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Neuron, ISSN 0896-6273, 2005, Volume 47, Issue 2, pp. 215 - 229
Accumulation of Na channels at the nodes of Ranvier is a prerequisite for saltatory conduction. In peripheral nerves, clustering of these channels along the... 
MYELINATED AXONS | PROTEIN | INITIAL SEGMENTS | PERIPHERAL NERVOUS-SYSTEM | BETA-IV-SPECTRIN | ADHESION MOLECULES | ANKYRIN-BINDING | IDENTIFICATION | NEUROSCIENCES | SODIUM-CHANNEL DISTRIBUTION | K+ CHANNELS | Cytoskeletal Proteins | Age Factors | Humans | Myelin-Associated Glycoprotein - metabolism | Cercopithecus aethiops | Sciatic Nerve - growth & development | Cell Adhesion Molecules - immunology | Phosphoproteins - metabolism | Ranvier's Nodes - metabolism | Ranvier's Nodes - ultrastructure | Cell Compartmentation | Sciatic Nerve - metabolism | Transfection - methods | Gene Expression Regulation, Developmental | Cloning, Molecular - methods | Sodium Channels - metabolism | Macromolecular Substances - immunology | Cell Adhesion Molecules, Neuronal - metabolism | Membrane Proteins - metabolism | Microfilament Proteins - metabolism | Spectrin - metabolism | Microscopy, Immunoelectron - methods | Protein Structure, Tertiary | Amino Acid Sequence | Blotting, Western - methods | Myelin Basic Protein - metabolism | Fluorescent Antibody Technique - methods | Cells, Cultured | Axons - metabolism | Rats | S100 Proteins - metabolism | Schwann Cells - metabolism | Cell Adhesion Molecules - metabolism | Claudins | Animals | Schwann Cells - ultrastructure | Ankyrins - metabolism | Blotting, Northern - methods | Neurofilament Proteins - metabolism | Macromolecular Substances - metabolism | Ganglia, Spinal - metabolism | Protein Binding - physiology | Receptors, Peptide - metabolism | Immunoglobulins | Proteins | Microscopy | Plasmids | Rodents | Ligands | Experiments | Binding sites
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2015, Volume 290, Issue 22, pp. 13875 - 13887
Journal Article
Journal of Biomedical Materials Research Part A, ISSN 1549-3296, 05/2006, Volume 77A, Issue 2, pp. 242 - 252
Development of biosynthetic conduits carrying extracellular matrix molecules and cell lines expressing neurotrophic growth factors represents a novel and... 
cell proliferation | fibronectin | cell culture | nerve tissue engineering | bioprosthesis | Cell proliferation | Cell culture | Bioprosthesis | Fibronectin | Nerve tissue engineering | PERIPHERAL-NERVE REGENERATION | STEM-CELLS | MATERIALS SCIENCE, BIOMATERIALS | RAT SCHWANN-CELLS | INJURED SPINAL-CORD | ADULT RATS | ENGINEERING, BIOMEDICAL | OLFACTORY ENSHEATHING GLIA | HUMAN BONE-MARROW | GROWTH-FACTORS | MARROW STROMAL CELLS | BASEMENT-MEMBRANE | Biocompatible Materials - metabolism | Stromal Cells - transplantation | Biocompatible Materials - chemistry | Nerve Regeneration - physiology | Collagen - chemistry | Ganglia, Spinal - cytology | Alginates - metabolism | Proteoglycans - chemistry | Schwann Cells - cytology | Neurites - ultrastructure | Cell Shape | Female | Hydrogels - metabolism | Olfactory Bulb - cytology | Animals, Newborn | Bone Marrow Cells - cytology | Stromal Cells - metabolism | Cells, Cultured | Proteoglycans - metabolism | Rats | Schwann Cells - transplantation | Materials Testing | Neurites - metabolism | Schwann Cells - metabolism | Rats, Sprague-Dawley | Fibronectins - metabolism | Collagen - metabolism | Animals | Alginates - chemistry | Laminin - chemistry | Laminin - metabolism | Bone Marrow Cells - metabolism | Drug Combinations | Hydrogels - chemistry | Stromal Cells - cytology | Cell Transplantation - methods | Proteoglycans/chemistry/metabolism | Cell Transplantation/methods | Collagen/chemistry/metabolism | Nerve Regeneration/physiology | Cells; Cultured | Bone Marrow Cells/cytology/metabolism | Rats; Sprague-Dawley | Animals; Newborn | Hydrogels/chemistry/metabolism | Biocompatible Materials/chemistry/metabolism | Stromal Cells/cytology/metabolism/transplantation | Alginates/chemistry/metabolism | Ganglia; Spinal/cytology | Olfactory Bulb/cytology | Neurites/metabolism/ultrastructure | Fibronectins/metabolism | Schwann Cells/cytology/metabolism/transplantation | Laminin/chemistry/metabolism
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, p. e47977
In recent years a growing debate is about whether botulinum neurotoxins are retrogradely transported from the site of injection. Immunodetection of cleaved... 
