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PLoS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, pp. e0170814 - e0170814
Chemotherapy-induced peripheral neuropathy (CIPN) and associated neuropathic pain is a debilitating adverse effect of cancer treatment. Current understanding... 
MACROPHAGE INFILTRATION | PAIN | INDUCED MECHANICAL ALLODYNIA | MULTIDISCIPLINARY SCIENCES | REGULATORY T-CELLS | DORSAL-ROOT GANGLIA | SPINAL-CORD | NERVE INJURY | SENSORY NEURONS | SATELLITE GLIAL-CELLS | PACLITAXEL | Hyperalgesia - chemically induced | Neuralgia - genetics | Spleen - immunology | Chemokine CCL2 - immunology | Male | Spleen - drug effects | Activating Transcription Factor 3 - immunology | T-Lymphocytes, Regulatory - pathology | Hyperalgesia - pathology | T-Lymphocytes, Regulatory - immunology | Neurofilament Proteins - genetics | Neuralgia - immunology | Chemokine CCL3 - genetics | Microglia - pathology | Lymph Nodes - drug effects | Activating Transcription Factor 3 - genetics | Receptors, Purinergic P2Y12 - genetics | Gene Expression | Receptors, Purinergic P2Y12 - immunology | Mice, Transgenic | Lymph Nodes - immunology | Spinal Cord - immunology | Hyperalgesia - immunology | T-Lymphocytes, Regulatory - drug effects | Hyperalgesia - genetics | Ganglia, Spinal - pathology | Mice | CD8-Positive T-Lymphocytes - immunology | Ganglia, Spinal - drug effects | CD8-Positive T-Lymphocytes - pathology | Lymph Nodes - pathology | Spinal Cord - drug effects | Chemokine CCL3 - immunology | Sensory Receptor Cells - immunology | Neuralgia - pathology | Microglia - immunology | Antineoplastic Agents - adverse effects | Spinal Cord - pathology | Neuralgia - chemically induced | Neurofilament Proteins - immunology | Spleen - pathology | Ganglia, Spinal - immunology | Sensory Receptor Cells - pathology | Microglia - drug effects | Mice, Inbred C57BL | Paclitaxel - adverse effects | Chemokine CCL2 - genetics | Animals | Sensory Receptor Cells - drug effects | CD8-Positive T-Lymphocytes - drug effects | Organoplatinum Compounds - adverse effects | Usage | Chemotherapy | Care and treatment | Oxalic acid | Polyneuropathies | Research | Diagnosis | T cells | Cancer | Spinal cord | Neurosciences | CD8 antigen | Carbon tetrachloride | Interleukin | Central nervous system | Transgenic | Nervous system | Diabetic neuropathy | Lymphocytes T | Cerebrospinal fluid | Peripheral neuropathy | Macrophages | Lymph nodes | Proteins | Neurotoxicity | Interleukin 4 | Pain | Dorsal root ganglia | Lymphocytes | Rodents | Sensory neurons | Tumor necrosis factor-TNF | Horns | Dorsal horn | Peripheral nervous system | Pain sensitivity | Spleen | Pain perception | Immune response | Cytokines | Granulocyte-macrophage colony-stimulating factor | Hypersensitivity | CCL3 protein | Interleukin 12 | Inflammation | CCL4 protein | Microglia | CD4 antigen | Studies | White blood cells | TNF inhibitors | Infiltration | Chemokines | Monocyte chemoattractant protein 1 | Index Medicus
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2006, Volume 21, Issue 5, pp. 599 - 616
Abstract Chronic constriction injury (CCI) of the sciatic nerve in rodents produces mechanical and thermal hyperalgesia and is a common model of neuropathic... 
