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Journal Article
Developmental Cell, ISSN 1534-5807, 10/2016, Volume 39, Issue 1, pp. 13 - 27
Journal Article
Immunity, ISSN 1074-7613, 08/2012, Volume 37, Issue 2, pp. 249 - 263
Inflammation-mediated neurodegeneration occurs in the acute and the chronic phases of multiple sclerosis (MS) and its animal model, experimental autoimmune... 
EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | NITRIC-OXIDE PRODUCTION | INHIBITION | MULTIPLE-SCLEROSIS | PATHWAY | O-GLYCANS | DIFFERENTIATION | IMMUNOLOGY | EXPRESSION | T-CELLS | NEURONAL DYSFUNCTION | Tumor Necrosis Factor-alpha - metabolism | Microglia - metabolism | Central Nervous System - metabolism | Galectin 1 - immunology | Leukocyte Common Antigens - metabolism | Encephalomyelitis, Autoimmune, Experimental - metabolism | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Galectin 1 - metabolism | NF-kappa B - metabolism | Central Nervous System - immunology | Multiple Sclerosis - physiopathology | Microglia - immunology | Female | Chemokine CCL2 - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Interleukin-6 - metabolism | Multiple Sclerosis - metabolism | Microglia - cytology | Mice, Inbred C57BL | Mice, Knockout | Polysaccharides - metabolism | Encephalomyelitis, Autoimmune, Experimental - therapy | Animals | Galectin 1 - therapeutic use | Cyclic AMP Response Element-Binding Protein - metabolism | Multiple Sclerosis - immunology | Protein Binding | Mice | Central Nervous System - physiopathology | Astrocytes - metabolism | Nitric Oxide Synthase Type II - metabolism | Multiple sclerosis | Nervous system diseases | Neurosciences | Phosphatases | Analysis | Proteins | Statistical analysis | Neurodegeneration | Rodents | Lectins | Regulation | Kinases | Phosphatase | Immune system | Antigens, CD45 | Astrocytes | Nitric Oxide Synthase Type II | Tumor Necrosis Factor-alpha | Interleukin-6 | Microglia | Multiple Sclerosis | Life Sciences | Encephalomyelitis, Autoimmune, Experimental | Polysaccharides | NF-kappa B | Central Nervous System | Immunology | Chemokine CCL2 | Cyclic AMP Response Element-Binding Protein | p38 Mitogen-Activated Protein Kinases | Galectin 1
Journal Article
American Journal of Obstetrics and Gynecology, ISSN 0002-9378, 02/2018, Volume 218, Issue 2, pp. S725 - S737
Fetal growth restriction is a major determinant of perinatal morbidity and mortality. Screening for fetal growth restriction is a key element of prenatal care... 
fetal biometry | models | screening | small for gestational age | human placental lactogen | stillbirth | alpha fetoprotein | biomarker | placental protein 13 | pregnancy-associated plasma protein-A | study design | fetal death | ultrasound | inhibin | randomized controlled trial | placental growth factor | human chorionic gonadotropin | review | prediction | placenta | soluble fms-like tyrosine kinase-1 | A-disintegrin and metalloprotease 12 | soluble endoglin | HUMAN CHORIONIC-GONADOTROPIN | PLASMA-PROTEIN-A | SERUM ALPHA-FETOPROTEIN | OBSTETRICS & GYNECOLOGY | UTERINE ARTERY DOPPLER | GESTATIONAL-AGE | CELL-FREE FETAL | PAPP-A | OUTCOME PREDICTION POP | CIRCULATING ANGIOGENIC FACTORS | EARLY-PREGNANCY | Fetal Growth Retardation - diagnostic imaging | ADAM12 Protein - metabolism | Humans | Placenta Growth Factor - metabolism | Pregnancy Proteins - metabolism | Placental Lactogen - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Estriol - metabolism | Placental Circulation | Female | Membrane Proteins - metabolism | Fetal Growth Retardation - diagnosis | alpha-Fetoproteins - metabolism | Biomarkers - metabolism | Prenatal Diagnosis | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Fetal Growth Retardation - metabolism | Laser-Doppler Flowmetry | Biometry | Chorionic Gonadotropin - metabolism | Pregnancy | Inhibins - metabolism | Ultrasonography, Prenatal | Endoglin - metabolism | Pregnancy-Associated Plasma Protein-A - metabolism | Galectins - metabolism
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, p. e57915
Obesity and type 2 diabetes are associated with increased production of Galectin-3 (Gal-3), a protein that modulates inflammation and clearance of glucose... 
