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PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 11, p. e15457
Spinal muscular atrophy (SMA) is a major genetic cause of death in childhood characterized by marked muscle weakness. To investigate mechanisms underlying... 
ANTERIOR HORN | PROTEIN | MOTOR-NEURON DEGENERATION | BIOLOGY | AXONAL NEUROPATHY | MICE | AMYOTROPHIC-LATERAL-SCLEROSIS | WERDNIG-HOFFMANN DISEASE | SENSORY NEURONS | END-PLATE | MOTONEURONS | Immunohistochemistry | Spinal Cord - metabolism | Neuromuscular Junction - metabolism | Electrophysiology | Spinal Nerve Roots - metabolism | Male | Muscle, Skeletal - metabolism | Axons - physiology | Ganglia, Spinal - physiopathology | Synapses - genetics | Hindlimb - physiopathology | Muscular Atrophy, Spinal - genetics | Spinal Nerve Roots - physiopathology | Female | Muscular Atrophy, Spinal - physiopathology | Disease Models, Animal | Proprioception - physiology | Motor Neurons - physiology | Muscular Atrophy, Spinal - metabolism | Receptors, Cholinergic - metabolism | Synapses - physiology | Hindlimb - innervation | Mice, Transgenic | Neuromuscular Junction - physiopathology | Disease Progression | Animals | Muscle, Skeletal - physiopathology | Mice | Spinal Cord - physiopathology | Ganglia, Spinal - metabolism | Analysis | Models | Spinal muscular atrophy | Neurosciences | Spinal cord | Disease | Circuits | Pathogenesis | SMN protein | Neurobiology | Defects | Proteins | Atrophy | Reduction | Rodents | Sensory neurons | Children | Quantitative analysis | Motor neurons | Neurons | Proprioception | Muscles | Amyotrophic lateral sclerosis | Muscle contraction | Microglia | Microscopy | Neuromuscular junctions | Glutamic acid transporter | Transporter | Synapses | Animal models
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2009, Volume 137, Issue 6, pp. 2084 - 2095.e3
Background & Aims The transient receptor potential (TRP) channel family includes transducers of mechanical and chemical stimuli for visceral sensory neurons.... 
Gastroenterology and Hepatology | SENSITIZATION | PELVIC COLONIC AFFERENTS | INFLAMMATORY PAIN | MICE | HYPERALGESIA | COLD | GASTROENTEROLOGY & HEPATOLOGY | IRRITABLE-BOWEL-SYNDROME | SENSORY NEURONS | BRADYKININ | HYPERSENSITIVITY | Hyperalgesia - chemically induced | Immunohistochemistry | Intestinal Mucosa - metabolism | Intestinal Mucosa - physiopathology | Mechanotransduction, Cellular - drug effects | Capsaicin - pharmacology | Neuroanatomical Tract-Tracing Techniques | Receptor, PAR-2 - metabolism | Colon - drug effects | Afferent Pathways - physiopathology | Colon - innervation | Male | Ganglia, Spinal - physiopathology | Nodose Ganglion - metabolism | Trinitrobenzenesulfonic Acid | Pelvis - innervation | RNA, Messenger - metabolism | Transient Receptor Potential Channels - deficiency | Action Potentials | Nodose Ganglion - physiopathology | Splanchnic Nerves - physiopathology | In Situ Hybridization | Colitis - chemically induced | Nodose Ganglion - drug effects | Colon - physiopathology | Female | Transient Receptor Potential Channels - metabolism | Bradykinin - pharmacology | Disease Models, Animal | Afferent Pathways - metabolism | Hyperalgesia - metabolism | Mice, Inbred C57BL | Reverse Transcriptase Polymerase Chain Reaction | Pressure | Stimulation, Chemical | Colon - metabolism | Hyperalgesia - physiopathology | Mice, Knockout | Animals | Colitis - physiopathology | Transient Receptor Potential Channels - genetics | TRPA1 Cation Channel | Colitis - metabolism | Splanchnic Nerves - metabolism | Mice | Ganglia, Spinal - drug effects | Pain Measurement | Ganglia, Spinal - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Nature Neuroscience, ISSN 1097-6256, 08/2013, Volume 16, Issue 8, pp. 1024 - 1031
Journal Article
Blood, ISSN 0006-4971, 2013, Volume 122, Issue 11, pp. 1853 - 1862
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2012, Volume 109, Issue 50, pp. 20602 - 20607
Osteoarthritis is one of the leading causes of chronic pain, but almost nothing is known about the mechanisms and molecules that mediate... 
Receptors | Pain | Neurons | Mice | Arthritis | Macrophages | Osteoarthritis | Behavioral neuroscience | Chemokines | Chronic pain | SYSTEM | NEUROPATHIC PAIN | CENTRAL SENSITIZATION | NOCICEPTIVE NEURONS | RAT | MULTIDISCIPLINARY SCIENCES | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | DORSAL-ROOT GANGLIA | SPINAL-CORD | MICE | EXPRESSION | Receptors, CCR2 - genetics | Receptors, CCR2 - physiology | Arthritis, Experimental - genetics | Chemokine CCL2 - immunology | Humans | Male | Pain - immunology | Ganglia, Spinal - physiopathology | RNA, Messenger - metabolism | Osteoarthritis - physiopathology | Arthritis, Experimental - pathology | Pain - genetics | Chemokine CCL2 - physiology | Osteoarthritis - pathology | Disease Models, Animal | Ganglia, Spinal - immunology | Arthritis, Experimental - physiopathology | Macrophages - pathology | Signal Transduction | Osteoarthritis - immunology | Mice, Inbred C57BL | RNA, Messenger - genetics | Chemokine CCL2 - genetics | Arthritis, Experimental - immunology | Osteoarthritis - genetics | Hyperalgesia - physiopathology | Mice, Knockout | Hyperalgesia - immunology | Animals | Hyperalgesia - genetics | Pain - physiopathology | Ganglia, Spinal - pathology | Receptors, CCR2 - deficiency | Prevention | Knee | Physiological aspects | Health aspects | Signal transduction | T cell receptors | Rodents | Biological Sciences
Journal Article
Neurology, ISSN 0028-3878, 04/2014, Volume 82, Issue 16, pp. 1410 - 1415
Journal Article
PLoS One, ISSN 1932-6203, 2016, Volume 11, Issue 3, p. e0150141
Journal Article