Psychiatry | Allergy and Immunology | MHC II | CD4+ macrophages | OX-42 (CD11b) | CD8+ T-cells | ED1 (CD68) | Neuroinflammation | Astrocyte | Macrophage | Microglia | MHC I | MICROGLIAL ACTIVATION | PRIMARY AFFERENT NEURONS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | macrophage | FACTOR RECEPTOR-1 | PAINFUL PERIPHERAL NEUROPATHY | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | NEUROSCIENCES | SENSORY NEURONS | neuroinflammation | CD4+macrophages | DIFFERENTIAL ACTIVATION | FACIAL MOTOR NUCLEUS | CD8+T-cells | SODIUM-CHANNEL EXPRESSION | microglia | astrocyte | Adaptation, Physiological | Gait - immunology | Sciatic Nerve - injuries | Male | Ganglia, Spinal - cytology | Activating Transcription Factor 3 - immunology | Sciatic Nerve - immunology | Axotomy | Sciatic Nerve - metabolism | Constriction, Pathologic - immunology | Inflammation Mediators - metabolism | Neuroglia - immunology | Neurons, Afferent - immunology | Sciatic Nerve - cytology | Spinal Cord - cytology | Calcitonin Gene-Related Peptide - metabolism | Macrophages - immunology | Ganglia, Spinal - immunology | Motor Neurons - immunology | Rats | Activating Transcription Factor 3 - metabolism | Rats, Sprague-Dawley | Motor Neurons - metabolism | Spinal Cord - immunology | Macrophages - metabolism | Animals | Analysis of Variance | Lymphocyte Subsets | Neuroglia - metabolism | Lumbar Vertebrae | Neurons, Afferent - metabolism | Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2012, Volume 109, Issue 50, pp. 20602 - 20607
Osteoarthritis is one of the leading causes of chronic pain, but almost nothing is known about the mechanisms and molecules that mediate... 
Receptors | Pain | Neurons | Mice | Arthritis | Macrophages | Osteoarthritis | Behavioral neuroscience | Chemokines | Chronic pain | SYSTEM | NEUROPATHIC PAIN | CENTRAL SENSITIZATION | NOCICEPTIVE NEURONS | RAT | MULTIDISCIPLINARY SCIENCES | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | DORSAL-ROOT GANGLIA | SPINAL-CORD | MICE | EXPRESSION | Receptors, CCR2 - genetics | Receptors, CCR2 - physiology | Arthritis, Experimental - genetics | Chemokine CCL2 - immunology | Humans | Male | Pain - immunology | Ganglia, Spinal - physiopathology | RNA, Messenger - metabolism | Osteoarthritis - physiopathology | Arthritis, Experimental - pathology | Pain - genetics | Chemokine CCL2 - physiology | Osteoarthritis - pathology | Disease Models, Animal | Ganglia, Spinal - immunology | Arthritis, Experimental - physiopathology | Macrophages - pathology | Signal Transduction | Osteoarthritis - immunology | Mice, Inbred C57BL | RNA, Messenger - genetics | Chemokine CCL2 - genetics | Arthritis, Experimental - immunology | Osteoarthritis - genetics | Hyperalgesia - physiopathology | Mice, Knockout | Hyperalgesia - immunology | Animals | Hyperalgesia - genetics | Pain - physiopathology | Ganglia, Spinal - pathology | Receptors, CCR2 - deficiency | Prevention | Knee | Physiological aspects | Health aspects | Signal transduction | T cell receptors | Rodents | Index Medicus | Biological Sciences
Journal Article
Neuroscience, ISSN 0306-4522, 09/2018, Volume 388, pp. 393 - 404
Painful neuropathic injuries are accompanied by robust inflammatory and oxidative stress responses that contribute to the development and maintenance of pain.... 
neuropathic pain | radiculopathy | inflammation | NSAID | phospholipase A2 | reactive oxygen species | phospholipase A | MECHANICAL ALLODYNIA | ASTROCYTE ACTIVATION | GLIAL ACTIVATION | NERVE ROOT COMPRESSION | ALZHEIMERS-DISEASE | BEHAVIORAL SENSITIVITY | NEUROSCIENCES | JOINT INJURY | ANIMAL-MODEL | RNA OXIDATION | Inflammation - pathology | Neurons - pathology | Spinal Nerve Roots - immunology | Oxidative Stress - physiology | Astrocytes - pathology | Male | Pain - immunology | Cervical Cord - drug effects | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Radiculopathy - pathology | Spinal Nerve Roots - injuries | Microglia - immunology | Radiculopathy - drug therapy | Astrocytes - immunology | Pain - drug therapy | Microglia - pathology | Peripheral Nervous System Diseases - pathology | Neurons - drug effects | Peripheral Nervous System Diseases - immunology | Disease Models, Animal | Astrocytes - drug effects | Ganglia, Spinal - immunology | Microglia - drug effects | Cervical Cord - pathology | Neurons - immunology | Cervical Cord - immunology | Antioxidants - pharmacology | Pain - pathology | Rats, Sprague-Dawley | Meloxicam - pharmacology | Animals | Ganglia, Spinal - pathology | Inflammation - prevention & control | Oxidative Stress - drug effects | Spinal Nerve Roots - pathology | Ganglia, Spinal - drug effects | Peripheral Nervous System Diseases - drug therapy | Radiculopathy - immunology | Inflammation - physiopathology | Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2003, Volume 100, Issue 14, pp. 8514 - 8519
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 08/2009, Volume 6, Issue 1, pp. 23 - 23
Background: Lyme neuroborreliosis (LNB) may present as meningitis, cranial neuropathy, acute radiculoneuropathy or, rarely, as encephalomyelitis. We... 