INDUCED GLOMERULAR INJURY | RECEPTOR-MEDIATED MECHANISMS | ACTIVATION | GALECTIN-3/AGE-RECEPTOR-3 KNOCKOUT MICE | LIPOXIDATION | MULTIDISCIPLINARY SCIENCES | FATTY LIVER | AGE-RECEPTOR | DIET-INDUCED OBESITY | INSULIN SENSITIVITY | EXPRESSION | Galectin 3 - metabolism | Glucose Tolerance Test - methods | Fibroblast Growth Factors - genetics | Monocytes - metabolism | PPAR gamma - metabolism | Obesity - genetics | Galectin 3 - deficiency | Fibroblast Growth Factors - metabolism | Inflammation - metabolism | Adiponectin - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Adiponectin - metabolism | Insulin - genetics | PPAR gamma - genetics | Liver - metabolism | Glucose - genetics | Lipase - metabolism | Transcription Factors - genetics | Galectin 3 - genetics | Adiposity - genetics | Mice, Knockout | Obesity - metabolism | Transcription Factors - metabolism | Insulin - metabolism | Animals | Diet | Insulin Resistance - genetics | Glucose - metabolism | Inflammation - genetics | Mice, Obese | Adiposity - physiology | Mice | Lipase - genetics | Type 2 diabetes | Obesity | Glucose metabolism | Blood sugar monitoring | Liver | Physiological aspects | Inflammation | Glucose | Glucose tolerance tests | Insulin | Statistics | Dextrose | Fibroblast growth factor | Oxidative stress | Adipose tissue | Microflora | Adipocytes | Galectin-3 | Adiponectin | Proteins | Microbiota | Hyperglycemia | Rodents | Thrombocytosis | Age | Adducts | Heart failure | Medical research | Fasting | Cytokines | Anemia | Diabetes mellitus | Metabolism | Glucose tolerance | Nutrition research | Pathology | Monocytes | Acute phase substances | Antibiotics | Neutrophilia | Diabetes | Kinesiology
Journal Article
Autophagy, ISSN 1554-8627, 04/2019, Volume 15, Issue 4, pp. 631 - 651
Macroautophagy/autophagy, a defense mechanism against aberrant stresses, in neurons counteracts aggregate-prone misfolded protein toxicity. Autophagy induction... 
autophagy | neurodegeneration | spinal and bulbar muscular atrophy | TFEB | Amyotrophic lateral sclerosis | motoneuron diseases | trehalose | lactulose | calcineurin | protein quality control | lysosomes | melibiose | galectin-3 | MITOCHONDRIAL BIOGENESIS | MUTANT ANDROGEN RECEPTOR | REGULATES AUTOPHAGY | AMYOTROPHIC-LATERAL-SCLEROSIS | SELECTIVE AUTOPHAGY | CELL BIOLOGY | MOUSE MODEL | PROTEIN B8 HSPB8 | MISFOLDED PROTEINS | POLYGLUTAMINE AGGREGATION | MEMBRANE PERMEABILIZATION | RNA, Small Interfering - genetics | Calcium - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Autophagosomes - enzymology | Lysosomes - enzymology | Amyotrophic Lateral Sclerosis - drug therapy | Autophagy - drug effects | Motor Neurons - pathology | Motor Neurons - ultrastructure | Neuroprotection - drug effects | Autophagosomes - drug effects | Calcineurin - genetics | Cell Nucleus - metabolism | Lysosomes - metabolism | Trehalose - analogs & derivatives | Cell Differentiation | Membrane Proteins - metabolism | Autophagy - genetics | Autophagy-Related Proteins - genetics | Sequestosome-1 Protein - metabolism | Induced Pluripotent Stem Cells - metabolism | Induced Pluripotent Stem Cells - pathology | Lysosomes - drug effects | Induced Pluripotent Stem Cells - enzymology | Unfolded Protein Response - genetics | Membrane Proteins - genetics | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - chemistry | Induced Pluripotent Stem Cells - ultrastructure | Signal Transduction - genetics | Autophagosomes - metabolism | Bulbo-Spinal Atrophy, X-Linked - metabolism | Motor Neurons - enzymology | Neuroprotection - genetics | Bulbo-Spinal Atrophy, X-Linked - drug therapy | Down-Regulation - genetics | Lysosomes - ultrastructure | Motor Neurons - metabolism | Animals | Signal Transduction - drug effects | Amyotrophic Lateral Sclerosis - metabolism | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Autophagy-Related Proteins - metabolism | Calcineurin - metabolism | RNA, Small Interfering - metabolism | Trehalose - pharmacology
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 3287 - 17
Leukocytes are coated with a layer of heterogeneous carbohydrates (glycans) that modulate immune function, in part by governing specific interactions with... 
