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Journal Article
PloS one, ISSN 1932-6203, 2010, Volume 5, Issue 11, pp. e15457 - e15457
Spinal muscular atrophy (SMA) is a major genetic cause of death in childhood characterized by marked muscle weakness... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Immunohistochemistry | Spinal Cord - metabolism | Neuromuscular Junction - metabolism | Electrophysiology | Spinal Nerve Roots - metabolism | Male | Muscle, Skeletal - metabolism | Axons - physiology | Ganglia, Spinal - physiopathology | Synapses - genetics | Hindlimb - physiopathology | Muscular Atrophy, Spinal - genetics | Spinal Nerve Roots - physiopathology | Female | Muscular Atrophy, Spinal - physiopathology | Disease Models, Animal | Proprioception - physiology | Motor Neurons - physiology | Muscular Atrophy, Spinal - metabolism | Receptors, Cholinergic - metabolism | Synapses - physiology | Hindlimb - innervation | Mice, Transgenic | Neuromuscular Junction - physiopathology | Disease Progression | Animals | Muscle, Skeletal - physiopathology | Mice | Spinal Cord - physiopathology | Ganglia, Spinal - metabolism | Analysis | Models | Spinal muscular atrophy | Neurosciences | Spinal cord | Disease | Circuits | Pathogenesis | SMN protein | Neurobiology | Defects | Proteins | Atrophy | Reduction | Rodents | Sensory neurons | Children | Quantitative analysis | Motor neurons | Neurons | Proprioception | Muscles | Amyotrophic lateral sclerosis | Muscle contraction | Microglia | Microscopy | Neuromuscular junctions | Glutamic acid transporter | Transporter | Synapses | Index Medicus | Animal models
Journal Article
Neurourology and urodynamics, ISSN 0733-2467, 01/2010, Volume 29, Issue 1, pp. 63 - 76
Aims To summarize the changes that occur in the properties of bladder afferent neurons following spinal cord injury... 
voltage‐gated ion channels | afferent neurons | neurotrophic factors | urinary bladder | neuropeptides | micturition | neuroplasticity | Neuroplasticity | Afferent neurons | Micturition | Urinary bladder | Voltage-gated ion channels | Neuropeptides | Neurotrophic factors | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Central Nervous System - metabolism | Spinal Cord Injuries - complications | gamma-Aminobutyric Acid - metabolism | Humans | Neuroanatomical Tract-Tracing Techniques | Urinary Bladder, Neurogenic - metabolism | Afferent Pathways - physiopathology | Urinary Bladder, Neurogenic - etiology | Ganglia, Spinal - physiopathology | Recovery of Function | Urinary Bladder - innervation | Potassium Channels - metabolism | Urinary Bladder, Neurogenic - physiopathology | Sodium Channels - metabolism | Urinary Bladder, Neurogenic - therapy | Spinal Cord Injuries - therapy | Afferent Pathways - metabolism | Reflex | Nerve Growth Factor - metabolism | Spinal Cord Injuries - metabolism | Nerve Fibers, Unmyelinated | Mechanotransduction, Cellular | Patch-Clamp Techniques | Animals | Neuronal Plasticity | Pituitary Adenylate Cyclase-Activating Polypeptide - metabolism | Spinal Cord Injuries - physiopathology | Central Nervous System - physiopathology | Urination | Nerve Fibers, Myelinated | Ganglia, Spinal - metabolism | Genetic Therapy - methods | Index Medicus | voltage-gated ion channels
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 1931-857X, 05/2011, Volume 300, Issue 5, pp. 1223 - 1234
...) following spinal cord injury (SCI) remains unknown. In this study, using a rat SCI model, we assessed the relevance of TRPA1 in OAB induced by SCI... 
Acrolein - analogs & derivatives | Spinal Cord - drug effects | Spinal Cord - metabolism | Spinal Cord Injuries - drug therapy | Calcium Channels - metabolism | Capsaicin - pharmacology | Spinal Cord Injuries - complications | Urodynamics - drug effects | Ganglia, Spinal - physiopathology | Spinal Cord Injuries - genetics | RNA, Messenger - metabolism | Urinary Bladder, Overactive - etiology | Urinary Bladder - innervation | Urinary Bladder, Overactive - physiopathology | Calcium Channels - genetics | Disease Models, Animal | Acrolein - pharmacology | Acetanilides - pharmacology | Oligonucleotides, Antisense - pharmacology | Spinal Cord Injuries - metabolism | TRPC Cation Channels | Urinary Bladder - metabolism | Purines - pharmacology | Rats | Urinary Bladder, Overactive - metabolism | Carbachol - pharmacology | Animals | Ankyrins - antagonists & inhibitors | Muscle Contraction - drug effects | Ankyrins - genetics | TRPA1 Cation Channel | Ankyrins - metabolism | Urinary Bladder, Overactive - genetics | Urinary Bladder - drug effects | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Urinary Bladder, Overactive - prevention & control | Ganglia, Spinal - drug effects | Ganglia, Spinal - metabolism | Urinary incontinence | Complications and side effects | Cell receptors | Physiological aspects | Spinal cord injuries | Research | Risk factors | Index Medicus
Journal Article
Molecular pain, ISSN 1744-8069, 01/2011, Volume 7, Issue 1, pp. 1744-8069-7-40 - 40
Journal Article
PloS one, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, pp. e102027 - e102027
In humans, spinal cord lesions induce not only major motor and neurovegetative deficits but also severe neuropathic pain which is mostly resistant to classical analgesics... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Cytokines - metabolism | Hot Temperature - adverse effects | Spinal Cord Injuries - complications | Rats | Male | Reverse Transcriptase Polymerase Chain Reaction | Activating Transcription Factor 3 - metabolism | Rats, Sprague-Dawley | Hyperalgesia - physiopathology | Nerve Tissue Proteins - metabolism | Animals | Analysis of Variance | Hyperalgesia - drug therapy | Phenols - therapeutic use | Hyperalgesia - etiology | Ketamine - therapeutic use | Pain Measurement | Real-Time Polymerase Chain Reaction | Morphine - therapeutic use | Ganglia, Spinal - metabolism | Analgesics | Intermediate filament proteins | Drugs | Neurosciences | Spinal cord | Transcription factors | Model testing | Gabapentin | Medical innovations | Morphine | Activation | Neuropathy | Spinal cord injury | Neuronal-glial interactions | Interleukin 6 | Pain | Dorsal root ganglia | Filaments | Coding | Surgery | Rodents | Cell cycle | Toll-like receptors | Anesthesia | Tumor necrosis factor-TNF | Lesions | Amitriptyline | Territory | Pain perception | Cytokines | Glial fibrillary acidic protein | Activating transcription factor 3 | Markers | Thorax | Pharmacology | Inflammation | Clonazepam | Pressure | Isoflurane | Ganglia | Microglia | Ketamine | Spinal cord injuries | Index Medicus | Life Sciences | Neurons and Cognition
Journal Article