BORRELIA-BURGDORFERI | TUMOR-NECROSIS-FACTOR | NONHUMAN PRIMATE MODEL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | RHESUS-MONKEY | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | CENTRAL-NERVOUS-SYSTEM | MENINGEAL INFLAMMATION | IMMUNOLOGY | CEREBROSPINAL-FLUID | NEUROSCIENCES | FACTOR-ALPHA | Lyme Neuroborreliosis - pathology | Neurons - pathology | Spinal Cord Diseases - physiopathology | Nerve Degeneration - physiopathology | Ganglia, Spinal - physiopathology | Gliosis - physiopathology | Macaca mulatta | Encephalitis - physiopathology | Lyme Neuroborreliosis - immunology | Spinal Cord - pathology | Leukocytosis - physiopathology | Lyme Neuroborreliosis - physiopathology | Neuroglia - immunology | Radiculopathy - physiopathology | Leukocytosis - microbiology | Gliosis - immunology | Ganglia, Spinal - immunology | Spinal Cord Diseases - immunology | Neurons - microbiology | Brain - physiopathology | Leukocytosis - immunology | Nerve Degeneration - microbiology | Neurons - immunology | Radiculopathy - microbiology | Encephalitis - immunology | Nerve Degeneration - immunology | Neuroglia - microbiology | Gliosis - microbiology | Spinal Cord - immunology | Meningitis - immunology | Animals | Apoptosis - immunology | Spinal Cord Diseases - microbiology | Ganglia, Spinal - pathology | Brain - pathology | Antibodies - blood | Chemokines - metabolism | Spinal Cord - physiopathology | Meningitis - microbiology | Meningitis - physiopathology | Brain - immunology | Encephalitis - microbiology | Radiculopathy - immunology | Prevention | Lyme disease | Usage | Care and treatment | Fluorescent antibody technique | Research | Immunofluorescence | T cells | Health aspects | Apoptosis | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 140, Issue 4, pp. 1283 - 1291.e2
Background & Aims The transient receptor potential (TRP) channels TRPV1 and TRPA1 have each been associated with regulation of efferent properties of primary... 
Gastroenterology and Hepatology | Analgesia | Nervous System | Pancreas | Pain Relief | AFFERENT NEURONS | NEUROGENIC INFLAMMATION | MOUSE | FACTOR FAMILY LIGANDS | VAGAL AFFERENTS | RECEPTOR | CLASSIFICATION | CHANNELS | PRIMARY SENSORY NEURONS | GASTROENTEROLOGY & HEPATOLOGY | CAPSAICIN | Transient Receptor Potential Channels - antagonists & inhibitors | Calcium - metabolism | Male | Ganglia, Spinal - cytology | Nodose Ganglion - cytology | Nodose Ganglion - metabolism | Pancreatitis - complications | Pancreatitis - drug therapy | Pancreas - innervation | TRPV Cation Channels - antagonists & inhibitors | Gene Expression - immunology | Pancreatitis - immunology | Behavior, Animal - drug effects | Acrylamides - pharmacology | Disease Models, Animal | Acetanilides - pharmacology | Acute Disease | Ganglia, Spinal - immunology | Abdominal Pain - etiology | TRPV Cation Channels - immunology | Purines - pharmacology | Mice, Inbred C57BL | Transient Receptor Potential Channels - immunology | Cells, Cultured | Nodose Ganglion - immunology | Pancreas - immunology | Abdominal Pain - drug therapy | TRPV Cation Channels - genetics | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Patch-Clamp Techniques | Animals | Transient Receptor Potential Channels - genetics | TRPA1 Cation Channel | Abdominal Pain - immunology | Mice | Ganglia, Spinal - metabolism | Index Medicus | Abridged Index Medicus | nervous system | pain relief | pancreas | analgesia
Journal Article