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Journal of Neurochemistry, ISSN 0022-3042, 03/2005, Volume 92, Issue 6, pp. 1508 - 1520
Journal Article
Journal of Neuropathology & Experimental Neurology, ISSN 0022-3069, 06/2015, Volume 74, Issue 6, pp. 500 - 511
Journal Article
Brain Research, ISSN 0006-8993, 2015, Volume 1622, pp. 377 - 385
Journal Article
Science, ISSN 0036-8075, 2/2011, Volume 331, Issue 6019, pp. 928 - 931
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 11, pp. e15457 - e15457
Spinal muscular atrophy (SMA) is a major genetic cause of death in childhood characterized by marked muscle weakness. To investigate mechanisms underlying... 
ANTERIOR HORN | PROTEIN | MOTOR-NEURON DEGENERATION | BIOLOGY | AXONAL NEUROPATHY | MICE | AMYOTROPHIC-LATERAL-SCLEROSIS | WERDNIG-HOFFMANN DISEASE | SENSORY NEURONS | END-PLATE | MOTONEURONS | Immunohistochemistry | Spinal Cord - metabolism | Neuromuscular Junction - metabolism | Electrophysiology | Spinal Nerve Roots - metabolism | Male | Muscle, Skeletal - metabolism | Axons - physiology | Ganglia, Spinal - physiopathology | Synapses - genetics | Hindlimb - physiopathology | Muscular Atrophy, Spinal - genetics | Spinal Nerve Roots - physiopathology | Female | Muscular Atrophy, Spinal - physiopathology | Disease Models, Animal | Proprioception - physiology | Motor Neurons - physiology | Muscular Atrophy, Spinal - metabolism | Receptors, Cholinergic - metabolism | Synapses - physiology | Hindlimb - innervation | Mice, Transgenic | Neuromuscular Junction - physiopathology | Disease Progression | Animals | Muscle, Skeletal - physiopathology | Mice | Spinal Cord - physiopathology | Ganglia, Spinal - metabolism | Analysis | Models | Spinal muscular atrophy | Neurosciences | Spinal cord | Disease | Circuits | Pathogenesis | SMN protein | Neurobiology | Defects | Proteins | Atrophy | Reduction | Rodents | Sensory neurons | Children | Quantitative analysis | Motor neurons | Neurons | Proprioception | Muscles | Amyotrophic lateral sclerosis | Muscle contraction | Microglia | Microscopy | Neuromuscular junctions | Glutamic acid transporter | Transporter | Synapses | Index Medicus | Animal models
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 1931-857X, 05/2011, Volume 300, Issue 5, pp. 1223 - 1234
The ankyrin-repeat transient receptor potential 1 (TRPA1) has been implicated in pathological conditions of the bladder, but its role in overactive bladder... 
Acrolein - analogs & derivatives | Spinal Cord - drug effects | Spinal Cord - metabolism | Spinal Cord Injuries - drug therapy | Calcium Channels - metabolism | Capsaicin - pharmacology | Spinal Cord Injuries - complications | Urodynamics - drug effects | Ganglia, Spinal - physiopathology | Spinal Cord Injuries - genetics | RNA, Messenger - metabolism | Urinary Bladder, Overactive - etiology | Urinary Bladder - innervation | Urinary Bladder, Overactive - physiopathology | Calcium Channels - genetics | Disease Models, Animal | Acrolein - pharmacology | Acetanilides - pharmacology | Oligonucleotides, Antisense - pharmacology | Spinal Cord Injuries - metabolism | TRPC Cation Channels | Urinary Bladder - metabolism | Purines - pharmacology | Rats | Urinary Bladder, Overactive - metabolism | Carbachol - pharmacology | Animals | Ankyrins - antagonists & inhibitors | Muscle Contraction - drug effects | Ankyrins - genetics | TRPA1 Cation Channel | Ankyrins - metabolism | Urinary Bladder, Overactive - genetics | Urinary Bladder - drug effects | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Urinary Bladder, Overactive - prevention & control | Ganglia, Spinal - drug effects | Ganglia, Spinal - metabolism | Urinary incontinence | Complications and side effects | Cell receptors | Physiological aspects | Spinal cord injuries | Research | Risk factors | Studies | Bladder | Pathology | Spinal cord | Gene expression | Rodents | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, pp. e102027 - e102027
In humans, spinal cord lesions induce not only major motor and neurovegetative deficits but also severe neuropathic pain which is mostly resistant to classical... 
