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PLoS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, pp. e0170814 - e0170814
Chemotherapy-induced peripheral neuropathy (CIPN) and associated neuropathic pain is a debilitating adverse effect of cancer treatment. Current understanding... 
MACROPHAGE INFILTRATION | PAIN | INDUCED MECHANICAL ALLODYNIA | MULTIDISCIPLINARY SCIENCES | REGULATORY T-CELLS | DORSAL-ROOT GANGLIA | SPINAL-CORD | NERVE INJURY | SENSORY NEURONS | SATELLITE GLIAL-CELLS | PACLITAXEL | Hyperalgesia - chemically induced | Neuralgia - genetics | Spleen - immunology | Chemokine CCL2 - immunology | Male | Spleen - drug effects | Activating Transcription Factor 3 - immunology | T-Lymphocytes, Regulatory - pathology | Hyperalgesia - pathology | T-Lymphocytes, Regulatory - immunology | Neurofilament Proteins - genetics | Neuralgia - immunology | Chemokine CCL3 - genetics | Microglia - pathology | Lymph Nodes - drug effects | Activating Transcription Factor 3 - genetics | Receptors, Purinergic P2Y12 - genetics | Gene Expression | Receptors, Purinergic P2Y12 - immunology | Mice, Transgenic | Lymph Nodes - immunology | Spinal Cord - immunology | Hyperalgesia - immunology | T-Lymphocytes, Regulatory - drug effects | Hyperalgesia - genetics | Ganglia, Spinal - pathology | Mice | CD8-Positive T-Lymphocytes - immunology | Ganglia, Spinal - drug effects | CD8-Positive T-Lymphocytes - pathology | Lymph Nodes - pathology | Spinal Cord - drug effects | Chemokine CCL3 - immunology | Sensory Receptor Cells - immunology | Neuralgia - pathology | Microglia - immunology | Antineoplastic Agents - adverse effects | Spinal Cord - pathology | Neuralgia - chemically induced | Neurofilament Proteins - immunology | Spleen - pathology | Ganglia, Spinal - immunology | Sensory Receptor Cells - pathology | Microglia - drug effects | Mice, Inbred C57BL | Paclitaxel - adverse effects | Chemokine CCL2 - genetics | Animals | Sensory Receptor Cells - drug effects | CD8-Positive T-Lymphocytes - drug effects | Organoplatinum Compounds - adverse effects | Usage | Chemotherapy | Care and treatment | Oxalic acid | Polyneuropathies | Research | Diagnosis | T cells | Cancer | Spinal cord | Neurosciences | CD8 antigen | Carbon tetrachloride | Interleukin | Central nervous system | Transgenic | Nervous system | Diabetic neuropathy | Lymphocytes T | Cerebrospinal fluid | Peripheral neuropathy | Macrophages | Lymph nodes | Proteins | Neurotoxicity | Interleukin 4 | Pain | Dorsal root ganglia | Lymphocytes | Rodents | Sensory neurons | Tumor necrosis factor-TNF | Horns | Dorsal horn | Peripheral nervous system | Pain sensitivity | Spleen | Pain perception | Immune response | Cytokines | Granulocyte-macrophage colony-stimulating factor | Hypersensitivity | CCL3 protein | Interleukin 12 | Inflammation | CCL4 protein | Microglia | CD4 antigen | Studies | White blood cells | TNF inhibitors | Infiltration | Chemokines | Monocyte chemoattractant protein 1 | Index Medicus
Journal Article
Anesthesiology, ISSN 0003-3022, 04/2016, Volume 124, Issue 4, pp. 934 - 944
Journal Article
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2013, Volume 123, Issue 4, pp. 1513 - 1530
Patients with cholestatic disease exhibit pruritus and analgesia, but the mechanisms underlying these symptoms are unknown. We report that bile acids, which... 
MEDICINE, RESEARCH & EXPERIMENTAL | GENE-RELATED PEPTIDE | INTRATHECAL MORPHINE | SYNAPTIC-TRANSMISSION | GPBAR1 TGR5 | LEUCINE-ENKEPHALIN | SPINAL-CORD | SUBSTANCE-P | CEREBROSPINAL-FLUID | SENSORY NEURONS | RAT MODEL | Spinal Cord - secretion | Bile Acids and Salts - pharmacology | Opioid Peptides - physiology | Spinal Cord - metabolism | Cholestasis - complications | Cholestasis - metabolism | Receptors, G-Protein-Coupled - metabolism | Capsaicin - pharmacology | Male | Receptors, G-Protein-Coupled - agonists | Pain - metabolism | Action Potentials | Pain Perception - drug effects | Gastrin-Releasing Peptide - secretion | Pain - etiology | Female | Pruritus - etiology | Neurons - metabolism | Neurons - drug effects | Single-Cell Analysis | Enkephalin, Leucine - secretion | Receptors, G-Protein-Coupled - physiology | Gene Expression | Mice, Inbred C57BL | Cells, Cultured | Rats | Bile Acids and Salts - metabolism | Organ Specificity | Opioid Peptides - metabolism | Rats, Sprague-Dawley | Ganglia, Spinal - secretion | Mice, Knockout | Macrophages - metabolism | Patch-Clamp Techniques | Pruritus - metabolism | Animals | Ganglia, Spinal - pathology | Dermis - pathology | Mice | Receptors, G-Protein-Coupled - genetics | Bile Acids and Salts - physiology | Ganglia, Spinal - drug effects | Ganglia, Spinal - metabolism | Complications and side effects | Jaundice, Obstructive | Bile acids | Pain | Development and progression | Pruritus | Health aspects | Cholestasis | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 09/2015, Volume 35, Issue 39, pp. 13487 - 13500
Journal Article
Experimental Neurology, ISSN 0014-4886, 07/2014, Volume 257, pp. 95 - 105
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2006, Volume 21, Issue 5, pp. 686 - 698
Abstract Paclitaxel is a commonly used cancer chemotherapy drug that frequently causes painful peripheral neuropathies. The mechanisms underlying this... 