ACTIVATION | ACIDIC PROTEIN GFAP | RAT | TOXIN | MULTIDISCIPLINARY SCIENCES | NEURONS | AXONAL-TRANSPORT | NEUROEXOCYTOSIS | GLIAL-CELLS | EXPRESSION | SNAP-25 | Immunohistochemistry | Analgesics - pharmacology | Sciatic Nerve - injuries | Spinal Cord - drug effects | Spinal Cord - metabolism | Neuralgia - metabolism | Analgesics - metabolism | Male | Acetylcholine - metabolism | Glial Fibrillary Acidic Protein - metabolism | Botulinum Toxins, Type A - pharmacology | Hindlimb - physiopathology | Sciatic Nerve - metabolism | Biological Transport | Neuralgia - prevention & control | Neuromuscular Agents - pharmacology | Peripheral Nerves - metabolism | Schwann Cells - drug effects | Neuromuscular Agents - metabolism | Cells, Cultured | Nuclear Proteins - metabolism | Schwann Cells - metabolism | Nerve Tissue Proteins - metabolism | Microscopy, Confocal | Animals | Hindlimb - drug effects | Cell Proliferation - drug effects | Mice | Synaptosomal-Associated Protein 25 - metabolism | Botulinum Toxins, Type A - metabolism | CD11b Antigen - metabolism | Hindlimb - metabolism | Astrocytes - metabolism | Ganglia, Spinal - metabolism | Care and treatment | Analysis | Pain | Cell proliferation | Spinal cord | Neurosciences | Complement receptor 3 | Neurobiology | SNAP receptors | Biology | Neuropathy | Proteins | Biological effects | Dorsal root ganglia | Analgesics | Botulinum Toxin Type A | Animal tissues | Fibroblasts | Neurotoxin A | Horns | Growth factors | Nerve endings | CD11b antigen | Neurotoxins | Pain perception | Astrocytes | Glial fibrillary acidic protein | Schwann cells | Injection | Retrograde transport | Ganglia | Quality of life | Councils | SNAP-25 protein | Acetylcholine | Toxins | Transport | Immunofluorescence | Sciatic nerve | Combinatorial analysis | Botulinum toxin
Journal Article
Neuron, ISSN 0896-6273, 04/2013, Volume 78, Issue 1, pp. 65 - 80
Mutations in VCP cause multisystem degeneration impacting the nervous system, muscle, and/or bone. Patients may present with ALS, Parkinsonism, frontotemporal... 
PATHOGENESIS | PARKIN | VALOSIN-CONTAINING-PROTEIN | INCLUSION-BODY MYOPATHY | DROSOPHILA MODEL | FRONTOTEMPORAL DEMENTIA | PAGETS-DISEASE | BONE | NEUROSCIENCES | P97 | DEGENERATION | Embryo, Mammalian | Humans | Ganglia, Spinal - cytology | Drosophila Proteins - metabolism | HSP72 Heat-Shock Proteins - genetics | Protein Tyrosine Phosphatases - genetics | Neurons - ultrastructure | Time Factors | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Neuromuscular Junction - genetics | Neurons - metabolism | Protein-Serine-Threonine Kinases - metabolism | Mitochondrial Membrane Transport Proteins - metabolism | Cell Cycle Proteins - metabolism | Enzyme Inhibitors - pharmacology | Ubiquitin-Protein Ligases - metabolism | Adenosine Triphosphatases - metabolism | Mutation - genetics | Leupeptins - pharmacology | GTP Phosphohydrolases - metabolism | Luminescent Proteins - genetics | Adenosine Triphosphatases - genetics | Ubiquitin-Protein Ligases - genetics | RNA, Small Interfering - metabolism | Immunoprecipitation | Neuromuscular Junction - metabolism | Valosin Containing Protein | Mitochondria - ultrastructure | Transfection | Mitochondria - genetics | Proton Ionophores - pharmacology | Cell Cycle Proteins - genetics | Microscopy, Electron, Transmission | Animals, Genetically Modified | Gene Expression Regulation - genetics | Drosophila | RNA, Small Interfering - pharmacology | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Nuclear Proteins - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Transcription Factors - genetics | Transcription Factors - metabolism | Animals | Proteins - metabolism | Drosophila Proteins - genetics | In Vitro Techniques | Ubiquitination - genetics | Luminescent Proteins - metabolism | Ubiquitin | Analysis | Genomics | Quality control | Amyotrophic lateral sclerosis | Genetic aspects | Mitochondrial DNA | Dementia | Neurons | Parkinsons disease | Biosynthesis | Digital cameras | Kinases | DNA repair | Proteins | Mitochondria | Brain research | Insects | Microscopy | Morphology | Mutation
Journal Article
Journal Article
Current Osteoporosis Reports, ISSN 1544-1873, 8/2017, Volume 15, Issue 4, pp. 255 - 270
We give an update on the etiology and potential treatment options of rare inherited monogenic disorders associated with arterial calcification and calcific... 
