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Molecular Medicine Reports, ISSN 1791-2997, 06/2018, Volume 17, Issue 6, pp. 7672 - 7682
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2004, Volume 279, Issue 37, pp. 38921 - 38935
We have recently demonstrated that osteopontin (OPN) induces nuclear factor kappaB (NFkappaB)-mediated promatrix metalloproteinase-2 activation through... 
BREAST-CANCER CELLS | SIGNALING PATHWAYS | PLASMINOGEN-ACTIVATOR | MATRIX-METALLOPROTEINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | GELATINASE-B | DOWN-REGULATION | TUMOR | NECROSIS-FACTOR-ALPHA | MELANOMA-CELLS | DIFFERENTIAL REGULATION
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 06/2016, Volume 24, Issue 18, pp. 145 - 1057
Aims: Media-to-intima migration of vascular smooth muscle cells (VSMCs) is critical to intimal thickening in atherosclerosis and restenosis after coronary... 
Original Research Communications | MATRIX | SUPEROXIDE ANIONS | PARAVENTRICULAR NUCLEUS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOCRINOLOGY & METABOLISM | DOWN-REGULATION | PROLIFERATION | SYMPATHETIC OUTFLOW | NF-KAPPA-B | HYPERTENSIVE-RATS | EXPRESSION | BLOOD-PRESSURE | Reactive Oxygen Species - metabolism | Vascular Diseases - pathology | Membrane Glycoproteins - metabolism | Muscle, Smooth, Vascular - metabolism | NADPH Oxidases - metabolism | Male | NF-kappa B - metabolism | Intercellular Signaling Peptides and Proteins - physiology | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinase 9 - genetics | Promoter Regions, Genetic | Signal Transduction | Cells, Cultured | Myocytes, Smooth Muscle - physiology | Enzyme Induction | NADPH Oxidase 2 | Rats, Sprague-Dawley | Protein Transport | Muscle, Smooth, Vascular - pathology | Animals | Tunica Intima - pathology | Carotid Arteries - pathology | Enzyme Activation | Vascular Diseases - metabolism | Cell Movement | Phosphorylation | Hyperplasia | Oxidase | Innovations | Smooth muscle | Matrix metalloproteinase | Arteries | NAD(P)H oxidase | Carotid arteries | Cell adhesion & migration | Vascular diseases | Transfection | Atherosclerosis | CYBB protein | Metalloproteinase | Inhibition | Carotid artery | Stents | Thickening | Translocation | Muscles | Rats | siRNA | Coronary angioplasty | Gene expression | Nuclear transport | Gelatinase B | Restenosis | Injury prevention | Ribonucleic acids | Arteriosclerosis | Cell migration | Index Medicus
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 05/2012, Volume 261, Issue 1, pp. 105 - 115
The induction of matrix metalloproteinase (MMP)-9 is particularly important for the invasiveness of various cancer cells. Osthole, a natural coumarin... 
NF-κB | Lung adenocarcinoma | Osthole | Migration | Invasion | MMPs | MMP-9 EXPRESSION | METASTASIS | MATRIX-METALLOPROTEINASE EXPRESSION | CNIDIUM-MONNIERI | BREAST-CANCER CELLS | NF-kappa B | ISOPENTENYLATED COUMARINS | PATHWAY | PROSTATE-CANCER | GROWTH | PHARMACOLOGY & PHARMACY | TOXICOLOGY | REGULATORY MECHANISM | Lung Neoplasms - drug therapy | Promoter Regions, Genetic | Adenocarcinoma - pathology | Medicine, Chinese Traditional | Matrix Metalloproteinase 2 - metabolism | Neoplasm Invasiveness | Humans | Lung Neoplasms - pathology | NF-kappa B - metabolism | RNA, Messenger - metabolism | Adenocarcinoma - drug therapy | Coumarins - pharmacology | Cell Movement - drug effects | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinase 9 - genetics | Fluorescent Antibody Technique | Cell Line, Tumor | Gene Expression Regulation, Neoplastic - drug effects | Binding Sites | NF-kappa B - drug effects | Index Medicus | Adenocarcinoma | Cell proliferation | Immunoprecipitation | Invasiveness | Tumor cells | Lung cancer | Matrix metalloproteinase | Tumor cell lines | Gene expression | Coumarin | Gelatinase B | Nuclear transport | Promoters | Western blotting | Gelatinase A | Traditional Chinese Medicine | NF- Kappa B protein | Proteolysis | Cell migration | TRANSLOCATION | CHROMATIN | LUNGS | METASTASES | TUMOR CELLS | 60 APPLIED LIFE SCIENCES | MUTANTS | INHIBITION | FLUORESCENCE | MESSENGER-RNA | PROMOTERS | DNA | CARCINOMAS | COUMARIN | PROTEINS
Journal Article
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 12/2017, Volume 232, Issue 12, pp. 3481 - 3495
IL‐33‐ST2L interaction increased MMP‐9 expression in RAW264.7 cells. ERK/CREB and NF‐κB cascades have an essential role in the induction of MMP‐9 in... 