LYN | IMMUNITY | PROTEIN | MULTIDISCIPLINARY SCIENCES | RED-CELLS | SIALIC ACIDS | BINDING | T-CELLS | CD22 | ANTIGEN | LYMPHOCYTES | Phosphorylation | Cell Proliferation | Leukocyte Common Antigens - metabolism | Humans | Receptors, Antigen, B-Cell - metabolism | Endocytosis | Lymphocyte Activation - immunology | Cell Nucleus - metabolism | Sialic Acid Binding Ig-like Lectin 2 - metabolism | src-Family Kinases - metabolism | Polysaccharides - chemistry | B-Lymphocytes - metabolism | Calcium Signaling | Cell Line | N-Acetylhexosaminyltransferases - metabolism | Signal Transduction | Lymphoid Tissue - metabolism | N-Acetylneuraminic Acid - metabolism | Polysaccharides - metabolism | Germinal Center - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - metabolism | Models, Biological | Protein Binding | Galectins - blood | Immunologic Memory | Amino Sugars - chemistry | Immunologic Factors - metabolism | Galectins - metabolism | Lymphocyte receptors | SHP-1 protein | Calcium | N-Acetylglucosinyldiphosphodolichol N-acetylglucosaminyltransferase | Activation | Leukocytes | B-cell receptor | CD45 antigen | Calcium signalling | Proteins | Galectin-9 | Cell activation | Polysaccharides | Germinal centers | Sugars | Binding | Carbohydrates | Immunological memory | Memory cells | Immune response | Tonsil | Immunoregulation | T cell receptors | N-glycans | Glycan | Membrane proteins | White blood cells | CD22 antigen | Lymphocytes B | Lectins
Journal Article
Stem Cells and Development, ISSN 1547-3287, 11/2014, Volume 23, Issue 21, pp. 2591 - 2599
Mesenchymal stromal cells (MSCs) are multipotent cells that possess broad immunomodulatory properties; the mechanisms underlying these properties have not been... 
Original Research Reports | MEDICINE, RESEARCH & EXPERIMENTAL | STEM-CELLS | INDUCE | EXOSOME | MICROVESICLES | PROTECT | CELL & TISSUE ENGINEERING | TRANSPLANTATION | B-CELLS | THERAPY | MEMBRANE-VESICLES | DIFFERENTIATION | HEMATOLOGY | INHIBIT | Leukocytes, Mononuclear - metabolism | Cell Proliferation | Galectin 1 - immunology | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Coculture Techniques | Humans | Galectin 1 - metabolism | Interferon-gamma - metabolism | Mesenchymal Stromal Cells - immunology | Intercellular Signaling Peptides and Proteins - metabolism | Mesenchymal Stromal Cells - cytology | Flow Cytometry | Lymphocytes - immunology | T-Lymphocytes - metabolism | Leukocytes, Mononuclear - immunology | Lysosomal-Associated Membrane Protein 1 - immunology | Interleukin-10 - metabolism | Cell-Derived Microparticles - metabolism | B-Lymphocytes - metabolism | Lymphocytes - metabolism | Transforming Growth Factor beta - immunology | B-Lymphocytes - cytology | Enzyme-Linked Immunosorbent Assay | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Lymphocytes - cytology | CD13 Antigens - metabolism | B-Lymphocytes - immunology | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | T-Lymphocytes - cytology | Cell-Derived Microparticles - immunology | Interferon-gamma - immunology | CD13 Antigens - immunology | Leukocytes, Mononuclear - cytology | Lysosomal-Associated Membrane Protein 1 - metabolism | T-Lymphocytes - immunology | Interleukin-10 - immunology | Transforming Growth Factor beta - metabolism | Intercellular Signaling Peptides and Proteins - immunology | Cell proliferation | Usage | Cytokines | Analysis
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 02/2017, Volume 37, Issue 2, pp. 258 - 263
Objective-To create a model of atherosclerosis using green fluorescent protein (GFP)-targeted monocytes/macrophages, allowing analysis of both endogenous... 
atherosclerosis | GFP | mouse | macrophage | monocyte | model | trafficking | CELLS | IN-VIVO | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | CCR2 | Macrophages - transplantation | Aortic Aneurysm - metabolism | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Cell Tracking - methods | Galectin 3 - metabolism | Male | Monocytes - metabolism | Green Fluorescent Proteins - genetics | Adoptive Transfer | Aorta - metabolism | Antigens, CD - genetics | Recombinant Fusion Proteins - metabolism | Aortic Diseases - metabolism | Receptors, IgG - metabolism | Antigens, CD - metabolism | Aortic Aneurysm - chemically induced | CD11c Antigen - metabolism | Monocytes - transplantation | Monocytes - pathology | Antigens, Differentiation - metabolism | Aortic Aneurysm - pathology | Angiotensin II | Aortic Diseases - pathology | Disease Models, Animal | Green Fluorescent Proteins - metabolism | Atherosclerosis - pathology | Genetic Predisposition to Disease | Macrophages - pathology | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Plaque, Atherosclerotic | Antigens, Differentiation, Myelomonocytic - genetics | Atherosclerosis - metabolism | Disease Progression | Aorta - pathology | Aortic Diseases - genetics | Macrophages - metabolism | Phenotype | Animals | Antigens, Differentiation, Myelomonocytic - metabolism | Apolipoproteins E - genetics | CD11b Antigen - metabolism | Aortic Aneurysm - genetics | 10030 | 10012 | 10188 | 10014 | Basic Sciences
Journal Article