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Neurourology and urodynamics, ISSN 0733-2467, 01/2010, Volume 29, Issue 1, pp. 63 - 76
Aims To summarize the changes that occur in the properties of bladder afferent neurons following spinal cord injury... 
voltage‐gated ion channels | afferent neurons | neurotrophic factors | urinary bladder | neuropeptides | micturition | neuroplasticity | Neuroplasticity | Afferent neurons | Micturition | Urinary bladder | Voltage-gated ion channels | Neuropeptides | Neurotrophic factors | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Central Nervous System - metabolism | Spinal Cord Injuries - complications | gamma-Aminobutyric Acid - metabolism | Humans | Neuroanatomical Tract-Tracing Techniques | Urinary Bladder, Neurogenic - metabolism | Afferent Pathways - physiopathology | Urinary Bladder, Neurogenic - etiology | Ganglia, Spinal - physiopathology | Recovery of Function | Urinary Bladder - innervation | Potassium Channels - metabolism | Urinary Bladder, Neurogenic - physiopathology | Sodium Channels - metabolism | Urinary Bladder, Neurogenic - therapy | Spinal Cord Injuries - therapy | Afferent Pathways - metabolism | Reflex | Nerve Growth Factor - metabolism | Spinal Cord Injuries - metabolism | Nerve Fibers, Unmyelinated | Mechanotransduction, Cellular | Patch-Clamp Techniques | Animals | Neuronal Plasticity | Pituitary Adenylate Cyclase-Activating Polypeptide - metabolism | Spinal Cord Injuries - physiopathology | Central Nervous System - physiopathology | Urination | Nerve Fibers, Myelinated | Ganglia, Spinal - metabolism | Genetic Therapy - methods | Index Medicus | voltage-gated ion channels
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 1931-857X, 05/2011, Volume 300, Issue 5, pp. 1223 - 1234
...) following spinal cord injury (SCI) remains unknown. In this study, using a rat SCI model, we assessed the relevance of TRPA1 in OAB induced by SCI... 
Acrolein - analogs & derivatives | Spinal Cord - drug effects | Spinal Cord - metabolism | Spinal Cord Injuries - drug therapy | Calcium Channels - metabolism | Capsaicin - pharmacology | Spinal Cord Injuries - complications | Urodynamics - drug effects | Ganglia, Spinal - physiopathology | Spinal Cord Injuries - genetics | RNA, Messenger - metabolism | Urinary Bladder, Overactive - etiology | Urinary Bladder - innervation | Urinary Bladder, Overactive - physiopathology | Calcium Channels - genetics | Disease Models, Animal | Acrolein - pharmacology | Acetanilides - pharmacology | Oligonucleotides, Antisense - pharmacology | Spinal Cord Injuries - metabolism | TRPC Cation Channels | Urinary Bladder - metabolism | Purines - pharmacology | Rats | Urinary Bladder, Overactive - metabolism | Carbachol - pharmacology | Animals | Ankyrins - antagonists & inhibitors | Muscle Contraction - drug effects | Ankyrins - genetics | TRPA1 Cation Channel | Ankyrins - metabolism | Urinary Bladder, Overactive - genetics | Urinary Bladder - drug effects | Spinal Cord - physiopathology | Spinal Cord Injuries - physiopathology | Urinary Bladder, Overactive - prevention & control | Ganglia, Spinal - drug effects | Ganglia, Spinal - metabolism | Urinary incontinence | Complications and side effects | Cell receptors | Physiological aspects | Spinal cord injuries | Research | Risk factors | Studies | Bladder | Pathology | Spinal cord | Gene expression | Rodents | Index Medicus
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