIRIN | CANCER INCIDENCE | PROSTAGLANDIN E-2 RECEPTORS | P-SELECTIN | PHARMACOLOGY & PHARMACY | EXPRESSION | Blood Platelets - pathology | Colonic Neoplasms - genetics | Gene Expression - drug effects | Humans | Galectin 3 - metabolism | Epithelial-Mesenchymal Transition - drug effects | Blood Platelets - enzymology | Cyclin B1 - metabolism | Epithelial-Mesenchymal Transition - genetics | Glycoproteins - pharmacology | Sulfones - pharmacology | Platelet-Derived Growth Factor - genetics | Colonic Neoplasms - metabolism | Lactones - pharmacology | Cyclooxygenase 2 - genetics | Lactose - pharmacology | Receptors, Collagen - genetics | Blood Platelets - drug effects | Binding Sites | Cyclooxygenase 2 Inhibitors - pharmacology | Dinoprostone - genetics | RNA, Messenger - genetics | Cyclin B1 - genetics | Cell Communication - genetics | Down-Regulation - drug effects | Galectin 3 - genetics | Dinoprostone - metabolism | HT29 Cells | Up-Regulation - drug effects | Platelet-Derived Growth Factor - metabolism | Receptors, Collagen - metabolism | Blood Platelets - metabolism | Galectin 3 - antagonists & inhibitors | Cell Communication - drug effects | Cyclooxygenase 2 - metabolism | Immunoglobulin Fc Fragments - pharmacology | Cell Line, Tumor | Colonic Neoplasms - blood | Colonic Neoplasms - enzymology | Index Medicus
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 08/2012, Volume 18, Issue 15, pp. 4037 - 4047
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2014, Volume 289, Issue 48, pp. 33481 - 33491
Lectin galactoside-binding soluble 3 binding protein (LGALS3BP, also called Mac-2 binding protein) is a heavily glycosylated secreted molecule that has been... 
BREAST-CANCER | CYCLOPHILIN-C | NK CELLS | MAC-2-BINDING PROTEIN | LUNG-CARCINOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNATE IMMUNE-RESPONSE | GROUP-B STREPTOCOCCUS | C-ASSOCIATED PROTEIN | SIALIC-ACID DIVERSITY | ANTIGEN 90K | Prostatic Neoplasms - metabolism | Tumor Escape | Colorectal Neoplasms - genetics | Humans | Sialic Acid Binding Ig-like Lectin 3 - immunology | Antigens, Neoplasm - biosynthesis | Neoplasm Proteins - immunology | Male | Neoplasm Proteins - metabolism | Prostatic Neoplasms - immunology | Prostatic Neoplasms - genetics | HEK293 Cells | Female | Neoplasm Proteins - genetics | Carrier Proteins - immunology | Colorectal Neoplasms - metabolism | Sialic Acid Binding Ig-like Lectin 3 - metabolism | Glycoproteins - genetics | Sialic Acid Binding Ig-like Lectin 3 - genetics | Antigens, Neoplasm - genetics | Prostatic Neoplasms - pathology | Antigens, Neoplasm - immunology | Carrier Proteins - biosynthesis | Immunologic Factors - immunology | Gene Expression Regulation, Neoplastic - immunology | Immunologic Factors - genetics | Glycoproteins - biosynthesis | Carrier Proteins - genetics | Glycoproteins - immunology | Animals | Colorectal Neoplasms - immunology | Up-Regulation - immunology | Biomarkers, Tumor - genetics | Mice | Colorectal Neoplasms - pathology | Biomarkers, Tumor - immunology | Biomarkers, Tumor - biosynthesis | Immunologic Factors - biosynthesis | Index Medicus | Galectin | Sialic Acid | LGALS3BP | Glycobiology | Tumor Immunology | Glycobiology and Extracellular Matrices | Siglecs | Cancer
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 03/2012, Volume 19, Issue 3, pp. 523 - 533
Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) induces apoptosis and preferentially kills tumor cells by engaging specific glycosylated death... 
apoptosis | TRAIL | galectin-3 | colon cancer | CANCER-CELLS | CARCINOMA CELLS | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOWN-REGULATION | PROLIFERATION | ENDOCYTOSIS | CELL BIOLOGY | INTERNALIZATION | IN-VIVO | COMPARTMENTALIZATION | TUMOR-CELL | Colonic Neoplasms - genetics | Adenocarcinoma - pathology | Apoptosis - drug effects | Colonic Neoplasms - drug therapy | Humans | Caspase 8 - metabolism | Galectin 3 - metabolism | Apoptosis - genetics | Protein Transport - drug effects | Receptors, TNF-Related Apoptosis-Inducing Ligand - metabolism | Death Domain Receptor Signaling Adaptor Proteins - genetics | Glycosylation - drug effects | Neoplasm Proteins - metabolism | Wnt Proteins - metabolism | Colonic Neoplasms - metabolism | Caspase 8 - genetics | Adenocarcinoma - metabolism | MAP Kinase Kinase 4 - metabolism | TNF-Related Apoptosis-Inducing Ligand - pharmacology | Wnt Proteins - genetics | Adenocarcinoma - genetics | Receptors, TNF-Related Apoptosis-Inducing Ligand - genetics | Neoplasm Proteins - genetics | Gene Silencing | Galectin 3 - genetics | TNF-Related Apoptosis-Inducing Ligand - metabolism | Adenocarcinoma - drug therapy | Protein Transport - genetics | Drug Resistance, Neoplasm - genetics | Wnt Signaling Pathway - drug effects | Wnt Signaling Pathway - genetics | Colonic Neoplasms - pathology | Cell Line, Tumor | MAP Kinase Kinase 4 - genetics | TNF-Related Apoptosis-Inducing Ligand - genetics | Death Domain Receptor Signaling Adaptor Proteins - metabolism | Drug Resistance, Neoplasm - drug effects | Index Medicus | Original Paper
Journal Article