Psychiatry | Allergy and Immunology | Interleukin-1 | Meninges | Glia | Macrophages | Tumor necrosis factor | Neuropathic pain | NERVOUS-SYSTEM | interleukin-1 | TNF RECEPTORS | NECROSIS-FACTOR-ALPHA | PAINFUL PERIPHERAL NEUROPATHY | ANTIINFLAMMATORY CYTOKINE | IMMUNOLOGY | glia | NEUROSCIENCES | SENSORY NEURONS | neuropathic pain | macrophages | tumor necrosis factor | INDUCED HYPERALGESIA | TAXOL | SPINAL-CORD GLIA | ALTERNATIVE ACTIVATION | meninges | Hyperalgesia - chemically induced | Interleukin-1beta - drug effects | Tumor Necrosis Factor-alpha - metabolism | Pain Threshold - physiology | Peripheral Nervous System Diseases - chemically induced | Spinal Cord - drug effects | Spinal Cord - metabolism | CD11b Antigen - drug effects | RNA, Messenger - analysis | Male | Ganglia, Spinal - cytology | Interleukin-10 - physiology | Meninges - metabolism | Neuroglia - drug effects | Peripheral Nervous System Diseases - prevention & control | Interleukin-1beta - metabolism | Interleukin-10 - administration & dosage | Plasmids - genetics | Pain Threshold - drug effects | Spinal Cord - cytology | Peripheral Nervous System Diseases - complications | Antineoplastic Agents, Phytogenic - adverse effects | Cytokines - immunology | Disease Models, Animal | Injections, Spinal | Hyperalgesia - prevention & control | Paclitaxel - adverse effects | Rats | Meninges - drug effects | Rats, Sprague-Dawley | Animals | Tumor Necrosis Factor-alpha - drug effects | Cytokines - drug effects | Interleukin-10 - genetics | Hyperalgesia - etiology | Neuroglia - metabolism | Receptors, Interleukin-1 - physiology | CD11b Antigen - metabolism | Ganglia, Spinal - drug effects | Receptors, Interleukin-1 - antagonists & inhibitors | Plasmids - administration & dosage | Ganglia, Spinal - metabolism | Genetic Therapy - methods | Genetic research | Gene expression | Gene therapy | Health aspects | Index Medicus
Journal Article
Journal Article
Journal of Pain, ISSN 1526-5900, 2014, Volume 15, Issue 7, pp. 712 - 725
Abstract This paper tests the contribution of the toll-like receptors, TLR4 in particular, in the initiation and maintenance of paclitaxel-related... 
Anesthesia & Perioperative Care | Pain Medicine | LPS-RS | DRG | Neuropathy | spinal cord | TRIF | TLR4 | MyD88 | CELLS | INFLAMMATORY HYPERALGESIA | NERVE INJURY | NEUROSCIENCES | CLINICAL NEUROLOGY | FACTOR-ALPHA | MECHANICAL HYPERSENSITIVITY | TUMOR-NECROSIS-FACTOR | IMMUNE-SYSTEM | CHRONIC CONSTRICTION INJURY | TNF-ALPHA | PAINFUL NEUROPATHY | Peripheral Nervous System Diseases - chemically induced | Male | Ganglia, Spinal - cytology | Paclitaxel - toxicity | Time Factors | Pain Threshold - drug effects | Neurons - metabolism | Peripheral Nervous System Diseases - pathology | Neurons - drug effects | Peripheral Nervous System Diseases - genetics | Disease Models, Animal | Physical Stimulation | Astrocytes - drug effects | Rats | Rats, Sprague-Dawley | Toll-Like Receptor 4 - metabolism | Polysaccharides - pharmacology | Antineoplastic Agents, Phytogenic - toxicity | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Animals | Signal Transduction - drug effects | Ganglia, Spinal - drug effects | Myeloid Differentiation Factor 88 - metabolism | Peripheral Nervous System Diseases - drug therapy | Astrocytes - metabolism | Ganglia, Spinal - metabolism | Chemotherapy | Analgesics | Peptides | Analysis | Paclitaxel | Biological response modifiers | Cancer | Neurons | Interferon | Health aspects | Mitogens | Index Medicus
Journal Article