interleukin‐33 (IL‐33) | macrophage | nuclear factor‐kappa B (NF‐κB) | matrix metalloproteinase‐9 (MMP‐9) | activator protein 1 (AP‐1) | matrix metalloproteinase-9 (MMP-9) | activator protein 1 (AP-1) | interleukin-33 (IL-33) | nuclear factor-kappa B (NF-κB) | nuclear factor-kappa B (NF-B) | 92-KDA GELATINASE | PHYSIOLOGY | MMP-9 EXPRESSION | IL-1 RECEPTOR | HUMAN MONOCYTES | CELL BIOLOGY | MATRIX METALLOPROTEINASES | RECEPTOR ACCESSORY PROTEIN | GENE-EXPRESSION | ALVEOLAR MACROPHAGES | TNF-ALPHA | NF-KAPPA-B | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Phosphorylation | Interleukin-33 - pharmacology | Transcription Factor AP-1 - genetics | Receptors, Interleukin-1 - genetics | Transcriptional Activation | Tumor Necrosis Factor-alpha - genetics | NF-kappa B - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Transcription Factor AP-1 - metabolism | Receptors, Interleukin-1 - agonists | Dose-Response Relationship, Drug | Transendothelial and Transepithelial Migration - drug effects | Matrix Metalloproteinase 9 - metabolism | Transfection | RNA Interference | Time Factors | Matrix Metalloproteinase 9 - genetics | Transcription, Genetic | Promoter Regions, Genetic | Macrophages - enzymology | Receptors, Interleukin-1 - metabolism | Cyclic AMP Response Element-Binding Protein - genetics | Animals | Signal Transduction - drug effects | Cyclic AMP Response Element-Binding Protein - metabolism | Protein Binding | Macrophages - drug effects | RAW 264.7 Cells | Mice | Interleukins | Macrophages | Protein binding | Chromatin | Transcription factors | Immunoprecipitation | Leukocyte migration | Interleukin | Activation | Matrix metalloproteinase | Blockage | Recruitment | Degradation | Proteins | Cell activation | Immunology | Cascades | Rodents | Interleukin 1 | Extracellular matrix | Metalloproteinase | Pretreatment | c-Fos protein | Translocation | NF-κB protein | Cytokines | Secretion | Activator protein 1 | Extracellular signal-regulated kinase | Cyclic AMP | Inflammation | Cyclic AMP response element-binding protein | Gene expression | Nuclear transport | Gelatinase B | Monocytes | Cell migration | Index Medicus
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 04/2019, Volume 120, Issue 4, pp. 5936 - 5948
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, pp. e77126 - e77126
Tumor necrosis factor-alpha (TNF alpha) activates both cell death and cell survival pathways. The activation of survival pathway renders most cancer cells... 
CARCINOMA-CELLS | CURCUMIN DIFERULOYLMETHANE | TUMOR-NECROSIS-FACTOR | BLOCKS INTERLEUKIN-1-BETA-INDUCED ACTIVATION | ONCOGENIC RAS | CONSTITUTIVE ACTIVATION | MULTIDISCIPLINARY SCIENCES | LEUKEMIA-CELLS | CYCLE ARREST | MULTIPLE-MYELOMA CELLS | LUTEOLIN | Vascular Endothelial Growth Factor A | Gene Expression | Pancreatic Neoplasms - metabolism | Apoptosis - drug effects | Humans | Apoptosis - genetics | JNK Mitogen-Activated Protein Kinases - metabolism | NF-kappa B - metabolism | Pancreatic Neoplasms - genetics | Enzyme Activation - drug effects | Transcription Factor RelA - genetics | Tumor Necrosis Factor-alpha - pharmacology | I-kappa B Kinase - antagonists & inhibitors | Proteolysis | Cell Line, Tumor | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Flavones - pharmacology | Genes, Reporter | Phosphorylation | Bcl-2 protein | Toxicity | c-Myc protein | Cytotoxicity | Activation | Myc protein | Cyclin D1 | Kinases | Anticancer properties | Angiogenesis | Signal transduction | Chinese medicine | Cell activation | Bcl-x protein | Rodents | Transcription activation | Tumor necrosis factor-TNF | Inhibition | Pretreatment | Vascular endothelial growth factor | Translocation | NF-κB protein | Cell survival | Survivin | Tumor necrosis factor-α | Gene expression | Survival | Nuclear transport | Molecular chains | Mcl-1 protein | Gelatinase B | Signaling | Cell death | Pancreatic cancer | Survival factor | Cyclooxygenase-2 | Cancer | Apoptosis | Index Medicus
Journal Article
Journal of Pharmacy and Pharmacology, ISSN 0022-3573, 08/2019, Volume 71, Issue 8, pp. 1255 - 1261
Objectives To investigate the therapeutic effects and mechanisms of S-nitrosoglutathione (SNG) on acute kidney injury (AKI) induced by lipopolysaccharide... 
acute kidney injury | nuclear factor‐κB | toll‐like receptor 4 | lipopolysaccharide | S‐nitrosoglutathione | INFLAMMATION | nuclear factor-kappa B | PHARMACOLOGY & PHARMACY | GELATINASE-ASSOCIATED LIPOCALIN | S-nitrosoglutathione | toll-like receptor 4 | SEPTIC PATIENTS | RAT MODEL | Kidneys | Cytokines |