GACI | Medicine & Public Health | Orthopedics | Phosphate | ABCC6 | Pyrophosphate | ENPP1 | PXE | Epidemiology | Arterial calcification | FAMILIAL TUMORAL CALCINOSIS | OF-FUNCTION MUTATION | SINGLETON-MERTEN SYNDROME | KLOTHO-DEFICIENT MICE | BASAL GANGLIA CALCIFICATION | MATRIX GLA PROTEIN | CONNECTIVE-TISSUE MINERALIZATION | CAUSE PSEUDOXANTHOMA ELASTICUM | HUTCHINSON-GILFORD-PROGERIA | ENDOCRINOLOGY & METABOLISM | SMOOTH-MUSCLE-CELLS | Humans | Dental Enamel Hypoplasia - drug therapy | Metacarpus - abnormalities | Basal Ganglia Diseases - drug therapy | Aortic Diseases - metabolism | Hyperostosis, Cortical, Congenital - metabolism | Vascular Calcification - metabolism | Cartilage Diseases - genetics | Osteoporosis - metabolism | Muscular Diseases - drug therapy | Osteoporosis - genetics | Abnormalities, Multiple - genetics | Enzyme Replacement Therapy | Calcinosis - drug therapy | Muscular Diseases - metabolism | Vascular Calcification - drug therapy | Hyperostosis, Cortical, Congenital - genetics | Aortic Diseases - genetics | Phosphates - metabolism | Basal Ganglia Diseases - metabolism | Hyperostosis, Cortical, Congenital - drug therapy | Pseudoxanthoma Elasticum - genetics | Interferons - metabolism | Hyperphosphatemia - drug therapy | Hyperphosphatemia - genetics | Pulmonary Valve Stenosis - metabolism | Dental Enamel Hypoplasia - genetics | Cartilage Diseases - metabolism | Abnormalities, Multiple - metabolism | Calcinosis - genetics | Metacarpus - metabolism | Diphosphates - metabolism | Odontodysplasia - metabolism | Vascular Calcification - genetics | Hyperphosphatemia - metabolism | Osteoporosis - drug therapy | Abnormalities, Multiple - drug therapy | Progeria - metabolism | Cartilage Diseases - drug therapy | Progeria - drug therapy | Basal Ganglia Diseases - genetics | Hand Deformities, Congenital - metabolism | Dental Enamel Hypoplasia - metabolism | Calcinosis - metabolism | Gaucher Disease - drug therapy | Pseudoxanthoma Elasticum - metabolism | Pulmonary Valve Stenosis - genetics | Gaucher Disease - metabolism | Pseudoxanthoma Elasticum - drug therapy | Progeria - genetics | Hand Deformities, Congenital - drug therapy | Aortic Diseases - drug therapy | Gaucher Disease - genetics | Hand Deformities, Congenital - genetics | Odontodysplasia - drug therapy | Odontodysplasia - genetics | Muscular Diseases - genetics | Pulmonary Valve Stenosis - drug therapy | Phosphates | Calcification | Medicine, Experimental | Medical research | Interferon
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 09/2011, Volume 194, Issue 5, pp. 751 - 764
The c-Jun N-terminal kinase (JNK) signaling pathway is essential for neuronal degeneration in multiple contexts but also regulates neuronal homeostasis. It... 
PROGRAMMED CELL-DEATH | SIGNALING PATHWAY | N-TERMINAL KINASE | WALLERIAN DEGENERATION | SYMPATHETIC NEURONS | ACTIVATED PROTEIN-KINASES | C-JUN | MICROTUBULE DYNAMICS | MICE LACKING | NERVE GROWTH-FACTOR | CELL BIOLOGY | RNA, Small Interfering - genetics | Caspase 9 - metabolism | Central Nervous System - metabolism | Apoptosis - drug effects | Homeodomain Proteins - metabolism | Caspase 3 - metabolism | Central Nervous System - pathology | Axons - physiology | Ganglia, Spinal - cytology | Central Nervous System - embryology | Neurons - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Protein Kinase Inhibitors - pharmacokinetics | Embryo, Mammalian - pathology | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Ganglia, Spinal - embryology | MAP Kinase Kinase Kinases - genetics | Mice, Transgenic | MAP Kinase Kinase Kinases - metabolism | Mice, Knockout | Central Nervous System - cytology | Embryo, Mammalian - embryology | Axons - pathology | Ganglia, Spinal - pathology | Nerve Growth Factor - deficiency | Mice | Protein Binding - physiology | Neurons - pathology | Phosphorylation | Spinal Cord - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Protein Transport - physiology | Extracellular Signal-Regulated MAP Kinases - metabolism | MAP Kinase Kinase Kinases - deficiency | Spinal Cord - pathology | Neurons - drug effects | Proto-Oncogene Proteins c-jun - genetics | Cells, Cultured | Axons - metabolism | Nerve Growth Factor - pharmacology | Mice, Inbred Strains | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Spinal Cord - embryology | Animals | Proto-Oncogene Proteins c-jun - metabolism | Adaptor Proteins, Signal Transducing - genetics | Apoptosis - physiology | Adaptor Proteins, Signal Transducing - metabolism | Physiological aspects | Neurons | Apoptosis
Journal Article
Journal Article
Nature Neuroscience, ISSN 1097-6256, 12/2005, Volume 8, Issue 12, pp. 1712 - 1719
Journal Article