DORSAL-HORN NEURONS | RECOVERY | INJURY MODEL | MICROGLIAL ACTIVATION | NEUROTROPHIC FACTOR | MULTIDISCIPLINARY SCIENCES | TACTILE ALLODYNIA | COLD ALLODYNIA | RECEPTOR | LOCOMOTOR FUNCTION | CENTRAL NEUROPATHIC PAIN | Cytokines - metabolism | Hot Temperature - adverse effects | Spinal Cord Injuries - complications | Rats | Male | Reverse Transcriptase Polymerase Chain Reaction | Activating Transcription Factor 3 - metabolism | Rats, Sprague-Dawley | Hyperalgesia - physiopathology | Nerve Tissue Proteins - metabolism | Animals | Analysis of Variance | Hyperalgesia - drug therapy | Phenols - therapeutic use | Hyperalgesia - etiology | Ketamine - therapeutic use | Pain Measurement | Real-Time Polymerase Chain Reaction | Morphine - therapeutic use | Ganglia, Spinal - metabolism | Analgesics | Intermediate filament proteins | Drugs | Neurosciences | Spinal cord | Transcription factors | Model testing | Gabapentin | Morphine | Activation | Neuropathy | Spinal cord injury | Neuronal-glial interactions | Interleukin 6 | Pain | Dorsal root ganglia | Filaments | Coding | Surgery | Rodents | Cell cycle | Toll-like receptors | Anesthesia | Tumor necrosis factor-TNF | Lesions | Amitriptyline | Territory | Pain perception | Cytokines | Glial fibrillary acidic protein | Activating transcription factor 3 | Markers | Thorax | Pharmacology | Inflammation | Clonazepam | Pressure | Isoflurane | Ganglia | Microglia | Ketamine | Spinal cord injuries | Index Medicus | Life Sciences | Neurons and Cognition
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 10/2011, Volume 31, Issue 42, pp. 15072 - 15085
Mechanisms of inflammatory pain are not fully understood. We investigated the role of TRPV1 (transient receptor potential subtype V1) and TNF-alpha, two... 
PROTEIN-KINASE-A | CAPSAICIN RECEPTOR | CENTRAL SENSITIZATION | LINOLEIC-ACID METABOLITES | DOCOSAHEXAENOIC ACID | SUPERFICIAL DORSAL-HORN | SUBSTANTIA-GELATINOSA | NECROSIS-FACTOR-ALPHA | PRIMARY SENSORY NEURONS | LONG-TERM POTENTIATION | NEUROSCIENCES | Acrylamides | Inflammation - chemically induced | Inflammation - pathology | Enzyme Inhibitors | Ganglia, Spinal - cytology | Excitatory Postsynaptic Potentials - drug effects | TRPV Cation Channels - deficiency | Excitatory Postsynaptic Potentials - physiology | Inflammation - complications | Receptors, Tumor Necrosis Factor, Type I - deficiency | TRPV Cation Channels - metabolism | Long-Term Potentiation - drug effects | Pain - drug therapy | Spinal Cord - pathology | TRPV Cation Channels - antagonists & inhibitors | Freund's Adjuvant - adverse effects | Neurons - physiology | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Pain - etiology | Receptors, Tumor Necrosis Factor - deficiency | Neurons - drug effects | Disease Models, Animal | Docosahexaenoic Acids - therapeutic use | Mice, Inbred C57BL | Bridged Bicyclo Compounds, Heterocyclic | Cells, Cultured | Excitatory Amino Acid Antagonists - pharmacology | Long-Term Potentiation - genetics | Pain - pathology | Mice, Knockout | Tumor Necrosis Factor-alpha - pharmacology | Patch-Clamp Techniques | Animals | Analysis of Variance | Mice | In Vitro Techniques | Pain Measurement | Index Medicus | primary sensory neurons | single-cell PCR | TRPA1 | docosahexaenoic acid | central sensitization | omega-3 polyunsaturated fatty acid | spontaneous EPSCs | long-term potentiation
Journal Article
Anesthesiology, ISSN 0003-3022, 04/2016, Volume 124, Issue 4, pp. 934 - 944
Journal Article
Journal Article